36351789 cerebral bleeding cva hemorrhage
TRANSCRIPT
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Cerebral bleeding
(CVA
hemorrhage)Presented by group 3 104
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Learning objectives
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A. General Objectives
This study aims to convey
familiarity and provide effective
nursing care to a patient diagnosedwith Cerebral Hemorrhage; HPN
and Seizure through understanding
the patient history, disease process
and management.
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B. Specific Objectives
At the end of the session, the students will be able
to:
Present a thorough assessment regarding Cerebral
Hemorrhage; HPN and Seizure, through NursingHealth History, Gordons Functional Health Pattern,
Physical Assessment, and the interpretation of the
laboratory examinations done on the patient.
Discuss the anatomy and physiology of the heart,
pathophysiology of the patients condition, usual
clinical manifestations and possible complications
of the condition.
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Enumerate the necessary
medications needed and be familiar
to its mode of action.
Formulate a workable nursing care
plan on the subjective and objectivecues gathered through nurse-
patient interaction to be able to help
the patient towards wellness.
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Introduction
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Cerebro-vascular accident
A stroke (sometimes called
a cerebrovascular accident (CVA)) is the
rapidly developing loss of brains function(s)
due to disturbance in the blood supply to the
brain.
is a "neurological deficit of cerebrovascular
cause that persists beyond 24 hours or isinterrupted by death within 24 hours"
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Classification of CVA
2 Types:
1. Ischemic
a. thrombosis cause by severeatherosclerosis.
b. embolic thrombus that travels
to the Circle of Willis.
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2. Hemorrhagic rupture of bloodvessels
Causes:- HPN
- aneurysm
- arterio-venous malformations
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Hemorrhagic strokes account for
15% of cerebrovascular disorders
and are primarily caused by anintracranial or subarachnoid
hemorrhage. Hemorrhagic strokes
are caused by bleeding into the
brain tissue, the ventricles, or the
subarachnoid space.
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Primary intracerebral hemorrhage from a
spontaneous rupture of small vessels accounts
for approximately 80% of hemorrhagic strokes
and is primarily caused by uncontrolledhypertension (Qureshi et al.,2001). Secondary
intracerebral hemorrhage is associated with
arteriovenous malformations (AVMs),intracranial aneurysms, or certain medications
(eg, anticoagulants and amphetamines)
(Qureshi et al., 2001).
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The patient with a hemorrhagic stroke can
present with a wide variety of neurologic
deficits, similar to the patient with ischemic
stroke. A comprehensive assessment will
reveal the extent of the neurologic deficits.
Many of the same motor, sensory, cranial
nerve, cognitive, and other functions that aredisrupted following ischemic stroke are
altered following a hemorrhagic stroke.
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In addition to the neurologic deficits that
are similar to ischemic stroke, the
patient with an intracranial aneurysm orAVM can have some unique clinical
manifestations. Rupture of an aneurysm
or AVM usually produces a sudden,unusually severe headache and often
loss of consciousness for a variable
period.
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At times, an aneurysm or AVM leaks
blood, leading to the formation of a clot
that seals the site of rupture. In thisinstance, the patient may show little
neurologic deficit. In other cases, severe
bleeding occurs, resulting in cerebraldamage followed rapidly by coma and
death.
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Prognosis depends on the neurologic
condition of the patient, age, associated
diseases, and the extent and location ofan intracranial aneurysm. Subarachnoid
hemorrhage from an aneurysm is a
catastrophic event with significantmorbidity and mortality. (Pfohman &
Criddle, 2001).
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Demographic data
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Name: R.O.D. Age: 52 yo Gender: Male
Civil Status: Married
Address: 109 Anabu 1-A Imus, Cavite
Birth date: 10/10/1957 Birth Place: Bicol
Religion: Roman Catholic
Language Spoken: Filipino
Admitting Diagnosis / Impression: CVA , hemorrhagic Admitting Physician: Dr. J. Sembrano
Source ofInformation: Wife
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NURSING HISTORY
ChiefComplaint: LOC
History of present illness: The patient is a
known cause of HPN was to maintained onClonodine and Lanoxine with poor
compliance few monitor PTC, patient was
noted to home loss of consciousness
GCS 3
Pupil 2-3 cm
CH Brudzinki
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Past Medical History
Hospitalization (Cause / organ
damage / Removed / Treatment):
Gallblader stone
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Family History
Parents: Mother died of stroke
Sibling: Hypertension
Spouse: None Children: None
Illness in the family similar to the patients:
Hypertension in the family
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Physical assesment
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Vital signs
The patient is afebrile with a
temperature of 36.5 C . Pulse
rate was 118 beats per minute.Patient is tachycardic.
Respiratory rate of 22 cycles perminute and his blood pressure
was 280/120 mmHg.
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General survey
The patient is in distress , bedridden
and has cardiopulmonary distress.
Patient is comatose GCS:3,incoherent and disoriented.
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Skin
The patient has pale complexion,
the texture smooth, skin turgor was
good, warm and dry.
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Head
head is normocephalic. The hair is fine,
equally distributed and has dandruff seen on
the scalp. The pupil size: 2 3mm and it
reacts to light sluggishly with unequal
constriction.
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Ears
The ears are symmetrical.There
were no discharges.
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Nose
There were no discharges and
mucosa is pink.
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Mouth
He has pale lips, the speech is not
assessed because the pt. is comatose
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NECK
The trachea is in the midline.
There are palpable cervical
lymph nodes. Patient neck is
rigid.
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Chest and lungs
The patient is tachypneic with 35 cycles
per minute with occasional dyspnea.
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abdomen
The bowel sounds are normoactive.
with positive dullness over the right
upper quadrant. There are notenderness.
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BACK ANDEXTREMITIES
The peripheral pulses are symmetrical, equal and faint in the
upper extremities. The nail beds are pallor. The muscle tone and
strength is cachexic. The bilateral lower extremities are weak and
have muscle dystrophy.
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Neurologic assessment
Motor response- 1
Eye response- 1
Verbal response- 1
------GCS 3
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Pathophysiology
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Laboratory studies
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Examination:CT scan: Cranial CT Scan plain
There is a massive hematoma seen in the cerebellum with intraventricular
hemorrhagic extravasations into the third ventricle the lateral ventricles are
dilated. There are also small malacic changes in both frontal periventricular
regions.
Remarks:
Massive cerebellar hematoma with intraventricular extravasations,
brainstem edema, obstructive hydrocephalus, chronic frontal
periventricular ischemic infarcts.
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Drug study
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Nursing care plan
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medicine
Comply with the treatment regimen.
Do not use any medicines, over-the-counter drugs, vitamins,
herbs, or food supplements without first talking to caregivers or
physician.
Do not quit taking your medicines until you discuss it with yourcaregiver. If you are taking medicine that makes you drowsy, do
not drive or use heavy equipment.
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Exercise
Regular exercises are good for the heart and lungs and could
stimulate proper circulation and oxygenation in the body.
Regular exercise should be minimal and basic in order not to
increase the workload of the heart
Follow your doctors recommendations for physical activity.
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Treatment
. Instructed client for strict compliance of treatment regimen.
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Hygiene
- Good oral hygiene and proper dental care apply to all age
groups but the needs of the elderly population can be slightly
different than the needs of the younger people. Client should also
observe regular hand and body hygiene to decrease the risk of
acquiring infection. Daily bath is recommended as well asfrequent hand hygiene, not only for the client but also for the
clients significant others.
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Out patient
Seek medical advice only from doctors
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Diet
Limiting the amount of fat to no more than 25 to 35% of daily
calories is recommended to promote good health.
. A low-fat diet also helps lower high total and LDL (the bad)
cholesterol levels, another risk factor for coronary artery disease.
Eating at least five servings of fruits and vegetables daily candecrease the risk of coronary artery disease.
The diet should contain the recommended daily requirements of
vitamins and minerals. Vitamin supplements are not considered
an acceptable substitute for a healthy diet
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Spirituality
Always pray for guidance and strength
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Final evaluation
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After the continuous treatment which consists of the medical and
the nursing management of the patient, a development of her
present health status is projected. Continuous administration of
therapy had partially alleviated the clients suffering from the
disease. Although disease process is incurable and has thetendency to attack again ones life, if the client will undergo
treatment, by some means this will help reduce the sufferings of
the client brought by the disease. But if the client will not subject
herself to any medical management, this could lead to further
complications; signs and symptoms of the disease will progressand will evidently manifested by the client. Furthermore, this
would worsen the condition and eventually leads to death.
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Evaluation of learning outcomes
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EVALUATION
After our thorough assessment, comprehensive
observation, interpretation of the patients laboratory results, the
nursing responsibilities of the prescribed medication, knowing the
factors that affect the disease process and the nursing
interventions rendered to our patient, we came up with thefollowing evaluations:
General:
The group was able to present all the accumulated information
about our patients case, duly diagnosed with Cerebrovascular
Accident Bleed, has improved our skills and knowledge pertainingon caring patients with the above mentioned disease.
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Specific:
1.) The incidence of CVA Bleed in global, national, and local
setting was determined.
2.) The inclusive assessment of the patient involving
biographical data, chief complaint upon admission, past andpresent medical history, personal, family and socio-economic
status as an apparent substantiation to the condition were
present.
3.) Review of systems through detailed but comprehensive
physical assessment was conducted.
4.) The significant diagnostic test and laboratory examinations,
comparing abnormal results from normal values with its
corresponding interpretation in relation to the current status of the
client was determined.
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5.) The predisposing and precipitating factors that contributed to
the disease process and present comprehensively in a written
and diagrammatic illustration were identified.
6.) The pathophysiology of CVA Bleed and its symptomatology
and complications of the client was discussed. 7.) The clients condition in the ICU from the day of admission
until the student nurses assessment was determined.
8.) The pharmacological management of CVA Bleed, and its
nursing considerations were enumerated.
9.) Three (3) prioritized health needs/ problems of the client and
formulate nursing plan as a framework of care identified and
applied.