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    HIGH RISK NEWBORNS

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    SGA Preterm, term, postterm

    (+) IUGR or failed to grow at expected rate

    Causes:

    Mothers nutrition (Adolescents)

    Placental anomaly

    Developmental defect

    Placental damage

    Systemic disease (DM)

    Smokers or use of narcotics

    Intrauterine infection

    Chromosomal abnormality

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    Assessment:

    Perinatal assessment

    FH than expectedUltrasound: small size

    BPP: Poor placental perfusion

    NSTPlacental grading

    AF amount

    Ultrasound exam

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    Appearance

    Small liver

    Poor skin turgor

    Large head

    Widely separated skull sutures

    Hair is dull

    Sunken abdomen Cord appears dry and stained yellow

    Better developed neurological responses, solecreases, ear cartilage

    Skull may be firmer Alert and active

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    Lab. Findingshct >65 70% exchange

    transfusion

    RBC blood viscosityacrocyanosis

    Hypoglycemia (

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    Nursing Diagnoses: Ineffective breathing pattern r/t underdeveloped body

    systems

    Resuscitation

    Observe RR and character

    Risk for Ineffective thermoregulation r/t lack of SC fat Control environment

    Risk for Impaired Parenting r/t Childs High Risk

    Status and Possible Cognitive or Neuro. Impairment

    from lack of Nutrition in Utero Discuss to parents ways to promote infants

    development

    Provide toys suitable for age

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    Assessment:

    Uterus is unusually large for the date ofpregnancy

    Sonogram Confirm

    NST assess placental perfusion

    Assess lung maturity by Amniocentesis CPD, Shoulder dystocia

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    Appearance:

    Immature reflexes

    Extensive bruising or birth injury

    Ecchymosis, jaundice, erythema

    Clavicle or cervical nerve injuries

    Asymmetry of anterior chest

    Unresponsive or dilated pupils

    Seizure

    Prominent caput succedaneum, cephalhematoma or Molding CV Dysfunction

    Polycythemia

    (+) Stress on the heart

    (+) Cyanosis Transposition of Great Vessels

    Hypoglycemia

    glucose to sustain the weight

    (+) DM mother glucose in utero insulin production continues up to 24 hrs of life Rebound hypoglycemia

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    Nursing Diagnoses:

    Ineffective Breathing Pattern r/t Possible Birthtrauma

    Risk for Imbalanced Nutrition Less than bodyrequirements r/t additional nutrition needed tomaintain weight and prevent hypoglycemia

    Breastfeed immediately

    Supplemental formula feedings

    Risk for Impaired Parenting r/t High risk status

    Needs the same developmental care

    Encourage parents to treat their baby as a fragileNB

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    PRETERM

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    PRETERM Causes:

    Low socio economic level Poor nutritional status

    Lack of prenatal care

    Multiple pregnancy

    Previous early birth

    Cigarette smoking

    Age of the mother

    Order of birth

    Closely spaced pregnancies

    Abnormalities of the mothers reproductive system Infections

    OB complications

    Early induction of labor

    Elective CS

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    Assessment:

    History:

    Pregnancy history

    Appearance: Appears small and underdeveloped

    Head is disproportionately large (3 cm or >chest)

    Skin is unusually ruddy

    Veins are easily noticeable

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    Acrocyanosis

    Covered with vernix caseosa

    Lanugo is usually extensive

    Few or no creases on soles of feet Eyes are small

    Myopia

    Immature ear cartilage, pinna falls forward

    Ears appears large in relation to head

    Less active, rarely cries

    (+) Cry; weak and high pitched

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    Potential Complications:

    Anemia

    Normochromic, normocytic anemia

    Reticulocyte count

    Pale,lethargic and anorectic

    Keep a record of the amount of blood drawn

    Give DNA recombinant erythropoietin BT, Vit. E and iron

    Kernicterus

    Acidosis

    albumin bind to indirect bilirubin

    (+) Jaundice phototherapy or exchangetransfusion

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    Persistent PDA

    surfactantblood from pulmonary artery tolungs Pulmonary artery PDA

    Hydrate

    Give Indomethacin or Ibuprofen

    Complication of Indomethacin: Oliguria

    monitor UO closelyPeriventricular/ Intraventricular Bleeding

    (+) Fragile capillaries and immature cerebralvascular development

    (+) Rapid change in cerebral BF capillariesrupture

    Hypoxia

    Pneumothorax

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    Nursing Diagnoses:

    Impaired Gas Exchange R/T Immature Pulmonary Function

    20 secs surfactant alveolar collapse

    (+) Breech expel meconium aspiration inflammation or pneumonia

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    Give mother O2

    Maternal analgesia and anesthesia

    Preterm must be resuscitated within 2 mins after

    birth

    Keep infant warm

    Carry out all procedures gently

    100% O2: 2 Dangers:

    Pulmonary edema

    Retinopathy of prematurity

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    Risk for Deficient Fluid Volume R/T Insensible WaterLoss at birth and small stomach capacity

    Normal glucose: 40-60 mg/100 ml

    Specific gravity: 1.003 1.030

    UO: 1 ml/kg/hr

    IVF 160-200 ml/kg/BW umbilical venous catheter

    Monitor weight, UO and specific gravity andelectrolytes

    Measure UO by weighing diapers

    Preterm: 40-100 ml/kg x 24 hrs ; 1.012

    Term: 10-20 ml/kg x 24 hrs ; 1.030

    Test urine for glucose and ketones

    Keep a record of all blood drawn

    Check for blood in stool

    Determine possible cause of hypovolemia

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    Risk for Imbalanced Nutrition Less than Body Requirements

    R/T Additional Nutrients Needed for Maintenance of rapid

    growth, possible sucking difficulty and small stomach Feeding Schedule

    IVF feeding may be delayed

    TPN

    Breast, gavage or bottle feeding Get CXR before feeding

    (+) Air in stomach

    Small, frequent feeding (1-2 ml every 2-3 hrs)

    Preterm: 115 140 cal/kg/ BW

    Term: 100 110 cal

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    Gavage Feeding

    (+) Gag reflex 32 weeks 32-34 weeks, ill, (+) RDS Gavage feeding

    Bottle feeding or breast feeding is graduallyintroduced Give softer nipple Observe preterm infant closely Offer pacifier

    Aspirate stomach secretions measure replace >2 ml not allowed (-) Digestion NEC

    Formula: 24 cal/oz preterm

    20cal/oz term Vit. K 0.5 ml Give Vit. E prevent hemolytic anemia

    Breastmilk: Prevents NEC

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    Ineffective Thermoregulations R/T Immaturity

    Keep NB warm during transportation Heat shield or plastic wrap

    Risk for Infection

    Linen and equipment must not be shared

    Staff members must be free of infection

    Hand washing and gowning

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    Risk for Impaired Parenting

    Rocking, singing and talking and gentle holding

    Kangaroo care

    Encourage the mother to express breastmilk

    Encourage mother to come to the hospital and hold thebaby before and after gavage or bottle feeding

    Photograph of baby

    Notes to convey messages from the baby to them can betaped to the incubator

    Sibling should not visit if they have colds or fever, (+)immunization, (-) exposure to communicable diseases

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    Deficient Diversional Activity (Lack of stimulation)

    Organize procedures Shield from noise and light

    Pain should be kept into minimum Look directly at an infant in the straight forward position Provide some talk time Gentle stroke an infants back

    Risk for disorganized infant behavior Modify environment; reduce stimuli Dim the lights; cover the incubator, turn infant to the side, contain

    body with rolled towels

    Offer non-nutritive sucking Maintain quiet hour

    Parental health-seeking behaviors Overprotection is not necessary Basic immunization

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    POST TERM Post term

    >42 weeks Placenta functions effectively for only 40 weeks

    (+) Postterm syndrome: SGA characteristics Dry Cracked (leather like)

    (-) Vernix Light weight meconium stained Fingernails have grown well Alertness = 2 weeks old

    (+) Difficulty establishing respiration Hypoglycemia SC tissue temperature regulation difficult Polycythemia, hct

    nutrition and O2 (+) Neurologic symptoms

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    RESPIRATORY DISTRESS

    SYNDROME Due to blood perfusion of lungs; surfactant (+) Hyaline like (fibrosis) membrane formed from

    an exudate of infants blood lines terminalbronchioles, alveolar ducts and alveoli

    prevents exchange of O2 and CO2 Pathophysiology:

    surfactant (+) areas of hypoinflation pulmonaryresistance blood shunts to foramen ovale and ductus

    arteriosus

    lung perfusion

    surfactant (+) Hypoxia, Co2 (+) Lactic acid acidosis

    vasoconstrictionpulmonary perfusionsurfactant production alveoli collapse withexpiration

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    Assessment: Lowbody temperature Nasal flaring

    Retractions Tachypnea (>60) Cyanosis Expiratory grunting distress

    Seesaw respiration Heart failure Pale, gray skin Periods of apnea Bradycardia Pneumothorax

    CXR: Diffuse pattern of radiopaque areas groundglass (haziness)

    Blood gas: Respiratory acidosis C/S: R/O -hemolytic group B strep

    May start Penicillin or Ampicillin + Gentamycin or

    Kanamycin

    Management:

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    Management:

    Surfactant replacement

    Sprayed into lungs by syringe or catheter by ET tube

    Head held upright and tilted downward

    AW should not be suctioned

    (+) Ventilator needs close observation

    O2 administration

    Continuous Positive Pressure (CPAP) or Assisted Ventilation

    with Positive End Expiratory Pressure (PEEP) Keep alveolifrom collaping

    Cx: Retinopathy of prematurity

    Management:

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    Management:

    Ventilation

    Normal I/E ratio: 1:2

    Infant ventilators: 2:1

    Complications:

    Pneumothorax

    Impaired CO

    ICP and arterial pressure

    Hemorrahge

    Limit fluid intakepulmonary artery pressure

    Indomethacin or Ibuprofen closure of PDA

    Complications:

    Renal function platelet function

    Gastric irritation

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    Additional Therapy

    Muscle relaxants Pancuronium (Pavulon) IVspontaneous respiratory

    function

    Pressure mechanical ventilation

    Pneumothorax

    Needs critical observation

    Frequent ABG

    Atropine and Prostigmine should be available

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    Liquid Ventilation

    Use of Perflourocarbons

    (+) O2 Perflourocarbons pick up and carry O2 distends

    the lungs

    exchange of O2 Can be used to deliver O2

    Nitric Oxide

    Cause of pulmonary vasodilation

    Prevention:

    Sonogram

    Document: Lecithin should exceed Sphingomyelin (2:1)

    MgSO4 or Terbutaline prevent preterm birth

    SteroidsLecithin

    Betamethasone 12-24 hrs; 24-34 wks AOG (takes effectsbefore 24-48 hrs)

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    MECONIUM ASPIRATIONSYNDROME

    (+) Meconium at 10 weeks AOG

    (+) Breech expel meconium in amniotic fluid

    (+) Hypoxia (+) Vagal reflex relaxation of rectal

    sphincter Appearance; Green to greenish black

    May be aspirated in utero or with 1st breath

    (+) Respiratory distress:

    (+) Inflammation of bronchioles Mechanical plugging

    surfactant production

    Hypoxemia, CO2, (+) shunting

    (+) secondary infection Pneumonia

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    Assessment:

    Apgar score

    Tachypnea, retractions, cyanosis

    Suction with bulb syringe or catheter while at theperineum

    Do not administer O2 under pressure

    Enlargement of AP diameter (barrel chest) ABG: pO2, pCO2

    CXR: Bilateral coarse infiltrates in the lungs, (+) spaces ofhyperaeration (honeycomb effect)

    Diaphragm pushed downward

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    Management:

    Amniotransfusion

    CS birth

    Tracheal suction, O2, assist ventilation

    Antibiotic therapy

    Observe closely for signs of trapping air in the alveoli

    Observe for signs of heart failure due to shunting ofblood from pulmonary artery to aorta (HR, respiratory

    distress)

    Maintain a temperature neutral environment

    Chest physiotherapy ECMO

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    SIDS Unexplained death in infancy

    Commons among:

    Infants of adolescent mother

    Closely spaced pregnancy

    Underweight and preterm infants Bronchopulmonary dysplasia

    Twins

    Narcotic dependents

    Peak age: 2-4 mos.

    Contributory Factors:

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    y Prolonged, unexplained apnea Viral respiratory or botulism Pulmonary edema Brainstem abnormality

    Neurotransmitter deficiency HR abnormalities Distorted familial breathing patterns arousal response surfactant Sleeping prone

    Infants are well nourished Slight head cold Dies with laryngospasm Blood flecked sputum or vomitus in mouth or on bed clothes Autopsy:

    Petechiae in the lungs Mild inflammation and congestion in respiratory tract

    Inform parents that the death was unexplained

    Give assurance that SIDS is a disease of infants

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    HYPERBILIRUBINEMIA Hemolytic Disease of the NB

    Rh incompatibility Mother: Rh (-)

    Fetus: Rh (+)

    Sensitization: Mother begins producing antibodiesagainst D antigen (72 hrs)

    2ndpregnancy: D antibody destroy fetal RBC Requires intrauterine transfusions

    May induce preterm labor

    Administer Phenobarbital to women speeds liver maturity

    ABO Incompatibility

    Mother: Type O Fetus: Type A or B or AB

    Not born anemic

    Hemolysis begins with birth; may continue up to 2 wks

    Preterm: Not affected

    Increase reticulocyte count

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    Assessment: Percutaneous umbilical blood sampling

    anti-Rh titer (Indirect Coombs test) Mother (+) Abs Fetal erythrocytes

    (-) Pale Enlarged liver and spleen (+) Edema Severe anemia Heart failure (Hydrops Fetalis)

    (+) Progressive jaundice (+) Preterm: (+) Hemolysis Liver cannot convert

    indirect to direct bilirubin (+) Breastfeeding: (+) PrenanediolProgesterone interferes with conjugation of indirect bilirubin

    Normal bilirubin: 0-3 mg/100ml >20mg/dl or 12 mg/dl in preterm Kernicterus

    Hypoglycemia Hgb

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    Management: Early feedingperistalsis

    Bilirubin incorporated into feces

    Phototherapy Specialized light: Quartz halogen, cool white day light

    or special blue fluorescent light

    12-30 inches above the bassinet or incubator

    Infant is undressed except for diaper Term NB: Bilirubin 15 mg/dl; Preterm: 10-12 mg/dl

    Eyes must always be covered

    Stool: Bright green, loose, irritating to skin; Urine: Darkcolored

    Assess skin turgor, I/O DHN

    Monitor axillary temperature

    Infant should be removed for feeding

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    Exchange Transfusion

    Aspirate stomach

    Umbilical vein is catheterized

    Draw small amounts of blood (2-10 ml) replace with

    equal amounts of donor blood

    Blood is exchanged slowly 1-3 hrs (automatic pumps)

    End: hct, bilirubin, Ca+, glucose, culture

    Repeat exchange transfusion

    Done for hyperbilirubinemia or polycythemia, bloodincompatibility, heart failure

    Keep NB warm

    Blood should be given at room temperature

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    Use only commercial blood warmers Albumin may be administered 1-2 hrs before Monitor rate of flow of albumin Blood type used: O-

    Monitor HR, RR and BP Blood contain acid-citrate-dextrose (ACD) as anticoagulant

    Ca acidosis Ca gluconate is given every 100 ml of blood

    Citrate-Phosphate Dextrose (preservative) hyperglycemia

    insulin

    hypoglycemia Heparinized blood interferes with clotting

    glucose hypoglycemia Give Protamine sulfate

    Observe infant for umbilical vessel bleeding

    (+) Redness or inflammation (+) infection

    Report changes with V/S Take and record glucose 1 hr after Monitor bilirubin 2 or 3 days after May administer erythropoietin

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    SEPSIS

    Early onset - birth to 7 days Pathogens: group B strep, E.coli, Klebsiella,

    Listeria

    Late onset - 7 to 28 days Pathogens: early onset pathogens PLUS

    Staph. aureus, Neisseria gonorrhea

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    Symptoms: poor feeding, vital signinstability, leukocytosis, leukopenia,thrombocytopenia, hypoglycemia,

    hyperbilirubinemia, altered consciousness Evaluation: CBC with diff (band/pmn>0.2

    or ANC

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    Ampicillin(100 mg/kg/24 hrs) PLUSgentamicin (3-5 mg/kg/24 hrs)

    Ampicillin(100 mg/kg/24 hrs) PLUScefotaxime (100-150 mg/kg/24 hrs)