pericarditis febi
TRANSCRIPT
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Pericarditis
Febrina RambuC 111 11 162
Pembimbing :
dr. Akhtar Fajar M, SpP, F!"A
#!$A%A&A' #A(AM RA')&A *+)AS &PA'!*RAA'&(!'!&
$A)!A' &AR#!-(-)! #A' -&*RA' AS&+(AR
FA&+(*AS -&*RA'
+'!RS!*AS "ASA'+##!'
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Anatm
Normal amount ofpericardial uid: 25-50 cc
Two layers:
Outer layer is theparietal pericardiumand consists of layersof brous and seroustissue
Inner layer is isceralpericardium andconsists of seroustissue only
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Pericardium
!ibroelastic sacconsistin" of 2layers
#isceral at
epicardial side
$arietal atmediastinalside
$ericardial uidformed fromultraltrate ofplasma
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pidemi3g Pericarditis
/.14 5 hspita3ied patients
04 5 patients admitted t
mergenc #epartment 5r nn7acute mcardia3 in5arctin chestpain
14 pericarditis patient is subc3inica3
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$ericarditis isinammation of
pericardial layer of
the heart% with orwithout uidacumulation in
percardial space
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C3assi8catin&'(T) $)*I'&*+ITI, lessthan . wee/s : brousa andefusif
,(1-&'(T) $)*I'&*+ITI, .wee/s . months :
'onstrictie and efusie-constrictie
'3*ONI' $)*I'&*+ITI,more than . months :constrictie% efusie% and
adhesie
IN!)'TIO(, $)*I'&*+ITI, :irus% pio"enic% tuberculosis%mi/oti/% etc
NON-IN!)'TIO(,$)*I'&*+ITI, : infarc
myocard acute% uremia%neoplasma% mi/sedema%cholestrol% trauma% acuteidiopati/% etc4
$)*I'&*+ITI, '&(,)+ 13$)*,)N,ITI#IT O*&(TOI6(N : rematic feer%cola"en ascular disease%dru"-induced% sindrom+ressler4
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AC+* PR!CAR#!*!S*he mst cmmn a9ictin 5 pericardium
Re5ers t inammatin 5 its 3aers
)TIO7O8 :IN!)'TIO(, : Idiopathic and iral pericarditis Tuberculosis pericarditis Nontuberculosis bacterial pericarditisNON-IN!)'TIO(, : $ericarditis followin" 6yocardial
Infarction (remic $ericarditis Neoplastic $ericarditis *adiation-induced pericarditis
$ericarditis associated with connectietissue disease
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'haracteri9ed by three sta"es:
47O'&7 #&,O+I7&TION ;IT3 T*&N,(+&TION O!$*OT)IN-$OO*% ')77-!*)) !7(I+ INTO$)*I'&*+I&7 ,$&')24IN'*)&,)+ #&,'(7&* $)*6)&1I7IT lea/ ofprotein to pericardial space
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PA*"-(-);
,)*O(, $)*I'&*+ITI, represents
the early inammatory response common to
all types of acute pericarditis
,)*O!I1*INO(, $)*I'&*+ITI,
,($$(*&TI#) or purulent
$)*I'&*+ITI,
3)6O**3&8I' $)*I'&*+ITI,
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C(!'!CA( FA*+RS
&(,'(7T&TION : sound& ,'*&T'3$)*I'&*+I&N!*I'TION *(1
In its full form, the rubconsist 3 components,corresponding to the phasesof greatest cardiac movement:Ventricular contractionVentricular relaxation
http://pericardial%20friction%20rub%20by%20-%20mp3pad.com.mp3/http://pericardial%20friction%20rub%20by%20-%20mp3pad.com.mp3/ -
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#iagnsis 5 Pericarditis:Presence 5 t
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7&1O*&TO*I(6 : serum transaminase till ?0unit4 '=61 not raise
*&+IO7O8 :Normal in patients with acute pericarditis unlesspericardial eAusion is present)nlar"ed cardiac silhouette *eCuires 200cc ofuid
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*RA*M'*
Treat underlyin" disease
1ed rest
&nal"esic
&nti-inamation
'olchicine recurrent
pericarditis or resistent
N,&I+ and chorticosteroid
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C"R-'!C PR!CAR#!*!S
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=&bnormalities occur durin" diastoleD systoliccontraction of the entricles is usually normal='linical features : fati"ue% hypotension% reeEtachycardia% Fu"ular enous distention%
hepatome"aly with ascites% and peripheral edema
'ON,T*I'TI#) $)*I'&*+ITI,
'hronic pericarditis with adhesion between parietaland isceral pericardium
6ostly seen in rheumatic disease
&+3),I#) $)*I'&*+ITI,
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Chest Radigraph
chcardigraphic
C*7scan Cardiac catheteriatin
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