pericarditis febi

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    Pericarditis

    Febrina RambuC 111 11 162

    Pembimbing :

    dr. Akhtar Fajar M, SpP, F!"A

    #!$A%A&A' #A(AM RA')&A *+)AS &PA'!*RAA'&(!'!&

    $A)!A' &AR#!-(-)! #A' -&*RA' AS&+(AR

    FA&+(*AS -&*RA'

    +'!RS!*AS "ASA'+##!'

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    Anatm

    Normal amount ofpericardial uid: 25-50 cc

    Two layers:

    Outer layer is theparietal pericardiumand consists of layersof brous and seroustissue

    Inner layer is isceralpericardium andconsists of seroustissue only

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    Pericardium

    !ibroelastic sacconsistin" of 2layers

    #isceral at

    epicardial side

    $arietal atmediastinalside

    $ericardial uidformed fromultraltrate ofplasma

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    pidemi3g Pericarditis

    /.14 5 hspita3ied patients

    04 5 patients admitted t

    mergenc #epartment 5r nn7acute mcardia3 in5arctin chestpain

    14 pericarditis patient is subc3inica3

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    $ericarditis isinammation of

    pericardial layer of

    the heart% with orwithout uidacumulation in

    percardial space

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    C3assi8catin&'(T) $)*I'&*+ITI, lessthan . wee/s : brousa andefusif

    ,(1-&'(T) $)*I'&*+ITI, .wee/s . months :

    'onstrictie and efusie-constrictie

    '3*ONI' $)*I'&*+ITI,more than . months :constrictie% efusie% and

    adhesie

    IN!)'TIO(, $)*I'&*+ITI, :irus% pio"enic% tuberculosis%mi/oti/% etc

    NON-IN!)'TIO(,$)*I'&*+ITI, : infarc

    myocard acute% uremia%neoplasma% mi/sedema%cholestrol% trauma% acuteidiopati/% etc4

    $)*I'&*+ITI, '&(,)+ 13$)*,)N,ITI#IT O*&(TOI6(N : rematic feer%cola"en ascular disease%dru"-induced% sindrom+ressler4

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    AC+* PR!CAR#!*!S*he mst cmmn a9ictin 5 pericardium

    Re5ers t inammatin 5 its 3aers

    )TIO7O8 :IN!)'TIO(, : Idiopathic and iral pericarditis Tuberculosis pericarditis Nontuberculosis bacterial pericarditisNON-IN!)'TIO(, : $ericarditis followin" 6yocardial

    Infarction (remic $ericarditis Neoplastic $ericarditis *adiation-induced pericarditis

    $ericarditis associated with connectietissue disease

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    'haracteri9ed by three sta"es:

    47O'&7 #&,O+I7&TION ;IT3 T*&N,(+&TION O!$*OT)IN-$OO*% ')77-!*)) !7(I+ INTO$)*I'&*+I&7 ,$&')24IN'*)&,)+ #&,'(7&* $)*6)&1I7IT lea/ ofprotein to pericardial space

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    PA*"-(-);

    ,)*O(, $)*I'&*+ITI, represents

    the early inammatory response common to

    all types of acute pericarditis

    ,)*O!I1*INO(, $)*I'&*+ITI,

    ,($$(*&TI#) or purulent

    $)*I'&*+ITI,

    3)6O**3&8I' $)*I'&*+ITI,

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    C(!'!CA( FA*+RS

    &(,'(7T&TION : sound& ,'*&T'3$)*I'&*+I&N!*I'TION *(1

    In its full form, the rubconsist 3 components,corresponding to the phasesof greatest cardiac movement:Ventricular contractionVentricular relaxation

    http://pericardial%20friction%20rub%20by%20-%20mp3pad.com.mp3/http://pericardial%20friction%20rub%20by%20-%20mp3pad.com.mp3/
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    #iagnsis 5 Pericarditis:Presence 5 t

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    7&1O*&TO*I(6 : serum transaminase till ?0unit4 '=61 not raise

    *&+IO7O8 :Normal in patients with acute pericarditis unlesspericardial eAusion is present)nlar"ed cardiac silhouette *eCuires 200cc ofuid

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    *RA*M'*

    Treat underlyin" disease

    1ed rest

    &nal"esic

    &nti-inamation

    'olchicine recurrent

    pericarditis or resistent

    N,&I+ and chorticosteroid

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    C"R-'!C PR!CAR#!*!S

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    =&bnormalities occur durin" diastoleD systoliccontraction of the entricles is usually normal='linical features : fati"ue% hypotension% reeEtachycardia% Fu"ular enous distention%

    hepatome"aly with ascites% and peripheral edema

    'ON,T*I'TI#) $)*I'&*+ITI,

    'hronic pericarditis with adhesion between parietaland isceral pericardium

    6ostly seen in rheumatic disease

    &+3),I#) $)*I'&*+ITI,

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    Chest Radigraph

    chcardigraphic

    C*7scan Cardiac catheteriatin

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