Download - Commentary

Transcript
Page 1: Commentary

[7] Penneys R, Wilkinson JH. Elevation of serum creatine kinasefollowing amputation of the leg. Surgery 1970;67:302-5.

[8] Shiraishi T. A new technique for exposure of the cervical spinelaminae: technical note. J Neurosurg 2002;96:S122-6.

[9] Sihvonen T, Herno A, Paljarvi L, et al. Local denervation atrophy ofparaspinal muscles in postoperative failed back syndrome. Spine1993;18:575-81.

[10] Spetzger U, Bertalanffy H, Reinges MH, et al. Unilateral laminotomyfor bilateral decompression of lumbar spinal stenosis: Part II. Clinicalexperiences. Acta Neurochir (Wien) 1997;139:397-403.

[11] Suwa H, Hanakita J, Ohshita N, et al. Postoperative changes inparaspinal muscle thickness after various lumbar back surgeryprocedures. Neurol Med Chir (Tokyo) 2000;40:151-4.

[12] Watanabe K, Hosoya T, Shiraishi T, et al. Lumbar spinousprocess-splitting laminectomy for lumbar canal stenosis: Technicalnote. J Neurosurg Spine 2005;3:405-8.

[13] Weiner BK, Fraser RD, Peterson M. Spinous process osteotomies tofacilitate lumbar decompressive surgery. Spine 1999;24:62-6.

[14] Weiner BK, Walker M, Brower RS. Microdecompression for lumbarspinal canal stenosis. Spine 1999;24:2268-72.

Commentary

Kim et al have demonstrated, via an elegant study, therelationship between operative muscle trauma and muscleinjury with accompanying atrophy. This information con-firms the need to continuously seek surgical approaches thatlimit muscle trauma. Kim et al have solidified the importanceof such strategies. For this, they are to be congratulated.

Edward C. Benzel, MDNeurosurgery, The Cleveland Clinic Foundation

Cleveland, OH 44106, USA

113K. Kim et al. / Surgical Neurology 69 (2008) 109–113

Top Related