commentary

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[7] Penneys R, Wilkinson JH. Elevation of serum creatine kinase following amputation of the leg. Surgery 1970;67:302-5. [8] Shiraishi T. A new technique for exposure of the cervical spine laminae: technical note. J Neurosurg 2002;96:S122-6. [9] Sihvonen T, Herno A, Paljarvi L, et al. Local denervation atrophy of paraspinal muscles in postoperative failed back syndrome. Spine 1993;18:575-81. [10] Spetzger U, Bertalanffy H, Reinges MH, et al. Unilateral laminotomy for bilateral decompression of lumbar spinal stenosis: Part II. Clinical experiences. Acta Neurochir (Wien) 1997;139:397-403. [11] Suwa H, Hanakita J, Ohshita N, et al. Postoperative changes in paraspinal muscle thickness after various lumbar back surgery procedures. Neurol Med Chir (Tokyo) 2000;40:151-4. [12] Watanabe K, Hosoya T, Shiraishi T, et al. Lumbar spinous process-splitting laminectomy for lumbar canal stenosis: Technical note. J Neurosurg Spine 2005;3:405-8. [13] Weiner BK, Fraser RD, Peterson M. Spinous process osteotomies to facilitate lumbar decompressive surgery. Spine 1999;24:62-6. [14] Weiner BK, Walker M, Brower RS. Microdecompression for lumbar spinal canal stenosis. Spine 1999;24:2268-72. Commentary Kim et al have demonstrated, via an elegant study, the relationship between operative muscle trauma and muscle injury with accompanying atrophy. This information con- firms the need to continuously seek surgical approaches that limit muscle trauma. Kim et al have solidified the importance of such strategies. For this, they are to be congratulated. Edward C. Benzel, MD Neurosurgery, The Cleveland Clinic Foundation Cleveland, OH 44106, USA 113 K. Kim et al. / Surgical Neurology 69 (2008) 109113

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Page 1: Commentary

[7] Penneys R, Wilkinson JH. Elevation of serum creatine kinasefollowing amputation of the leg. Surgery 1970;67:302-5.

[8] Shiraishi T. A new technique for exposure of the cervical spinelaminae: technical note. J Neurosurg 2002;96:S122-6.

[9] Sihvonen T, Herno A, Paljarvi L, et al. Local denervation atrophy ofparaspinal muscles in postoperative failed back syndrome. Spine1993;18:575-81.

[10] Spetzger U, Bertalanffy H, Reinges MH, et al. Unilateral laminotomyfor bilateral decompression of lumbar spinal stenosis: Part II. Clinicalexperiences. Acta Neurochir (Wien) 1997;139:397-403.

[11] Suwa H, Hanakita J, Ohshita N, et al. Postoperative changes inparaspinal muscle thickness after various lumbar back surgeryprocedures. Neurol Med Chir (Tokyo) 2000;40:151-4.

[12] Watanabe K, Hosoya T, Shiraishi T, et al. Lumbar spinousprocess-splitting laminectomy for lumbar canal stenosis: Technicalnote. J Neurosurg Spine 2005;3:405-8.

[13] Weiner BK, Fraser RD, Peterson M. Spinous process osteotomies tofacilitate lumbar decompressive surgery. Spine 1999;24:62-6.

[14] Weiner BK, Walker M, Brower RS. Microdecompression for lumbarspinal canal stenosis. Spine 1999;24:2268-72.

Commentary

Kim et al have demonstrated, via an elegant study, therelationship between operative muscle trauma and muscleinjury with accompanying atrophy. This information con-firms the need to continuously seek surgical approaches thatlimit muscle trauma. Kim et al have solidified the importanceof such strategies. For this, they are to be congratulated.

Edward C. Benzel, MDNeurosurgery, The Cleveland Clinic Foundation

Cleveland, OH 44106, USA

113K. Kim et al. / Surgical Neurology 69 (2008) 109–113