36351789 cerebral bleeding cva hemorrhage

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    Cerebral bleeding

    (CVA

    hemorrhage)Presented by group 3 104

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    Learning objectives

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    A. General Objectives

    This study aims to convey

    familiarity and provide effective

    nursing care to a patient diagnosedwith Cerebral Hemorrhage; HPN

    and Seizure through understanding

    the patient history, disease process

    and management.

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    B. Specific Objectives

    At the end of the session, the students will be able

    to:

    Present a thorough assessment regarding Cerebral

    Hemorrhage; HPN and Seizure, through NursingHealth History, Gordons Functional Health Pattern,

    Physical Assessment, and the interpretation of the

    laboratory examinations done on the patient.

    Discuss the anatomy and physiology of the heart,

    pathophysiology of the patients condition, usual

    clinical manifestations and possible complications

    of the condition.

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    Enumerate the necessary

    medications needed and be familiar

    to its mode of action.

    Formulate a workable nursing care

    plan on the subjective and objectivecues gathered through nurse-

    patient interaction to be able to help

    the patient towards wellness.

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    Introduction

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    Cerebro-vascular accident

    A stroke (sometimes called

    a cerebrovascular accident (CVA)) is the

    rapidly developing loss of brains function(s)

    due to disturbance in the blood supply to the

    brain.

    is a "neurological deficit of cerebrovascular

    cause that persists beyond 24 hours or isinterrupted by death within 24 hours"

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    Classification of CVA

    2 Types:

    1. Ischemic

    a. thrombosis cause by severeatherosclerosis.

    b. embolic thrombus that travels

    to the Circle of Willis.

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    2. Hemorrhagic rupture of bloodvessels

    Causes:- HPN

    - aneurysm

    - arterio-venous malformations

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    Hemorrhagic strokes account for

    15% of cerebrovascular disorders

    and are primarily caused by anintracranial or subarachnoid

    hemorrhage. Hemorrhagic strokes

    are caused by bleeding into the

    brain tissue, the ventricles, or the

    subarachnoid space.

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    Primary intracerebral hemorrhage from a

    spontaneous rupture of small vessels accounts

    for approximately 80% of hemorrhagic strokes

    and is primarily caused by uncontrolledhypertension (Qureshi et al.,2001). Secondary

    intracerebral hemorrhage is associated with

    arteriovenous malformations (AVMs),intracranial aneurysms, or certain medications

    (eg, anticoagulants and amphetamines)

    (Qureshi et al., 2001).

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    The patient with a hemorrhagic stroke can

    present with a wide variety of neurologic

    deficits, similar to the patient with ischemic

    stroke. A comprehensive assessment will

    reveal the extent of the neurologic deficits.

    Many of the same motor, sensory, cranial

    nerve, cognitive, and other functions that aredisrupted following ischemic stroke are

    altered following a hemorrhagic stroke.

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    In addition to the neurologic deficits that

    are similar to ischemic stroke, the

    patient with an intracranial aneurysm orAVM can have some unique clinical

    manifestations. Rupture of an aneurysm

    or AVM usually produces a sudden,unusually severe headache and often

    loss of consciousness for a variable

    period.

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    At times, an aneurysm or AVM leaks

    blood, leading to the formation of a clot

    that seals the site of rupture. In thisinstance, the patient may show little

    neurologic deficit. In other cases, severe

    bleeding occurs, resulting in cerebraldamage followed rapidly by coma and

    death.

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    Prognosis depends on the neurologic

    condition of the patient, age, associated

    diseases, and the extent and location ofan intracranial aneurysm. Subarachnoid

    hemorrhage from an aneurysm is a

    catastrophic event with significantmorbidity and mortality. (Pfohman &

    Criddle, 2001).

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    Demographic data

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    Name: R.O.D. Age: 52 yo Gender: Male

    Civil Status: Married

    Address: 109 Anabu 1-A Imus, Cavite

    Birth date: 10/10/1957 Birth Place: Bicol

    Religion: Roman Catholic

    Language Spoken: Filipino

    Admitting Diagnosis / Impression: CVA , hemorrhagic Admitting Physician: Dr. J. Sembrano

    Source ofInformation: Wife

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    NURSING HISTORY

    ChiefComplaint: LOC

    History of present illness: The patient is a

    known cause of HPN was to maintained onClonodine and Lanoxine with poor

    compliance few monitor PTC, patient was

    noted to home loss of consciousness

    GCS 3

    Pupil 2-3 cm

    CH Brudzinki

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    Past Medical History

    Hospitalization (Cause / organ

    damage / Removed / Treatment):

    Gallblader stone

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    Family History

    Parents: Mother died of stroke

    Sibling: Hypertension

    Spouse: None Children: None

    Illness in the family similar to the patients:

    Hypertension in the family

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    Physical assesment

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    Vital signs

    The patient is afebrile with a

    temperature of 36.5 C . Pulse

    rate was 118 beats per minute.Patient is tachycardic.

    Respiratory rate of 22 cycles perminute and his blood pressure

    was 280/120 mmHg.

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    General survey

    The patient is in distress , bedridden

    and has cardiopulmonary distress.

    Patient is comatose GCS:3,incoherent and disoriented.

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    Skin

    The patient has pale complexion,

    the texture smooth, skin turgor was

    good, warm and dry.

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    Head

    head is normocephalic. The hair is fine,

    equally distributed and has dandruff seen on

    the scalp. The pupil size: 2 3mm and it

    reacts to light sluggishly with unequal

    constriction.

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    Ears

    The ears are symmetrical.There

    were no discharges.

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    Nose

    There were no discharges and

    mucosa is pink.

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    Mouth

    He has pale lips, the speech is not

    assessed because the pt. is comatose

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    NECK

    The trachea is in the midline.

    There are palpable cervical

    lymph nodes. Patient neck is

    rigid.

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    Chest and lungs

    The patient is tachypneic with 35 cycles

    per minute with occasional dyspnea.

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    abdomen

    The bowel sounds are normoactive.

    with positive dullness over the right

    upper quadrant. There are notenderness.

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    BACK ANDEXTREMITIES

    The peripheral pulses are symmetrical, equal and faint in the

    upper extremities. The nail beds are pallor. The muscle tone and

    strength is cachexic. The bilateral lower extremities are weak and

    have muscle dystrophy.

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    Neurologic assessment

    Motor response- 1

    Eye response- 1

    Verbal response- 1

    ------GCS 3

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    Pathophysiology

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    Laboratory studies

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    Examination:CT scan: Cranial CT Scan plain

    There is a massive hematoma seen in the cerebellum with intraventricular

    hemorrhagic extravasations into the third ventricle the lateral ventricles are

    dilated. There are also small malacic changes in both frontal periventricular

    regions.

    Remarks:

    Massive cerebellar hematoma with intraventricular extravasations,

    brainstem edema, obstructive hydrocephalus, chronic frontal

    periventricular ischemic infarcts.

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    Drug study

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    Nursing care plan

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    medicine

    Comply with the treatment regimen.

    Do not use any medicines, over-the-counter drugs, vitamins,

    herbs, or food supplements without first talking to caregivers or

    physician.

    Do not quit taking your medicines until you discuss it with yourcaregiver. If you are taking medicine that makes you drowsy, do

    not drive or use heavy equipment.

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    Exercise

    Regular exercises are good for the heart and lungs and could

    stimulate proper circulation and oxygenation in the body.

    Regular exercise should be minimal and basic in order not to

    increase the workload of the heart

    Follow your doctors recommendations for physical activity.

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    Treatment

    . Instructed client for strict compliance of treatment regimen.

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    Hygiene

    - Good oral hygiene and proper dental care apply to all age

    groups but the needs of the elderly population can be slightly

    different than the needs of the younger people. Client should also

    observe regular hand and body hygiene to decrease the risk of

    acquiring infection. Daily bath is recommended as well asfrequent hand hygiene, not only for the client but also for the

    clients significant others.

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    Out patient

    Seek medical advice only from doctors

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    Diet

    Limiting the amount of fat to no more than 25 to 35% of daily

    calories is recommended to promote good health.

    . A low-fat diet also helps lower high total and LDL (the bad)

    cholesterol levels, another risk factor for coronary artery disease.

    Eating at least five servings of fruits and vegetables daily candecrease the risk of coronary artery disease.

    The diet should contain the recommended daily requirements of

    vitamins and minerals. Vitamin supplements are not considered

    an acceptable substitute for a healthy diet

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    Spirituality

    Always pray for guidance and strength

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    Final evaluation

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    After the continuous treatment which consists of the medical and

    the nursing management of the patient, a development of her

    present health status is projected. Continuous administration of

    therapy had partially alleviated the clients suffering from the

    disease. Although disease process is incurable and has thetendency to attack again ones life, if the client will undergo

    treatment, by some means this will help reduce the sufferings of

    the client brought by the disease. But if the client will not subject

    herself to any medical management, this could lead to further

    complications; signs and symptoms of the disease will progressand will evidently manifested by the client. Furthermore, this

    would worsen the condition and eventually leads to death.

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    Evaluation of learning outcomes

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    EVALUATION

    After our thorough assessment, comprehensive

    observation, interpretation of the patients laboratory results, the

    nursing responsibilities of the prescribed medication, knowing the

    factors that affect the disease process and the nursing

    interventions rendered to our patient, we came up with thefollowing evaluations:

    General:

    The group was able to present all the accumulated information

    about our patients case, duly diagnosed with Cerebrovascular

    Accident Bleed, has improved our skills and knowledge pertainingon caring patients with the above mentioned disease.

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    Specific:

    1.) The incidence of CVA Bleed in global, national, and local

    setting was determined.

    2.) The inclusive assessment of the patient involving

    biographical data, chief complaint upon admission, past andpresent medical history, personal, family and socio-economic

    status as an apparent substantiation to the condition were

    present.

    3.) Review of systems through detailed but comprehensive

    physical assessment was conducted.

    4.) The significant diagnostic test and laboratory examinations,

    comparing abnormal results from normal values with its

    corresponding interpretation in relation to the current status of the

    client was determined.

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    5.) The predisposing and precipitating factors that contributed to

    the disease process and present comprehensively in a written

    and diagrammatic illustration were identified.

    6.) The pathophysiology of CVA Bleed and its symptomatology

    and complications of the client was discussed. 7.) The clients condition in the ICU from the day of admission

    until the student nurses assessment was determined.

    8.) The pharmacological management of CVA Bleed, and its

    nursing considerations were enumerated.

    9.) Three (3) prioritized health needs/ problems of the client and

    formulate nursing plan as a framework of care identified and

    applied.