special edition impuls magazine

1

Association Periodical

Contents

im PULSSpecial Edition July 2011Relevant News of the

www.vereinigung-schwingungsmedizin.de

Special Edition

Structure and Application of the newRayonex Analysis and

Harmonising System (RAH)

Rayocomp PS 10: from program version 02.36Rayocomp PS 1000 polar: from program version 93

Token fee: € 20

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Masthead

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1 Foreword to the offprint . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 4

2 The basic ideas behind the RAH . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 5-8

3 The design of the RAH . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 9

4 The structure of the RAH . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 10-12

5 Use of the RAH . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 12-16

6 Application in the Rayocomp PS 10 and PS 1000 polar . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 17-19

7 Integration of new programs . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 20-21

8 Appendix I: Currently available programs . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 22-61

9 Appendix II: Organ-specific meridian and pathogen tables . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 62-68

10 Appendix III: Information on bacteria, viruses, parasites and fungi . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 69-99

11 Appendix IV: Use of bioresonance for detoxification purposes. . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 100-111

12 Appendix V: The new analysis support by means of test protocols . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 112-170

Table of contents

4

1 Foreword to the offprint

The mission of the Vereinigung zur Förderung der Schwin-

gungsmedizin e.V. (VFS - Association for the Promotion of

Vibrational Medicine) is firmly anchored in its name: for

over 10 years, the VFS has been pursuing the objective

of helping as many people as possible to appreciate the

benefits of vibrational medicine and, in particular, bioreso-

nance according to Paul Schmidt.

Although classic, orthodox medicine has neither accepted

nor recognised the effect of bio-energetic vibrations, an

increasing number of therapists at both national and inter-

national level successfully use this method every year: over

5,500 therapists in Germany alone.

The VFS has already published its history in several offprints.

We are of the opinion that it is particularly important, since

the new Rayonex analysis and harmonisation system –

abbreviated to RAH – combines a great deal of benefits and

the architecture of the method is designed such that it can

win over the majority of people.

In the form of this offprint, we would like to present the

basic ideas behind this method, to explain the design and,

ultimately, to give tips for the use of both the Rayocomp PS

10 and the Rayocomp PS 1000 polar.

A note for non-members of the VFS: there is a registration

form on the last page. You can use this to join the Association

for the Promotion of Vibrational Medicine. As a member,

you will receive two or three times a year the association’s

journal, IMPULS, which always keeps you up to date with

the latest happenings in the world of vibrational medicine.

Furthermore, you can participate in the annual congress of

the Association for the Promotion of Vibrational Medicine.

We would be delighted if you were to take us up on our

offer and to support us in the form of your membership.

Thanks in advance!

The editorial office of the Association for the Promotion

of Vibrational Medicine

5

2 The basic ideas behind the RAH

One of the very special advantages of bioresonance accor-

ding to Paul Schmidt and vibrational medicine is its multi-

tude of uses. Here is a list of them:

• recognition of causal influences

• allergy testing and allergy harmonisation

• acupuncture vibrational medicine

• organ-specific analysis and harmonisation

• consideration of the psyche

• recognition of pathogen-caused illnesses

• testing of the individual physical energetics

• the integration of bodily secretions such as blood, urine,

saliva and stool

• qualification of water or environmental influences

• development of energy-optimised preparations

• test of preparations and medicines

• use on animals and plants

It is easy to comprehend the resulting range of applications

and the automatically growing number of therapists who,

over time and with a great deal of experience, have become

experts in a particular field. Furthermore, a frequently

observed phenomenon is that, thanks to successes in very

specific illnesses, therapists have often become experts

in these very specific fields of application in vibrational

medicine.

Today, for instance, we have experts whose entire surgeries

are designed to meet the requirements of the treatment

of pathogen-caused illnesses, and experts in the fields of

detoxification, the energy balance, emotional disbalances,

etc. In the past, these experts have created therapy pro-

grammes on the individual topics which have been applied

successfully for years: some of them thousands of times

over. One of the claims of the RAH is therefore to bring

together the best programmes of experienced therapists.

Here is an example: Dr med. Yayama, Japanese specialist

in the use of vibrational medicine, created programs for his

surgery close to Fukuoka with which he can stimulate the

targeted production of adenosine triphosphate – ATP – in

the cell structures of various organs. Any work carried out

in the cells – irrespective of whether this work is of a che-

mical, osmotic or mechanical nature - requires energy. ATP

provides this energy. It is fair to say that ATP is used as the

basic source of energy for all the energy-consuming pro-

cesses of living things. It was Dr med. Yayama’s idea to find

frequency patterns for the individual organs with which, via

the bioresonance devices (RPS 1000 polar and RPS 10) the

production of ATP is selectively stimulated. According to his

– easily comprehensible – experience, it doesn’t make much

sense to treat living things if they don’t have the energy to

get the regulation going.

A basic requirement of the RAH can be derived from this

example – namely the integration of many experts who

make available their best programs for general use. It is

often said that very successful therapists are not prepared

to universally share their knowledge. The RAH is evidence

that the opposite can also be true!

The example also shows, however, that future expert systems

– such as the RAH – always depend on the integration of

many therapists. Frequently, the created programs are the

life’s work of the therapist in question. Due to the time limit

alone, it isn’t possible for one single individual to manage

something of this magnitude on his or her own. The RAH is

an open system which, following a relevant examination,

also provides new therapists and their programs with a

platform for general use. This open approach is expressly

supported by the Association for the Promotion of

Vibrational Medicine, since one of our objectives is to bring

together the experiences of successful therapists.

6

The panel of experts of the new RAH at the first meeting in the Sauerland Pyramids.

But who decides which programmes are included in the system?

• The provision of expert programs is an inestimable

advantage, especially for the inexperienced new-comer

to the world of vibrational medicine. This means that the

therapist does not have to work out the information from

scratch – instead, the therapist can take advantage of

programs which have been tried and tested thousands

of times over. This means that therapists new to the

world of vibrational medicine are able to successfully use

vibrational medicine much earlier than used to be the

case.

• It is the nature of an open system that it keeps on grow-

ing. The Association for the Promotion of Vibrational Me-

dicine has positively noted that many experts have made

A panel of experts, comprising the therapists, the Associa-

tion for the Promotion of Vibrational Medicine, representa-

tives from Germany and abroad, development engineers, the

therapy centre and the School of Non-medical Practitioners

was formed in the Sauerland Pyramids. Its objective is to

take into consideration as many different interests as possi-

ble, at the same time forming a decision-making committee

which assesses and integrates new suggestions.

It offers further advantages:

their programs available to the RAH in an astonishingly

short time. There are currently over 1249 expert programs

for analysis and harmonisation available. The integration

of many experts means that development is expected to

be very dynamic.

• A further, inestimable advantage of an open system is the

wide application range. Each and every therapist places

individual demands on an analysis and harmonisation

system. However, if numerous therapists contribute their

approaches to an expert system, the user can pursue just

as many therapeutic approaches. This will fill an incre-

asing number of people with the enthusiasm to use the

RAH, and vibrational medicine on the whole.

Prerequisite: the programs must have successfully proven

their practical value over an extended period, and relevant

progress reports must be available. Following relevant tests,

the panel of experts will then decide on the integration of

new programs.

7

The arrangement of frequencies in the RAH

Reduction of the harmonisation time

It is time to delve a bit further into the RAH itself. So-called

programs for analysis and harmonisation purposes are used

in the RAH. These are compilations of frequencies which

are not applied in succession: instead, they are applied as

a whole, as frequency patterns. Here is an example: if you

examine a precious stone using bioresonance according to

Paul Schmidt for its typical frequencies, you can find 18

various frequencies, for instance. By using bioresonance

according to Paul Schmidt, you could use one after the other,

including for harmonisation purposes. RAH takes a different

approach. It applies the 18 frequencies of the precious

stones in one frequency pattern. This allows you to come

much closer to the nature of the stone as a whole. Hence,

when programs are mentioned in conjunction with RAH,

Experience shows that the body has a great deal of work to

perform when wide frequency spectrums are offered during

the harmonisation. In order to achieve treatment times

which are as short as possible in the surgery, however, a

new transfer system for the frequency spectrums has been

developed for the RAH.

In the RAH, frequency structures are transferred by means

of so-called transfer values which correspond to the Schu-

It was of special importance to the VFS to create the

guidelines for the integration of new programs as quickly

as possible. Please see Chapter 7 to this special publication

for the guidelines.

it always refers to the compilations of various frequencies

which, seen as a whole, best reflect the nature of the relevant

structure. If, for example, program 42.20 (nasal sinuses) is

set, this always refers to the frequency spectrum which

describes the structure. Please don’t mix these up! 42.20 is

not the individual frequency – it is the indenture number of

the program which covers the frequency spectrums.

If, for example program 42.20 is used for the analysis, one

frequency spectrum is always tested. This enables a clearer

description of the structure than the former description.

Double occupancy of individual frequencies is now a thing

of the past.

man frequency range. Experience shows that the length of

the harmonisation process can be reduced to almost a third

if the transfer values are amended during harmonisation.

This is comparable with an acoustic signal that, after a long

time, you no longer hear or, if you do, then you perceive it as

considerably weaker. If, however, you permanently amend

the pitch, new attention signals for the organism keep on

being produced. The following diagram makes this clear:

8

Reduction of the harmonisation time by attention signals

On the left-hand axis, you can see the therapeutic intensity.

On the bottom axis, you can see the harmonisation time.

If you now transfer a frequency pattern with unchanged

transfer values to the organism, the therapeutic intensity

is reduced over time. Hence, longer harmonisation times

are required. In RAH, on the other hand, the transfer values

are permanently changed, maintaining the high therapeutic

intensity of the frequency spectrums. The result: a consi-

derable reduction of the harmonisation time Some wide fre-

quency spectrums can be harmonised in just a few minutes.

A further yet only indirect wish of many therapists has been

realised with the new system It is now possible to visualise

the results of the analysis in the Rayocomp PS 1000 polar.

The interferences discovered can be visually shown to the

patient and corresponding print-outs can be made.

Let’s discuss the design of the RAH now.

9

3 The design of the RAH

The new system has a clear medical structure, both with

regard to the analysis and the harmonisation.

The design and the structure go back to Rayonex’s own

training programme by the School of Non-medical Practiti-

oners. There, the teaching material has been completely re-

vised. At Rayonex, training to become a non-medical prac-

titioner includes the additional training of bioresonance

according to Paul Schmidt.

The design of the RAH also includes the cause-orientated

approach as well as, in particular, energetics.

The next page contains a diagram which explains the basic

design.

If you look at the design as a whole, right at the front you’ll

find the program for the preparation of the analysis (fur-

ther information to follow in this offprint). This is followed

by programs for energetics, in particular for vitalising, for

the energy balance, for initial control, for polarity and the

chakras. Programs for the meridian courses are also part of

this category.

The field of energetics is part of the extensive field of causes.

Programs pertaining to e-smog, geopathics, the acid-base

balance, vital substances, harmful substances, bacteria, vi-

ruses, parasites and fungi are located here.

You then find programs pertaining to the physiology (fre-

quency patterns of healthy structures) and to pathology

(frequency patterns of sick structures). This is depicted al-

ternately. Even program numbers, such as 30.00, descri-

be physiological structures. The following odd program

numbers (31.00) describe the pathological structures.

There then follow the 70s programs which depict the cause-

orientated system programs. Although the following pages

will go into this in more detail, let’s say this for now: all

programs commencing with the number 70 are so-called

system programs. For the pertinent selected field, these

contain the relevant physiological structures, the involved

meridians, the potential pathogens and relevant immune-

stabilising frequency structures.

The structure is rounded off by programs for pain, the

psyche, for stress management, and programs for the teeth

/ milk teeth and by special programs for the analysis of

Bach flower extracts, Schüssler’s tissue salts and frequency

structures on the periodic system of the elements (PSE).

The training material of the non-medical practitioner training

in the Sauerland Pyramids served as the model for the RAH.

10

EnergeticsVitalisation, energy, polarity, pre-control, chakras 01.00 ff.

Meridian courses 02.00 ff.

Analysis preparation 00.00 ff.

Psyche 72.00 ff.

Stress 75.00 ff.

Bach flower extracts 81.00 bis 81.38

Schüssler tissue salts 82.00 bis 82.27

Causes

Electrosmog 04.00 ff.

Geopathy 05.00 ff.

Acid-base balance 06.00 ff.

Vital substances 07.00 ff.

Harmful substances 08.00 ff.

Bacteria I / II 20.05 ff. / 21.05 ff.

Viruses I / II 22.05 ff. / 23.05 ff.

Parasites I / II 24.05 ff. / 25.05 ff.

Fungi I / II 26.05 ff. / 27.05 ff.

Cause-orientated system therapy

70.10 bis 70.46

Physiology and pathology

Cell and tissue 30.00 ff. / 31.00 ff.

Blood 32.00 ff. / 33.00 ff.

Immune system 34.00 ff. / 35.00 ff.

Lymphatic system 36.00 ff. / 37.00 ff.

Cardiovascular system 38.00 ff. / 39.00 ff.

Heart 40.00 ff. / 41.00 ff.

Respiratory tracts 42.00 ff. / 43.00 ff.

Kidneys / urinary organs 44.00 ff. / 45.00 ff.

Digestive system 46.00 ff. / 47.00 ff.

Liver, gall-bladder, pancreas 48.00 ff. / 49.00 ff.

Metabolism 50.00 ff. / 51.00 ff.

Locomotor system 52.00 ff. / 53.00 ff.

Nervous system 54.00 ff. / 55.00 ff.

Visual organ 56.00 ff. / 57.00 ff.

Hearing organ / vestibular organ 58.00 ff. / 59.00 ff.

Skin / hair 62.00 ff. / 63.00 ff.

Hormone system 64.00 ff. / 65.00 ff.

Female sex organs 66.00 ff. / 67.00 ff.

Male sex organs 68.00 ff. / 69.00 ff.

4 The structure of the RAH

Periodic table of the elements 85.00 bis 86.04

Teeth / milk teeth 76.00 ff. / 77.00 ff.

Pain 71.00 ff.

Own programs 95.00 ff.

11

How was the design in the bioresonance devices Rayocomp PS 10 and the Rayocomp PS 1000 polar realised?

Let’s now take a closer look at one individual program structure, e.g. that of electrosmog.

Here is the excerpt from the program structure:

04.00 Electrosmog, total

04.10 Electric and magnetic alternating fields

04.20 Pulse-modulated radiation, total

04.21 Mobile communications

04.22 UMTS

04.23 DECT (cordless telephone)

04.24 WLAN

04.25 Bluetooth

04.26 Satellite transmission

04.27 Wi Max

04.30 Radiation, protection

Example: the program structure for e-smog

The program number for e-smog is 04.00. The description

is “electrosmog, total”. The information “total” means that

all following frequency structures are contained within the

04.00. Hence, the frequency structures of 04.10, of 04.20

and the sub-programs 04.21 to 04.27 as well as 04.30. This

design brings with it great advantages. When the program

number 04.00 is tested and does not need to be harmonised,

we can do without all further tests of the sub-programs. If,

for instance, a stress on 04.00 can be tested via 04.10 to

see whether the problem is caused by electric or magnetic

alternating fields, or even via 04.20 to see if the e-smog

problem has been caused by pulse-modulated radiation.

Program number 04.20 also has the number suffix: “total”.

The objective was the integration of the new RAH in both

the Rayocomp PS 10 and the Rayocomp PS 1000 polar.

Change-over of the hardware is necessary for operating

the RAH in the Rayocomp devices. To this end, the devices

need to spend a few days in the Rayonex factory. Devices

delivered after 01.07.2009 already have the new hardware

integrated into them, which means that only the module

needs to be activated in order for it to work.

This means that this program number contains all following

programs. If there is a problem here, you can very soon find

out via the individual programs (04.21 to 04.27) whether

the problem has been caused by a DECT telephone (cordless

phone) via program number 04.23.

However, the structure also enables direct testing of pro-

gram number 04.23 if there is an indication that this is ne-

cessary.

By means of the design presented, testing from approximate

to fine is supported, the objective always being to reduce

the necessary tests. The presented structure enables the

therapist to decide the depth at which he or she tests.

This type of program structure is reflected in all fields. Hence

program number 56.00 describes the entire physiology of

the eyes, the sub-groups, the corresponding differentiated

fields of the eye (chambers of the eye, skins, musculature,

nerves, etc.)

Please see Appendix I of this offprint for a precise depiction

of all currently available programs and their sub-programs.

There are also some depicted in grey. These are currently in

the development phase, and will be available in the next

versions of the RAH.

It is very easy to see from the exterior of the Rayocomps

whether the new hardware has already been integrated -

the word “Evolution” is visible on the side of the Rayocomp.

If you can read the word “Evolution” on the side of the

Rayocomps, this means that the hardware necessary for

operating the RAH has already been integrated.

12

5 Use of the RAH

With the following, we would like to display a central

theme, based on which the RAH can be used.

Please note: by means of the descriptions of the use of

the RAH, we are by no means claiming that the method

described here is the only or the only correct one. Naturally,

the individual programs of the RAH can be combined in

radiesthetic precision work. However, this is only one pos-

sible use which is not viable for all therapists. A doctor, for

instance, will not have the time to perform an extensive

range of tests in his surgery. Similarly, therapists will use

the RAH in completely different ways, depending on the

main focus of their treatment. And that is ideal, since one

of the objectives of the RAH is a wide application range.

The RAH distinguishes between the analysis in which in-

terferences can be determined, and harmonisation, whose

function is to selectively stimulate self-regulation mecha-

nisms.

Analysis within the RAH commences with the preparation

for the analysis. This ensures that the patient doesn’t show

any resonance to the transfer values of the RAH. Should a

linear motion be observed on the Rayotensor during testing

of the program 00.00 (analysis preparation), this program is

to be harmonised until a rotation is observed on the Rayo-

tensor. Only then should the actual test commence. Analysis

preparation is therefore a precondition for accurate tests.

Otherwise, it would be possible for all other tests to indi-

cate resonances: however, these wouldn’t come from the

program, but rather from the transfer frequencies required

for the transfer of the programs.

The actual analysis in the RAH commences with the test

on the energetic programs. Patients are frequently not able

to undergo therapy, since the organism cannot convert

the regulations set in motion by the harmonisation. It is

not without good reason that program number 01.00

Vitalisation, total is right at the top of the RAH. It is here

The RAH with all analysis and harmonisation programs is

available in the Rayocomp PS 1000 polar.

The RAH is supported with three modules in the Rayocomp

PS 10. The more economical module 8 only includes hig-

her programs and their frequency structures, whereas the

module 9 makes available all available programs and their

frequency spectrums. The new module M10 also makes

available extensive analysis options and test protocols: see

Appendix V. Appendix I contains a list of all programs and a

list of which programs belong to which module.

13

When is a stress to be categorised as strong?

The cause-orientated therapy approach is the most sustainable

If you set, for example, program 01.00 Vitalisation, total,

the accompanying frequency spectrum is transferred to the

body. First of all, by means of the Rayotensor set at polari-

sator setting N, you test to see whether there is a rotation

(= harmonisation not necessary) or a linear motion (= har-

monisation necessary). If a linear motion is displaced, you

can set the Polarisator to the bipolar function by means of

the button on the Rayotensor. The frequency spectrum has

a greater intensity to the body in this mode. If the line-

ar motion remains, a strong interference is present and we

know that special attention needs to be paid to the tested

program. This course of action is applicable to each of the

other programs. This means that, by means of the polarisa-

tor, it is possible to determine whether interference is weak

or strong and, hence, requires closer observation.

Let’s return to program 01.00 Vitalisation, total. If a strong

interference is present, it is advisable to also test program

The energetics testing is followed by the extensive field

of causal influences. With this, the RAH follows on the

philosophy of Paul Schmidt, who coined the following

phrase back in around 1980: the cause-orientated therapy

approach is the most sustainable

With the accompanying program main groups of 04

(electrosmog) to 27 (fungi), the approach according to Paul

Schmidt is supported. Basic stresses in the organism can

be detected via these programs. Here is an example: there

is no point wanting to treat headaches, migraines or sleep

disorders if no test has been carried out to see whether the

patient suffers from the effects of electrosmog. There have

been numerous cases whereby the actual reason behind

this type of illness has been a radio alarm clock placed

point 02.00 Acupuncture meridians, total. If this also indi-

cates a strong interference, then it is possible to find out

which meridian is strongly disturbed.

For such patients, harmonisation only with program 01.00

Vitalisation, total, 02.00 Acupuncture meridians, total and

program 31.10 ATP production, total, is recommended in

order to ensure that the organism is once more in the posi-

tion to react to a follow-up therapy. This case occurs more

frequently than you’d think! There are therapists who are of

the opinion that the sole activation of the body’s energetics

suffices, since then the organism alone begins to regulate.

Back to the three programs: they should be harmonised for

10 minutes each, resulting in a total harmonisation time of

30 minutes.

Irrespective of which therapeutic approach is favoured, the

RAH offers the right programs for many therapy approaches.

on a bedside cabinet, emitting over 600nT of magnetic

alternating field to the patient the whole night. For this

reason, take the cause-orientated program part during the

test very seriously. In this case too, via testing on the bipolar

polarisator setting, you can detect a strong interference and

selectively attempt to block this. Irrespective of whether the

stress is caused by electrosmog, geopathy, the acid-base

balance, a lack of vital substances, harmful substances or

caused by pathogens. There are many programs available

which support the cause-orientated therapy approach.

Naturally, the cause-orientated programs serve the main

purpose of analysis, since it makes no sense to harmonise

geopathy, for instance, without rectifying the cause.

Such treatment wouldn’t be sustainable. However, after

eliminating the interferences, it would be a good idea to

that the current energetic status can be clarified. If testing

shows a strong blockade, it makes sense to first of all

intensively boost the body’s energetics and to wait with

the actual treatment. Incidentally, the elaboration of the

vitalisation program was done mainly by Wilhelm Hömberg,

member of the board of the Association for the Promotion

of Vibrational Medicine.

14

Why was such a design selected?

Because it provides the structure necessary for the applica-

tion. With it, it is possible on the one hand to test all areas

and, thanks to the testing of the main programs, to exclude

certain organ zones, hence performing a quick yet efficient

basic test.

Frequently, however, patients come with a very specific pro-

blem and would like to have a solution to only this problem,

such as otitis media (inflammation of the middle ear). To

this end, we would like to describe an example of how to

proceed with the RAH in testing and harmonisation.

harmonise the organism precisely in line with the frequency

spectrums on which it had been disturbed for a long time.

Let’s now come to the pathogen programs. The RAH has

a multitude of programs of bacteria, viruses, parasites and

fungi. Always testing every pathogen on every patient

would be hard to realise in practice. Since there is prac-

tically no patient without some pathogenic bacteria, a vi-

rus, a parasite or a fungus, foundation work by the therapy

centre in Melbeck carried out by Ms HP Schußmann and Dr

med. Schußmann on a total of 26,000 patients determined

in which organ / in which organ zones which pathogens

are present. The results of this work were integrated in the

RAH. Ask a therapist today which pathogen usually occurs

in which organs, practically all therapists would have to ad-

mit that they don’t have adequate knowledge to answer this

question. This topic is of extreme importance for this. In this

respect, it is to be seen as positive that the RAH in this area

offers particular support.

Later on in this offprint, a precise description is given of

which pathogens occur in which organs. Frequently, the

testing of many hundreds of pathogens can be reduced to

just a few. The testing of all pathogen programs is always

a good idea if required by the symptoms or if, for reasons

of development, further experiences need to be collected.

Please note: bioresonance frequently shows up pathogens

which can no longer be proven in the organism by orthodox

medicine. Frequently this is a matter of existing energetic

interferences due to a past incidence of pathogens.

Let’s now proceed to the programs concerned with

physiology and pathology. It was pointed out at the start

that the programs pertaining to physiology describe

frequency patterns of healthy structures and that the

programs pertaining to pathology describe frequency

patterns of ill structures. The numbers of the programs have

been designed alternately. Even program numbers, such

as 30.00, describe physiological structures. The following

odd program numbers (31.00) describe the pathological

structures.

A very special program number is cell and tissue which, in

addition to the physiology, contain all detoxification pro-

grams which have arisen from the groundwork done by HP

Gerhard G. Rögele. The entire, aforementioned ATP programs

by Dr med. Yayama are also covered by this. Naturally, the

programs by Dr med. Ulrich – tried and tested thousands of

times – are also reflected in the corresponding parts of the

RAH. Ms HP Rögele contributed special programs pertaining

to the locomotor system and to female illnesses.

In the event of pain in the hearing organ, you can start

with program group 58 straight away, and test to see which

physiological structure is affected in the ear. At the start,

program 58.00 Hearing organ, total is tested. If, in this case,

a linear motion appears on the Rayotensor, then it is evident

that an energetic structure in the hearing organ is affected.

By the way: at this early point, via testing on the bipolar

function, it is possible to gain information about whether

the stress is strong or weak (this has already been descri-

bed in detail above). If you continue testing, via the sub-

programs of program 58, it is possible to find out precisely

where the main problem is: in this case, it is in the middle

15

How would you design the harmonisation process based on these measuring results?

Here is a recommendation:

01.00 Vitalisation, total 5 minutes

02.16 Small intestine meridian 2 minutes

02.18 Kidney meridian 2 minutes

31.10 ATP production overall 3 minutes

58.30 Middle ear, total 5 minutes

then the detected pathogens:

At resonance on N: 3 minutes

At resonance on +/-: 5 minutes

31.50 Detoxification basic program 5 minutes

01.00 Vitalisation, total 2 minutes

ear: program 58.30 shows a linear motion on the bipolar

function.

Now a test needs to be carried out to discover the types of

pathogen in the middle ear. As already mentioned, Appendix

II of this offprint contains the assignment of the pathogens

to the individual organ zones.

The following pathogens are given for the hearing organ,

for instance:

20.12 Beta-haemolytic streptococcus

20.22 Streptococcus mitis

21.88 Rickettsias

22.12 Cytomegalovirus (CMV)

22.13 Epstein-Barr viruses (EBV)

22.15 Herpes simplex

22.17 Herpes zoster

22.64 Chikungunya

23.81 Viruses N.N.

25.62 Dermatophagoides (dust mite)

25.86 Pneumocystis jiroveci (carinii)

26.12 Aspergillus niger

26.41 Aflatoxin

Each and every pathogen now needs testing on the bioreso-

nance devices. Here, too, the same principle applies: if the

Rayotensor shows a linear motion, a stress is present in the

organism. If you then wish to precisely localise the tissue

area, then you hold a spherical detector connected to the

bioresonance device, for example, to the ear, and then test

for a linear motion on the Rayotensor.

Tip: if you work with a Rayocomp PS 10, you can copy Ap-

pendix II and use it as a template where you can enter the

measuring results. If you use a Rayocomp PS 1000 polar, the

measuring results can be printed out.

Recommendation: if there is a stress on N in subsequent

harmonisation lasting at least 3 minutes, there is a linear

motion on the bipolar function – i.e. a strong stress. In this

case, harmonisation lasting at least 5 minutes needs to be

performed. Ideally, the harmonisation times are determined

radiesthetically.

This provides clear information and the causes of the illness

which of course can go even deeper, since there is a rea-

son why pathogens managed to position themselves in the

middle ear, e.g. a disbalanced acid-base balance, malnutri-

tion, the psyche, or further reasons.

The compilations always commence with vitalisation and

the relevant meridians, which are contained in Appendix I of

this offprint. There then follows a program on ATP produc-

tion. Since there is no specific ATP program for the hearing

organ, the higher program ATP Production, total is selected.

There then follows the appropriate physiological program

part. In the case named above, this is the middle ear. There

then follow the programs for the pathogens to be harmoni-

sed. Due to the endotoxins which can arise by the harmo-

nisation of the pathogens in the organism, a detoxification

always follows. In this case too, the universal detoxification

program with the number 31.50 is selected. This is all roun-

ded off by the vitalisation program.

16

In this way, individual program compilations can also be

determined for other organs / regulation areas.

In addition to the physiology programs, the RAH offers

a multitude of frequency patterns which describe the

pathological processes in the body. If, for instance, a fracture

is suspected, it is possible to find out if the bone is broken

via the program 53.11 and a spherical detector connected

to the bioresonance device. The test can be considerably

supported via the pathological frequency patterns in

particular. These programs include the classics, such as

open wounds (program number 31.80) and the teaching

program by Dr med. Ulrich (program number 75.19) for

aiding concentration, especially amongst children.

Please note that the main numbers of the pathological

programs are empty. Can you remember? The frequency

patterns of the entire sub-programs are always summarised

under the physiological main programs (e.g. 32.00 Blood

physiology). However, this doesn’t make any sense for

pathology programs. Hence the program 33.00 Blood

pathology, for example, is empty, and not suitable for use.

It would have been possible to summarise pathological

sub-programs, but that wouldn’t have made any sense from

a therapeutic point of view.

The pathology programs can be used for both analysis and

for harmonisation purposes. Naturally, there are limits here,

too. A program entitled Morbus Alzheimer can be helpful

for analysis purposes and for the general differentiation. It

can also superbly support the organism, and stabilisation of

the illness’s progress can be achieved. Despite this, it cannot

be expected that such degenerative illnesses can be solved

with the application of one single program. A deeper, more

specific analysis and harmonisation process is necessary in

this case.

We would like to point out another new program point

within the RAH which has just been integrated in the

program: teeth. To date, there were no physiological

frequency patterns for the individual teeth. The integration

of these programs will make it possible to analyse the teeth

of the upper jaw, of the lower jaw in their entirety, as well

as individual teeth.

Let’s turn to the further possible applications of the RAH,

and we arrive at the 70s programs which have a very special

feature of their own. The development of these programs is

attributed to Ms HP Schußmann and Dr med. Schußmann.

She wrote about this: “Each cause-orientated system

program contains all pathogens detected by use which

have been examined in the past 8 years. They are complete

programs which contain all pathogens which we have ever

found in the corresponding organ system. The same thing

applies to the frequency patterns for the supply of energy, the

transfer frequencies, the physiological frequency patterns

of the affected organ systems and the immune system. This

makes it possible to successfully treat the patient without

making the distinction between pathogens of a bacterial,

viral, parasitical or fungal nature. In this respect, it is not

possible to make a mistake if precise tests cannot be carried

out. Even doctors not familiar with this examination method

can carry out a cause-orientated treatment and have

content, long-term healthy patients”.

This makes it clear that the 70s programs of the RAH

have been conceived not for testing purposes, but for

harmonisation purposes only. If these programs are used,

harmonisation should last at least 30 minutes.

The 70s programs are followed by four further areas assigned

specific programs. On the one hand, programs on the psyche

and stress reduction and teeth / milk teeth are found here.

In conclusion, the RAH contains special programs with the

objective of supporting further therapeutic approaches. By

means of the special programs, for example, it is possible to

test out the right Bach’s flower extracts or the appropriate

Schüssler tissue salts for a patient.

17

6 Application in the Rayocomp PS 10 and PS 1000 polar

The RAH has been integrated into bioresonance devices to

date: one, the mobile Rayocomp PS 10. Two, the Rayocomp

PS 1000 polar professional device.

Since an increasing number of therapists now uses the mo-

bile Rayocomp PS 10 for treating patients at home, all pro-

grams available in the RAH can also be used in the Rayo-

comp PS 10. For home treatment, the patient rents a Rayo-

comp PS 10 and the RAH, and receives programs drawn up

by the therapists for the duration of the treatment. The

advantages: the patients don’t have to go to the surgery.

Furthermore, it is possible to relocate treatment, lightening

the load on the surgery. The new Rayonex “Green Card” in

particular is making the application of treatment at home

much easier:

the idea behind the new RAH “Green Card” is as simple as

it is brilliant. Whereas, in the Rayocomp PS 1000 polar and

in the Rayocomp PS 10, the programs to be harmonised

were determined by means of the RAH, these had to be

noted or printed out for future use. By means of the new

RAH “Green Card”, these program compilations can now be

saved on a special memory card, the “Green Card”. This now

enables, for instance, the saving of the programs tested in

the Rayocomp PS 1000 on a RAH “Green Card”, which can

then be given to their patient for his or her use at home

on the Rayocomp PS 10. Via the RAH “Green Card”, both

devices work hand in hand, as it were.

The “Green Card” further optimally unites bioresonance

according to Paul Schmidt and the RAH. Since, in parallel to

the use of RAH programs, single frequencies of bioresonance

according to Paul Schmidt (e.g. from an individual range

value test) are often used, engineers at Rayonex found a way

to store on the “Green Card” both individual frequencies (up

to 500 various values) and RAH programs. Additionally, a

special function has been added to the Rayocomp PS 1000

polar under the main menu item, and to the Rayocomp PS

10 under the main menu. If you insert a RAH “Green Card”

there, an automatic harmonisation run is launched. First

of all the individual frequencies stored on the RAH “Green

Card” are harmonised for 30 seconds each and then the RAH

programs saved on the same card. This enables patients to

use a “Green Card” compiled by therapists at home, at the

touch of a button.

Rayocomp PS 10

RAH “Green Card“

Rayocomp PS 1000 polar

18

Rayocomp PS 1000 polarThe Rayocomp PS 1000 polar is the high-end bioresonance

device by Rayonex GmbH. The new RAH has been realised

correspondingly comprehensively and easily in the device.

There is both an analysis function as well as a harmonisa-

tion function. The option for intuitive testing described at

the start has been achieved via clear information on the

display. Naturally, the results can also be printed out.

The visualisation of the analysis integrated in this device

can be particularly helpful for the patient. If for instance

a problem in the visual organ (e.g. the conjunctiva) is de-

tected, this can be indicated on the display of the Rayocomp

PS 1000 polar (see picture). This enables a very graphic ex-

planation of the problems. In addition, the RAH in the Rayo-

comp PS 1000 polar offers a very special type of analysis. By

means of the operating button on the Rayotensor, you can

quickly access the sub-menus, changing from the overall

structures to the detailed structures of the organs. The RAH

also makes available a special function which enables the

pathogen-specific testing and the equally fast change-over

to the harmonisation function.

Rayocomp PS 10The RAH is now supported with three modules in the Rayo-

comp PS 10.

Module 8 only contains higher frequency structures: in

the current program version this amounts to 191 various

frequency structures. On the other hand, module 9 has all

available frequency spectrums – currently amounting to

1249!

Module 10 is the highest-quality module in the Rayocomp

PS 10 which, in addition to module 9, not only provides all

programs but also extensive test functions. The very spe-

cial feature: at the touch of a button, all RAH program

numbers necessary for organ areas and specific illnesses

are prepared for testing.

These test protocols (see Appendix V for more detailed

information) create a guide for both the analysis and the

harmonisation process with the RAH. A further objective of

the test protocols is to clarify as precisely as possible the

energetic deficits. To do this, it is not necessary to type in

the RAH program numbers individually: it is ready to launch

the test at the touch of a button. The test results are then

ready for harmonisation purposes and for storing on the

RAH “Green Card”.

The level test familiar from the Rayocomp PS 1000 polar is

also supported in module M 10. If a higher RAH program

is selected, the subordinate RAH programs can be selected

and tested via the operating button of the Rayotensor. In

order to provide you with an overview of which programs

can be found in which module, this information has been

added to the list of programs in Appendix I.

The use of the RAH in the Rayocomp PS 10 is conceivably

easy. Under the menu item, the programs in the appendix

can be entered via the relevant program number. It can be

set for a session of up to 200 various programs with various

time settings. Once the start button has been activated, the

frequency spectrums are generated and output so that they

can be transferred to the organism via appropriate detec-

tors. The programs in the Rayocomp PS 10 can be used for

both analysis and harmonisation purposes.

19

Detected interferences can be indicated on the display of the Rayocomp PS 1000 polar.

Detected interferences can be indicated on the display of

the Rayocomp PS 1000 polar. The picture displays the visual

organ.

Naturally, the test protocols on various illnesses mentioned

at the beginning (see Appendix V) as well as the use of the

RAH “Green Card” are also available in the Rayocomp PS

1000 polar.

20

We wish you a every successwhen using the RAH

7 Integration of new programs

The new RAH already has over 1,000 various programmes (frequency structures) which can be used for analysis and harmonisation purposes. These programs have been provi-ded by a large number of therapists.

Frequently, the created programmes are the life’s work of the therapist in question. Due to the time limit alone, one single individual cannot manage something of this magni-tude. The RAH is an open system which, following a rele-vant examination, provides new therapists and their pro-grams with a platform for general application.

It is an inestimable advantage for all users should as many therapists as possible make their programs available, since each and every therapist makes his or her individual de-mands on an analysis and harmonisation system. It is only if numerous therapists contribute their approaches to an expert system that the user can pursue as many therapeutic approaches. This will fill an increasing number of people with the enthusiasm to use the RAH, and vibrational medi-cine on the whole.

The Rayocomp PS 1000 polar offers the option of compiling separate programs in order to enable selective testing and application. This provides the necessary platform for the development and application of separate, new programs, further enhancing the fact that new programs submitted by new therapists are expressly welcome.

But who decides which programs are included in the RAH? A panel of experts, comprising the therapists, the Association for the Promotion of Vibrational Medicine, representatives from Germany and abroad, development engineers, the therapy centre and the School of Non-medical Practitioners was formed in the Sauerland Pyramiden. Its objective is to take into consideration as many different interests as possible, at the same time forming a decision-making committee which assesses and integrates new suggestions.

Preconditions for new programs:

the programs must have successfully proven their practical value over an extended period, and relevant progress reports must be available. The new program should cover a new area of application. The panel of experts is to evaluate the new programs.

For the integration of a new program into the RAH, a Word document with the following content is required:

• The therapist’s address

• The name of the program

• Brief description of the main area of application of the program

• How long the program has already been in use

• How many patients have already been treated using this program

• Frequency basic values of the program (required for programming, will not be published)

• A minimum of three progress reports

Please see the Appendix for a template.

Please send the new program to the Paul-Schmidt Academy, keyword: panel of experts. Please see the template for the address. From there, the new program will be sent to the other therapists on the panel of experts for testing. Should the results be satisfactory for the new program, it will be provided to all users in the next RAH program update.

21

The therapist’s address

The name of the program

Brief description of the main area of application of the program

How long has the program already been in use?

How many patients have already been treated using this program?

Frequency basic values of the program

2: 3: 4: 5:

6: 7: 8: 9: 10:

11: 12: 13: 14: 15:

16: 17: 18: 19: 20:

21: 22: 23: 24: 25:

26: 27: 28: 29: 30:

31: 32: 33: 34: 35:

36: 37: 38: 39: 40:

41: 42: 43: 44: 45:

46: 47: 48: 49: 50:

51: 52: 53: 54: 55:

56: 57: 58: 59: 60:

61: 62: 63: 64: 65:

66: 67: 68: 69: 70:

71: 72: 73: 74: 75:

Please submit further frequency basic values: use a separate sheet!

Number of attached progress reports

Send to: Paul-Schmidt-Akademie- Panel of experts -Sauerland-Pyramiden 157368 LennestadtGERMANYTelefax: +49 2721 6006-66

Integration of a new program in the RAH

22

8 Appendix I: Currently available programs.

PS 10 M8 PS 10 M9 PS 10 M10 Polar

00.00 Analysis preparation • • • •


01.00 Vitalisation, complete • • • •

01.10 Energy charging • • •

01.20 Equalisation of polarity • • •

01.30 Pre-control • • •

01.40 Chakras, complete • • •

01.41 Vertex Chakra • • •

01.42 Forehead Chakra • • •

01.43 Throat Chakra • • •

01.44 Heart Chakra • • •

01.45 Spleen Chakra • • •

01.46 Navel Chakra • • •

01.47 Root Chakra • • •


02.00 Channels of acupuncture, complete • • • •

02.11 Lung channel • • •

02.12 Colon channel • • •

02.13 Stomach channel • • •

02.14 Spleen channel • • •

02.15 Heart channel • • •

02.16 Channel of the small intestines • • •

02.17 Bladder channel • • •

02.18 Kidney channel • • •

02.19 Liver channel • • •

02.20 Channel of the heart and circulation • • •

02.21 Sanjiao channel • • •

02.22 Gall bladder channel • • •

02.23 Channel of the Governor Vessel • • •

02.24 Channel of the Conception Vessel • • •

Note: The gray marked programmes are still being developped and will be available in the following versions of RAH. The additional description “T” refers to an available test protocol.

23


04.00 Electro-magnetic pollution, complete • • • •

04.10 Alternating electric and magnetic fields • • •

04.20 Pulse-modulated irradiation, complete • • •

04.21 Mobile phones • • •

04.22 UMTS • • •

04.23 DECT (wireless telephone) • • •

04.24 WLAN • • •

04.25 Bluetooth • • •

04.26 Satellite transmission • • •

04.27 Wi Max • • •

04.30 Radiation, protection • • •


05.00 Geopathic disorders, complete • • • •

05.10 Underground water • • •

05.20 Shiftings • • •

05.30 Global grids • • •

05.31 Hartmann grid line • • •

05.32 Hartmann grid intersection • • •

05.33 Curry grid line • • •

05.34 Curry grid intersection • • •

05.35 Benker grid line • • •

05.36 Benker grid intersection • • •


06.00 Acid-alkali balance, complete • • • •

06.10 Connective tissue • • •

06.20 Pancreas • • •

06.30 Liver • • •

06.40 Small intestines • • •


07.00 Vital substances, complete • • • •

07.10 Minerals, complete • • • •

07.11 Calcium • • •

07.12 Potassium • • •

07.13 Magnesium • • •

07.14 Sodium • • •

07.20 Trace elements • • • •

07.21 Iron • • •

24


07.22 Zinc • • •

07.23 Copper • • •

07.24 Manganese • • •

07.25 Molybdenum • • •

07.26 Iodine • • •

07.27 Cobalt • • •

07.28 Chromium • • •

07.29 Selenium • • •

07.30 Vitamins, fat-soluble, complete • • • •

07.31 Vitamin A • • •

07.32 Vitamin D • • •

07.33 Vitamin E • • •

07.34 Vitamin K • • •

07.35 Vitamin K1 • • •

07.36 Vitamin K2 • • •

07.40 Vitamins, water-soluble, complete • • • •

07.41 Vitamin C • • •

07.42 Vitamin B1, thiamine • • •

07.43 Vitamin B2, riboflavin • • •

07.44 Vitamin B3, niacin • • •

07.45 Vitamin B5, pantothenic acid • • •

07.46 Vitamin B6, pyridoxine • • •

07.47 Vitamin B7, biotin • • •

07.48 Vitamin B9, folic acid • • •

07.49 Vitamin B12, cobalamin • • •

07.50 Vitamin B17, laetril • • •

07.60 Probiotic bacteria, complete • • • •

07.61 Lactobacillus rhamnosus • • •

07.62 Enterrococcus faecium • • •

07.63 Bifidobacterium lactis • • •

07.64 Bifidobacterium longum • • •

07.65 Lactococcus lactis • • •

07.66 Lactobacillus sporogenes • • •

07.67 Lactobacillus casei • • •

07.68 Lactobacillus plantarum • • •

07.69 Lactobacillus acidophilus • • •

07.70 Bifidobacterium infantis • • •

07.71 Lactobacillus salivarius • • •

07.80 Fatty acides, complete • • • •

07.81 Monocarbylic acids • • •

25


20.00 Bacteria, complete • • • •

20.05 Bacteria I, complete • • • •

20.10 Coccobacilli, complete • • • •

20.11 Alpha streptococcus • • •

20.12 Beta haemolytic streptococci • • •

20.13 Eikanella corrodens • • •

20.14 Gaffkya tetragena • • •

20.15 Meningococcus • • •

20.16 MRSA multidrug-resistant V • •


08.00 Harmful substances, complete • • • •

08.10 Heavy metals, complete • • •

08.11 Palladium • • •

08.12 Silver • • •

08.13 Cadmium • • •

08.14 Platinum • • •

08.15 Gold • • •

08.16 Mercury • • •

08.17 Lead • • •

08.50 Pesticides, complete • • • •

08.51 Fungicides (fungus) • • •

08.52 Herbicides (weeds) • • •

08.53 Insecticides (insects) • • •

08.54 Molluscicides (snails) • • •

08.55 Nematicides (threadworms) • • •

08.56 Rodenticides (rodents) • • •

08.57 Vermicides (parasitic worms) • • •

08.58 Miticides (mites) • • •

08.80 Genotoxines • • • •

08.81 Psorine • • •

08.82 Medorrhine • • •

08.83 Luesine • • •

08.84 Tuberculin • • •


07.82 Saturated fatty acids • • •

07.83 Monounsaturates • • •

07.84 Polyunsaturates • • •

07.85 Essential fatty acids • • •

26


20.17 Neisseria gonorrhoea • • •

20.18 Staphylococci • • •

20.19 Staphylococcus aureus • • •

20.20 Streptococcus • • •

20.21 Streptococcus lactis • • •

20.22 Streptococcus mitis • • •

20.23 Streptococcus pneumoniae • • •

20.24 Streptococcus pyogenes • • •

20.25 Streptococcus sp. • • •

20.26 Veillonella dispar • • •

20.40 Rod-shaped bacteria, complete • • • •

20.41 Actinobacillus (suis) V • • •

20.42 Actinomyces israelii • • •

20.43 Arcanobacterium pyogenes • • •

20.44 Bacilli • • •

20.45 Bacillus anthracis V • • •

20.46 Bacillus cereus • • •

20.47 Bacteroides fragilis • • •

20.48 Bordetella bronchiseptica • • •

20.49 Bordetella pertussis • • •

20.50 Brucella abortus V • • •

20.51 Brucella melitensis V • • •

20.52 Brucella suis V • • •

20.53 Coxiella burnetii V • • •

20.54 Clostridia • • •

20.55 Clostridium botulinum V • • •

20.56 Clostridium feseri V • • •

20.57 Clostridium perfringens • • •

20.58 Clostridium septicum • • •

20.59 Clostridium tetani V • • •

20.60 Corynebacterium diphteriae • • •

20.61 Corynebacterium xerosis • • •

20.62 Cytophaga rubra • • •

20.63 Erysipelotrix rhusiopathiae V • • •

20.64 Eubacterium suis • • •

20.65 Francisella tularensis V • • •

20.66 Gardnerella vaginalis • • •

20.67 Haemophilus influenzae • • •

20.68 Haemophilus parasuis V • • •

20.69 Helicobacter pylori • • •

27


21.05 Bacteria II, complete • • • •

21.10 Enterobacteriaceae, complete • • • •

21.11 Enterobacter aerogenes • • •

21.12 Erwinia amylovora • • •

21.13 Erwinia carotavora • • •

21.14 Escherichia coli • • •

21.15 Klebsiella pneumoniae • • •

21.16 Proteus mirabilis • • •

21.17 Proteus vulgaris • • •

21.18 Salmonellae • • •

21.19 Salmonella enteritidis • • •

21.20 Salmonella paratyphi • • •

21.21 Salmonella typhi • • •

21.22 Serratia marcescens • • •

21.23 Shigella dysenteriae • • •

21.24 Shigella flexneri • • •

21.25 Shigella sonnei • • •

21.26 Yersiniae • • •

21.27 Yersinia enterocolitica • • •

21.50 Mycoplasmae, complete • • • •

21.51 Mycoplasma • • •

21.52 Mycoplasma agalactiae V • • •

21.53 Mycoplasma capricolum • • •

21.54 Mycoplasma mycoides V • • •

21.60 Spirochaetae, complete • • • •


20.70 Lactobacillus acidophilus • • •

20.71 Lawsonia intracellularis • • •

20.72 Legionella • • •

20.73 Listeria monocytogenes V • • •

20.74 Malleomyces mallei V • • •

20.75 Mycobacteria phlei • • •

20.76 Mycobacteria tuberculosis • • •

20.77 Nocardiae V • • •

20.78 Nocordia asteroids • • •

20.79 Pasteurellae V • • •

20.80 Pasteurella multocida V • • •

20.81 Propionobacterium acnes • • •

20.82 Pseudomonas aeruginosa • • •

28


21.61 Borellia • • •

21.62 Brachyspira V • • •

21.63 Leptospira canicola V • • •

21.64 Leptospira grippotyphosa V • • •

21.65 Leptospira icterohaemorrhagiae • • •

21.66 Leptospira interrogans • • •

21.67 Leptospira pomona V • • •

21.68 Leptospirae (suis) V • • •

21.69 Treponema pallidum • • •

21.80 Intracellular bacteria (cell parasites), complete • • • •

21.81 Anaplasma marginale • • •

21.82 Chlamydiae • • •

21.83 Chlamydiae (feline) V • • •

21.84 Chlamydia ovis V • • •

21.85 Chlamydia psittaci V • • •

21.86 Chlamydia trachomatis • • •

21.87 Cowdia rumantium V • • •

21.88 Rickettsiae • • •

21.90 Other bacteria • • • •

21.91 Laryngeal 1 bacteria • • •

21.92 Borellia toxin • • •

21.93 Caries bacterium • • •

21.94 PIA Porcine intestinal adenomatosis V • • •

21.95 Pain-producing bacteria • • •

21.96 Tuberculinum burnetti • • •


22.00 Viruses, complete • • • •

22.05 Viruses I, complete • • • •

22.10 Double-strain DNA viruses, complete • • •

22.11 Adenovirus • • •

22.12 Cytomegalovirus (CMV) • • •

22.13 Epstein-Barr virus (EBV) • • •

22.14 Hepatitis B virus • • •

22.15 Herpes simplex • • •

22.16 Herpes simplex (feline) V • • •

22.17 Herpes zoster • • •

22.18 Human papilloma virus (HPV) • • •

22.19 Papilloma virus • • •

22.40 Single-strain DNA viruses, complete • • • •

29


22.41 Panleucopenia virus V • • •

22.42 Parvoviruses (suis) V • • •

22.43 Porcine circovirus V • • •

22.60 Single-strain RNA viruses, positive-strain RNA genome, complete

• • • •

22.61 AE virus V • • •

22.62 BVD virus V • • •

22.63 Calciviruses (feline) V • • •

22.64 Chikungunya • • •

22.65 Coronaviruses (feline) V • • •

22.66 Coronaviruses (suis) V • • •

22.67 Coxsackie virus B-1 • • •

22.68 Coxsackie virus B-4 • • •

22.69 EAV virus • • •

22.70 Duck hepatitis virus V • • •

22.71 Enteroviruses • • •

22.72 FHV viruses (feline herpes virus) V • • •

22.73 FSME • • •

22.74 Hepatits A virus • • •

22.75 Hepatitis C virus • • •

22.76 CSF virus V • • •

22.77 FMD virus V • • •

22.78 Norovirus • • •

22.79 PRRS viruses (suis) V • • •

22.80 Rhinovirus • • •

22.81 SVD virus V • • •

22.82 Tobacco mosaic virus • • •

22.83 Teschen virus V • • •

22.84 VES virus V • • •


23.05 Viruses II, complete • • • •

23.10 Negative-strain RNA genome, unsegmented, complete

• • • •

23.11 Borna virus • • •

23.12 Equine influenza virus V • • •

23.13 Highly pathogenic avian influenza virus V • • •

23.14 Measles virus • • •

23.15 Mumps virus • • •

23.16 Parainfluenza • • •

30


23.17 Porcine influenza virus V • • •

23.30 Negative-strain RNA genome, segmented, complete • • • •

23.31 H1N1 • • •

23.32 H5N1 • • •

23.33 Influenza virus A and B • • •

23.50 Double-strain RNA viruses, complete • • • •

23.51 Bluetongue viruses V • • •

23.52 FCo viruses V • • •

23.53 FeL viruses V • • •

23.54 FI viruses V • • •

23.55 Retroviruses • • •

23.56 Rotaviruses • • •

23.57 Rotaviruses (suis) V • • •

23.70 Wart frequencies, complete • • • •

23.71 Seborrhoeic warts • • •

23.72 Molluscums contagiosum • • •

23.73 Condolymas • • •

23.74 Flat warts • • •

23.75 Verrucas • • •

23.76 Juvenile warts • • •

23.77 Flat warts • • •

23.78 Common warts • • •

23.79 Warts N.N. warts recurrent • • •

23.80 Other viruses, complete • • • •

23.81 Viruses N.N. • • •


24.00 Parasites, complete • • • •

24.05 Parasites I, complete • • • •

24.10 Hookworms, complete • • •

24.11 Ancylostoma brasiliense • • •

24.12 Ancylostoma caninum • • •

24.13 Gyrodactylus • • •

24.20 Eelworms/intestinal roundworms/pinworms compl. • • • •

24.21 Ascaris megalocephala • • •

24.22 Dirofilaria immitis (heart worm) • • •

24.23 Enterobius vermicularis • • •

24.24 Haemonchus contortus • • •

24.25 Loa loa • • •

24.26 Macracanthorhynchus • • •

31


24.27 Onchocerca volvulus (tumor) • • •

24.28 Enterobius worms • • •

24.29 Passalurus ambiguous (rabbit worm) • • •

24.30 Stephanurus dentalus • • •

24.31 Strongyloides (filariform) • • •

24.32 Trichinella spiralis (muscle) • • •

24.33 Trichuris sp. • • •

24.40 Capillariae, complete • • • •

24.41 Capillaria hepatica (liver) • • •

24.50 Trematodes / leeches, complete • • • •

24.51 Clonorchis sinesi • • •

24.52 Cryptocotyle lingua • • •

24.53 Echinostoma revolutum • • •

24.54 Eurytrema pancreaticum • • •

24.55 Fasciola heptica • • •

24.56 Fasciolopsis buski • • •

24.57 Fischoedrius elongatus • • •

24.58 Gastrothylax elongatus • • •

24.59 Hasstile sig. tricolor • • •

24.60 Metagonimus Yokogawai • • •

24.61 Paragonimus Westermani • • •

24.62 Prosthogonimus macro. • • •

24.63 Schistosoma haematica • • •

24.64 Schistosoma masoni • • •

24.65 Urocleidus • • •

24.80 Tapeworms, complete • • • •

24.81 Echinococcus granulosus • • •

24.82 Echinococcus multicolaris • • •

24.83 Taenia pisiformis • • •

24.84 Taenia saginata • • •

24.85 Taenia solium • • •


25.05 Parasites II, complete • • • •

25.10 Protozoa, complete • • • •

25.11 Balantidia • • •

25.12 Balantidium coli • • •

25.13 Besnoitia (lung) • • •

25.14 Blepharisma • • •

25.15 Chilomastix cysts (rat) • • •

32


25.16 Chilomonas • • •

25.17 Coccidia (suis) V • • •

25.18 Coccidia (canis) V • • •

25.19 Dientamoeba fragilis • • •

25.20 Encephalitozoon cuniculi V • • •

25.21 Endolimax nana • • •

25.22 Endolimax tropica • • •

25.23 Entamoeba coli trophozoi • • •

25.24 Entamoeba gingivalis • • •

25.25 Entamoeba histolytica tro. • • •

25.26 Giardia lamblia (troph.) • • •

25.27 Iodamoeba bütschlii • • •

25.28 Iodamoeba bütschlii tropica • • •

25.29 Leishmania brasiliensis • • •

25.30 Leishmania donovani • • •

25.31 Leishmania mexicana • • •

25.32 Leishmania tropica • • •

25.33 Leucocytozoon • • •

25.34 Myxobolus cerebralis • • •

25.35 Naegleria fowleri • • •

25.36 Plasmodium cynomolgi • • •

25.37 Plasmodium falciparum • • •

25.38 Plasmodium vivax • • •

25.39 Sarcocystis • • •

25.40 Toxoplasma gondii • • •

25.41 Trichomonas vaginalis • • •

25.42 Trypanosoma brucei • • •

25.43 Trypanosoma cruzi (brain) • • •

25.44 Trypanosoma equip. • • •

25.45 Tryanosoma gambiense • • •

25.46 Trypanosoma lewisi • • •

25.47 Trypanosoma rhodesiens • • •

25.60 Mites / ticks, complete • • • •

25.61 Acarus siro • • •

25.62 Dermatophagoides (dust mite) • • •

25.63 Demodex canis V • • •

25.64 Demodex folliculorum (hair follicle mite) • • •

25.65 Neotrombicula autumnalis (harvest mite) V • • •

25.66 Notoedres cati V • • •

25.67 Ornithonyssus (bird mite) • • •

33


27.05 Fungi II, complete • • • •

27.10 Yeast fungus, complete • • • •

27.11 Candida albicans • • •


26.00 Fungi, complete • • • •

26.05 Fungi I, complete • • • •

26.10 Mould fungi, complete • • • •

26.11 Aspergillus fumigatus • • •

26.12 Aspergillus niger • • •

26.13 Aspergillus ochraceus • • •

26.14 Cladosporium herbarum • • •

26.15 Geotrichum candidum • • •

26.16 Monilia albicans • • •

26.17 Mucor mucedo • • •

26.18 Mucor racemosus • • •

26.19 Penicillinum camemberti • • •

26.20 Penicillinum frequentans • • •

26.21 Penicillinum notatum • • •

26.22 Penicillinum roqueforti • • •

26.23 Pullulania pullulans • • •

26.24 Scopulariopsis brevic. • • •

26.25 Torulpsis glabratis • • •

26.40 Mould fungus toxines, complete • • • •

26.41 Aflatoxin • • •

26.42 Griseofulvin • • •

26.43 Helminthosporium dermatoideum (Cytochalasin B)

• • •

26.44 Sterigmatocaptin • • •

26.45 Zearealenon • • •


25.68 Sarcoptes scabei (scabies) • • •

25.80 Other parasites, complete • • • •

25.81 Echinoporyphium recurvatum • • •

25.82 Hypodereum conoideum • • •

25.83 Stigeoclonium • • •

25.84 Troglodytella abrasseri • • •

25.85 Blood parasites • • •

25.86 Pneumocystis carinii • • •

34


27.12 Candida crusei • • •

27.13 Candida dattila • • •

27.14 Candida famata • • •

27.15 Candida glabrata • • •

27.16 Candida guilliermondii • • •

27.17 Candida kefyr • • •

27.18 Candida lusitaniae • • •

27.19 Candida parapsilosis • • •

27.20 Candida stellatoidea • • •

27.21 Candida tropicalis • • •

27.22 Candida viswanthii • • •

27.23 Cryptococcus neoformans • • •

27.24 Malasseziae V • • •

27.25 Malassezia furfur • • •

27.26 Rhodotorula ruba • • •

27.27 Saccaromyces cerevisiae • • •

27.28 Sporothrix schenkii • • •

27.29 Torulpsis glabratis • • •

27.30 Trichosporum capitatum • • •

27.31 Trichosporon cutaneum • • •

27.50 Filamentous fungi / dermatophytes, dimorphic fungi, complete

• • • •

27.51 Coccidioides immitis V • • •

27.52 Microsporum canis • • •

27.53 Microsporum gypseum • • •

27.54 Trichophyton cutaneum • • •

27.55 Trichophyton mentagro • • •

27.56 Trichophyton rubrum • • •

27.57 Trichophyton terrestre • • •

27.58 Trichophyton verrucosum (trichophytia) • • •

27.59 Zymonema farciminosus • • •

27.70 Mycetozoa, complete • • • •

27.71 Arcyria • • •

27.72 Lycogala • • •

27.73 Stemonitis • • •

27.80 Ascomycota, complete • • • •

27.81 Secale cornutum • • •

27.82 Ergot • • •

27.90 Other fungi, complete • • • •

27.91 Tryptophanum • • •

35


30.00 Cells and tissue, physiology, complete • • • •

30.10 Cell nucleus • • •

30.20 Cell membrane • • •

30.30 Cytoplasm • • •

30.40 Organelles, complete • • •

30.41 Endoplasmatic reticulum • • •

30.42 Mitochondria • • •

30.43 Golgi apparatus • • •

30.44 Ribosomes • • •

30.45 Lysosomes • • •

30.65 Epithelial tissues, complete • • •

30.66 Surface epithelium • • •

30.67 Ciliated epithelium • • •

30.68 Glandular epithelium • • •

30.69 Sensory epithelium • • •

30.70 Connective tissues, complete • • •

30.71 Collagenic tissues • • •

30.72 Elastic tissues • • •

30.73 Fat tissues • • •

30.74 Cartilage tissues • • •

30.75 Bone tissues • • •

30.80 Nerve tissues, complete • • •

30.81 Nerve cells • • •

30.82 Astrocytes • • •

30.83 Oligodendrocytes • • •

30.90 Mucous membranes, complete • • •

30.91 Mucous membranes, head • • •

30.92 Mucous membranes, trunk • • •

30.93 Mucous membranes, genital organs • • •


31.00 Cell and tissue, pathology (empty)

31.10 ATP production, complete • • • •

31.11 ATP production, lung • • •

31.12 ATP production, colon • • •

31.13 ATP production, stomach • • •

31.14 ATP production, pancreas • • •

31.15 ATP production, heart • • •

31.16 ATP production, small intestines • • •

31.17 ATP production, urinary bladder • • •

36


31.18 ATP production, prostate gland • • •

31.19 ATP production, testicles • • •

31.20 ATP production, uterus • • •

31.21 ATP production, uterine cervix • • •

31.22 ATP production, ovaries • • •

31.23 ATP production, kidney • • •

31.24 ATP production, thymus • • •

31.25 ATP production, lymph • • •

31.26 ATP production, adrenal gland • • •

31.27 ATP production, gall bladder • • •

31.28 ATP production, biliary tract • • •

31.29 ATP production, liver • • •

31.30 ATP production, spleen • • •

31.31 ATP production, eyes • • •

31.32 ATP production, parathyroid gland • • •

31.33 ATP production, thyroid gland • • •

31.34 ATP production, cerebelum • • •

31.35 ATP production, cerebrum • • •

31.36 ATP production, mammary gland • • •

31.37 ATP production, bone marrow • • •

31.38 ATP production, skin • • •

31.39 ATP production, vessels • • •

31.40 ATP production, muscles • • •

31.41 ATP production, bones • • •

31.50 Detoxication, basic program • • • •

31.51 Detoxication, blood system • • •

31.52 Detoxication, lymphatic system • • •

31.53 Detoxication, acidosis • • •

31.54 Detoxication, extra-cellular • • •

31.55 Detoxication, intra-cellular • • •

31.56 Detoxication, mucous membrane • • •

31.57 Detoxication, lung • • •

31.58 Detoxication, stomach • • •

31.59 Detoxication pancreas • • •

31.60 Detoxication liver • • •

31.61 Detoxication intestines • • •

31.62 Detoxication kidney • • •

31.63 Detoxication bladder • • •

31.64 Detoxication woman / female-specific • • •

31.65 Detoxication skin • • •

37


32.00 Blood physiology, complete • • • •

32.05 Stem cells of the bone marrow • • •

32.06 Formation of blood (haematopoiesis) • • •

32.07 Blood plasma • • •

32.10 Erythrocytes • • •

32.11 Iron storage (ferritin) • • •

32.20 Leukocytes, complete • • •

32.21 Lymphocytes • • •

32.22 Monocytes • • •

32.23 Macrophages • • •

32.24 Neutrophil granulocytes • • •

32.25 Eosinophil granolocyte • • •

32.26 Basophil granolocyte • • •

32.27 Phagocytes • • •

32.28 T helper cells • • •

32.29 Regulatory T cells • • •

32.30 Thrombocytes • • •

32.31 Fibrinolysis • • •

32.40 Blood coagulation system • • •

32.41 Coagulation factor I • • •

32.42 Coagulation factor II • • •

32.43 Coagulation factor III • • •

32.44 Coagulation factor IV • • •

32.45 Coagulation factor V • • •

32.46 Coagulation factor VI • • •

32.47 Coagulation factor VII • • •

32.48 Coagulation factor VIII • • •


31.66 Detox of endotoxins • • •

31.67 Detox of exotoxins • • •

31.70 Degeneration cell tissue • • • •

31.80 Open wounds/wound healing • • • •

31.81 Scar interference suppression • • • •

31.82 Care after operations • • • •

31.83 Dupuytren’s contracture • • • •

31.84 Myomata • • •

31.85 Cysts • • •

31.86 Fistulae • • •

31.87 Oedemata • • • •

38


35.00 Immune system, pathology (empty)

35.10 Raising the defence capacity, basic program • • • •

35.11 Raising the unspecific defence • • • •

35.12 Raising the specific defence • • • •

35.13 Phagocytosis • • • •

35.20 Allergy, complete • • • T • T35.21 Allergy type I • • •


34.00 Immune system physiology, complete • • • •

34.10 Interleukins • • •

34.20 Cytokines • • •

34.30 Lymphokines • • •


33.00 Blood, pathology (empty)

33.10 Haemorrhagic anaemia • • • •

33.20 Anaemia caused by a disorder of the erythropoesis, complete

• • •

33.21 Renal anaemia • • •

33.22 Aplastic anaemia • • •

33.23 Anaemia caused by the myelodysplatic syndrome (MDS)

• • •

33.24 Iron-deficiency anaemia • • •

33.25 Vitamin B12 deficiency anaemia • • •

33.26 Vitamin B6 deficiency anaemia • • •

33.27 Folic acid deficiency anaemia • • •

33.28 Vitamin C deficiency anaemia • • •

33.29 Protein deficiency anaemia • • •

33.50 Degeneration bone marrow • • •

33.55 Inflammation bone marrow • • •

33.60 Oxygen supply / utilisation improvement • • • •

33.70 Polycythaemia • • •


32.49 Coagulation factor IX • • •

32.50 Coagulation factor X • • •

32.51 Coagulation factor XI • • •

32.52 Coagulation factor XII • • •

32.53 Coagulation factor XIII • • •

39


35.22 Allergy type II • • •

35.23 Allergy type III • • •

35.24 Allergy type IV • • •

35.30 Fructose intolerance • • •


36.00 Lymphatic system physiology, complete • • • •

36.10 Lymphatic tracts • • •

36.20 Lymph nodes • • •

36.40 Tonsils • • •

36.50 Thymus gland • • •

36.60 Spleen • • •

36.70 Peyer’s patches • • •

36.80 Appendix • • •


37.00 Lymphatic system, pathology (empty)

37.10 Lymph vessel inflammation • • •

37.11 Lymph vessel degeneration • • •

37.12 Lymphadenitis, swelling of a lymph node • • •

37.13 Lymph flow disorder • • •

37.14 Tonsillitis, acute • • • •

37.15 Lymphatic oedema • • • •

37.30 Spleen, strengthening the organ function • • • •

37.40 Thymus gland, strengthening the organ function • • • •

37.50 Appendicitis • • •

37.60 Symptomatic detoxication, complete • • • •

37.61 Detoxication, rheumatic toxines • • •

37.62 Detoxication, vaccination lesions • • •

37.70 Detoxication, metals • • • •

37.71 Detoxication, palladium • • •

37.72 Detoxication, cadmium • • •

37.73 Detoxication, mercury • • •

37.74 Detoxication, platinum • • •

37.75 Detoxication, copper • • •

36.76 Detoxication, nickel • • •

37.77 Detoxication, lead • • •

40


40.00 Heart physiology, complete • • • •

40.10 Heart layers, complete • • •

40.11 Pericardium • • •

40.12 Epicardium • • •

40.13 Myocardium • • •

40.14 Endocardium • • •

40.20 Heart interior, complete • • •

40.21 Right atrium • • •

40.22 Right ventricle • • •

40.23 Left atrium • • •

40.24 Left ventricle • • •

40.30 Cardiac valves, complete • • •

40.31 Tricuspid valve • • •

40.32 Pulmonary valve • • •

40.33 Mitral valve • • •

40.34 Aortic valve • • •

40.40 Conduction system, complete • • •

40.41 Sinus node • • •

40.42 Atrioventricular node • • •

40.43 Conduction system • • •

40.44 Atrioventricular bundle of His • • •


39.00 Circulatory system, pathology (empty)

39.10 Arterial impairment of the blood supply • • • •

39.15 Atherosclerosis • • •

39.20 Venous impairment of the blood supply (varicosis) • • • •

39.30 Inflammation of the blood vessels • • •

39.40 Degeneration of the blood vessels • • •

39.50 Blood pressure regulatory disorder • • •

39.60 High blood pressure (hypertonia) • • T • T39.65 Renal hypertension • • •

39.70 Low blood pressure (hypotonia) • • •


38.00 Circulatory system physiology, complete • • • •

38.10 Arteries • • •

38.40 Blood pressure receptors of the carotid artery • • •

38.50 Veins • • •

38.80 Capillaries • • •

41


41.00 Heart, pathology (empty)

41.10 Strengthening of the myocardium • • •

41.11 Strengthening of the heart capacity • • • •

41.20 Cardiac insufficiency, left • • •

41.30 Cardiac insufficiency, right • • •

41.40 Angina pectoris • • •

41.50 Psychogenic heart disorder • • •


40.50 Interventricular septum • • •

40.60 Apex of heart • • •


42.00 Respiratory system physiology, complete • • • •

42.10 Nose/olfactory organ, complete • • •

42.11 Dorsal tubinate • • •

42.12 Upper nasal meatus • • •

42.13 Central nasal meatus • • •

42.14 Lower nasal meatus • • •

42.15 Nasal mucous membrane • • •

42.16 Olfactory nerve • • •

42.17 Posterior nares • • •

42.20 Paranasal sinuses, complete • • •

42.21 Frontal sinuses • • •

42.22 Sphenoidal sinuses • • •

42.23 Bony ethmoidal cells • • •

42.24 Maxillary sinus • • •

42.30 Throat • • •

42.40 Larynx, complete • • •

42.41 Epiglottis • • •

42.42 Thyroid cartilage • • •

42.43 Cricoid cartilage • • •

42.44 Vocal ligaments • • •

42.50 Windpipe • • •

42.60 Bronchus, complete • • •

42.61 Main bronchus • • •

42.62 Lobar bronchus • • •

42.63 Segmental bronchus • • •

42.70 Lung • • •

42.71 Alveoles (air sacks) • • •

42


45.00 Kidney/urinary organs, pathology (empty)

45.05 Kidney failure • • •


44.00 Kidney/urinary organs, physiology complete • • • •

44.10 Kidney, complete • • •

44.11 Renal pelvis • • •

44.12 Renal calices • • •

44.13 Renal papilae • • •

44.14 Renal medulla • • •

44.15 Renal cortex • • •

44.16 Renal hilus • • •

44.17 Renal glomerul • • •

44.20 Urinary organs, complete • • •

44.21 Ureter • • •

44.22 Urinary bladder • • •

44.23 Urethra • • •

44.24 Sphincter • • •


43.00 Respiratory system, pathology (empty)

43.10 Cough • • • •

43.11 Rhinitis, acute (common cold) • • • •

43.12 Nasal polyps • • •

43.13 Bronchitis, acute • • • •

43.14 Bronchitis, chronic • • •

43.15 Sinusitis, acute • • • •

43.16 Sinusitis, chronic • • •

43.17 Pharyngitis • • •

43.18 Laryngitis • • •

43.20 Bronchial asthma • • T • T43.30 Mucoid degeneration • • •

43.40 Pleuritis sicca/exsudativa • • •

43.50 Pneumonia, bacterial • • •

43.51 Pneumonia, atypical • • •


42.80 Pleura • • •

42.81 Pulmonary pleura (pleura visceralis) • • •

42.82 Costal pleura (pleura parietalis) • • •

43


46.00 Digestive system, physiology complete • • • •

46.10 Oral cavity/tongue, complete • • •

46.11 Oral cavity • • •

46.12 Tongue • • •

46.13 Salivary glands • • •

46.14 Parotid gland • • •

46.15 Submandibular gland • • •

46.16 Sublingual gland • • •

46.20 Oesophagus • • •

46.30 Stomach • • •

46.31 Stomach glands • • •

46.32 Cardia • • •

46.33 Body • • •

46.34 Fundus • • •

46.35 Sphincter pylori • • •

46.38 Peritoneum • • •

46.40 Small intestines, complete • • •

46.41 Duodenum • • •

46.42 Jejunum • • •

46.43 Ileum • • •

46.44 Intestinal villi • • •

46.50 Colon, complete • • •

46.51 Appendix • • •

46.52 Vermicular appendix • • •

46.53 Ascending colon • • •


45.10 Glomerulonephritis • • •

45.11 Membranous glomerulonephritis • • •

45.12 Tubulo-interstitial glomerulonephritis • • •

45.15 Nephrosis (protein-losing kidney) • • •

45.16 Glomerulopathy • • •

45.20 Renal artery stenosis • • •

45.25 Nephrolithiasis (kidney stones) • • •

45.30 Pyelonephritis (pyelitis and kidney infection) • • •

45.35 Cystitis (inflammation of the bladder) • • T • T45.40 Urethritis (inflammation of the urethra) • • •

45.45 Diabetic nephropathy (diabetic glomerulosclerosis) • • •

45.50 Renal diabetes • • •

45.80 Water removal • • • •

44


46.54 Transverse colon • • •

46.55 Descending colon • • •

46.56 S-shaped colon • • •

46.59 Intestinal flora • • •

46.60 Straight bowel • • •

46.70 Anus • • •


47.00 Digestive system pathology (empty)

47.10 Oesophagitis • • •

47.20 Gastritis, acute • • • T • T47.30 Gastritis, chronic • • • T • T47.31 Gastritis, A type • • •

47.32 Gastritis, B type • • •

47.33 Gastritis, C type • • •

47.40 Gastric ulcer • • • •

47.45 Duodenal ulcer • • •

47.50 Crohn’s disease • • T • T47.60 Ulcerative colitis • • T • T47.70 Colon irritable (irritable bowel syndrome) • • •

47.80 Intestinal polyps • • •


48.00 Liver – gall – pancreas, physiology complete • • • •

48.10 Liver, complete • • •

48.11 Right liver lobe • • •

48.12 Left liver lobe • • •

48.13 Small liver lobe • • •

48.14 Hepatocytes (liver cells) • • •

48.15 Liver sinusoids • • •

48.16 Kupffer star cells • • •

48.20 Gall, complete • • •

48.21 Gall bladder • • •

48.22 Bile ducts • • •

48.30 Pancreas, complete • • •

48.31 Head of the pancreas • • •

48.32 Body of the pancreas • • •

48.33 Tail of the pancreas • • •

48.34 Pancreatic tract • • •

48.35 Islet cells • • •

45


49.00 Liver – gall – pancreas, pathology (empty)

49.10 Hepatitis • • •

49.15 Degeneration of the liver • • •

49.30 Bile formation disorder • • •

49.34 Bile flow disorder • • •

49.37 Inflammation of the gall bladder / tract • • •

49.38 Gallstones • • •

49.50 Pancreas, exocrine functional disorder • • •


50.00 Metabolism, physiology complete • • • •

50.10 Protein metabolism • • •

50.20 Carbohydrate metabolism • • •

50.30 Fat metabolism • • •


51.00 Metabolism, pathology (empty)

51.10 Protein metabolism disorder • • • •

51.11 Prions • • • •

51.20 Carbohydrate metabolism disorder • • • •

51.30 Fat metabolism disorder • • • •

51.40 Diabetes mellitus • • T • T51.50 Gout • • T • T


52.00 Musculoskeletal system, physiology complete • • • •

52.05 Bone cells • • •

52.06 Myelocytes • • •

52.07 Osteoblasts • • •

52.08 Osteocytes • • •

52.09 Osteoclasts • • •

52.10 Skeleton, complete • • •

52.11 Skeleton skull • • •

52.12 Skeleton shoulder • • •

52.13 Skeleton – upper extremities • • •

52.14 Skeleton hands • • •

52.15 Skeleton chest • • •

52.16 Skeleton hips / lower extremities • • •

52.17 Skeleton feet • • •

52.20 Musculature, complete • • •

46


53.00 Musculoskeletal system, pathology (empty)

53.11 Bone injury / fracture • • • •

53.12 Inflammation of the bone • • •

53.21 Sprain (distorsion) • • • •

53.22 Haematoma / bruise • • • •

53.23 Muscle tension • • • •

53.24 Injury of the muscle / fibre rupture • • •

53.25 Inflammation of the muscle • • •

53.26 Ligament injury • • • •


52.21 Musculature / ligaments head / neck • • •

52.22 Musculature / ligaments upper extremities/ trunk

• • •

52.23 Diaphragm • • •

52.24 Musculature / ligaments hands • • •

52.25 Musculature / ligaments lower extremities • • •

52.26 Tendons • • •

52.27 Peritenons • • •

52.28 Muscles of the pelvic floor / perineum • • •

52.30 Backbone, complete • • •

52.31 Cervical spine (C1 – C7) • • •

52.32 Thoracic spine (Th1 – Th12) • • •

52.33 Lumbar spine (L1 – L5) • • •

52.34 Sacral bone / coccyx bone • • •

52.40 Spinal discs, complete • • •

52.41 Spinal discs of the cervical spine (C1 – C7/Th1)

• • •

52.42 Spinal discs of the thoracic spine (Th1/Th2 – Th12/L1)

• • •

52.43 Spinal discs of the lumbar spine (L1/L2 – L5)

• • •

52.50 Bursa (knee) • • •

52.60 Joint, complete • • •

52.61 Capsular ligament • • •

52.62 Synovial fluid • • •

52.63 Periosteum • • •

52.64 Periochondrium • • •

52.65 Menisci • • •

52.66 Chondrogenesis • • •

47


53.27 Stretched ligament • • • •

53.28 Inflammation of a ligament / tendon sheath inflammation

• • • •

53.29 Inguinal hernia • • •

53.30 Amyotrophic lateral sclerosis / muscle atrophy • • •

53.31 Carpal tunnel syndrome • • • •

53.41 Backbone pain / tension • • • •

53.42 Backbone, degeneration of spinal discs • • •

53.51 Joint injury • • •

53.52 Joint inflammation (arthritis) • • • T • T53.53 Joint degeneration (arthrosis) • • •

53.54 Shortage of hyaluronic acid • • •

53.61 Bursa injury • • •

53.62 Bursitis • • •

53.70 Backaches, complete • • • •

53.71 Backache cervical spine • • •

53.72 Backache thoracic spine • • •

53.73 Backache lumbar spine • • •

53.80 Osteoporosis • • • •

53.81 Osteomalacia / rachitis • • •

53.82 Sciatica • • • •

53.83 Lumbago • • • •

53.84 Fibromyalgia • • T • T


54.00 Nervous system physiology, complete • • • •

54.10 Central nervous system, complete • • • •

54.11 Brain, cortex, nuclei • • •

54.12 Interbrain, midbrain • • •

54.13 Lobe of cerebrum, corpus callosum, cerebelum, pons

• • •

54.14 Pyramidal and extrapyramidal system • • •

54.15 Medulla oblongata (elongated marrow) • • •

54.16 Spinal marrow • • •

54.17 Blood-cerebrospinal fluid barrier • • •

54.18 Cerebrospinal fluid-brain barrier • • •

54.19 Cerebrospinal fluid • • •

54.20 Peripheral nervous system, complete • • • •

54.21 Cranial nerve I (olfactory nerve) • • •

54.22 Cranial nerve II (optic nerve) • • •

48


55.00 Nervous system, pathology (empty)

55.10 Sleep-onset insomnia (9-11pm) – often hormonal disorders

• • • •

55.20 Sleep-maintenance insomnia time 1 (11pm-01am early waking)

• • • •

55.21 Sleep-maintenance insomnia time 2 (01-03am early waking)

• • • •

55.22 Sleep-maintenance insomnia time 3 (03am – 05am early waking)

• • • •

55.30 Alzheimer’s disease • • T • T55.31 Parkinson’s disease • • T • T55.40 Neuritis • • •

55.41 Neuralgia • • •

55.42 Nerve degeneration • • •

55.43 Multiple Sclerosis • • •

55.44 Restless-leg-syndrome • • •

55.45 ADD/ADHD • • •

55.50 Cerebral concussion • • • •

55.55 Headache • • • •

55.60 Migraine • • • T • T


56.00 Organ of vision, physiology complete • • • •

56.10 Lachrymal gland, nasal duct, complete • • •


54.23 Cranial nerve III (oculomotor nerve) • • •

54.24 Cranial nerve IV (trochlear nerve) • • •

54.25 Cranial nerve V (trigeminal nerve) • • •

54.26 Cranial nerve VI (abducens nerve) • • •

54.27 Cranial nerve VII (facial nerve) • • •

54.28 Cranial nerve VIII (vestibulocochlear nerve) • • •

54.29 Cranial nerve IX (glossopharyngeal nerve) • • •

54.30 Cranial nerve X (vagus nerve) • • •

54.31 Cranial nerve XI (accessory nerve) • • •

54.32 Cranial nerve XII (hypoglossal nerve ) • • •

54.35 Nerve ganglia • • • •

54.36 Ischiadic nerve • • •

54.50 Autonomic nervous system • • •

54.60 Psychosomatic control • • •

49


57.00 Eye, pathology (empty)

57.10 Retinal detachment • • •

57.20 Cataract • • •

57.30 Glaucoma • • •

57.40 Wet macular degeneration • • T • T57.41 Dry macular degeneration • • T • T57.50 Hordeolum • • • •

57.51 Chalazion • • • •

57.52 Conjunctivitis • • • •


58.00 Acoustic organ, physiology complete • • • •

58.10 Auricle, complete • • •

58.11 Auricle • • •

58.12 Ear cartilage • • •


56.11 Lachrymal gland • • •

56.12 Nasal duct • • •

56.13 Lachrymal point • • •

56.14 Lachrymal sac • • •

56.20 Chambers of the eye, complete • • •

56.21 Front eye chamber • • •

56.22 Rear eye chamber • • •

56.30 Layers, complete • • •

56.31 Conjunctiva • • •

56.32 Cornea • • •

56.33 Iris • • •

56.34 Retina • • •

56.35 Choroid • • •

56.36 Dermis • • •

56.40 Lens, pupil, vitreous body, complete • • •

56.41 Lens • • •

56.42 Pupil • • •

56.43 Vitreous body • • •

56.50 Musculature, nerve, socket of the eye • • •

56.60 Visual nerves, complete • • •

56.61 Visual nerve • • •

56.62 Yellow spot • • •

56.63 Blind spot • • •

50


59.00 Acoustic organ / organ of equilibrium, pathology (empty)

59.10 Tinnitus • • • T • T59.20 External otitis • • •

59.21 Otitis media, acute (acute ear) • • • •

59.30 Menière’s disease • • •

59.40 Acute hearing loss • • •


62.00 Skin / hair, physiology complete • • • •

62.10 Skin, complete • • •

62.11 Epidermis • • •

62.12 Dermis • • •

62.13 Subcutis • • •

62.14 Fatty tissue • • •

62.15 Melanocytes (melanin forming cells) • • •

62.16 Keratinocytes • • •

62.20 Skin glands, complete • • •

62.21 Sebaceous gland • • •

62.22 Sweat gland • • •

62.50 Hair • • •

62.60 Nails, complete • • •

62.61 Onychogenesis, basic structure • • •


58.20 External ear, complete • • •

58.21 Cartilaginous part • • •

58.22 Bony part • • •

58.23 Earwax glands • • •

58.30 Middle ear, complete • • •

58.31 Ear drum, tympanic membrane • • •

58.32 Hammer, maleus • • •

58.33 Anvil, incus • • •

58.34 Stirrup / oval window • • •

58.35 Tympanum / Eustachian tube • • •

58.40 Inner ear, complete • • •

58.41 Semicircular canals • • •

58.42 Cochlea • • •

58.43 Acoustic nerve and nerve of equilibrium (nerve VIII)

• • •

51


63.00 Skin / hair, pathology (empty)

63.10 Psoriasis • • •

63.20 Neurodermatitis • • • T • T63.30 Contact dermatitis (allergic) • • •

63.40 Urticaria • • •

63.50 Epidermatomycoses • • •

63.60 Lichen • • •


64.00 Hormonal system, physiology complete • • • •

64.05 Pineal gland (epiphysis) • • •

64.06 Melatonin hormone • • •

64.10 Hypothalamus • • •

64.11 Sleeping-waking-centre • • •

64.12 Thermoregulation centre • • •

64.13 Oxytocic hormone • • •

64.14 Antidiuretic hormone • • •

64.15 Gonadotropin releasing hormone • • •

64.20 Hypophysis • • •

64.21 ACTH (from anterior lobe of the hypophysis) • • •

64.22 STH (from anterior lobe of the hypophysis) • • •

64.23 Melanotropin (MSH) • • •

64.24 Follicle stimulating hormone (FSH) • • •

64.25 Luteinising hormone • • •

64.27 Histamine • • •

64.28 Dopamine • • •

64.29 Serotonin • • •

64.30 Thyroid gland • • •

64.35 Para-thyroid gland • • •

64.40 Thymus gland • • •

64.50 Adrenal medulla • • •

64.55 Adrenal cortex • • •

64.60 Kidney • • •

64.65 Erythropoietin • • •

64.70 Pancreas • • •

64.80 Ovary • • •

64.81 Oestrogens • • •

64.82 Progesterone / gestagens • • •

64.85 Testicles • • •

64.86 Testosterone • • •

52


66.00 Female sexual organs, physiology complete • • • •

66.10 External female genitalia • • •

66.11 Greater labia • • •

66.12 Clitoris • • •

66.13 Lesser labia • • •

66.14 Bartholin’s glands • • •

66.15 Mammary glands with mamillae • • •

66.16 Lactiferous glands • • •

66.17 Lactiferous tubules • • •

66.18 Prolactin • • •

66.19 Colostrum • • •


65.00 Hormonal system, pathology (empty)

65.10 Female hormonal balance, basic regulation • • • •

65.20 Male hormonal balance, basic regulation • • • •

65.30 Hypothalamus • • •

65.31 Anterior lobe of pituitary • • •

65.32 Posterior lobe of pituitary • • •

65.33 Thyroid gland hyperfunction • • •

65.34 Thyroid gland hypofunction • • •

65.35 Parathyroid gland, hyperfunction • • •

65.36 Parathyroid gland, hypofunction • • •

65.37 Hyperfunction of the adrenal cortex • • •

65.38 Hypofunction of the adrenal cortex • • •

65.39 Hyperfunction of the adrenal medulla • • •

65.40 Hypofunction of the adrenal medulla • • •

65.45 Premenstrual syndrome • • • •

65.50 Menstruation programs, complete • • • •

65.51 Amenorrhea • • •

65.52 Oligomenorrhea • • •

65.53 Polymenorrhea • • •

65.54 Hypermenorrhea • • •

65.55 Hypomenorrhea • • •

65.56 Metrorrhagia • • •

65.60 Menopause complaints • • • T • T65.61 Female gonad, exocrine functional disorder • • •

65.62 Female gonad, exocrine functional disorder • • •

65.65 Male gonad, exocrine functional disorder • • •

65.66 Male gonad, exocrine functional disorder • • •

53


67.00 Female sexual organs, pathology (empty)

67.10 Salpingitis • • • •

67.20 Ovariitis • • • •

67.30 Endometriosis • • T • T67.40 Mastitis • • •

67.50 Vaginitis • • •


68.00 Male sexual organs, physiology complete • • • •

68.10 External male genitalia • • •

68.11 Scrotum • • •

68.12 Penis • • •

68.20 Internal male genitalia • • •

68.21 Testicles • • •

68.22 Epididymis • • •

68.23 Spermatic duct • • •

68.24 Seminal vesicle • • •

68.25 Cowper’s glands • • •

68.26 Prostate gland • • •

68.27 Spermatic cord • • •


66.30 Internal female genitalia • • •

66.31 Ovaria • • •

66.32 Oviducts • • •

66.33 Uterus • • •

66.34 Placental barrier • • •

66.35 Amniotic fluid • • •

66.36 Vagina • • •


69.00 Male sexual organs, pathology (empty)

69.10 Prostate gland, functional disorder • • •

69.20 Potency-enhancing • • •

69.30 Prostatitis • • • •


70.00 Cause-oriented system therapy

70.10 Nervous system • • • •

70.11 Hair and scalp • • • •

54


72.00 Psyche • • • •

72.10 Depression • • • •

72.11 Episode of depression • • •

72.12 Recurring depressive disorders • • •

72.13 Continuous affective disorders • • •

72.14 Cyclothymia • • •

72.15 Dysthymia • • •


71.00 Pain (empty)

71.11 Pain receptors • • •

71.50 Pain relief • • • •


70.12 Eye system • • • •

70.13 Tongue, oral cavity, salivary glands • • • •

70.14 Teeth, jawbone, mouth • • • •

70.15 Acoustic organ, organ of equilibrium • • • •

70.16 Upper respiratory system • • • •

70.17 Lung system • • • •

70.18 Heart • • • •

70.19 Digestive organs • • • •

70.20 Liver, gall, pancreas • • • •

70.21 Kidneys, ureter • • • •

70.22 Female organs • • • •

70.23 Male organs • • • •

70.24 Skin system • • • •

70.25 Artery and vein system • • • •

70.26 Musculature I • • • •

70.27 Musculature II • • • •

70.28 Skeleton I • • • •

70.29 Skeleton II • • • •

70.40 Borreliosis, Rickettsioses • • • •

70.41 Helicobacter pylori infection • • • •

70.42 Infectious mononucleosis, acute • • • •

70.43 Infectious mononucleosis, chronic • • • •

70.44 Cytomegaly, chronic • • • •

70.45 Migraine, headache, insomnia, psychic state of unbalance, pathogen-oriented

• • • •

70.46 Influenza • • • •

55


75.00 Stress • • • •

75.10 Stress reduction • • • •

75.15 Weight reduction • • • •

75.16 Giving up smoking • • • •

75.17 Giving up an addiction • • • •

75.18 Meteorosensitivity • • • •

75.19 Learning program / concentration enhancement • • • •

75.20 Mental stress • • • •


76.00 Teeth, physiology, total • • • •

76.10 Teeth, superior maxilla (adult) • • • •

76.11 Tooth 11 • • •

76.12 Tooth 12 • • •

76.13 Tooth 13 • • •

76.14 Tooth 14 • • •

76.15 Tooth 15 • • •

76.16 Tooth 16 • • •

76.17 Tooth 17 • • •

76.18 Tooth 18 • • •

76.21 Tooth 21 • • •

76.22 Tooth 22 • • •

76.23 Tooth 23 • • •

76.24 Tooth 24 • • •

76.25 Tooth 25 • • •

76.26 Tooth 26 • • •

76.27 Tooth 27 • • •

76.28 Tooth 28 • • •

76.30 Teeth, inferior maxilla (adult) • • • •

76.31 Tooth 31 • • •

76.32 Tooth 32 • • •

76.33 Tooth 33 • • •

76.34 Tooth 34 • • •

76.35 Tooth 35 • • •

76.36 Tooth 36 • • •


72.16 Adaptation disorders • • •

72.17 Phobic neuroses • • •

72.18 Panic attacks • • •

56


76.37 Tooth 37 • • •

76.38 Tooth 38 • • •

76.41 Tooth 41 • • •

76.42 Tooth 42 • • •

76.43 Tooth 43 • • •

76.44 Tooth 44 • • •

76.45 Tooth 45 • • •

76.46 Tooth 46 • • •

76.47 Tooth 47 • • •

76.48 Tooth 48 • • •

76.50 Milk teeth, upper jaw (child) • • • •

76.51 Milk tooth 51 • • •

76.52 Milk tooth 52 • • •

76.53 Milk tooth 53 • • •

76.54 Milk tooth 54 • • •

76.55 Milk tooth 55 • • •

76.61 Milk tooth 61 • • •

76.62 Milk tooth 62 • • •

76.63 Milk tooth 63 • • •

76.64 Milk tooth 64 • • •

76.65 Milk tooth 65 • • •

76.70 Milk teeth, lower jaw (child) • • • •

76.71 Milk tooth 71 • • •

76.72 Milk tooth 72 • • •

76.73 Milk tooth 73 • • •

76.74 Milk tooth 74 • • •

76.75 Milk tooth 75 • • •

76.81 Milk tooth 81 • • •

76.82 Milk tooth 82 • • •

76.83 Milk tooth 83 • • •

76.84 Milk tooth 84 • • •

76.85 Milk tooth 85 • • •


77.00 Teeth, pathology (empty)

77.05 Cyst of the jaw • • •

77.10 Toothache • • • •

77.11 Toothache, acute • • • •

77.15 Parodontitis • • •

77.20 Parodontosis • • •

57


81.00 Bach flowers, complete • • • •

81.01 Agrimony • • •

81.02 Aspen • • •

81.03 Beech • • •

81.04 Centaury • • •

81.05 Cerato • • •

81.06 Cherry Plum • • •

81.07 Chestnut Bud • • •

81.08 Chicory • • •

81.09 Clematis • • •

81.10 Crab Apple • • •

81.11 Elm • • •

81.12 Gentian • • •

81.13 Gorse • • •

81.14 Heather • • •

81.15 Holly • • •

81.16 Honeysuckle • • •

81.17 Hornbeam • • •

81.18 Impatiens • • •

81.19 Larch • • •

81.20 Mimulus • • •

81.21 Mustard • • •

81.22 Oak • • •

81.23 Olive • • •

81.24 Pine • • •

81.25 Red Chestnut • • •

81.26 Rock Rose • • •

81.27 Rock Water • • •

81.28 Scleranthus • • •

81.29 Star of Bethlehem • • •

81.30 Sweet Chestnut • • •

81.31 Vervain • • •

81.32 Vine • • •

81.33 Walnut • • •


77.25 Gingivitis • • •

77.30 Apical granuloma • • •

77.35 Dental caries (prophylaxis) • • •

77.40 Teething problems (milk teeth) • • • •

58


82.00 Schuessler salts, complete • • • •

82.01 Calcium fluoratum • • •

82.02 Calcium phosphoricum • • •

82.03 Ferrum phosphoricum • • •

82.04 Potassium chloratum • • •

82.05 Potassium phosphoricum • • •

82.06 Potassium sulfuricum • • •

82.07 Magnesium phosphoricum • • •

82.08 Sodium chloratum • • •

82.09 Sodium phosphoricum • • •

82.10 Sodium sulfuricum • • •

82.11 Silicea • • •

82.12 Calcium sulfuricum • • •

82.13 Potassium arsenicum • • •

82.14 Potassium bromatum • • •

82.15 Potassium jodatum • • •

82.16 Lithium chloratum • • •

82.17 Manganum sulfuricum • • •

82.18 Calcium sulfuratum • • •

82.19 Cuprum arsenicosum • • •

82.20 Potassium aluminium sulfuricum • • •

82.21 Zincum chloratum • • •

82.22 Calcium carbonicum • • •

82.23 Sodium bicarbonicum • • •

82.24 Arsenicum jodatum • • •

82.25 Aurum chloratum natronatum • • •

82.26 Selenium • • •

82.27 Potassium bichromicum • • •


85.00 Periodic Table of the Elements (PT), total • • • •

85.01 Hydrogen (H) • • •

85.02 Helium (He) • • •


81.34 Water Violet • • •

81.35 White Chestnut • • •

81.36 Wild Oat • • •

81.37 Wild Rose • • •

81.38 Willow • • •

59


85.03 Lithium (Li) • • •

85.04 Beryllium (Be) • • •

85.05 Boron (B) • • •

85.06 Carbon (C) • • •

85.07 Nitrogen (N) • • •

85.08 Oxygen (O) • • •

85.09 Fluor (F) • • •

85.10 Neon (Ne) • • •

85.11 Sodium (Na) • • •

85.12 Magnesium (Mg) • • •

85.13 Aluminium (Al) • • •

85.14 Silicon (Si) • • •

85.15 Phosphor (P) • • •

85.16 Sulphur (S) • • •

85.17 Chlorine (Cl) • • •

85.18 Argon (Ar) • • •

85.19 Potassium (K) • • •

85.20 Calcium (Ca) • • •

85.21 Scandium (Sc) • • •

85.22 Titan (Ti) • • •

85.23 Vanadium (V) • • •

85.24 Chromium (Cr) • • •

85.25 Manganese (Mn) • • •

85.26 Iron (Fe) • • •

85.27 Cobalt (Co) • • •

85.28 Nickel (Ni) • • •

85.29 Copper (Cu) • • •

85.30 Zinc (Zn) • • •

85.31 Gallium (Ga) • • •

85.32 Germanium (Ge) • • •

85.33 Arsenic (As) • • •

85.34 Selenium (Se) • • •

85.35 Bromine (Br) • • •

85.36 Krypton (Kr) • • •

85.37 Rubidium (Rb) • • •

85.38 Strontium (Sr) • • •

85.39 Yttrium (Y) • • •

85.40 Zirconium (Zr) • • •

85.41 Niobium (Nb) • • •

85.42 Molybdenum (Mo) • • •

60


85.43 Technetium (Tc) • • •

85.44 Ruthenium (Ru) • • •

85.45 Rhodium (Rh) • • •

85.46 Palladium (Pd) • • •

85.47 Silver (Ag) • • •

85.48 Cadmium (Cd) • • •

85.49 Indium (In) • • •

85.50 Tin (Sn) • • •

85.51 Antimony (Sb) • • •

85.52 Tellurium (Te) • • •

85.53 Iodine (J) • • •

85.54 Xenon (Xe) • • •

85.55 Caesium (Cs) • • •

85.56 Barium (Ba) • • •

85.57 Lanthanum (La) • • •

85.58 Cerium (Ce) • • •

85.59 Praseodymium (Pr) • • •

85.60 Neodymium (Nd) • • •

85.61 Promethium (Pm) • • •

85.62 Samarium (Sm) • • •

85.63 Europium (Eu) • • •

85.64 Gadolinium (Gd) • • •

85.65 Terbium (Tb) • • •

85.66 Dysprosium (Dy) • • •

85.67 Holmium (Ho) • • •

85.68 Erbium (Er) • • •

85.69 Thulium (Tm) • • •

85.70 Ytterbium (Yb) • • •

85.71 Lutetium (Lu) • • •

85.72 Hafnium (Hf) • • •

85.73 Tantalum (Ta) • • •

85.74 Tungsten (W) • • •

85.75 Rhenium (Re) • • •

85.76 Osmium (Os) • • •

85.77 Iridium (Ir) • • •

85.78 Platinum (Pt) • • •

85.79 Gold (Au) • • •

85.80 Mercury (Hg) • • •

85.81 Thallium (Tl) • • •

85.82 Lead (Pb) • • •

61


85.83 Bismuth (Bi) • • •

85.84 Polonium (Po) • • •

85.85 Astatine (At) • • •

85.86 Radon (Rn) • • •

85.87 Francium (Fr) • • •

85.88 Radium (Ra) • • •

85.89 Actinium (Ac) • • •

85.90 Thorium (Th) • • •

85.91 Protactinium (Pa) • • •

85.92 Uranium (U) • • •

85.93 Neptunium (Np) • • •

85.94 Plutonium (Pu) • • •

85.95 Americium (Am) • • •

85.96 Curium (Cm) • • •

85.97 Berkelium (Bk) • • •

85.98 Californium (Cf) • • •

85.99 Einsteinium (Es) • • •

86.00 Fermium (Fm) • • •

86.01 Mendelevium (Md) • • •

86.02 Nobelium (No) • • •

86.03 Lawrencium (Lr) • • •

86.04 Rutherfordium (Rf) • • •

62

38.00 Circulatory system physiology, complete

corresponding meridians N +/- corresponding germs N +/-

02.15 Heart channel 21.88 Rickettsiae

02.19 Liver channel 24.51 Clonorchis sinesi

25.15 Chilomastix cysts (rat)

25.16 Chilomonas

25.85 Blood parasites

27.10 Yeast fungus, complete

40.00 Heart physiology, complete


02.20 Channel of the heart and circulation 20.22 Streptococcus mitis

21.88 Rickettsiae

24.22 Dirofilaria immitis (heart worm)

24.51 Clonorchis sinesi

25.15 Chilomastix cysts (rat)

25.16 Chilomonas


25.86 Pneumocystis carinii

42.00 Respiratory system physiology, complete


02.11 Lung channel 20.12 Beta haemolytic streptococci

02.12 Colon channel 20.19 Staphylococcus aureus

02.14 Spleen channel 20.22 Streptococcus mitis

02.17 Bladder channel 20.23 Streptococcus pneumoniae

02.21 Sanjiao channel 20.24 Streptococcus pyogenes

02.22 Gall bladder channel 20.44 Bacilli

20.49 Bordetella pertussis

20.67 Haemophilus influenzae

20.72 Legionella

20.76 Mycobacteria tuberculosis

21.15 Klebsiella pneumoniae

21.86 Chlamydia trachomatis

21.91 Laryngeal 1 bacteria

9 Appendix II: Organ-specific meridian and pathogen tables

63

44.00 Kidney/urinary organs, physiology complete


02.12 Colon channel 20.66 Gardnerella vaginalis

02.17 Bladder channel 21.14 Escherichia coli

02.18 Kidney channel 21.16 Proteus mirabilis

02.22 Gall bladder channel 21.17 Proteus vulgaris

24.63 Schistosoma haematica

24.64 Schistosoma masoni

24.65 Urocleidus

25.41 Trichomonas vaginalis



27.11 Candida albicans

46.00 Digestive system, physiology complete


02.13 Stomach channel 20.22 Streptococcus mitis

02.14 Spleen channel 20.69 Helicobacter pylori

02.19 Liver channel 21.11 Enterobacter aerogenes

02.21 Sanjiao channel 21.19 Salmonella enteritidis

02.22 Gall bladder channel 21.20 Salmonella paratyphi

02.24 Channel of the Conception Vessel 21.21 Salmonella typhi

21.23 Shigella dysenteriae

21.93 Caries bacterium


22.11 Adenovirus


22.13 Epstein-Barr virus (EBV)


22.17 Herpes zoster

22.67 Coxsackie virus B-1


22.80 Rhinovirus

23.16 Parainfluenza

23.33 Influenza virus A and B

23.81 Viruses N.N.

24.21 Ascaris megalocephala



26.41 Aflatoxin

64


22.78 Norovirus

23.56 Rotaviruses

24.21 Ascaris megalocephala

24.23 Enterobius vermicularis

24.28 Enterobius worms

24.31 Strongyloides (filariform)

24.54 Eurytrema pancreaticum

24.56 Fasciolopsis buski

24.58 Gastrothylax elongatus

24.63 Schistosoma haematica

24.64 Schistosoma masoni

24.84 Taenia saginata

24.85 Taenia solium

25.35 Naegleria fowleri


48.00 Liver - gall - pancreas, physiology complete


02.19 Liver channel 20.69 Helicobacter pylori

02.22 Gall bladder channel 22.14 Hepatitis B virus

02.23 Channel of the Governor Vessel 22.74 Hepatits A virus

22.75 Hepatitis C virus

24.41 Capillaria hepatica (liver)

24.54 Eurytrema pancreaticum

24.55 Fasciola heptica

24.58 Gastrothylax elongatus

24.81 Echinococcus granulosus

24.82 Echinococcus multicolaris

26.41 Aflatoxin

52.00 Musculoskeletal system, physiology complete


02.12 Colon channel 20.22 Streptococcus mitis

02.16 Channel of the small intestines 20.76 Mycobacteria tuberculosis

02.17 Bladder channel 21.27 Yersinia enterocolitica

02.19 Liver channel 21.61 Borellia

02.22 Gall bladder channel 21.86 Chlamydia trachomatis

21.88 Rickettsiae

21.95 Pain-producing bacteria

21.96 Tuberculinum burnetti

65

54.00 Nervous system physiology, complete


02.15 Heart channel 20.22 Streptococcus mitis

02.17 Bladder channel 21.61 Borellia

02.18 Kidney channel 21.88 Rickettsiae

02.19 Liver channel 21.95 Pain-producing bacteria

02.20 Channel of the heart and circulation 22.12 Cytomegalovirus (CMV)



22.17 Herpes zoster

22.64 Chikungunya



23.11 Borna virus

23.56 Rotaviruses

23.81 Viruses N.N.


25.64 Demodex folliculorum (hair follicle mite)



26.41 Aflatoxin





22.17 Herpes zoster

22.64 Chikungunya



23.56 Rotaviruses

23.81 Viruses N.N.

24.32 Trichinella spiralis (muscle)

24.33 Trichuris sp.

24.61 Paragonimus Westermani

24.62 Prosthogonimus macro.




51.11 Prions

66

58.00 Acoustic organ, physiology complete


02.16 Channel of the small intestines 20.12 Beta haemolytic streptococci

02.18 Kidney channel 20.22 Streptococcus mitis

21.88 Rickettsien




22.17 Herpes zoster

22.64 Chikungunya

23.81 Viruses N.N.




26.41 Aflatoxin

62.00 Skin / hair, physiology complete


02.11 Lung channel 05.00 Geopathic disorders, complete

02.12 Colon channel 20.12 Beta haemolytic streptococci

02.14 Spleen channel 20.13 Eikanella corrodens

02.18 Kidney channel 20.19 Staphylococcus aureus

56.00 Organ of vision, physiology complete


02.19 Liver channel 20.12 Beta haemolytic streptococci

02.22 Gall bladder channel 20.19 Staphylococcus aureus


21.88 Rickettsiae




22.17 Herpes zoster

22.64 Chikungunya



25.14 Blepharisma





67

66.00 Female sexual organs, physiology complete


02.13 Stomach channel 20.12 Beta haemolytic streptococci

02.14 Spleen channel 20.19 Staphylococcus aureus

02.18 Kidney channel 20.21 Streptococcus lactis

02.24 Channel of the Conception Vessel 20.22 Streptococcus mitis

20.23 Streptococcus pneumoniae

20.24 Streptococcus pyogenes


02.19 Liver channel 20.21 Streptococcus lactis




20.25 Streptococcus sp.

20.42 Actinomyces israelii

20.46 Bacillus cereus

20.47 Bacteroides fragilis

20.66 Gardnerella vaginalis

20.70 Lactobacillus acidophilus

20.81 Propionobacterium acnes

21.12 Erwinia amylovora

21.13 Erwinia carotavora

21.16 Proteus mirabilis

21.17 Proteus vulgaris

21.22 Serratia marcescens

21.23 Shigella dysenteriae



22.17 Herpes zoster

22.82 Tobacco mosaic virus

23.70 Wart frequencies, complete

23.81 Viruses N.N.


25.64 Demodex folliculorum (hair follicle mite)

25.67 Ornithonyssus (bird mite)

25.68 Sarcoptes scabei (scabies)

25.84 Troglodytella abrasseri

26.05 Fungi I, complete

27.05 Fungi II, complete

68

76.00 Teeth, physiology, total


02.11 Lung channel 20.22 Streptococcus mitis

02.12 Colon channel 21.93 Caries bacterium

02.13 Stomach channel 22.15 Herpes simplex

02.14 Spleen channel 22.17 Herpes zoster

02.15 Heart channel 24.52 Cryptocotyle lingua

02.16 Channel of the small intestines 26.05 Fungi I, complete

02.17 Bladder channel 27.05 Fungi II, complete

02.18 Kidney channel 63.60 Lichen

02.19 Liver channel

02.22 Gall bladder channel

68.00 Male sexual organs, physiology complete


02.18 Kidney channel 20.12 Beta haemolytic streptococci

02.19 Liver channel 20.19 Staphylococcus aureus

02.24 Channel of the Conception Vessel 20.21 Streptococcus lactis






22.17 Herpes zoster

22.18 Humanes Papilloma Virus (HPV)





21.86 Chlamydia trachomatis


22.17 Herpes zoster

22.18 Human papilloma virus (HPV)



69

10 Appendix III: Information on bacteria, viruses, parasites and fungi

BacteriaProgram-no. Description

20.00 Bacteria, complete Bacteria are tiny, single-cell beings, so-called micro-organisms which

multiply by means of single transverse division. Bacteria can have a diffe-

rent appearance (morphology) which can be divided into three basic forms:

cocci, bacilli and helical bacteria. Bacteria can be passed on via air, water,

soil and bodily substances such as blood, stool, urine and bodily secretions.

Many bacteria are very useful for us people, such as in the intestinal flora.

Others, on the other hand, can result in acute illnesses. Bacterial infections

usually start off localised, at one specific point. They can spread to the

entire body.

20.05 Bacteria I, complete This program contains all bacteria from the program groups 20 and 21.

20.10 Coccobacilli, complete This contains all bacteria from the program group 20.

20.11 Alpha streptococcus Infections with streptococci occur very frequently. Depending on the type

of streptococci, infectious diseases such as scarlet fever, angina, meningitis,

otitis media, wound infections or even urinary tract infections can occur.

20.12 Beta haemolytic streptococci See above

20.13 Eikanella corrodens These bacteria are part of the normal flora of the oral cavity and of the

upper respiratory tract. An infection occurs following a bite from a dog or

a person. The consequences of such an infection could be illnesses such as

endocarditis and meningitis.

20.14 Gaffkya tetragena Infections with these bacteria result in illnesses of the respiratory tracts.

20.15 Meningococcus These are transferred from person to person by means of air-borne infec-

tions, such as by coughing or sneezing at somebody, or by kissing. In the

case of a weakened immune system, caused for instance by other infec-

tions, the bacteria multiply, get into the mucous membranes and cause

meningitis and blood poisoning.

20.16 MRSA multidrug-resistant V Originally methicillin-resistant Staphylococcus aureus, called after an

antibiotic no longer used these days, whose resistance was first observed

during the 1960s. Its biological properties are no different to the antibiotic

sensitive staphylococcus aureus strains.

70


20.16 MRSA multidrug-resistant V MRSA strains produce a changed penicillin binding protein. This makes

them resistant to all beta-lactam antibiotics (penicillin, cephalosporins

and carbapenem). Sources of infection: invasive catheters (dialysis shunt),

wound drainage, burns, chronic skin lesions.

20.17 Neisseria gonorrhoea The pathogens of gonorrhoea, a sexually transmitted disease. The patho-

gens are passed on during sexual intercourse or by means of a smear infec-

tion.

20.18 Staphylococci As pathogens, these bacteria populate the skin and mucous membranes of

people and animals, and also occur in the environment, such as on food.

20.19 Staphylococcus aureus These pathogens occur very frequently in the case of wound infections,

abscesses and boils.

20.20 Streptococcus Infections caused by streptococci bacteria range from mild infections such

as impetigo (skin infection), tonsillitis and sore throats through to toxic

shock syndrome and to nectrotizing fasciitis.

20.21 Streptococcus lactis See above

20.22 Streptococcus mitis See above

20.23 Streptococcus pneumoniae See above

20.24 Streptococcus pyogenes See above

20.25 Streptococcus sp. See above

20.26 Veillonella dispar As part of the normal flora, these bacteria live in the upper respiratory

tracts, in the intestinal tract and in the vagina of the organism. In the

event of unfavourable milieu changes in the organism, they are found in

combination with other bacteria as well as in the case of endocarditis,

inflammation of the joints and abscesses.

20.40 Rod-shaped bacteria, complete

20.41 Actinobacillus (suis) V This pathogen is found mainly in pigs.

Infections caused by this pathogen display the following symptoms: pain,

increased heart frequencies, fever, states of shock, weakened immune

systems.

20.42 Actinomyces israelii This pathogen causes actinomycosis (ray fungus). The actinomycosis is a

bacterial mixed infection. In the event of injury of the mucous membrane,

the germ penetrates deeper layers of tissue: occurrence in the CNS, the

lung (by breathing it in) and the skin is rare.

20.43 Arcanobacterium pyogenes These bacteria frequently cause serious clinic mastitis. The pathogen can

be passed on by flies. Entry points to the organism are offered by wound

infections, teat injuries, udder infections and abscesses.

71


20.44 Bacilli The bacillus genus is found in its natural habitat, the ground. Infections can

be caused by infected animals or animal products with spores. The patho-

gens can also be breathed in.

20.45 Bacillus anthracis V These pathogens cause so-called anthrax (cutaneous anthrax, pulmonary

anthrax). The bacteria live in the ground for decades in spore form. General-

ly, they enter the body via skin injuries or by breathing them in.

20.46 Bacillus cereus This bacterium occurs natural in the ground, making it one of the most

common cultivable soil bacteria. It is a bacterium which is poisonous to

food and which occurs particularly frequently in rice plants. The spores

which occur in raw rice survive the cooking process, and multiply. The

toxins can cause both vomitting and diarrhoea.

20.47 Bacteroides fragilis These pathogens are part of the physiological flora of humans and animals.

They occur frequently in mixed infections such as in peritonitis, gynaecolo-

gical infections (e.g. Fallopian tube or ovary), aspiration pneumonia, sinusi-

tis and brain abscesses. Infections are mainly endogenous, i.e. originate

from the physiological flora of our own bodies.

20.48 Bordetella bronchiseptica This pathogen causes illnesses to the upper respiratory tracts in mammals

and rodents, such as bronchitis and pneumonia. It is implicated in the

rhinotracheitis complex as well as in dogs.

20.49 Bordetella pertussis Bacterium of the genus bordetella attributed great medical importance as

the pathogen of whooping cough. The source of infection for whooping

cough are sick people who cough out the pathogen. There is no such thing

as a healthy germ carrier. Furthermore, transmission of the pathogen might

also occur by means of contaminated objects since the pertussis bacterium

can survive outside of the organism for several days.

20.50 Brucella abortus V Pathogen of brucellosis.

20.51 Brucella melitensis V Pathogen of Mediterranean fever, Malta fever, undulant fever. Symptomes:

fever, sickness, swelling of the liver, spleen and lymph nodes. Brucella

can survive for several weeks in unpasturised milk and cheese made from

unpasturised milk: this ability to survive presents it with its main path of

infection. Infected animals (faeces, urine) can be a source of infection for

farmers and vets.

20.52 Brucella suis V A pathogen form which particularly affects pigs.

72


20.53 Coxiella burnetii V Pathogen of Q fever. The pathogen is widespread throughout the world,

and can be spread by sheep or even by pets such as dogs and cats, as well

as by cows and goats to people. The actual carrier when this illness is

passed between animals is the tick: ingesting infected faeces or milk can

also cause it to spread. Infection in humans is aerobic, e.g. via breathing in

infected dust.

20.54 Clostridia These bacteria occur everywhere (ubiquitous), especially in soil and in the

digestive tract of higher living things. Their toxins can trigger various infec-

tion illnesses, e.g. anaerobic cellulitis, gas gangrene and tetanus.

20.55 Clostridium botulinum V This bacterium is a soil dweller. When multiplying, a toxin forms, a botuli-

numtoxin, which is the cause behind an illness known by the name of

botulism. When deprived of oxygen, such as in closed tins or in raw ham,

if the food is not refrigerated, the bacterium can multiply and form toxins

which can cause food poisoning. Since the pathogens are soil dwellers,

most cases of contamination are found in tins of vegetables.

20.56 Clostridium feseri V This pathogen causes blackleg, a non-infectious, acute, highly febrile,

sometimes endemic animal plague. The natural sources of infection for the

animals are fodder and water infected with spores of the blackleg patho-

gen, or wound infections.

20.57 Clostridium perfringens Along with other clostridia, it belongs to the group of gas gangrene bacillus:

it is the most frequent pathogen of gas gangrene. Furthermore, the bacteri-

um is a frequent cause of nectrotizing pneumonia, gangrenous cholecysti-

tis, of sepsis or other non-specific infections. Clostridium perfringens can

cause infections of the central nervous system. In animals, the illnesses

caused by clostridium perfringens toxins are known as enterotoxaemia.

20.58 Clostridium septicum These bacteria are the pathogens of the para-blackleg illness. This illness

is a feverish, usually fatal infectious disease whose clinical picture cannot

be distinguished from that of blackleg. Also caused by clostridium septi-

cum is abomasum para-blackleg of sheep (Nordic Bradsot). The pathogen is

pathogenic for all domesticated mammals, humans and pigeons.

20.59 Clostridium tetani V The reservoirs of this pathogen are soil and wood as well as the excrement

of cows and other species of animal. Open wounds can quickly become

infected with the bacterium, therefore resulting in tetanus. Clinical

symptoms start with headaches and increased reflexes.

73


20.59 Clostridium tetani V Spasmophilia of a painful and violent nature occurs.

20.60 Corynebacterium diphteriae This bacterium is the pathogen for diphtheria. It is spread via direct contact

from person to person with infected people, usually via droplets, less

frequently via contaminated objects. Pain in the throat and pharynx area,

difficulty swallowing and shortness of breath can be the first symptoms of

an infection. The toxin of the bacteria affects the whole body, damaging

above all the heart, the kidneys, the supraranal glands, motor nerves

and the liver. Independent of the effect of the toxin, skin infections and

endocarditis have also been observed.

20.61 Corynebacterium xerosis This pathogen belongs to the family of coryneform bacteria, and can result

in skin infections, pneumonia and inflammation of the pharyngeal mucosa.

20.62 Cytophaga rubra Bacteria which live in the soil. Infection is generally via direct contact or

via contact with contaminated objects.

20.63 Erysipelotrix rhusiopathiae V Causes an illness known in animals as swine erysipelas and in people as

erysipeloid. Turkeys and pigs are most frequently affected. The pathogen

is in the earth, in water and waste water, as well as in rotting animal

material. The pathogen can also survive in a dry state and in pickled, salted

or smoked meats. The infection is transmitted mainly via skin injuries, yet

is can also be transmitted orally. Endocarditis, arthritis and necrosis of the

skin can occur.

20.64 Eubacterium suis These pathogens cause cystitis in pigs, for instance.

20.65 Francisella tularensis V This pathogen causes the frequently deadly illness tularaemia in wild

rodents. The illness can be passed on to humans. An infection can be

caused by infectious rodents or indirectly via blood-sucking ectoparasites

such as mosquitoes, fleas and lice. Dirty water or breathing in the bacteria

can also cause an infection. Symptoms: fever, weakness, swollen lymph

nodes, conjunctivitis, lung abscesses, mediastinitis, meningitis, pericarditis

and / or osteomyelitis.

20.66 Gardnerella vaginalis In small quantities, these bacteria are present as part of the normal vaginal

flora. In increased numbers, they are the most detected pathogens in bacte-

rial vaginitis. The germ can ascend to the upper genital tract, resulting in

serious infections. Long-term inflammation can result in infertility.

74


20.67 Haemophilus influenzae This bacterium lives exclusively in the mucous membranes, especially in

those of the upper respiratory system (nose, throat, windpipe), where it

causes inflammatory illnesses (epiglottitis, bronchitis, pneumonia). These

germs are passed on as airborne infections: they only have a short survival

time outside of the mucous membranes.

20.68 Haemophilus parasuis V Pathogen of Glässer’s disease in pigs. Febrile polyserositides and poly

arthritides are the main characteristics of the illness. However, the patho-

gen is also observed on the mucous membranes of animals that are not

sick. It is an airborne infection.

20.69 Helicobacter pylori Infections caused by these bacteria are held responsible for many stoma-

ch ailments accompanied by increased secretion of gastric acid. Stomach

ulcers and ulcers of the duodenum can also arise as the consequence of an

infection with this bacterium. A long-term infection can have carcinogenic

consequences. The transmission path has not been definitely clarified. The

bacterium appears to spread by means of a faeces – oral path. This means,

the bacterium is excreted via stool, and ingested via water or dirty food.

The possibility of the bacterium being spread by bluebottles is currently

under discussion.

20.70 Lactobacillus acidophilus This bacteria is found in the flora of the mouth, the digestive tract and the

vagina / in men, in the extended area just in front of the urethral orifice.

Lactobacillus acidophilus is considered a probiotic bacterium.

20.71 Lawsonia intracellularis These bacteria are pathogenic for pigs, and cause the diarrhoea illness

porcine proliferative enteritis. The bacteria have also been detected in

horses, sheep and rodents.

20.72 Legionella Is found in samples of soil and stretches of water. As a source of infection

for humans, it is found in hot water pipes with insufficiently heated water

(< 70°C), air-conditioning systems and cooling towers. Infections with this

pathogen cause legionnaire’s disease, a form of pneumonia with fever,

diarrhoea, headache and disorientation. Pontiac fever, a further illness

caused by infection with the pathogen, is an acute illness with coughs and

colds.

20.73 Listeria monocytogenes V This pathogen is not restricted to certain host organisms, and is distribu-

ted throughout the environment, e.g. in the ground, in stretches of water,

and on plants. It is found on animals as well as on birds and fish. Between

about 1 and 10% of all people are probably also infected, and excrete the

pathogens via stool.

75


20.73 Listeria monocytogenes V In the event of an infection, monocytes in the blood are multiplied. The

illness is known as listeriosis in people and animals. The most frequent

infection path is by eating unclean food.

20.74 Malleomyces mallei V This is a pathogenic burkholderia type which can cause glanders in both

people and animals.

20.75 Mycobacteria phlei These pathogens can result in inflammation of the lungs and the eyes. They

are widely spread in plants, the ground and dust.

20.76 Mycobacteria tuberculosis This is a bacterium from the family of mycobacteria, the most important

pathogen for tubercolosis It is an airborne infection. The main entry point is

the lung. Animals always become infected by people infected with tubercu-

losis disease. In animals, it is usually only a quickly healing, local process. In

such cases, it is advisable to examine people looking after such animals for

tuberculosis. Small mammals such as dogs and cats, and perhaps parrots,

can become a dangerous, permanent source of infection following infec-

tion with tubercolosis disease.

20.77 Nocardiae V Occur all over in the grund and in damp biotopes. Infection is spread via the

respiratory tracts or via skin wounds.

20.78 Nocordia asteroids These pathogens are found in the ground and in damp biotopes. The follow-

ing illnesses are possible: nocardiosis (bronchopneumonia, lung abscess),

sepsis, brain abscesses, abscesses in the kidneys and musculature, cutane-

ous or subcutaneous abscesses, lymphocutaneous syndrome).

20.79 Pasteurellae V Infections with this pathogen are known as pasteurellosis. Such infections

frequently take on the form of sepsis, but also of an infection of the respi-

ratory tracts or the intestinal tract.

20.80 Pasteurella multocida V These pathogens can be transmitted via cat bites or scratches. Infections in

the area of the respiratory tracts and the intestinal tract are possible. The

illness is known as pasteurellosis and occurs in both mammals and birds.

20.81 Propionobacterium acnes Also known as propionic acid bacteria: they are known by the holes formed

in many types of cheese. They are normally found on the skin. If the natural

balance of the skin bacteria shifts and, for instance, the skin bacteria are

joined by staphylococcus aureus, these pathogens can have a very negative

effect on the infection, worsening it. Increased propion bacterium acnes

pathogens have been detected in endocarditis, ulcers of the cornea and

septic arthritis.

76


20.82 Pseudomonas aeruginosa This pathogen is a so-called hospital germ. It is a widespread ground and

water germ which occurs in damp milieus. It is also present in tap water,

hand basins, showers, toilets, dishwashers, dialysis equipment, medication

and disinfectants. It can poison food. Since it is very resistant, it sureves

in distilled water. Even the use of some disinfectants does not guarantee

sure protection against the pathogen. It can trigger pneumonia in the case

of cystic fibrosis, infections of the urinary tract, enterocolitis, meningitis,

otitis external or infections on burn wounds. This pathogen also plays a

major role in infectious diseases in the field of veterinary medicine.

21.05 Bacteria II, complete This contains all bacteria from the program group 21.

21.10 Enterobacteriaceae, complete Nitrobacteria or enterobacteriaceae (currently the only family in the

genus of enterobacteriales) are a large group of bacteria. According to the

phylogenetic system, they belong to the phylum (strain) of proteobacte-

ria, where they form their own family. The name enterobacteria is derived

from the Greek enteron (intestines) since many of them typically live in the

intestines. However, this family also includes many free-living species of

bacteria which do not live in the intestines.

21.11 Enterobacter aerogenes These bacteria occur in almost all habitats, including in the human

intestines. They are part of the normal intestinal flora. Several species of

this genus can be pathogens of illnesses. Infections of the urinary tract,

meningitis and infections of the respiratory tract are possible.

21.12 Erwinia amylovora Pathogen of the so-called fire blight. The pathogen is spread via contami-

nated plant material, packaging material, cutting tools and migratory birds.

At close range, the bacteria slime is spread by wind, rain, insects, small

mammals, birds and people. The bacteria penetrate the plant tissue during

the active growth phase of the plant via natural entry points. There are

the further, following infection paths: flower infection (the most frequent

variant), shoot infection, infection from re-activated infestation locations.

The dangerous infection time is during the spring and the summer, especial-

ly during the flowering season if the weather is hot and humid: the flowers

are then the main infection locations.

21.13 Erwinia carotavora Many species of erwinia break down plant left-overs: however, they are

also involved in the cause of plant diseases or are seen as pests of stored

food. Erwinia carotovora (new name: pectobacterium carotovorum) causes

blackleg in potatoes. Some species have been found on animals and people:

however, their role as pathogen has never been determined.

77


21.14 Escherichia coli Bacterium which occurs in the intestines of humans and animals. E. coli is

a part of the intestinal flora. Outside of the intestine, however, E. coli can

cause infections since it is then located in the wrong area of the organism.

Infections of the urinary tract, peritonitis or meningitits in new-borns

(infection during birth) can be secondary diseases of an infection.

21.15 Klebsiella pneumoniae Bacterium of the genus Klebsiella which can cause pneumonia, amongst

other things. Pneumobacillus occurs practically everywhere, especially in

the human intestinal flora. The bacteria can trigger illnesses in people with

a weakened immune system. Illnesses frequently caused by pneumobacillus

are: infections of the upper respiratory tract, pneumonia, hospital pneumo-

nia (spreading by air-conditioning), infections of the urinary tract, blood

poisoning, meningitis.

21.16 Proteus mirabilis These bacteria are pathogens which also occur frequently in healthy people

and do not necessarily cause illnesses. However, should the immune system

become weakened, they following illnesses caused by this bacterium can

occur: infections of the urinary tract, wound infections, pneumonia and

sepsis. In the event of infections of the urinary tract caused by proteus

mirabilis, the pH value of the urine can be increased, favouring bladder

stones.

21.17 Proteus vulgaris See above

21.18 Salmonellae These bacteria occur globally in warm-blooded and cold-blooded animals,

in people and in habitats outside of living beings. The bacteria cause

illnesses in humans and animals. Salmonellosis can be spread from animal

to human, or even vice versa. Infections caused by food are frequent. We

distinguish between enteritis and typhus / paratyphoid salmonella. The

most important illnesses caused by salmonella are: diarrhoea with vomiting

caused by salmonella enteritidis, salmonella typhimurium, etc., salmonello-

sis or salmonella enteritis, typhoid caused by salmonella typhi, paratyphus

caused by salmonella paratyphi. Typhoid and paratyphoid are systemic

illnesses (affect several organs) with the intestines part of the symptoms.

21.19 Salmonella enteritidis See above

21.20 Salmonella paratyphi See above

21.21 Salmonella typhi See above

78


21.22 Serratia marcescens Bacteria of this species occur in the ground, on plants and in water: it is

only occasionally that they are detected in the gastrointenstinal tract or the

upper respiratory tracts of healthy people. Serratia marcescens is, above all,

a pathogen of hospital infections. Those patients with weakened defences

can develop wound infections, infections of the kidneys and urinary tracts,

infections of the respiratory tract as well as sepsis, endocarditis, meningitis

and prosthesis infections. Particular sources of infection are contaminated

catheters and infusion solutions.

21.23 Shigella dysenteriae This bacterium is named after its discoverer, the Japanese microbiologist

Kiyoshi Shiga, as well as the main symptom of diarrhoea which occurs as

part of this infection (dysentery). The pathogens cause stomachache and

diarrhoea. Sources of infection: uncleaned food, drinking water polluted

with faeces. The form shigella dysenteriae also forms a neurotoxin.

21.24 Shigella flexneri Causes diarrhoea. These specific pathogens are also named in conjunction

with several cases of cot death.

21.25 Shigella sonnei These species, also known as Kruse-Sonne bacteria, are the most frequent

shigella in Central Europe in particular, and cause harmless summer

diarrhoea, particularly in children.

21.26 Yersiniae Yersinia enterocolitica and Yersinia pseudotuberculosis can cause infec-

tions in humans and animals. The pathogens are orally ingested, causing

enteritis.

21.27 Yersinia enterocolitica Following oral infection with this pathogen, an acute case of enteritis or

enterocolitis occurs. Diarrhoea (mainly in toddlers), pseudo-appendicitis,

colicky stomachache, fever, nausea, bloody stool and inflammation in the

throat area are possible. Raw or incompletely heated pork (mince meat and

raw sausage) are the main sources of infection for the infection of human

yersiniosis.

21.50 Mycoplasmae, complete Mycoplasmas are bacteria characteristic for the lack of a cell wall. They are

the smallest bacteria possible of multiplying outside of cells.

21.51 Mycoplasma As parasitic bacteria, mycoplasmas are the cause for numerous illnesses

in human, animals and plants. Amongst other things, the pathogens cause

chronic infections, tracheobronchitis, pharyngitis, meningitis and middle

ear infections.

21.52 Mycoplasma agalactiae V Pathogen of the contagious agalactia in small ruminants (sheep, goat).

It usually causes unnoticed inflammation of the udder with reduced milk

output, rarely inflammation of the joints or conjunctiva (conjunctivitis).

21.53 Mycoplasma capricolum Pathogen of contagious caprine pleuropneumonia in goats (CCPP).

79


21.54 Mycoplasma mycoides V Pathogen of the notifiable contagious bovine pleuropneumonia in cattle.

21.60 Spirochaetae, complete

21.61 Borellia The most frequently occurring type of borrelia in Germany and Europe

is borrelia burgdorferi. This pathogen causes so-called Lyme disease. The

borrelia are spread mainly via ticks; however, infection caused by mosqui-

toes cannot be ruled out.

21.62 Brachyspira V Some species are pathogenic. Alls species occur in the intestines of various

animals (e.g. of pigs) and people.

21.63 Leptospira canicola V Leptospira are divided into main hosts, i.e. species of animals to which the

relevant species of bacteria has adapted itself and which are the actual

pathogen reservoirs, and reservoir hosts which are only rarely infected by

the species of pathogen. Dogs are the main hosts of leptospira canicola.

Leptospira are secreted by the infected animals via urine. Infection occurs

by means of contact via the skin or mucous membranes. The principal

means of transmission is the ingestion of water contaminated with rat

urine (puddles). During the acute phase, the pathogen spreads throughout

the blood and then colonises the various organs, such as the liver, spleen,

kidney and lymph nodes. Symptoms: anorexia, vomiting, fever, difficu-

lt breathing, sometimes jaundice (icterus), bleeding (haemorrhages) and

tissue defects on the mucous membranes of the mouth, muscles tremors or

bloody stool as the result of a serious bout of gastroenteritis.

21.64 Leptospira grippotyphosa V Leptospira cause general infections. Erthrocytes are damaged: as a result,

anaemia, icterus and haemoglobinuria occur. The central nervous system,

blood vessels and other organs are damaged by endotoxins. Leptospira

grippotyphosa can cause leptospirosis in cattle, sheep, goats, pigs and dogs.

21.65 Leptospira icterohaemorrhagiae Pathogen of Weil disease, also known as icterohaemorrhagic fever. In Europe,

it mainly occurs in people who come into contact with infectious material,

such as rat urine: however, pigs and dogs are also pathogen reservoirs. The

infection path is via the ingestion of contaminated, dirty waste water or

soil, via the softened or not intact skin, or via the mucous membrane. It can

also get into the body via the respiratory tracts. Symptoms: sudden, high

fever, headache, pains in the arms and legs. During the course of the illness,

jaundice, meningitis, inflammation of the kidneys or heart can occur.

21.66 Leptospira interrogans With these pathogens, the dog is the reservoir host: the main host in the

brown rat.

21.67 Leptospira pomona V The main hosts are cattle and pigs.

21.68 Leptospirae (suis) V The main host of this pathogen is the pig.

80


21.69 Treponema pallidum Pathogen of syphilis (also known as lues, lues venerea, hard chancre and

the French pox). It is an infectious disease which belongs to the group of

sexually transmitted diseases. Symptoms: painless ulcers on the mucous

membranes, swelling of the lymph nodes. In some of the infected patients,

the course of the disease is chronic, characterised by multiple infestation

of the skin and organs. In the terminal stage, destruction of the central

nervous system can occur.

21.80 Intracellular bacteria (cell parasites),

complete

Bacteria colonise very different habitats: many species of bacteria live in

the cells of other living beings.

21.81 Anaplasma marginale Penetrates the red blood corpuscles of the host, and multiplies there.

The presence of the parasites in the red blood corpuscles stimulates the

organism of the animal to destroy the red blood cells. This wide-spread

destruction of the red blood corpuscles results in anameia, fever, weight

loss, shortness of breath. The pathogens are spread by ticks: however,

contaminated hypodermic needles, operating instruments, mosquitoes and

horseflies can also result in infection.

21.82 Chlamydiae These pathogens are cell parasites. Chlamydia cause mainly diseases

(chlamydiosis) of the mucous membranes of the eye area, the respiratory

tract area and genital area, sometimes with serious consequences such as

blindness or infertility. Infection with this pathogen is via direct contact,

contaminated objects or, for example, flies.

21.83 Chlamydiae (feline) V A form of the pathogen which occurs mainly in cats.

21.84 Chlamydia ovis V Pathogen of the enzootic abortion of the sheep.

21.85 Chlamydia psittaci V Pathogen of the parrot disease, psittacosis, ornithosis.

21.86 Chlamydia trachomatis This pathogen causes a sexually transmitted disease in the urinogenital

tract which remains undetected in two-thirds of women, since there are

no symptoms. In men, it occasionally causes inflammation of the urethra

with a clear discharge and which is otherwise free of symptoms. However,

left untreated, the infection can result in infertility. Various strains of

the pathogen can cause eye infections or acute conjunctivitis, so-called

swimming pool conjunctivitis, since it is also frequently transmitted via

swimming pool water, urethritis (inflammation of the mucous membrane

of the urethra) and cervititis (inflammation of the cervix). There are also

strains which can cause lymphogranuloma venereum.

81


21.87 Cowdia rumantium V This pathogen causes the illness “heartwater”. Domestic and wild ruminants

are affected. The pathogen is spread by ticks of the amblyomma genus.

Affected mammals are cattle, sheep, goats, antilopes and buffalo. The

name of the illness is derived from the symptoms of the arising illness.

Fluid accumulates in the heart / in the lung.

21.88 Rickettsiae Bacteria, parastical organisms which are found in many ticks, fleas, mites

and lice, which transmit them. Infections are known as rickettsias. This

includes fleckfieber, rickettsial pox, Brill’s disease, boutonneuse fever

(Mediterranean tick fever) and Rocky Mountain spotted fever.

21.90 Other bacteria

21.91 Laryngeal 1 bacteria Pathogenic bacteria from the area of the larynx.

21.92 Borellia toxin Neurotoxins (toxins) produced by borrelia.

21.93 Caries bacterium Bacteria always found in conjunction with caries infections in patients.

21.94 PIA Porcine intestinal adenomatosis V Thickening and plication of the mucous membrane of the intestines of pigs.

21.95 Pain-producing bacteria These are bacteria which haven’t been classified.

21.96 Tuberculinum burnetti A nosode from tubercular lung tissue or tubercular cavities.

82

VirusesProgram-no. Description

22.00 Viruses, complete This program contains all viruses from the program groups 22 and 23.

22.05 Viruses I, complete This contains all viruses from the program group 22.

22.10 Double-strain DNA viruses, complete

22.11 Adenovirus These pathogens can cause numerous illnesses. Particularly affected are

the respiratory tract (influenza), infections of the eyes (conjunctivities) and

of the intestinal tract (diarrhoea). The infection is airborne or is passed on

as a smear infection.

22.12 Cytomegalovirus (CMV) Multiplies following oral infection via saliva or other bodily fluids in the

salivary glands. From there, the viruses are transported via the blood to

organs such as the liver, spleen, lung, bone marrow and the kidneys. Perma-

nent infections of the affected organs can occur as primary infection or

even in a latent form many years after the initial infection.

22.13 Epstein-Barr virus (EBV) An infection, mainly via droplets, saliva, genital secretions or blood cells,

transplants, result in a life-long infection. Symptoms: fever, swelling of the

lmyph nodes, coating on the tonsils. Pathogen of Pfeiffer’s disease.

22.14 Hepatitis B virus Possible illnesses are inflammation of the liver (hepatitis), cirrhosis of the

liver, liver cell carcinoma. Infection with the pathogen is parenteral and

sexual, i.e. via the blood or other bodily fluids of an infected patient. The

entry points are usually tiny injuries on the skin or the mucous membrane.

22.15 Herpes simplex These pathogens are characterised by the fact that they can stay in the host

for life. Following initial infection, the virus genome stays in the body for

the rest of the host’s life. The immune status of the host has a great influ-

ence on the virus reactivation. Herpes simplex viruses are globally present:

man is their only natural host as reservoir. Since the herpes simplex virus

is acquired via contact with saliva and smear infections from infancy in

normal contact with the family, it is very widespread in the population. A

further source of infection is contact via the mucous membrane. Illnesses

caused by the herpes simplex viruses: gingivostomatitis (inflammation of

the mucous membrane in the mouth), herpes labiales, herpes encephalitis,

keratokonjunctivitis and many other illnesses.

22.16 Herpes simplex (feline) V This pathogen causes infections in cats.

22.17 Herpes zoster Causes two different symptoms: upon the initial infection varicella

(chickenpox), upon reactivation, herpes zoster (shingles). The virus is trans-

mitted via the air or via the contents of a small blister. Contact with a

person who is ill is the most frequent source of infection.

83


22.18 Human papilloma virus (HPV) Is an oncogenetic virus. This species of the viruses are seen in conjunction

with certain cancers (cervical carcinomas, angogenital carcinomas). The

viruses are transmitted mainly via sexual contact and skin injuries.

22.19 Papilloma virus Causes warts in the organism. The viruses are transmitted via direct contact.

22.40 Single-strain DNA viruses, complete

22.41 Panleucopenia virus V Panleucopenia is a frequently fatal, viral cat-scratch disease. It is also

known as cat-bite fever, felinosis, infectious enteritis of the cat, cat plague.

The pathogen enters the body via contact with infectious material (faeces,

nasal secretion, urine) through the mucous membrane of the nose and

mouth.

22.42 Parvoviruses (suis) V The parvovirosis is caused by porcine parvovirus (PPV). It occurs globally in

pigs. If the infection occurs in the first three weeks of gestation, either all

or most of the embryos die, and are resorbed.

22.43 Porcine circovirus V Porcine circovirus (PCV) type 2 is a virus which occurs in pigs.

22.60 Single-strain RNA viruses, positive-

strain RNA genome, complete

22.61 AE virus V Avian encephalomyelitis, contagious chick encephalomyelitis. A highly

contagious illness with nervous symptoms in chicks. Infected layers are

the reservoir for the transmission of the virus via the egg or shortly after

hatching. The viruses are also transmitted via faeces. Chicken, turkeys,

pheasants, also ducks, pigeons and guinea fowl can also fall victim to the

virus.

22.62 BVD virus V Bovine virus diarrhoea / mucosal disease (BVD/MD) is caused by the bovine

virus diarrhoea virus (BVDV). This is a very frequent viral illness complex

amongst cattle.

22.63 Calciviruses (feline) V Known for years, feline calicivirus (FCV) is a pathogen of diseases of the

upper respiratory tracts in cats. Transmission is mainly aerogen via secre-

tions from the nose and throat area containing viruses.

22.64 Chikungunya These pathogens are transmitted via mosquitoes. Symptoms: Fever and

pains in the joints.

22.65 Coronaviruses (feline) V Infectious chest and peritonitis in cats (FIP) is a feline disease caused by

coronaviruses. These viruses usually cause a harmless intestinal infection in

cats. However, in a small percentage of infected cats, FIP can arise.

84


22.65 Coronaviruses (feline) V If a cat lives with other cats or encounters other cats outdoors, it picks

up the virus via its mouth and nose by scratching in the litter tray or by

sniffing and licking (other cats or objects and clothing).

22.66 Coronaviruses (suis) V These pathogens occur in pigs, where they cause infections.

22.67 Coxsackie virus B-1 Infection with this pathogen is caused by unclean water and food: airborne

infection or smear infection are also possible. Illnesses: cold, viral meningi-

tis, myocarditis, hand, foot and mouth disease.

22.68 Coxsackie virus B-4 See above

22.69 EAV virus Equine arteritis viral infection, EAV, earlier, acute sepsis, equine influen-

za, equine distemper, pink eye or erysipelas. Infection is via aerosols from

the respiratory tract, via the urine of acutely infected animals or venerally

during mating.

22.70 Duck hepatitis virus V This pathogen comes from the family of hepatitis viruses and is found in

the presence of liver disease of fowl.

22.71 Enteroviruses Transmission of all species of virus which are part of the genus enterovirus

is mainly via the faecal-oral path: however, the infection path of some

pathogens is airborne. Also possible is the transmission of the virus via

the placenta. Polio, infections of the upper respiratory tracts, colds, inten-

stinal illnesses, febrile generalised exanthem, haemorrhagic conjunctivi-

tis, myocarditis, pericarditis, hepatitis, meningitis and encephalitis can be

caused by the pathogen.

22.72 FHV viruses (feline herpes virus) V FHV is one of the pathogens of the infections of the cat’s upper respi-

ratory tracts complex. The virus mainly causes symptoms in the form of

rhinotracheitis in kittens. Infected animals are virus carriers for the rest of

their lives, acting as a carrier and source of infection for susceptible cats.

Furthermore, there is a connection with the feline cytomegalovirus.

22.73 FSME The pathogens are transmitted via ticks. Symptoms of an infection: influ-

enza, meningitis, encephalitis, radiculitis, paralysis.

22.74 Hepatits A virus Infection with these viruses is via a faecal-oral path (ingestion of contami-

nated food). The course of the illness is usually acute: chronicity as in other

hepatitis infections does not occur.

22.75 Hepatitis C virus Is transmitted via blood. After an infection, there are hardly any direct

consequences: damage to the liver is chronic. Once detected, it is frequent-

ly not possible to trace the path of the infection. Cirrhosis of the liver and

liver carcinoma are possible.

85


22.76 CSF virus V Although it is related to other pathogens, the swine fever pathogen (classic

swine fever) cannot be transmitted to other species of animal or humans.

Infection of the pigs is via direct contact with sick animals or via unclean

vehicles and equipment, clothing or waste food.

22.77 FMD virus V Foot and mouth disease is a highly-contagious viral disease in cattle and

pigs. Deer, goats and sheep, as well as elephants, rats and hedgehogs

can also become infected. Horses are not susceptible to foot and mouth

disease. A person can occasionally become infected. The disease can be

transmitted via contact and smear infection during direct contact with

infected animals, with contaminated stables or animal transport vehicles.

Infection via the air is possible. People who have contact with infected

animals should have their clothing disinfected. Fodder additives which

contain infected animal products and animal products such as cheese and

meet can harbour the virus. Cows can be infected with foot and mouth

disease from infected bulls during insemination.

22.78 Norovirus This pathogen results in acute gastroenteriditis. Sudden vomiting and

diarrhoea are typical symptoms of an infection. The viruses are excepti-

onally infectioius and can be detected in stool weeks later. Infection via

contaminated objects, smear infection.

22.79 PRRS viruses (suis) V The pathogen results in infections of the respiratory tracts of pigs.

22.80 Rhinovirus This pathogen causes infections known colloquial as the sniffles or a cold.

Transmission is airborne (coughing or sneezing), yet contaminated hands

or objects can also result in an infection. Via the mucous membranes, the

viruses enter the organism and result in generalised infections.

22.81 SVD virus V This pathogen causes a disease similar to foot and mouth disease in pigs.

22.82 Tobacco mosaic virus The tobacco mosaic virus causes the economically significant mosaic

disease in tobacco. Many agricultural crops and ornamental plants can be

infected. The virus is very easily transmitted, such as via direct contact

between plants, via sap, via seeds in the case of some plants. Compared

to many other plant viruses, it is extremely heat-resistant. Due to these

properties, it is probably one of the most widespread viruses in the world.

86


22.83 Teschen virus V Pathogen of the Teschen disease (contagious paralysis of pigs, polioence-

phalomyelitis enzootic suum, poliomyelitis suum). The contagious paralysis

of the pig is poliomyelitis of pigs of all ages, and is characterised by a short,

acute stage and following, typical paralysis. The disease has similarities

with poliomyelitis in people.

22.84 VES virus V This pathogen is the pathogen of the vesicular rash in pigs. Clinically, it

cannot be distinguished from foot and mouth disease.

23.05 Viruses II, complete This contains all viruses from the program group 23.

23.10 Negative-strain RNA genome, unseg-

mented, complete

23.11 Borna virus Borna disease or contagious encephalitis and multiple sclerosis of the

spinal cord of solipeds, is transmitted via this virus. The brain and the

spinal cord of horses and sheep in particularly are affected Symptoms:

behavioural changes, movement disorders and impairment to sensitivity

and sensorium such as keeping away from the herd / flock, depression,

lowered head posture, in some cases increased hyperkinesia, in some cases

aggressiveness towards others, in some cases nervousness, reduced partici-

pation in the animal’s environment, spasms and salivation. In the terminal

stages, paralysis with rowing motions, febrile attacks. The natural infection

is probably transmitted via the mucous membrane of the upper respiratory

tracts, the throat or the olfactory mucosa. It is now presumed that people

can also become infected with it. Symptoms: depression, abnormal behavi-

our.

23.12 Equine influenza virus V Equine influenza, also known as horse flu or Hoppengarten cough, is an

acute, highly contagious illness of the upper and lower respiratory tracts of

the horse, caused by equine influenza virus type A. In addition to indirect

transmission, the pathogen is mainly transmitted by air by the animals

coughing. Characteristic symptoms such as intermittent febrile phases

(temperature of up to 41°C), watery-serous nasal discharge, a dry cough,

lack of appetite and apathy can occur. During the course of the illness,

laryngitis, bronchitis, bronchiolitis or even viral pneumonia can develop.

Some horses, especially performance horses, display muscle weakness, a

stiff gait and, frequently, myocarditis and myocardium insufficiency.

23.13 Highly pathogenic avian influenza

virus V

Fowl plague is a serious general illness, especially in chickens, turkeys and

quails, as well as in numerous free-living species of bird. Basically, the same

infection paths as with other influenza viruses is observed.

87


23.13 Highly pathogenic avian influenza

virus V

The viruses are airborne, and are breathed in via the air, or are spread via

particles of faeces on clothing and equipment. The acute form of fowl plague

is manifested in signs of general weakness, dull, tangled feathers, a high

fever, breathing through the open beak, oedema on the head, neck, comb,

wattles, legs and feet, blue colour of the skin and the mucous membranes,

watery-slimy and greenish diarrhoea, and neurological disorders (abnormal

head posture, mobility disorders). The chronic course of the illness results

in a reduction of laying capacity, the eggs are thin-walled or have no shell.

23.14 Measles virus The pathogen is transmitted by means of airborne infections directly from

person to person. Following infection, typical measles exathem occurs (red

spots on the skin), fever and general weakness. Pneumonia and meningitis

can result in severe cases.

23.15 Mumps virus This is the pathogen of an infectious disease which mainly affects the

salivary glands. Meningitis or testitis (orchitis) can occur as complications.

The pathogen is transmitted via direct contact or via an airborne disease.

23.16 Parainfluenza Global virus which mainly attacks the respiratory tracts, results in a cold in

adults, and can result in serious symptoms in infants and toddlers including

pneumonia (pseudo croup). The pathogens are transmitted via airborne

infection, smear infection and contaminated objects.

23.17 Porcine influenza virus V Swine flue was first observed in 1918, at the same time as the major flu

pandemic amongst humans. Swine flu now occurs globally. Symptoms: the

animals experience shortness of breath, painful coughing and a short-term

increase of temperature of up to 42°C. Due to the high fever, sows that

become ill during gestation can experience spontaneous abortions or give

birth to small, weak piglets. Infections are spread by permanent carriers.

23.30 Negative-strain RNA genome, seg-

mented, complete

23.31 H1N1 Also known as human influenza or the Spanish flu.

23.32 H5N1 Also known as bird flu.

23.33 Influenza virus A and B These viruses and the illnesses they cause exist globally. In humans, influ-

enza viruses multiply in the respiratory tract of an infected person. Accor-

ding to studies, human influenza viruses favour cilia-less epithelial cells. In

contrast, in birds, the flu virus mainly multiplies in the intestinal epithelial

cells. These genuses also include the pathogens of influenza or “real” flu.

88


23.33 Influenza virus A and B These pathogens are responsible for infectious diseases which are general-

ly known as “flu”. Various types of virus of this species have occurred

frequently in recent years. The infection paths are airborne infection and

direct contact with infected objects.

23.50 Double-strain RNA viruses, complete

23.51 Bluetongue viruses V The bluetongue virus (BTV) causes a blue tongue in ruminants.

23.52 FCo viruses V The cause of a FIP disease in cats is a mutation of an actually harmless

intestinal virus. It is called the feline corona virus, abbreviated to FcoV. This

intestinal virus is widespread. If it makes the animal sick at all, it results

in mild diarrhoea and short-term lack of appetite. The fatal variant of FIP

develops from a mutation of the virus. The pathogen is destroyed by the

organism, but causes the formation of anti-bodies. The anti-bodies join

with other proteins to make “immune complexes”. This causes inflamma-

tion of the blood vessels, and vessel fluid enters the abdominal and chest

cavities or in the pericardium. Local infections are also possible.

23.53 FeL viruses V

23.54 FI viruses V Feline immune deficiency virus, also known as cat AIDS. This virus is mainly

secreted in saliva and transmitted via bite wounds during fights. Since the

virus destroys the immune system, infected cats are also more prone to

“normal” illnesses. Particularly frequent signs for the presence of a cat AIDS

infection are inflammation of the gums, wounds which heal poorly, and

chronic problems with the bladder.

23.55 Retroviruses These viruses are omnipresent amongst vertebrates. They infect mammals,

birds, amphibians, reptiles and fish: however, they are mainly very restricted

very specifically to their host. HIV and HTLV-1 are known to cause illnesses

in people.

23.56 Rotaviruses An infection with these viruses results in gastroenteritis, also known as

travellers’ diarrhoea. Rotaviruses are mainly transmitted via smear infec-

tions (faecal-oral) or via contaminated water (water contaminated with

rotaviruses) and food. Although the viruses do not multiply in the respira-

tory tract, during the acute phase they can also be secreted via secretions

from the respiratory tracts which makes it possible for them to be trans-

mitted by air. The virus is very easily transmitted: only 10 particles of the

virus suffice to infect a child. Infection is practically only possible from

person to person.

89


23.57 Rotaviruses (suis) V Worldwide, rotaviruses cause more than 70% of serious cases of diarrhoea

amongst people and animals and, hence, are the most frequent cause of

intestinal infections. This pathogen is found particularly in pigs.

23.70 Wart frequencies, complete Warts are predominantly caused by so-called papillom viruses, of which

there are over 100 different types. One exception is seborrhoeic warts,

whose cause remains unclarified. After an incubation time of a few days

through to several months, they develop on the surface of the skin as

slightly raised growths. Warts can occur practically anywhere on the body:

however, they are mainly found on the hands and the feet. Depending on

which part of the body warts occur and their appearance, they are divided

into the following:

23.71 Seborrhoeic warts It is currently unclear how they arise. Found all over the body.

23.72 Molluscums contagiosum Also known as “swimming pool warts”. These are not actually warts, although

they resemble them. They are little lumps the size of a pin head to that of

a pea, with smooth, frequently shiny surface. They usually have a dent in

the middle, and occur all over the body, particularly on the arms, hands,

fingers and the upper body. In contrast to other warts of the molluscum

contagiosum virus (MCV) from the family of poxvirida, a double-stranded

DNA virus (dsDNA), they are caused by smear infection or contact infection.

23.73 Condolymas These occur on the genitals and in the anal region, and are transmitted

during sexual intercourse.

23.74 Flat warts Also known as “flat warts”. Flat, round or multi-cornered growths, usually

soft, skin coloured to yellowish-grey or even brown with a diameter of

between 1 and 5mm. Its surface is usually dull and finely spotted. They can

occur anywhere on the body: however, they are mostly seen on the face or

the wrists, the backs of the hands and fingers, or on the outer parts of the

lower arms. Infection is via smear infection.

23.75 Verrucas An unpleasant form of the wart is the sole or pinhead wart. Its aculeiform

character can cause extreme pain when walking.

23.76 Juvenile warts A further form of warts is juvenile warts, whose form is flat. It is usually

children that are affected by this type of wart.

23.77 Flat warts Filiform growths, especially on the face. They are transmitted via smear

infection.

23.78 Common warts Also known under the names common wart and simple warts. These occur

particularly frequently on the hands, the fingers, the edges of nails and on

the soles of the feet.

90


23.79 Warts N.N. warts recurrent Recurrent warts. These are warts whose pathogens have not been clearly

classified.

23.80 Other viruses, complete

23.81 Viruses N.N. These pathogens have not been clearly classified.

91

ParasitesProgram-no. Description

24.00 Parasites, complete This program contains all parasites from the program groups 24 and 25.

24.05 Parasites I, complete This contains all parasites from the program group 24.

24.10 Hookworms, complete

24.11 Ancylostoma brasiliense Hookworm, which occurs mainly in cats and dogs. As a parasite, it colonises

the intestines. Can also infect people via larvae which bore their way into

the skin: perorale infection is also possible. In animals, the infection can be

transmitted via the mother’s milk (lactogen). Symptoms: anaemia, weight

loss, diarrhoea, pneumonia, changes to the skin.

24.12 Ancylostoma caninum

24.13 Gyrodactylus A genus of flatworm from the class of hookworms.

24.20 Eelworms/intestinal roundworms/

pinworms compl.

24.21 Ascaris megalocephala Ascaris worms belong to the threadworms. Infections in people and animals

are via the consumption of eggs from the environment. Symptoms: cough,

fever, asthmatic attacks, intestinal and gall-bladder ailments are possible.

24.22 Dirofilaria immitis (heart worm) A threadworm which is the pathogen of the heartworm disease in dogs. The

infectious third-stage larva is transmitted by mosquitoes. The heartworm

develops from the larva. Symptoms: conditional problems, heart problems.

24.23 Enterobius vermicularis This parasitical threadworm is the most frequent intestinal helminth, occurs

globally. Both animals and people can become infected.

24.24 Haemonchus contortus A threadworm which mainly infests small ruminants. It is ingested orally

by the animals and, consequently, parasitical gastritis occurs. Symptoms:

intestinal problems, diarrhoea, anaemia.

24.25 Loa loa A threadworm also known as eye worm and Loa. The parasite is the

pathogen of loaiasis (Calabar swellings). During its migration through the

organism, it also appears in the eye. It is transmitted percutaneously by

horseflies of the genus chrysops.

24.26 Macracanthorhynchus Acanthocephala. The parasite lives in the intestines. Infections can occur

via ingesting infested larvae or larvae from the soil. Symptoms: diarrhoea,

intestinal bleeding.

24.27 Onchocerca volvulus (tumor) Threadworm and pathogen of river blindness.

24.28 Enterobius worms Also maggot, threadworm or seat worm. The parasitical threadworm is the

most frequent intestinal helminth, occurs globally. It affects both people

and animals. The infected eggs of the pathogen are ingested orally or

inhaled. Symptoms: extreme itchiness in the anal region.

92


24.29 Passalurus ambiguus Rabbit worm. This is a species of roundworm which mainly colonises the

intestines of rabbits. Symptoms: anaemia, intestinal problems, weight loss.

24.30 Stephanurus dentalus Also known as kidney worm. Belongs to the family of threadworms.

24.31 Strongyloides (filariform) These pathogens belong to the strongyloid threadworms. Pathogen of stron-

gyloidiasis. The infection is transmitted percutaneously via larvae, directly

in the host. Symptoms: itchy skin, inflammation of the skin, breathing diffi-

culties, vomiting and bloody diarrhoea. 24.32 Trichinella spiralis (muscle):

parasitical threadworms. Infection is oral, e.g. by consuming minced pork

or uncooked pork. The illness is known as trichinosis. Symptoms: stomach-

ache, nausea, vomiting and diarrhoea.

24.33 Trichuris sp. Whipworm. The pathogen belongs to threadworms, the illness is known as

trichuriasis. This is a gastro-intestinal complaint. Infection occurs follow-

ing the oral ingestion of eggs containing larvae. Symptoms: vomiting,

diarrhoea, anaemia.

24.40 Haarwürmer gesamt

24.41 Capillaria hepatica (Leber) A hairworm which lives in the liver of mammals. Secreted eggs of rodents

are a potential source of infection. Symptoms: epigastric complaints,

enlargement of the liver.

24.50 Saugwürmer/Egel gesamt

24.51 Clonorchis sinensis Chinese fluke. Belongs to the trematoda. Definitive hosts are fish-

eating mammals (cats) and man. Symptoms: epigastric complaints, liver

complaints.

24.52 Cryptocotyle Trematode. Infection via the consumption of raw fish. Symptoms: diarrhoea,

vomiting, gastro-intestinal problems.

24.53 Echinostoma revolutum A flatworm or leech which lives as an intestinal parasite in birds.

24.54 Eurytrema pancreaticum Trematode or leech. Mainly found in the area of the pancreas.

24.55 Fasciola hepatica Liver fluke. Via consumption of watercress, plant stalks or blades of grass,

the larvae enter the organism of man or animal. Following consumption,

they migrate to the liver, where the problems start to develop. Symptoms:

Epigastric complaints, gastro-intestinal problems, liver insufficiency,

anaemia, increased body temperature.

24.56 Fasciolopsis buski Intestinal fluke. The infection occurs via the consumption of water plants

such as water chestnuts or water spinach. Manchurian wild rice, eaten raw,

is frequently infected with the pathogen. Symptoms: epigastric complaints,

digestive problems, fever.

93


24.57 Fischoedrius elongatus Also known under the name of Opisthorchis felineus.

24.58 Gastrothylax elongatus A worm that can be found in the stomachs of sheep and cattle.

24.59 Hasstile sig. tricolor Rabbit leech.

24.60 Metagonimus Yokogawai Consumption of bladder worms: the leech migrates to the intestines.

Symptoms: problems in the digestive tract, diarrhoea, anaemia.

24.61 Paragonimus Westermani Lungworm. A tremadote which, as a parasite, infects people and mammals.

It is the pathogen of paragonimiasis. An oral infection is caused by raw

shellfish. Most frequently, the leech becomes encapsulated in the lung.

Symptoms: fever, coughing, epigastric complaints. If it migrates to the

brain, epilepsy is possible.

24.62 Prosthogonimus macro. This pathogen belongs to the trematoda. Oral ingestion is via the pond

snail. It is mainly chickens which are affected. Symptoms: inflammation of

the cloacae and of the Fallopian tubes.

24.63 Schistosoma haematica Bilharzia worm. Pathogen of the schistosomiasis. Infection is via contami-

nated water or snails. Depending on the species, it is mainly the intestine

or the bladder of the organism which is affected. Symptoms: fever, cough,

headache, enlargement of the liver or the spleen.

24.64 Schistosoma masoni See above

24.65 Urocleidus A trematode which attaches itself to the gills of the white perch.

24.80 Tapeworms, complete

24.81 Echinococcus granulosus Three-membered dog tapeworm. Infections in humans are via the peroral

ingestion of the eggs. In the liver and lung, large blisters filled with fluid

are formed from the eggs. Carcinoma-like metastasises can frequently be

found in the liver.

24.82 Echinococcus multicolaris Fox tapeworm, see above

24.83 Taenia pisiformis A tapeworm which mainly infects dogs, foxes and cats.

24.84 Taenia saginata Beef tapeworm. Also occurs in the human organism. Cattle function as an

intermediate host.

24.85 Taenia solium Pork tapeworm also occurs in the human organism. The pig functions as an

intermediate host.

25.05 Parasites II, complete This contains all parasites from the program group 25.

25.10 Protozoa, complete Protozoa are protozoon with a nucleus and cell organelles. Many proto-

zoa have flagella which are used for movement purposes. They are very

adaptable in various living conditions. Amoeba, for instance, are able to

constantly change their shape.

94


25.11 Balantidia Parasites which colonise the mucous membrane of the intestine and

destroy it.

25.12 Balantidium coli A single-celled organism which occurs in the digestive tract of animals.

Rarely, they also infect people. Symptoms: diarrhoea, intestinal bleeding.

25.13 Besnoitia (lung) Single-celled organisms. Pathogen of besnoitiosis. This is an ailment of

the skin, hypodermis, mucous membrane and other tissues. Symptoms:

swelling of the lymph nodes, subcutaneous swelling, miscarriages, infer-

tility, diarrhoea.

25.14 Blepharisma Blepharisma japonicum is a single-celled organism and belongs to the

group of ciliates. It is found in stagnant stretches of water.

25.15 Chilomastix cysts (rat) A parasite which is found in both people and animals. It lives in the appen-

dix and in the colon. Symptoms: diarrhoea.

25.16 Chilomonas A genus of cryptophyts. These are single-celled, microscopically small algae

which occur in fresh and sea water. They move with the aid of two flagella

through the water and can be coloured red, blue or brown.

25.17 Coccidia (suis) V Coccidia are microscopically small, spore-forming, single-celled parasites

which infect the intestinal tract of animals. Coccidia are obligate intra-

cellular parasites: this means that they live and reproduce within one cell.

Coccidiosis is the name of the illness which is caused by the coccidia infec-

tion. Possible sources of infection are contaminated faeces or swallowing

infected tissue. Bloody diarrhoea is a classic symptom of the illness.

25.18 Coccidia (canis) V See above: particularly young animals with weakened immune systems are

infected by these parasites.

25.19 Dientamoeba fragilis A widespread parasite of the colon. Symptoms: if the host organism is

weakened, diarrhoea and epigastric complaints can occur.

25.20 Encephalitozoon cuniculi V Encephalitozoon cuniculi (earlier, also known as Nosema cuniculi) is an

obligate inter-cellular parasitical, single-celled organisms which live in the

kidney, brain and other organs. It belongs to the microsporoses: however,

the precise, systematic position of this parasite has not been definitively

clarified. It is the pathogen of encephalitozoonosis, a disease which occurs

mainly in rabbits, old world mice and canines, which can also be transmit-

ted to people suffering from immunodeficiency.

25.21 Endolimax nana Type of amoeba in the colon.

25.22 Endolimax tropica Type of amoeba in the colon.

25.23 Entamoeba coli trophozoi A species of amoeba found in the gastro-intestinal tract.

95


25.24 Entamoeba gingivalis Can be found in the gingival pockets of the teeth. Results in gum diseases.

Transmitted by kissing or by using the same cutlery.

25.25 Entamoeba histolytica tro. Cause of amoebic dysentery (a type of diarrhoea).

25.26 Giardia lamblia (troph.) This parasite is the pathogen of giardiasis in people: however, it also affects

mammals and birds. The infection spreads via contaminated surface water

or via contact with flies. Symptoms: swollen belly, pressure pain in the area

of the navel, diarrhoea, weight loss.

25.27 Iodamoeba bütschlii Amoeba that live in the colon.

25.28 Iodamoeba bütschlii tropica Amoeba that live in the colon.

25.29 Leishmania brasiliensis Pathogen of visceral leishmaniasis, cutaneous leishmaniasis, mucocutane-

ous leishmaniasis. The pathogens multiply in the blood of macrophages.

They are also known as intercellular parasites. The pathogens are transmit-

ted by psychodidae (phlebotomidae).

25.30 Leishmania donovani See above

25.31 Leishmania mexicana See above

25.32 Leishmania tropica See above

25.33 Leucocytozoon This pathogen is transmitted percutaneously via bites from blackflies. It is

mainly birds which are affected. In the leucocytes, the parasites migrate

through the entire organism.

25.34 Myxobolus cerebralis Pathogen of coenurosis in trout. The intermediate host is tubifex, a mud

worm which lives in the mud in the bottom of ponds.

25.35 Naegleria fowleri Pathogen of PAME (Primary Amoebic Meningoencephalitis), purulent

meningitis. Infection comes from bathing in polluted stretches of water.

It is via the nasal mucous membranes that the pathogens get into the

organism. Symptoms: fever, nausea, vomiting, stiff neck.

25.36 Plasmodium cynomolgi Belongs to the genus of sporozoa. Pathogens of this genus cause malarial

illnesses, amongst other things. The pathogen is transmitted via mosqui-

toes. Symptoms: fever, in phases, anaemia, seizures.

25.37 Plasmodium falciparum See above

25.38 Plasmodium vivax See above

25.39 Sarcocystis Sarcosporidia are parasites of the muscles and the intestines. The patho-

gens can be found in the musculature of cattle and pig. The animals are

infected via contaminated fodder. It is by consuming infected meat that

the pathogens also enter the organism of people. They colonise the small

intestine. Symptoms: vomiting, diarrhoea, fever.

96


25.40 Toxoplasma gondii Pathogen of toxoplasmosis. Infection is oral via cat faeces, infected meat

from sheep and pigs. It can cause symptoms of the central nervous system,

gait disturbances, diarrhoea and vomiting in cats. In people, the symptoms

of an infection are rather inconspicuous, similar to colds. Complications

to the infection only occur during pregnancy: the unborn child could be

harmed.

25.41 Trichomonas vaginalis Pathogen of trichomoniasis. The single-celled organism lives on mucous

membranes (particularly in the genital area) of people: the source of infec-

tion is direct contact between people.

25.42 Trypanosoma brucei Pathogen of the Chagas’ disease and sleeping sickness. Infection is percu-

taneous, via insects that sting and bite. Symptoms: fever, swelling of the

lymph nodes, pains in the arms and legs.

25.43 Trypanosoma cruzi (brain) See above

25.44 Trypanosoma equip. See above

25.45 Tryanosoma gambiense See above

25.46 Trypanosoma lewisi See above

25.47 Trypanosoma rhodesiens See above

25.60 Mites / ticks, complete Mites belong to the family of arachnida. There are around 50,000 known

species. As parasites, several of them cause problems for both people and

animals. For example the dust mites whose secretions can cause allergies.

Or the mange mites which can cause mange or scratches (skin illnesses) to

develop. A suborder of the mite is the so-called tick which is feared as the

carrier of CEE and borreliosis.

25.61 Acarus siro The flour mite is seen as a pest of stored food. The ingredients of foodstuffs

undergo negative changes when infested by mites.

25.62 Dermatophagoides (dust mite) By secreting these mites, allergic symptoms and, for instance asthma, can

occur.

25.63 Demodex canis V In the event of a heavy incidence or in the case of a weakened immune

system, this mite causes canine demodicosis, a parasitical skin disease in

dogs. It can be localised or affect the whole body. In older animals, demodi-

cosis only occurs in conjunction with disorders of the immune system. In

young animals, the cause of the disease has not yet been fully clarified.

Deomicosis usually starts with hair loss, and without itchiness. Later on, a

bacterial secondary infection can cause skin changes, through to suppura-

ting dermatitis (pyodermia).

25.64 Demodex folliculorum (hair follicle

mite)

In dogs with a weakened immune system, a typical skin disease occurs (see

above). Generally, it is harmless in people.

97


25.65 Neotrombicula autumnalis (harvest

mite) V

This parasite belongs to the class of arachnids. Their larvae live as parasites,

they mainly infest mice, but also dogs, domestic cats, people and other

mammals. The grass mite is also known as the autumn grass mite, harvest

mite, hay mite, autumn louse and grass louse. The mites’ larvae cause

trombiculiasis. Itchiness, reddening of the skin and itchy wheals (similar to

mosquito bites, but in larger numbers) occur.

25.66 Notoedres cati V A species of mite which, as a parasite, colonises the skin on the heads of

cats, causing so-called head mange. The pathogens can also occasionally

be transmitted to people (pseudo-mange) or ear mange such as in hedge-

hogs.

25.67 Ornithonyssus (bird mite) This ectoparasite is mainly found in birds: however, people and birds can

also be infected. Bacteria, viruses and blood parasites are transmitted by

the mites. Symptoms: severe itchiness.

25.68 Sarcoptes scabei (scabies) This pathogen belongs to the genus of mites. It lives as a parasite on the

skin of mammals, where it makes bore holes on the epidermis. The so-called

sarcoptic mange in mammals is known as the “mange” when a person is

infected with it. Symptoms: itchiness, formation of scabs on the skin.

25.80 Other parasites, complete

25.81 Echinoporyphium recurvatum A leech suspected of parasitizing the pancreas.

25.82 Hypodereum conoideum Parasitical worms.

25.83 Stigeoclonium A green alga.

25.84 Troglodytella abrasseri


25.86 Pneumocystis carinii According to a generally accepted definition, an ascomycota (see 27.81-

27.82). Due to the frequency spectrum, we classify it as a parasite from an

energetic point of view.

98

FungiProgram-no. Description

26.00 Fungi, complete This program contains all fungi from the program groups 26 and 27.

26.05 Fungi I, complete This contains all fungi from the program group 26.

26.10 Mould fungi, complete

26.11–26.25 Moulds occur almost everywhere. As a rule, the spores can be found in

the air. If mould spores occur in large quantities, they can cause allergies

in certain cases. Moulds and their spores can result in serious illnesses in

people and animals with a weakened immune system.

26.40 Mould fungus toxines, complete

26.41–26.45 Mould fungus toxines (myco-

toxins)

Under certain conditions, such as the optimum temperature, appropriate

humidity, adequate food supply and in appropriate development phases

of fungi, mycotoxins are formed. Emitted into indoor air, they can result

in unspecific health problems, amongst other things. Headache, pains in

the arms and legs, irritation or inflammation of the mucous membrane,

increased susceptibility to infections are possible. If these mycotoxins are

consumed via food, food poisoning can occur.

27.05 Fungi II, complete This contains all fungi from the program group 27.

27.10 Yeast fungus, complete

27.11–27.31 Yeast fungus Like many other micro-organisms, yeast fungi are part of a healthy body

flora. However, should yeast fungi suddenly multiply uninhibited, they pre-

sent a danger for the healthy organism: infections result. Such an impe-

diment to the balance can, for example, be caused by taking anti-biotics

or by a chronic disease such as diabetes mellitus. In the case of a yeast

fungus infection (candidiasis), large numbers of fungi infect the mucous

membranes of the organism, mainly in warm and humid places. Yeast fungi

are transmitted by direct contact (sexual intercourse) or by contact with

contaminated objects (towels).

27.50 Filamentous fungi / dermatophytes,dimorphic fungi, complete

27.50 Filamentous fungi / dermatophytes,

dimorphic fungi

Dermatophytes are hyphomycetes which cause a specific fungal infection

of the skin (dermatophytosis). The fungi nest in the top layer of the skin,

and feed from the keratin from the dead skin cells. Several fungi are also

able to remove keratin from the skin themselves. The organism reacts by

means of inflammation of the skin. Changes to the skin (perfectly circular),

trichoclasia or hair loss are visible signs of an infection. Dermatophytes

can be transmitted via contact between people or between animal and a

person. Contact with contaminated objects (shoes in the case of athlete’s

foot) can also result in an infection.

99

FungiProgram-no. Description

27.70 Mycetozoa, complete

27.71–27.73 Mycetozoa Slime moulds can be found in different places: in piles of leaves or

brushwood, compost heaps, grass, dead plant components and moss.

Various species only occur during the spring thaw in the mountains. They

are neither animal nor plant, however they are not a true fungus either.

27.80 Ascomycota, complete

27.81–27.82 Ascomycota Ascomycota are responsible for numerous infections in humans and

animals. However, they are also used in the field of medicine and in the

production of food.

Health problems occur when they are directly ingested: the symptoms range

from reactions in the gastro-intestinal tract through to hallucinations.

27.90 Other fungi, complete

27.91 Tryptophanum

100

11 Appendix IV: Use of bioresonance for detoxification purposes

Living nature functions exclusively by means of intelligent

regulation, and never by means of the control of its systems.

In the sense of correctly understood natural healing which,

according to my observations and my experiences, also

includes bioresonance according to Paul Schmidt, control-

ling therapy is not necessary and hence does not need to be

carried out. Naturally, this does not apply to the treatment

of acute illnesses and emergencies! Therapy should always

be designed such that the organism is always free to react,

or that reactivity is further improved.

In developing the RAH detoxification programs, the real

objective was to rectify a dysfunction of the inner regula-

tion. This type of therapy and this approach to illness are

what make the RAH detoxification programs so unique.

From the point of view of natural healing, the organ’s

function and the organ itself bear the following relation-

ships. Nature doesn’t recognise organs – it only recognises

functions. The function always uses the organ as its tool.

This was the intellectual starting point for all RAH detoxi-

fication programs. In all RAH detoxification programs, care

was taken to ensure that, first of all, the relevant organ

is stimulated and/or its reactivity is improved. The diver-

sion, removal and detoxification via the relevant organ or

system of organs or the entire regulation cycle, such as is

the case with program RAH 31.53, is the second pillar of

this basic concept. The third pillar is the protection of the

organ against the ensuing toxins to be secreted.

Earlier, therapists saw the cause of many illnesses in the

accumulation of unwanted substances in the body. These

toxins can be of an exogenous type, i.e. coming from

the outside, or of an endogenous type, i.e. formed by the

organism itself. Initially, these toxins poison the blood, then

the so-called matrix, or basic substance, of the body. These

toxins prevent the harmonious functions of the organism.

This results in various defensive reactions, since the body

endeavours to liberate itself from toxic substances. The

multifarious reactions, such as a cold, bronchitis, eczema,

ulcers, diarrhoea, perspiration, etc. therefore arise, as does

the body’s endeavour to detoxify itself.

Hippocrates of Kos ca. 460 BC on the Greek Aegean island

of Kos, † ca. 370 BC in Larissa, Thessaly, is considered the

most famous physician from Antiquity. He described this in

the following words:

Illnesses do not come to us out the blue. They are developed

from daily sins against Nature. When enough sins have

accumulated, illnesses will suddenly appear.

Philippus Theophrastus Aureolus Bombast von Hohen-

heim, baptised Theophrastus Bombast von Hohenheim,

known as Paracelsus, * probably on 10 November 1493 in

Egg, close to Einsiedeln; † 24 September 1541 in Salzburg,

was a physician, alchemist, astrologist, mystic, lay theolo-

gian and philosopher. He continues this line of thought. He

further points out that the powers of good health lie within

man himself, describing it thus:

Nature is the first physician: man the second.

Thomas Sydenham, 10 September 1624 in Wynford Eagle

close to Dorchester, Dorset; † 29 December 1689 in London,

was an English physician. He is also known as the “English

Hippocrates”. Amongst other works, Sydenham presented a

series of classic descriptions of infectious diseases. It is in

1686 that he first describes chorea minor, named after him.

He first described the differences between rheumatism and

gout in his works in 1683. Sydenham says:

The illness is nothing apart from the endeavours of nature

which, in order to keep the ill person, does its very best to free

the patient from the pathogenic substances.

Yoshimasu Tōdō * 1702 in Yamaguchi, province of

Aki (corresponds approximately to the present-day:

101

Hashimoto-chō, Naka-ku, Hiroshima); † 1773 in Heian-kyō,

Kyōto) was the first-born son of the surgeon and

obstetrician Hatakeyama Shigemune. His first name was

Tamenori, yet he was called Shūsuke. Later, he changed his

surname to that of his birthplace Hiroshima in Yoshimasu,

and his first name to the place of residence of his most

significant supporter and friend, Yamawaki Toyo in Tōdō.

Due to his legendary successes, Yoshimasu became one of

the most famous physicians and medical scientists in Japan,

and is considered the leading authority of kampo medicine

of his time. His Yoshima formula for weight reduction is

well-know, for instance.

In line with the then generally accepted expert medical

opinion that illnesses arise as the result of a dysfunctional

energy cycle, he considered toxins from the outside as the

trigger of a “systemic imbalance” which can be rebalanced

by using medicine as an antidote.

Hahnemann discovers homoeopathy. Initial suggestion of

medicine of bio-information. The materials of an active

agent cannot be proven in highly diluted homoeopathic

remedies.

Hans-Heinrich Reckeweg * 9 May 1905 in Herford / Prussi-

an province of Westphalia, † 13 June 1985 in Baden-Baden,

founder of homotoxology, a modification and further

development of homoeopathy. Along with the Austrian

physician Pischinger, he is one of the first to point out the

importance of the matrix or of the pluripotent tissue known

as the extracellular space or Pischinger space. Reckeweg

continues this idea through to illness and health, postula-

ting:

That illness is the expression of biologically suitable defence

processes against exogenous and endogenous toxins. This

is what he calls homotoxins. As a result, good health is the

state of being free of homotoxins or of damage caused by

homotoxins.

In 1976, Paul Schmidt and his discoveries take the first step

towards treating people by applying exclusively vibrational

medicine or information or bio-information.

The treatment methods, including the RAH detoxification

programs, which resulted from these ways of looking at

things, hence comprise helping the body to free itself from

the harmful substances. As an integral concept, the supply

of toxins – including those in food – is to be regulated in

order to make the source of the harmful substances dry up.

This clear and simple logic – reflected in the main therapy

points of prevention by means of removal, detoxification,

regulation and immune modulation, by means of the treat-

ment of simple acute illnesses by improving symptoms and

boosting the immune system and by means of the treatment

of chronic illnesses by immune modulation, the strengthe-

ning of organs and metabolism activation – has relieved

and healed sick people for centuries. These main aspects of

therapy maintain their value.

The specific compilation of the RAH detoxification programs

does justice to these connections. This further increases the

value of all treatments by using bioresonance therapy.

102

Mesenchyme – cell – milieu or physical milieu

There is an ideal compilation of the matrix, of the inner

milieu, which guarantees the good functioning of the

organism. Any too-large quantitative or qualitative devia-

tion from the constitution of the physical milieu results in

illness.

If, as the result of excess food or the ingestion of alcohol or

medication, the physical milieu is exceptionally overloaded

with toxins, this does not have any dramatic consequences,

since the body is able to detoxify itself in order to, in turn,

re-establish the ideal composition of the physical milieu.

If, however, these deviations become commonplace, even

provoked daily, then the body’s ability to re-establish a

healthy balance is quickly overtaxed. And so waste products

collect in the blood, for instance, and are deposited in the

vascular walls. The diameter of the vessels is reduced. As

a result, the blood is concentrated – it thickens. Blood

circulation becomes increasingly poorer, and the exchange

between blood and the matrix and, hence, the cell itself,

slows down and worsens. Waste products regularly elimina-

ted from the cells collect in the tissues (excess substances)

instead of quickly leaving the organism. The organs can no

longer perform their work correctly (assimilation and dissi-

milation). The liver and the kidneys are overworked in their

cleaning of the bodily fluids. All processes are disturbed.

This is true at cell level, at organ level and in the matrix.

These disturbances are manifested in the form of enzyme

dysfunctions and other various biochemical reactions, as

well as in the immune system such as the white blood

corpuscle.

The diagnosis and the therapy reflect the correctness of the

above in that, for all illnesses, we can observe the 3 phases

of secretion, deposition and degeneration.

During the secretion phase, the organism is still in the

position to help itself by means of secretion processes, such

as by means of a cold or diarrhoea.

During the phase of deposition, due to the aforementioned

reasons, toxins have been deposited in the matrix since

secretion is no longer possible. Either because the secretion

organs are overworked, because their function has been

weakened, or because the influx of toxins is too great.

During phase 3, degeneration and all its feared conse-

quences occur. The vicariation effect describes the illness.

Progressive vicariation: illnesses move from outside to

inside, from less vital to vital organs. The prognosis is not

good.

Regressive vicariation: illnesses move from inside to outside,

from vital to less vital organs. The prognosis is good.

104

The harmful substances and influences or effects on man

can be divided into:

• Physical influences,

• Chemical influences,

• Biological / physiological influences

• Psychological influences.

Physically harmful influences are, for instance, the climate,

air conditioning and ventilation systems, lighting, noise,

electrosmog, vibrations, ergonomics at work, colours in

your environment and, not least of all, geopathic stresses.

Chemically harmful influences are general pollution,

solvents, biocides, formaldehyde, cleaning agents, dust /

fine dust, ozone, carbon dioxide, VOC emissions (volatile

organic compound) and odours.

Biological influences are those to which man is physiolo-

gically subjected: these include fungi, bacteria, viruses and

parasitical stresses.

On the whole, we underestimate psychological influences.

The research results from the field of pyschoneuroim-

munology indicate the relationship between the psyche, the

nervous system and the immune system. Essentially, these

are being overworked or underworked, a lack of the exertion

of influence with regard to non-observance of individual

intentions. A lack of communication – including between

couples – harassment at work, private problems and, above

all, any type of illness, can literally poison a person.

The basic system of detoxification

Lymphatic system

The anatomical structures are lymph nodes, lymph vessels

and lymphatic fluid. The special functional aspect of the

traditional medical view is that of a disposal system yet also

of a supply system. The lymphatic system is the compensa-

tion system of the venous system. In the regulation cycle of

life, therefore, each venous insufficiency causes lymphatic

intoxication to start with. Hence, the RAH detoxification

programs focus especially on the lymphatic system.

Vegetative nerve system

All basic functions are regulated by the vegetative nerve

system. This also applies to all eliminating, detoxifying

processes. In the RAH detoxification programs, the main

focus is on ensuring a balanced, vegetative prevailing mood

in the organism. The vegetative nerve system represents

the functional unit of very different systems. These systems

ensure that we are alive, and that we stay alive. It is for

this reason that these systems react instinctively to stimu-

lation. A person enjoys good health when the organism

replies adequately to these stimuli. An allergy, for instance,

presents an inadequate stimulus response by intoxication.

The natural healing way of looking at things does not limit

the vegetative nerve system to the sympathic nervous

system, the parasympathic nervous system and intramural

system in its entirety as a neurovegetative system. Functio-

nally, it also belongs to the hormone system subordinate

to the pituitary gland. It is the endocrine vegetative nerve

system. This endocrine vegetative nerve system regulates

the aforementioned basic functions, i.e. breathing, the heart

action, digestion, metabolism, secretion, the water balance,

tissue tone and the state of stimulation. For the develop-

ment of the RAH detoxification programs, I took guidance

from the old, natural medical wisdom of “there is no illness

with a normal state of stimulation”.

105

The RAH detoxification programs In detail:

RAH 31.50

The basic program contains the central frequency of detoxi-

fication. It is a summary of all detoxification functions of

the body. Due to the complexity, the effectiveness is unspe-

cific. At the beginning of the treatment, it is considerab-

ly more gentle than the specific other RAH detoxification

programs. Very good for application on older people, very ill

people and at the start of treatment.

RAH 31.51

Detoxification of the blood system. The blood system is of

vital importance to all detoxification processes. The blood is

the central means of transport of material in the organism.

The contents of the program include the improvement

of the flowing properties, improved blood formation and

regeneration. It is good to apply this program in the case

of deficiencies, in the event of acute illnesses and during

convalescence.

RAH 31.52

Detoxification of the lymph system The lymphatic fluid

could be described as the sister of blood. Cinderella, in a

certain respect. It is via the lymphatic system that all waste

produced by the organism, from dead bacteria through

to waste metabolism products, is removed. The program

includes the protection of the system and improved

lymphatic drainage. It is the basic program of extracellular

detoxification. Improvement of the detoxification

performance at non-organic level.

Colloid system or the system of basic regulation

The basic regulation takes place in the basic substance. The

basic substance is the end point or, depending on the way

of looking at things, the starting point of all biorhythms,

all formation and degradation processes, of the endocrine

system, the central nervous system, the blood system and of

the lymphatic system. The basic regulation is the synthesis

of basic substance, collagen and fibroblasts. Fibroblasts are

the cells which occur in the connective tissue. They synthe-

sise the intercellular substance as well as the collagen and

the proteoglycan. These proteoglyans are the filters in the

matrix.

Elimination systems

For the sake of completeness, the individual elimination

systems need to be mentioned. In the RAH detoxification

programs, these systems are compiled in a functional-

ly practical manner. The systems are, individually, the ENT

mucous membranes, the mucous membranes, the saliva

glands in the mouth, the lungs, the stomach, the pancreas,

the liver, the bowels, the kidneys, the bladder, the uterus

in women, the urogential tract and the skin. This was then

followed by a practical compilation of the individual organs,

systems of organs and regulation cycles (removal systems)

in the RAH detoxification programs analogous to the embry-

onic blastodermic layers and gastrulation.

We have now been through the foundation of a cause-

orientated bioresonance therapy. By means of the detoxi-

fication programs of the RAH, we can positively stimulate

all detoxification and removal regulation cycles and all

detoxification organs. We then act in line with an old Asian

proverb which says:

If you want to chase the tiger (illness) from the house (body),

first of all you need to open all doors and windows (removal

systems and detoxification organs) before you nip him in the

tail (therapy). It usually then leaves of its own accord (good

health).

106

RAH 31.53

Detoxification of acidosis. Cases of acidosis arise from

clogging up of the matrix. It is difficult to diagnose acidosis

of the cell itself, especially if the matrix itself is not hypera-

cidic. All substances of the healthy matrix are in solution. In

the case of hyperacidity, the substances are in a type of gel

status. They aren’t available to the organism or, if they are,

only under difficult conditions. These tougher conditions

are accompanied by an increased expenditure of energy.

A symptom of hyperacidity is therefore chronic lethargy.

The Acidosis program covers both types of hyperacidity.

The program should only be used as part of the therapy

once the detoxification organs are working well. The use of

RAYOBASE can also be considered at this point. Depending

on the type of hyperacidity, it can be easily combined with

RAH programs 31.54 and 31.55.

RAH 31.54

Detoxification, extracellular is a basic detoxification

program, and affects the entire matrix. It is always a good

combination with all other detoxification programs.

RAH 31.55

Detoxification, intracellular is the basic program for all

chronic illnesses, from rheumatism through to cancer. In

this case, intoxification has already infiltrated the cell. This

program is rarely used at the beginning of bioresonance

therapy. A good combination is to use it in conjunction with

RAH 31.52 Detoxification of the lymphatic system.

RAH 31.56

Detoxification of mucous membranes. The mucous

membranes are delimitating organs. The program strengthens

both the function and the mucous membrane itself. An

extensive area of application is allergies which manifest

themselves on the skin and the mucous membranes.

RAH 31.57

Detoxification of the lung. The lung is responsible for the

elimination of gases, in particular the acidic carbon dioxide

and the ammonia from purine metabolism. This program

is the little brother of program RAH 31.53 Detoxification

of acidosis. The program actively protects the lung when,

during detoxification, increased acid reduction occurs. It

can also be used as a component of asthma treatment.

RAH 31.58

Detoxification of the stomach. In addition to the skin, the

stomach is the all-round genius when it comes to secre-

tion. It secretes all types of acid. However, it also ensures

that enough buffer substances are available. The program

aids both. Above all, it strengthens the stomach as an organ

itself. The program can be used in the event of both hyper-

acidity and hypoacidity of the stomach.

RAH 31.59

Detoxification of the pancreas. Functionally, the pancreas is

upstream of our most important detoxification organ, the

liver The main objective of this program is the protection of

the pancreas and the good functioning of the organ. Many

toxins are also secreted via the very alkaline pancreas. This

program is helpful for all illnesses of the pancreas and the

liver. In order to relieve the liver, the program can also be

used very harmoniously with program RAH 31.60 Detoxifi-

cation of the liver.

RAH 31.60

Detoxification of the liver. Like all organ-related detoxifi-

cation programs, the main focus is on the protection of the

organ in the event of multiple detoxification. The liver makes

it possible to excrete all substances. The detoxification of

the liver program is the central detoxification program. It is

very suitable for the start of all types of therapy.

RAH 31.61

Detoxification of the intestines. The intestines, especially

the colon, have practically limitless power when it comes to

detoxification. Acids, alkalis, water and minerals can all be

excreted. When creating program RAH 31.61 Detoxification

of the intestines, great attention was paid to the stimula-

tion of the organ. Use throughout the duration of the thera-

py is recommended, and it can be used again and again. It

is very effective in the case of diarrhoea, intestinal mycosis

and colitis.

RAH 31.62

Detoxification of the kidney. The kidney is very thorough

when it comes to detoxification, yet it is also extremely

sensitive. The kidney secretes highly toxic substances such

as uric acid. Any stimulation of the kidney function and

kidney activity can damage the kidney by serious inflam-

107

mation. That is why the main objective of this program is to

protect the organ that is the kidney. It can be used for all

chronic ailments.

RAH 31.63

Detoxification of the bladder. Pimples on the skin and itchy

skin means that detoxification via the bladder has not be

carried out correctly. In this case program 31.63 Detoxifica-

tion of the bladder is to be used, especially for pruritus, acne

vulgaris, psoriasis and inflammation of the bladder.

RAH 31.64

Detoxification female / female-specific. Menstruation gives

women a great detoxification potential. This function is

limited at the start of the menopause. The main focus of

this program is to enable the transition to the menopause

without the need for hormone supplements or medication.

RAH 31.65

Detoxification of the skin. This program is the second side

of the coin of program 31.63 Detoxification of the bladder.

This program improves the skin function as such. The main

focus is on the organ of the skin, since the skin frequently

only displays problems of other organs. It is of particular use

in the case of allergies.

RAH 31.66

Detoxification of endotoxins. This program has been

especially developed to optimise the detoxification cascades

of the organism following acute illnesses, infections and

operations. In a multiplied, improved condition, endotoxins

are sent for secretion. It is particularly applicable following

each course of antibiosis and for treating allergies.

RAH 31.67

Detoxification of exotoxins. Via the detoxification cascades,

the exotoxins in the organism are released and secreted.

This is particularly true following acute illnesses and infec-

tions. It is recommended right after the removal of amalgam

from the teeth, and also as a non-specific detoxification

program. It is suitable for use at the start of a treatment,

since the reactivity of the patient manifests itself.

Sample therapy for selected illnesses

Basically, all RAH detoxification programs can be used at

any phase of an illness (humoral phase, matrix phase and

cellular phase). The program compilation predestines many

RAH detoxification programs for the appropriate phases.

This can be the clue behind successful treatment. It isn’t a

statement about the value of a RAH detoxification program.

The value / effectiveness manifests itself on the patient

during the treatment. All RAH detoxification programs can

be easily combined with each other.

Illnesses progress from the humoral phase through the

matrix phase, right through to the cellular phase.

The following are recommended as detoxification programs

during the humoral phases:

RAH 31.50 Detoxification basic program

RAH 31.51 Detoxification blood system

RAH 31.52 Detoxification lymph system

RAH 31.54 Detoxification extra-cellular

RAH 31.56 Detoxification mucous membranes

RAH 31.67 Detoxification of exotoxins


during the matrix phases:

108

RAH 31.53 Detoxification acidosis

RAH 31.55 Detoxification intra-cellular

RAH 31.57 Detoxification lung

RAH 31.64 Detoxification female / female-specific


during the humoral phases:


Sample therapies and possible approaches, paying special

attention to the RAH detoxification programs:

acute, not chronic urinary tract infection

The illness is in the humoral phase stage. Hence, possible

RAH detoxification programs are:







Since the illness in the humoral phase has progressed to the

inflammation phase,

program RAH 31.52 Detoxification lymph system is particu-

larly appealing.

The specific program for the urinary tract infection is

program RAH 31.56 Detoxification mucous membranes.

2-3 detoxification programs usually suffice for one therapy

session.

The possible therapy settings for the RAH detoxification

programs for the treatment of an acute urinary tract infec-

tion are:

1. RAH 31.50 Detoxification basic program

or


or


or


and

2. RAH 31.52 Detoxification lymph system

and

3. RAH 31.56 Detoxification mucous membranes

A cause-oriented treatment of an acute urinary tract infec-

tion could take on the following sample program compila-

tion:

Organ strengthening

RAH 01.30 Pre-control,

RAH 02.17 Bladder meridian,

RAH 07.22 Zinc,

RAH 21.14 Colon bacillus

(In 80% of all cases, gram-negative bacilli from the intesti-

nal flora are associated with an acute urinary tract infection,

but also gram-positive cocci, mycoplasma, urea plasma,

yeasts, chlamydia and viruses).

Modulation of the immune system

RAH 35.10 Increased defences, basic program

Detoxification




Therapy damage caused by medication

RAH 30.00 Cell and tissue, physiology, total

RAH 30.40 Organelles, total

RAH 34.00 Immune system physiology, total


RAH 65.10 Female hormone balance basic regulation, or

RAH 65.20 Male hormone balance basic regulation


Removal of the relevant toxins by means of bioresonance

109

Recurrent virus infections



RAH 31.10 ATP production overall

RAH 22.05 Viruses I, total

RAH 23.05 Viruses II, total


RAH 36.00 Lymphatic system physiology, total


Precanceroses



RAH 30.41 Endoplasmic reticulum

RAH 30.42 Mitochondria

RAH 30.43 Golgi apparatus

RAH 30.44 Ribosomes

RAH 30.45 Lysosomes / lysozymes

RAH 31.50–31.67 Detoxification programs (following

testing or related to the relevant organ)

RAH physiology of the tested organ, e.g. 45.00 Kidney

RAH 31.25 ATP production lymph

RAH 32.20 Leucocytes total


RAH 36.50 Thymus

RAH 36.60 Spleen

Toxic liver damage

RAH 31.60 Detoxification liver

RAH 31.59 Detoxification pancreas



RAH 48.10 Liver, total

Migraines



RAH 35.11 Increase of non-specific defence

RAH 35.12 Increase of specific defence


RAH 54.10 Central nervous system, total

RAH 45.80 Boosting diuresis (dehydration)

RAH 33.60 Oxygen supply / improvement of the utilisation



Chronic eczema




RAH 31.65 Detoxification skin

RAH 31.63 Detoxification bladder

RAH 31.62 Detoxification kidney

RAH 62.10 Skin, total

RAH 44.10 Kidney, total

RAH 35.20 Allergy, total

RAH 30.20 Cell membrane

Asthma bronchial


RAH 31.66 Detoxification endotoxins





RAH 31.81 Scarring

Bronchitis





RAH 35.10 Increased defences

RAH 31.80 Open wounds / wound healing

RAH 07.22 Zinc

110

Duodenal ulcer and gastric ulcer



RAH 07.22 Zinc


RAH 33.55 Inflammation of bone marrow



RAH 54.00 Nervous system physiology, total

RAH 64.00 Hormone system physiology, total

RAH 20.00-21.96 Bacteria (following testing)

Arthrosis



RAH 07.22 Zinc



RAH 53.53 Arthrosis

RAH 65.10 Female hormone balance basic regulation

or

RAH 65.20 Male hormone balance basic regulation

Lymphatic diathesis / lymphatisum



RAH 07.22 Zinc







RAH 30.43 Golgi apparatus

RAH 30.44 Ribosomes

RAH 30.45 Lysosomes


RAH 37.13 Lymphatic drainage disorder

Allergy

RAH 35.20 Allergy, total

RAH 36.00 Lymphatic system physiology, total

RAH 44.10 Kidney, total



Gout

RAH 51.10 Gout

Can also be used:






111

Summary

Anamnesis is the be-all and end-all of any treatment. Via

testing, via the RAH, we gain a sharper view of our patients.

From both a diagnostic and a therapeutic point of view, this

gives us an inestimable lead in the cause-orientated detec-

tion of illnesses. The living conditions of the patient are

always important, as is his or her family situation, earlier

illnesses, vaccinations, habits and modalities. The entire

therapy using RAH is based on three equally important

pillars. One pillar is the strengthening of the ill organ, the

other pillar is the improvement and aid of the defences for

the organism, and the third pillar is detoxification.

Detoxification is the very first step towards healing, since

a sick cell or basic function can only recover in a healthy

environment. Treatment of the matrix is hence indispensa-

ble with the RAH detoxification programs.

Treat the pillars of the therapy creatively – for the well-

being of your patients.

Gerhard G. Rögele, HP

112

12 Appendix V: The new analysis support by means of test protocols

The idea behind the test protocols was born in the

non-medical school of the Paul Schmidt Akademie. There,

one of the things students learn is which organ structures

and regulation areas are to be taken into consideration for

which illnesses. This also applies to energetic testing using

the RAH. Hence in the case of hypertension (high blood

pressure), for instance, the kidney has to be taken into

consideration since it produces the enzyme renin, which

increases blood pressure. Or the hormone system, since

it has an enormous influence on metabolism and blood

pressure. It is precisely these links that the new analysis

support takes into consideration. If, for instance, you select

the frequency structure of the RAH program 39.60 Hyper-

tension, at the touch of a button you can have the device

display the RAH programs linked to the symptoms. This can

exceed 50 different areas in some cases. In the program

version available since April 2011 for the Rayocomp PS

1000 polar and the Rayocomp PS 10, 21 sets of symptoms

are already supported to allow extensive energetic tests.

1. 35.20 Allergy, total

2. 39.60 High blood pressure (hypertension)

3. 43.20 Asthma bronchial

4. 45.35 Cystitis (inflammation of the bladder)

5. 47.20 Gastritis, acute

6. 47.30 Gastritis, chronic

7. 47.50 Crohn’s disease

8. 47.60 Ulcerative colitis

9. 51.40 Diabetes mellitus

10. 51.50 Gout

11. 53.52 Arthritis

12. 53.84 Fibromyalgia

13. 55.30 Alzheimer’s disease

14. 55.31 Parkinson’s disease

15. 55.60 Migraines

16. 57.40 Humid maculadegeneration

17. 57.41 Dry maculadegeneration

18. 59.10 Tinnitus

19. 63.20 Neurodermatitis

20. 65.60 Menopause symptoms

21. 67.30 Endometriosis

For each of these 21 test protocols, there is a very detailed

description of why the appropriate program should be taken

into consideration for the illness at hand. The assignment

and the description of the areas to be individually tested are

the work of the principal of the Paul Schmidt Akademie, Ms

HP Bettina Shipper, herself a member of the RAH panel of

experts, and lecturer of such.

The structure of the following test protocols is orientated

towards the cause-orientated structure of bioresonance

according to Paul Schmidt. First of all the energetics are

tested, e.g. the vitalisation and the Meridians associated

with the target illness. There then follows a suggestion

of the possible causal influences, starting with e-smog,

through deficiencies, right through to harmful substances.

Then, a test of the pathogens associated with the illness

is recommended (created by Ms HP Schußmann and Dr

Schußmann). Then the relevant ATP programs by Dr Yayama

from Japan. There then follows the extensive area of

programs pertaining to physiology and pathology. This is

followed by a recommended test of the relevant detoxifica-

tion programs developed by Mr HP Rögele.

These test protocols (see Appendix V for more detailed

information) create a guide for both the analysis and the

harmonisation process with the RAH. A further objective of

the test protocols is to clarify as precisely as possible the

energetic deficits.

The test protocols can be used in the RAH module of the

Rayocomp PS 1000 polar and in the M10 module of the

Rayocomp PS 10.

The test results can also be archived via an RAH “Green

Card”.

113

35.20 Allergy, totalProgram No. / description Explanation Time

00.00 Analysis preparation This program is only used during analysis. Rotation on the Rayo-

tensor: measurement can commence, linear motion: first of all,

harmonise the program until a rotation is visible on the Rayo-

tensor.

0 min.

01.00 Vitalisation, total Test to ascertain if the energy balance is disturbed / too weak. 5 min.

02.11 Lung meridian Meridians associated with the target illness. 2 min.

02.12 Colon meridian 2 min.

02.13 Stomach meridian 2 min.

02.14 Spleen meridian 2 min.

02.15 Heart meridian 2 min.

02.17 Bladder meridian 2 min.

02.18 Kidney meridian 2 min.

02.19 Liver meridian 2 min.

02.21 Meridian of triple burner 2 min.

02.22 Gall-bladder meridian 2 min.

02.24 Conception vessel meridian 2 min.

31.11 ATP production lung These ATP programs are to be taken into consideration for the

illness.

5 min.

31.31 ATP production eyes 5 min.

31.38 ATP production skin 5 min.

31.39 ATP production vessels 5 min.

04.00 Electrosmog, total By means of the named programs, the cause-orientated treat-

ment approach is supported.

5 min.

05.00 Geopathic stresses, total 5 min.

06.00 Acid-base balance, total 5 min.

07.00 Vital substances, total 5 min.

08.00 Harmful substances, total 5 min.

32.20 Leucocytes total White blood corpuscles are responsible for defence tasks and,

in the event of an allergy, the reactivity of the immune system

is changed compared to the substances known as allergens. The

lymphocytes, a group of leucocytes, form antibodies and prema-

ture reactions of the immune system as well as delayed reactions

can occur (allergy type I-IV).

5 min.

34.00 Immune system physiology, total The immune system is changed in the event of allergic reactions,

and needs to be supported.

5 min.

35.20 Allergy, total In the case of an allergic reaction, various reponses by the

immune system can occur. Three mainly anti-body dependent

premature reactions (allergy type I to III) and a lymphocyte-

dependent delayed reaction (allergy type IV) can occur.

5 min.

114


36.00 Lymphatic system physiology, total The lymphatic system with its lymphatic organs reacts very early

to allergic reactions in the body, and it is an important part of

testing.

5 min.

37.12 Lymphadenitis, swelling of the lymph

node

In the case of a defensive reaction of the immune system, the

lymph nodes react very prematurely, usually regionally at first.

5 min.

37.30 Spleen organ function fortification The spleen is an important lymphatic organ which breaks down

the blood corpuscles.

5 min.

38.10 Arteries Allergies can cause arteries to undergo inflammation changes. 5 min.

38.50 Veins Allergies can cause veins to undergo inflammation changes. 5 min.

39.30 Inflammation of the blood vessels In the case of allergic reactions, inflammation of the entire

vascular system can occur.

5 min.

42.00 Respiratory tracts physiology, total The upper and the lower respiratory tracts are usually affected in

the case of allergies, which can cause constriction and shortness

of breath.

5 min.

43.10 Cough, acute Coughing is a physical reaction which can also occur as a defen-

sive reaction in the case of allergies.

5 min.

43.20 Asthma bronchial Asthma bronchial can be caused by an allergic disposition of the

body.

5 min.

43.30 Mucous congestion Mucous congestion, constriction and coughing are frequent re-

actions in the case of allergies.

5 min.

46.00 Digestive system physiology, total In the case of allergic illnesses, the digestive system reacts since

this accommodates small lymph nodes, such as those in the final

part of the small intestine, the ileum, which provide the immune

defence system.

5 min.

56.00 Visual organ physiology, total Inflammatory reactions of the eyes, especially of the conjuncti-

va, can occur in conjunction with allergies.

5 min.

62.00 Skin / hair physiology, total Skin reactions, reddening and swelling of the skin are part of

allergic illnesses.

5 min.

64.10 Hypothalamus CRH, a stimulating hormone, is produced in the hypothalamus.

At the first hormone level, CRH determines the later formation

and release of cortisol from the adrenal cortex. Cortisol regulates

and reduces the reactions of the immune system in the event of

inflammation and allergic reactions.

5 min.

64.20 Pituitary gland It is in the anterior lobes of the hypophysis that ACTH is formed:

in turn, this stimulates the adrenal cortex to form cortisol and

release it into the blood. Cortisol reduces the defence reaction

and readiness of the body.

5 min.

64.55 Adrenal cortex The adrenal cortex produces cortisol, which has anti-allergenic

and immunosuppressive effects.

5 min.

20.12 Beta-haemolytic streptococcus From an energetic point of view, these pathogens require parti-

cular consideration.

5 min.

115


20.13 Eikenella corrodens From an energetic point of view, these pathogens require parti-


5 min.

20.19 Staphylococcus aureus 5 min.

20.21 Streptococcus lactis 5 min.

20.22 Streptococcus mitis 5 min.

20.23 Streptococcus pneumoniae 5 min.

20.24 Streptococcus pyogenes 5 min.

20.25 Streptococcus sp. 5 min.

20.42 Actinomyces israelii 5 min.

20.44 Bacilli 5 min.

20.46 Bacillus cereus 5 min.

20.47 Bacteroides fragilis 5 min.

20.49 Bordetella pertussis 5 min.

20.66 Gardnerella vaginalis 5 min.

20.67 Haemophilus influenzae 5 min.

20.69 Helicobacter pylori 5 min.

20.70 Lactobacillus acidophilus 5 min.

20.72 Legionella pneumophila 5 min.

20.76 Mycobacterium tuberculosis 5 min.

20.81 Propionibacterium acnes 5 min.

21.11 Enterobacter aerogenes 5 min.

21.12 Erwinia amylovora 5 min.

21.13 Erwinia carotovora 5 min.

21.15 Klebsiella pneumoniae 5 min.

21.16 Proteus mirabilis 5 min.

21.17 Proteus vulgaris 5 min.

21.19 Salmonella enteritidis 5 min.

21.20 Salmonella paratyphi 5 min.

21.21 Salmonella typhi 5 min.

21.22 Serratia marcescens 5 min.

21.23 Shigella dysenteriae 5 min.

21.86 Chlamydia trachomatis 5 min.

21.88 Rickettsias 5 min.

21.91 Bacteria laryndiale 5 min.

21.93 Caries bacteria 5 min.

22.11 Adenovirus 5 min.

22.12 Cytomegalovirus (CMV) 5 min.

22.13 Epstein-Barr virus (EBV) 5 min.

22.15 Herpes simplex 5 min.

116


22.17 Herpes zoster From an energetic point of view, these pathogens require parti-


5 min.

22.64 Chikungunya 5 min.

22.67 Coxsackie virus B-1 5 min.


22.78 Norovirus 5 min.

22.80 Rhino virus 5 min.

22.82 Tobacco mosaic virus 5 min.

23.16 Parainfluenza 5 min.

23.33 Influenza A and B virus 5 min.

23.56 Rota viruses 5 min.

23.70 Warts, total 5 min.

23.81 Viruses N.N. 5 min.

24.21 Ascaris megalocephala 5 min.

24.23 Enterobius vermicularis 5 min.

24.28 Oxyuria 5 min.

24.31 Strongyloides (filariform) 5 min.

24.51 Clonorchis sinensis 5 min.

24.54 Eurythrema pancreaticum 5 min.

24.56 Fasciolopsis buski 5 min.

24.58 Gastrothylax elongates 5 min.

24.63 Schistosoma haematica 5 min.

24.64 Schistosoma mansoni 5 min.

24.84 Taenia saginata 5 min.

24.85 Taenia solium 5 min.

25.14 Blepharisma 5 min.

25.15 Chilomastix cysts (rat) 5 min.

25.16 Chilomonas 5 min.

25.35 Naegleria fowleri 5 min.

25.62 Dermatophagoides (dust mite) 5 min.

25.64 Demodex folliculorum (follicular

mite)

5 min.

25.67 Ornithonyssus (bird mite) 5 min.

25.68 Sarcoptes scabiei (scabies) 5 min.

25.84 Troglodytella abrasseri 5 min.

25.86 Pneumocystis jiroveci (carinii) 5 min.

26.05 Fungi I, total 5 min.

26.12 Aspergillus niger 5 min.

26.41 Aflatoxin 5 min.

117

Own notice of 35.20 Allergy, total


27.05 Fungi II, total From an energetic point of view, these pathogens require parti-


5 min.

27.10 Yeast fungi, total 5 min.

27.11 Candida albicans 5 min.

31.52 Detoxification lymph system The detoxification programs opposite should be taken into con-

sideration for the illness.

5 min.

31.56 Detoxification mucous membranes 5 min.

31.62 Detoxification kidney 5 min.

31.65 Detoxification skin 5 min.

01.00 Vitalisation, total In order to implement the specified regulation pulses, the body

requires energy. For this reason, the vitalisation program is al-

ways used once more during the harmonisation process.

2 min.

118

39.60 High blood pressure (hypertension)Program no. / description Explanation Time

00.00 Preparation of analysis This program is only used during analysis. Rotation at the rayo-

tensor: Measurement can start, Linear movement: Start harmo-

nizing the program until rotation can be seen at the rayotensor

0 min.

01.00 Vitalization in general Tests to see if the energy balance is disturbed / weak. 5 min.

02.14 Spleen meridian Meridian in relation with the target disease. 2 min.





02.20 Cardiovascular meridian 2 min.

02.22 Gallbladder meridian 2 min.

31.15 ATP production heart These ATP programs should be considered in connection with the

target disease.

5 min.

31.23 ATP production kidney 5 min.


04.00 Electrosmog in general These programs support the cause-oriented treatment approach. 5 min.

05.00 Geopathic stress in general 5 min.

06.00 Acid-base balance in general 5 min.

07.00 Vital substances in general 5 min.

08.00 Harmful substances in general 5 min.

38.10 Arteries Hypertension is located in the arterial vessel system and is ag-

gravated by deposits in the arteries.

5 min.

39.10 Arterial circulatory disorders Arterial circulatory disorders are a consequence of hypertension,

they begin in the small arteries and later also extend to the lar-

ger arteries.

5 min.

39.40 Blood vessel degeneration Deposits and ateriosclerosis change the inner layers of arteries,

which encourages hypertension.

5 min.

39.50 Blood pressure regulation disorders Hypertension leads to disorders of the blood pressure regulation. 5 min.

39.60 High blood pressure (hypertension) Hypertension 5 min.

39.65 Renal hypertension The kidneys, to be exact, diseases of the kidneys, can be the

cause of hypertension. The kidneys produce renin, an enzyme

that increases the blood pressure.

5 min.

40.13 Myocard The layers of the heart, more exactly, the myocard, the heart

muscle layer, generates pressure in the heart and the outgoing

arteries. High pressure causes thickening of the myocard and

damage in the long term.

5 min.

40.30 Heart valves in general Diseases of the heart valves can cause high blood pressure or if

hypertension existed before, the heart valves can be damaged.

5 min.

119

39.60 High blood pressure (hypertension)Program No. / description Explanation Time

41.10 Strengthening of the heart muscle Hypertension causes the heart muscle to do more work and be-

come thicker in the course of time.

5 min.

41.11 Improving the performance of the

heart

Existing hypertension requires the heart to do more work, which

with time can lead to cardiac insufficiency.

5 min.

41.20 Left heart insufficiency Hypertension causes stress, in particular, of the left side of the

heart from where the oxygen-rich blood is pumped throughout

the body. The muscles of the left heart are overloaded and lack

sufficiency in the course of time.

5 min.

44.10 Kidney in general The kidneys produce renin, an enzyme that increases the blood

pressure. Diseases of the kidneys can be the cause of high blood

pressure, so called renal hypertension. Always consider the kid-

neys as potential cause of hypertension.

5 min.

64.10 Hypothalamus Some hormonal glands influence the blood pressure: The hypo-

thalamus produces stimulating and inhibiting hormones which

cause the anterior pituitary gland and the posterior pituitary

gland to produce further hormones. These, in turn, are carried by

the bloodstream, e.g., to the thyroid, which has a major impact

on the body’s metabolism and on blood pressure. The adrenals

produce adrenalin in the adrenal medulla, a stress hormone that

increases the blood pressure whereas the adrenal cortex produ-

ces cortisone that binds water in the body and thereby increases

the blood volume and the blood pressure in the body. The kidneys

produce renin, which raises the blood pressure. The posterior pi-

tuitary gland produces, for example, ADH, antidiuretic hormone,

which also contributes to higher blood pressure in connection

with the release of renin in the kidneys, by retaining water in the

body, thereby increasing the blood volume.

5 min.

64.20 Pituitary gland 5 min.

64.55 Adrenal cortex 5 min.

20.22 Streptococcus mitis From the angle of energy, these causal agents should be given

particular attention in connection with the target disease.

5 min.


21.14 Escherichia coli 5 min.



21.88 Rickettsia 5 min.

24.22 Dirofilaria immitis (heart worm) 5 min.

24.51 Clonorchis sinensis 5 min.



120

Own notice of 39.60 High blood pressure (hypertension)

39.60 High blood pressure (hypertension)Program No. / description Explanation Time

24.65 Urocleidus From the angle of energy, these causal agents should be given

particular attention in connection with the target disease.

5 min.

25.15 Chilomastix cysts (rat) 5 min.

25.16 Chilomonas 5 min.

25.41 Trichomonas vaginalis 5 min.

25.85 Blood parasites 5 min.


27.10 Mould fungi in general 5 min.


31.66 Detoxification endotoxins These detoxification programs should be considered in connec-

tion with the target disease.

5 min.

31.67 Detoxification exotoxins 5 min.

01.00 Vitalization in general The body needs energy to implement the regulation impulses it

receives. For this reason, the vitalization program is always cho-

sen at the end, in connection with harmonization.

2 min.

121

43.20 Asthma bronchialProgram No. / description Explanation Time




tensor.

0 min.








31.11 ATP production lung These ATP programs are to be taken into consideration for the

illness.

5 min.



Zinc is very important for the immune system and for many en-

zymatic conversion processes in the body.

5 min.




07.22 Zinc 5 min.


31.81 Scarring Scars can be a cause / a trigger for asthma bronchial, and need

to be harmonised before therapy commences.

5 min.

34.00 Immune system physiology, total The immune system is generally weakened in the case of asthma

bronchial and should be included in the testing and harmonisa-

tion processes.

5 min.

35.10 Increased defences, basic program Since asthma bronchial can be triggered or worsened by infec-

tions, it is important to strengthen and increase the body’s de-

fence system.

5 min.

35.20 Allergy, total Asthma bronchial can be caused by allergies: in the presence of

a linear motion on the Rayotensor, please use a testing kit for

differentiated further testing.

5 min.

40.13 Myocardium In the case of chronic lung diseases, the heart muscle, the myo-

cardium, is particularly strained on the right side since it needs

to pump blood to the lung in the face of increased resistance.

5 min.

40.22 Right ventricle The right ventricle is faced with increased stress due to the in-

creased pressure which builds up in the lung with chronic lung

disease, and is enlarged over time.

5 min.

41.10 Strengthening of the cardiac muscles Asthma bronchial places a particular strain on the right side of

the myocardium, which thickens at a later date.

5 min.

122


41.30 Right ventricular failure Long-term asthma bronchial can cause weakening of the right

side of the heart, with a weakening of the heart muscles in the

advanced stage.

5 min.

42.60 Bronchial tubes, total Asthma bronchial affects the bronchial tubes. 5 min.

42.70 Lungs Asthma bronchial affects the lung tissue with pulmonary alve-

olus.

5 min.

43.10 Cough, acute Coughing is one of the main symptoms of asthma bronchial. 5 min.

43.20 Asthma bronchial Asthma bronchial 5 min.

43.30 Mucous congestion Mucous congestion in the bronchial tubes is one of the three

main criteria of the symptoms of asthmas bronchial alongside

spasms (muscular cramps) of the bronchial tubes and accumula-

tion of fluid, bronchial oedema.

5 min.

75.10 Stress reduction Stress factors can trigger or fortify asthma bronchial. 5 min.

75.20 Emotional stress Emotional stress, particularly long-term stress, can trigger or

worsen asthma bronchial.

5 min.



5 min.





20.44 Bacilli 5 min.

20.49 Bordetella pertussis 5 min.

20.67 Haemophilus influenza 5 min.

20.72 Legionella pneumophila 5 min.


21.15 Klebsiella pneumoniae 5 min.


21.91 Bacteria laryndiale 5 min.

22.11 Adenovirus 5 min.




22.17 Herpes zoster 5 min.



123


22.80 Rhino Virus From an energetic point of view, these pathogens require parti-


5 min.

23.16 Parainfluenza 5 min.

23.33 Influenza A und B Virus 5 min.

23.81 Viren N.N. 5 min.


25.86 Pneumocystis jiroveci (carinii)



31.53 Entgiftung Acidose The detoxification programs opposite should be taken into con-


5 min.

31.55 Entgiftung intrazellulär 5 min.

31.57 Entgiftung Lunge 5 min.

31.66 Entgiftung Endotoxine 5 min.

31.67 Entgiftung Exotoxine 5 min.

01.00 Vitalisierung gesamt In order to implement the specified regulation pulses, the body



2 min.

Own notice of 43.20 Asthma bronchial

124

45.35 Cystitis (inflammation of the bladder)Program No. / description Explanation Time




tensor.

0 min.


02.12 Colon meridian Meridians associated with the target illness. 2 min.





31.17 ATP production bladder These ATP programs are to be taken into consideration for the

illness.

5 min.




5 min.





35.10 Increased defences, basic program Since inflammation of the bladder is frequently caused by pa-

thogens or thermal stimuli such as the cold, it is very important

to strengthen and increase the body’s overall defences.

5 min.

44.10 Kidney, total Inflammation of the bladder can be caused and triggered by a

descending infection of the kidney.

5 min.

44.20 Urinary organs, total Bladder infection can be caused by a descending infection of the

urethra or an ascending infection of the urethra.

5 min.

45.35 Cystitis (inflammation of the bladder) Inflammation of the bladder 5 min.

45.40 Urethritis (inflammation of the ure-

thra)

Inflammation of the urethra can result in an ascending infection

of the bladder.

5 min.

20.66 Gardnerella vaginalis From an energetic point of view, these pathogens require parti-


5 min.

21.14 Colon bacillus 5 min.





24.65 Urocleidus 5 min.




125

45.35 Cystitis (inflammation of the bladder)Program No. / description Explanation Time

27.11 Candida albicans From an energetic point of view, these pathogens require parti-


5 min.

31.51 Detoxification, blood When it comes to an infection of the bladder, a difference is

made between the acute and the chronic forms.

Hence, there are various testing options in the detoxification

programs.

The detoxification programs opposite should be taken into con-


5 min.

31.52 Detoxification lymph system 5 min.

31.54 Detoxification extra-cellular 5 min.

31.56 Detoxification mucous membranes 5 min.


31.63 Detoxification bladder 5 min.





2 min.

Own notice of 45.35 Cystitis (inflammation of the bladder)

126

47.20 Gastritis, acuteProgram No. / description Explanation Time




tensor.

0 min.


02.13 Stomach meridian Meridians associated with the target illness. 2 min.






31.13 ATP production stomach These ATP programs are to be taken into consideration for the

illness.

5 min.



A problem with the stomach, such as a case of gastritis, can re-

sult in problems absorbing vitamin B12. The intrinsic factor from

the stomach cells necessary for the absorption of vitamin B12

via the small intestine is no longer adequately formed. Vitamin

B12 is vital for the formation of new cells, especially for blood

corpuscles.

5 min.




07.49 Vitamin B12, cobalamin 5 min.


46.30 Stomach In the case of gastritis, various layers and areas of the stomach

can be affected.

5 min.

46.40 Small intestine, total Dysfunctions in the small intestine, especially in the duodenum,

can be the cause of gastritis.

5 min.

47.20 Gastritis, acute Acute gastritis 5 min.

47.31 Gastritis, type A Type A gastritis is known as auto-immune gastritis: anti-bodies

are formed against the stomach cells and the intrinsic factor.

5 min.

47.32 Gastritis, type B Type B gastritis is a bacterial form caused by the helicobacter

bacterium: it is also the most common type of gastritis.

5 min.

47.33 Gastritis, type C Type C gastritis is the chemo-toxic form of gastritis, which is

caused by the return of bile from the duodenum to the stomach.

5 min.

20.22 Streptococcus mitis From an energetic point of view, these pathogens require parti-


5 min.






127

47.20 Gastritis, acuteProgram No. / description Explanation Time

21.23 Shigella dysenteriae From an energetic point of view, these pathogens require parti-


5 min.

















31.58 Detoxification stomach The detoxification programs opposite should be taken into con-


5 min.

31.66 Detoxification endotoxins 5 min.





2 min.

Own notice of 47.20 Gastritis, acute

128

47.30 Gastritis, chronicProgram No. / description Explanation Time




tensor.

0 min.








31.13 ATP production stomach These ATP programs are to be taken into consideration for the

illness.

5 min.



A problem with the stomach, such as a case of gastritis, can re-

sult in problems absorbing vitamin B12. The intrinsic factor from

the stomach cells necessary for the absorption of vitamin B12

via the small intestine is no longer adequately formed. Vitamin

B12 is vital for the formation of new cells, especially for blood

corpuscles.

5 min.






46.30 Stomach In the case of gastritis, various layers and areas of the stomach

can be affected.

5 min.

46.40 Small intestine, total Dysfunctions in the small intestine, especially in the duodenum,

can be the cause of gastritis.

5 min.

47.30 Gastritis, chronic Chronic gastritis 5 min.

47.31 Gastritis, type A Type A gastritis is known as auto-immune gastritis: anti-bodies

are formed against the stomach cells and the intrinsic factor.

5 min.

47.32 Gastritis, type B Type B gastritis is a bacterial form caused by the helicobacter

bacterium: it is also the most common type of gastritis.

5 min.

47.33 Gastritis, type C Type C gastritis is the chemo-toxic form of gastritis, which is

caused by the return of bile from the duodenum to the stomach.

5 min.



5 min.






129

47.30 Gastritis, chronicProgram No. / description Explanation Time

21.23 Shigella dysenteriae From an energetic point of view, these pathogens require parti-


5 min.

















31.58 Detoxification stomach The detoxification programs opposite should be taken into con-


5 min.






2 min.

Own notice of 47.30 Gastritis, chronic

130

47.50 Crohn’s diseaseProgram No. / description Explanation Time




tensor.

0 min.








31.12 ATP production colon These ATP programs are to be taken into consideration for the

illness.

5 min.

31.16 ATP production small intestine 5 min.



In the case of chronic, inflammatory intestinal diseases, the ab-

sorption of vital substances such as vitamin B12 in the final part

of the small intestine is disrupted.

The intestinal flora is usually in a state of disbalance caused by

the digestion and absorption disorders.

5 min.





07.60 Probiotic bacteria, total 5 min.


31.70 Degeneration cellular tissue Due to its chronic course which occurs in phases, Crohn’s disease

tends to cause hardening of cell tissue. This means that the risk

of a malignant intestinal disease increases in the case of a long-

term disease.

5 min.

32.20 Leucocytes total Leucocytes are responsible for unspecific and specific defence

which is usually impaired by immunological regulatory disorders

in the case of inflammatory intestinal diseases.

5 min.

34.00 Immune system physiology, total In the presence of inflammatory intestinal diseases, the immune

system is weakened, since an auto-immune disease is usually

the cause.

5 min.

35.10 Increased defences, basic program The entire defence needs stabilising and increasing in the pre-

sence of inflammatory intestinal diseases.

5 min.

35.20 Allergy, total In the case of inflammatory intestinal diseases, the auto-immune

processes can cause allergic reactions.

5 min.

36.00 Lymphatic system physiology, total The lymphatic system needs to be strengthened in the case of

inflammatory diseases and, consequently, lymphatic drainage

stimulated, and toxins removed.

5 min.

131


46.00 Digestive system physiology, total In the presence of Crohn’s disease, changes to the mucous

membranes in the mouth, in the stomach, in the small intestine

and in the colon are common. It is usually the final part of the

small intestine and parts of the colon which are affected.

5 min.

47.50 Crohn’s disease Crohn’s disease 5 min.

48.00 Liver, gall-bladder, pancreas physiolo-

gy, total

Absorption disorders and indigestion throughout the whole

intestines can cause inflammation of and diseases to the gall-

bladder and the pancreas.

5 min.

52.00 Locomotor system physiology, total In the presence of inflammatory intestinal diseases, auto-immune

factors can also cause arthritis.

5 min.

56.30 Skin, total The sclera can also be affected by auto-immune factors. 5 min.

64.10 Hypothalamus Strengthening of the hypothalamus aids the production of corti-

sol at the first hormone level. Cortisol has an anti-inflammatory

and immunosuppressive effect.

5 min.

64.20 Pituitary gland The anterior lobes of the pituitary gland produce ACTH, which

activates the adrenal cortex.

5 min.

64.55 Adrenal cortex The adrenal cortex produces cortisol. 5 min.

72.00 Psyche Emotional ordeals worsen the inflammatory intestinal diseases

/ can cause flare-ups. Mental illnesses can also occur following

the illness.

5 min.

75.10 Stress reduction Stress factors are a very frequent trigger of flare-ups. 5 min.

75.20 Emotional stress Emotional stress can be the main trigger as well as reinforcer of

inflammatory intestinal illnesses.

5 min.



5 min.
















132


24.58 Gastrothylax elongates From an energetic point of view, these pathogens require parti-


5 min.









5 min.

31.61 Detoxification intestines 5 min.




2 min.

Own notice of 47.50 Crohn’s disease

133

47.60 Ulcerative colitisProgram No. / description Explanation Time




tensor.

0 min.









illness.

5 min.




In the case of chronic, inflammatory intestinal diseases, the ab-

sorption of vital substances such as vitamin B12 in the final part

of the small intestine is disrupted.

The intestinal flora is usually in a state of disbalance caused by

the digestion and absorption disorders.

5 min.





07.60 Probiotic bacteria, total 5 min.


31.70 Degeneration cellular tissue Due to its chronic course which occurs in phases, ulcerative co-

litis tends to cause hardening of cell tissue. This means that the

risk of a malignant intestinal disease increases in the case of a

long-term disease.

5 min.

32.20 Leucocytes total Leucocytes are responsible for unspecific and specific defence

which is usually impaired by immunological regulatory disorders

in the case of inflammatory intestinal diseases.

5 min.

34.00 Immune system physiology, total In the presence of inflammatory intestinal diseases, the immune

system is weakened, since an auto-immune disease is usually

the cause.

5 min.

35.10 Increased defences, basic program The entire defence needs stabilising and increasing in the pre-

sence of inflammatory intestinal diseases.

5 min.

35.20 Allergy, total In the case of ulcerative colitis, the auto-immune processes can

cause allergic reactions.

5 min.

36.00 Lymphatic system physiology, total The lymphatic system needs to be strengthened in the case of

inflammatory diseases and, consequently, lymphatic drainage

stimulated, and toxins removed.

5 min.

134


46.00 Digestive system physiology, total In the presence of ulcerative colitis, the entire colon can be af-

fected: however, it is normally the final parts of the colon. Very

frequent bloody, mucous diarrhoea occurs.

5 min.

47.60 Ulcerative colitis Ulcerative colitis 5 min.


gy, total

Absorption disorders and indigestion throughout the whole

intestines can cause inflammation of and diseases to the gall-

bladder and the pancreas.

5 min.

52.00 Locomotor system physiology, total In the presence of inflammatory intestinal diseases, auto-immune

factors can also cause arthritis.

5 min.

56.30 Skin, total The sclera can also be affected by auto-immune factors. 5 min.

64.10 Hypothalamus Strengthening of the hypothalamus aids the production of corti-

sol at the first hormone level. Cortisol has an anti-inflammatory

and immunosuppressive effect.

5 min.

64.20 Pituitary gland The anterior lobes of the pituitary gland produce ACTH, which

activates the adrenal cortex.

5 min.

64.55 Adrenal cortex The adrenal cortex produces cortisol. 5 min.

72.00 Psyche Emotional ordeals worsen the inflammatory intestinal diseases

/ can cause flare-ups. Mental illnesses can also occur following

the illness.

5 min.

75.10 Stress reduction Stress factors are a very frequent trigger of flare-ups. 5 min.

75.20 Emotional stress Emotional stress can be the main trigger as well as reinforcer of

inflammatory intestinal illnesses.

5 min.



5 min.

















135


24.63 Schistosoma haematica From an energetic point of view, these pathogens require parti-


5 min.








5 min.

31.61 Detoxification intestines 5 min.




2 min.

Own notice of 47.60 Ulcerative colitis

136

51.40 Diabetes mellitusProgram No. / description Explanation Time




tensor.

0 min.


02.19 Liver meridian Meridians associated with the target illness. 2 min.


02.23 Gouvernor vessel meridian 2 min.

31.14 ATP production pancreas These ATP programs are to be taken into consideration for the

illness.

5 min.

31.15 ATP production heart 5 min.


31.31 ATP production eyes 5 min.





5 min.





34.00 Immune system physiology, total Due to the metabolic disease, the immune system of diabetics is

weakened over the long term, and requires regular fortification.

5 min.

35.10 Increased defences, basic program The bodily defences of the diabetic need to be strengthened due

to the weakened immune system caused by the illness.

5 min.

38.10 Arteries The chronic glycometabolism disease of diabetes mellitus is fre-

quently accompanied by increased deposits in the arterial ves-

sels. These manifest themselves initially in smaller and, later, in

larger arteries, with circulatory problems and corresponding se-

condary diseases.

5 min.

39.10 Circulatory problem, arterial Circulatory problems with the arteries and their secondary di-

seases such as hardening of the arteries, hypertension and coro-

naries are encouraged by diabetes mellitus.

5 min.

39.60 High blood pressure (hypertension) In the presence of diabetes mellitus, increased deposits in the

arteries result initially in hardening of the arteries and, conse-

quently, to high blood pressure.

5 min.

40.13 Myocardium The myocardium, the layer of the heart muscle, produces the

pressure in the heart and in the arteries. High blood pressure

means that the myocardium thickens, damaging it over the long

term.

5 min.

137


41.10 Strengthening of the cardiac muscles Hypertension results in more pressure on the heart muscle, with

thickening of it, and to premature weakening.

5 min.

44.10 Kidney, total The kidneys have a very finely structured arterial supply, which

means that this organ reacts by means of dysfunctions in the

event of circulatory problems. In diabetes patients, kidney di-

seases are amongst the most frequent secondary diseases and

should be taken into consideration during the testing and har-

monisation processes.

5 min.

44.17 Kidney corpuscle The kidney corpuscles are the part in the kidneys in which filtra-

tion of the blood is performed. Deposits and high blood pressure

in these tiniest of arteries result in losses of function and, later,

to inflammation and diseases of the kidneys. Degeneration of

the kidney function tissue as well as loss of the organ’s function

occur as long-term consequences.

5 min.

48.35 Islet cells Islet cells – B-cells, to be more precise – are located in the pan-

creas. These cells produce insulin. Insulin reduces blood sugar

and is not produced in sufficient quantities in the diabetic.

In the case of type I diabetes, the causes for the loss of function

of the B-cells are in the destruction of these cells, usually by

auto-immune processes and inflammation. In the case of type

II diabetes, the cause is a long-term depletion of B-cells with

insulin resistance.

5 min.

50.20 Carbohydrate metabolism In the presence of diabetes mellitus, the carbohydrate metabo-

lism is disrupted and unbalanced.

5 min.

51.20 Carbohydrate metabolism dysfunction In the presence of diabetes mellitus, this is a carbohydrate me-

tabolism dysfunction with disruptions of the insulin production

in the B-cells of the pancreas.

5 min.

51.40 Diabetes mellitus Glycometabolism disease 5 min.

54.20 Peripheral nerve system, total In the diabetic, due to circulatory problems in the arteries, later

on in the illness the nerves, particularly the peripheral nerve

system, are affected.

5 min.

55.42 Nerve degeneration Due to the advanced circulatory problems of the arteries, the

nerve cells are increasingly destroyed, accompanied by degene-

ration, especially in the extremities – the hands and feet – as

well as those sensory organs finely supplied with blood.

5 min.

56.30 Skin, total Sclera, in particular the retina and the choroid coat, are extreme-

ly affected by circulatory problems caused by diabetes mellitus.

5 min.

138


56.40 Lens, pupil, vitreous bodies, total The lens and the vitreous body are also impaired by diabetes

mellitus.

5 min.

57.10 Separation of the retina Separation of the retina is one of the most frequent secondary

diseases experienced by the diabetic.

5 min.

57.20 Cataract Clouding and degeneration of the lens are worsened by diabetes

mellitus.

5 min.

57.30 Glaucoma Increased eye pressure is a possible secondary disease of the di-

abetic.

5 min.

62.10 Skin, total Due to the weakened immune system of the diabetic, premature

dermatomycoses and other inflammatory skin diseases occur.

5 min.

64.70 Pancreas As a hormone-producing organ, the pancreas of diabetes pati-

ents must be tested and harmonised in order to supply the best

possible support to the hormonal functionality.

5 min.

20.69 Helicobacter pylori The detoxification programs opposite should be taken into con-


5 min.

22.14 Hepatitis B virus 5 min.

22.74 Hepatitis A virus 5 min.

22.75 Hepatitis c virus 5 min.

24.41 Capillaria hepatica (liver) 5 min.


24.55 Fasciola hepatica 5 min.


24.81 Echinococcus granulosus 5 min.

24.82 Echinococcus multilocularis 5 min.


31.59 Detoxification pancreas The detoxification programs opposite should be taken into con-


5 min.





2 min.

Own notice of 51.40 Diabetes mellitus

139

51.50 GoutProgram No. / description Explanation Time




tensor.

0 min.



02.16 Small intestine meridian 2 min.




31.23 ATP production kidney These ATP programs are to be taken into consideration for the

illness.

5 min.

31.40 ATP production muscles 5 min.

31.41 ATP production bones 5 min.



5 min.





32.00 Blood physiology total Causes of gout can also be diseases of the blood-producing

system with increased cell destruction. Increased uric acid is

formed, especially by the increased degeneration of erythrocytes

(red blood corpuscles) in the presence of anaemia and haemolysis.

5 min.

33.60 Oxygen supply / improvement of the

utilisation

With gout, the articular cartilage is particularly affected: it can-

not be supplied by its own blood vessels, it is supplied functio-

nally via diffusion. Hence, improvement to the supply of oxygen

and the removal of uric acid from the body are important objec-

tives of the treatment.

5 min.

34.00 Immune system physiology, total In the event of metabolic diseases, the immune system needs to

be boosted, since inflammatory changes occur to the entire joint.

5 min.

35.10 Increased defences, basic program In the event of inflammation, the unspecific and specific defence

should be boosted.

5 min.

35.20 Allergy, total Metabolic diseases can further auto-immune processes, and

allergic reactions can occur.

5 min.

39.10 Circulatory problem, arterial The deposit of uric acid can cause circulatory problems in the

body, especially in the cartilaginous joint parts.

5 min.

39.60 High blood pressure (hypertension) With gout, the protein metabolism disease, uric acid is deposited

in the vascular walls of the arteries, which can result in harde-

ning of the arteries and high blood pressure.

5 min.

140


40.13 Myocardium In the case of chronic gout, the uric acid crystals can be deposi-

ted in the body, e.g. in the heart muscle, the myocardium (gout

heart).

5 min.

44.10 Kidney, total Kidney diseases can result in dysfunctions of the kidneys which,

in turn, contribute towards increased deposits of uric acid in the

body. The uric acid crystals can be deposited in the case of the

chronification of gout, e.g. in the kidneys (gout kidneys). Fre-

quent attendant illnesses of gout are kidney stones (uric acid

stones).

5 min.


gy, total

Gout causes frequent attendant illnesses such as lipid metabo-

lism disorders, diabetes mellitus and damage to the liver.

5 min.

50.10 Protein metabolism Gout is a protein metabolism disease. 5 min.

50.20 Carbohydrate metabolism Carbohydrate metabolism is frequently unbalanced in the pre-

sence of gout, and can result in diabetes mellitus.

5 min.

50.30 Lipid metabolism Gout is frequently accompanied by a lipid metabolism disorder,

which is usually caused by malnutrition (adiposity).

5 min.

51.50 Gout Protein metabolism disease (gout) 5 min.

52.00 Locomotor system physiology, total With gout, deposits of uric acid occur in the large and the small

joints, especially in the joint of the big toe, the ankle joint, knee,

shoulder, hand and finger joints, as well as in the bursae.

5 min.

56.00 Visual organ physiology, total In the case of chronic gout, the uric acid can also manifest itself

as gout tophi on the eyelids.

5 min.

58.00 Hearing organ, total In the case of chronic gout, uric acid can also be deposited as

gout tophi on the outer ear and on the ear cartilage.

5 min.

62.10 Skin, total In the case of chronic gout, deposits of uric acid in the skin can

occur.

5 min.

75.15 Weight reduction Gout is usually accompanied by excess weight or even obesity. 5 min.



5 min.


21.27 Yersinia enterocolitica 5 min.

21.61 Borrelia 5 min.



21.95 Pain bacteria 5 min.

21.96 Tuberculin burnetii 5 min.





141


22.64 Chikungunya From an energetic point of view, these pathogens require parti-


5 min.





24.32 Trichinella spiralis (muscle) 5 min.

24.33 Trichuris sp. 5 min.

24.61 Paragonimus westermani 5 min.

24.62 Prosthogonimus macro. 5 min.



51.11 Prions 5 min.

31.53 Detoxification acidosis The detoxification programs opposite should be taken into con-


5 min.





2 min.

Own notice of 51.50 Gicht

142

53.52 ArthritisProgram No. / description Explanation Time




tensor.

0 min.







31.40 ATP production muscles These ATP programs are to be taken into consideration for the

illness.

5 min.

31.41 ATP production bones 5 min.



Zinc is very important for the immune system and for many en-

zymatic conversion processes in the body.

5 min.




07.22 Zinc 5 min.



utilisation

Since arthritis occurs in the cartilage part of the joint and this is

not supplied with blood – instead, it is functionally only supplied

via diffusion – targeted oxygen supply is an important objective

of the therapy and harmonisation.

5 min.

34.00 Immune system physiology, total The immune system needs to be boosted in the case of

inflammatory illnesses.

5 min.

35.10 Increased defences, basic program In the event of inflammation, the unspecific and specific defence

should be boosted.

5 min.

35.20 Allergy, total Arthritis can be triggered by an allergic reaction, and be encou-

raged by auto-immune processes in the body.

5 min.

46.40 Small intestine, total Inflammation of the joints can be accompanied by an

inflammatory disease of the small intestine (e.g. as is the case

with Crohn’s disease).

5 min.

46.50 Colon, total Inflammation of the joints can be accompanied by an

inflammatory disease of the colon (e.g. as is the case with

ulcerative colitis).

5 min.

50.10 Protein metabolism Arthritis occurs in conjunction with gout (protein metabolism

disease).

5 min.

50.20 Carbohydrate metabolism Arthritis can occur in conjunction with diabetes mellitus (carbo-

hydrate metabolism).

5 min.

51.40 Diabetes mellitus Diabetes also encourages circulatory problems in the joint, and

encourages an inflammatory change in the joint.

5 min.

143


51.50 Gout Gout, due to the deposit of uric acid in the joints, results in ar-

thritis.

5 min.

52.00 Locomotor system physiology, total Arthritis affects the entire locomotor system, especially the

bones, the musculature and the ligamentous apparatus.

5 min.

53.51 Joint injury Arthritis can be caused by the incidence of trauma. 5 min.

53.52 Arthritis Arthritis 5 min.

53.53 Arthrosis Arthritis can result in joint degeneration with a change to the

bone and cartilage substance. Consequently, extensive damage

to the joints accompanied by restricted movements can occur.

5 min.

53.54 Hyaluronic acid deficiency Hyaluronic acid is a component of synovia fluid and of bone. A

deficit of synovia encourages premature arthritis.

5 min.



5 min.







21.96 Tuberculin burnetii 5 min.

















51.11 Prions 5 min.

31.50 Detoxification basic program The detoxification programs opposite should be taken into con-


5 min.



144

Own notice of 53.52 Arthritis





2 min.

145

53.84 FibromyalgiaProgram No. / description Explanation Time

00.00 Analysevorbereitung This program is only used during analysis. Rotation on the Rayo-



tensor.

0 min.

01.00 Vitalisierung gesamt Test to ascertain if the energy balance is disturbed / too weak. 5 min.

02.12 Dickdarm-Meridian Meridians associated with the target illness. 2 min.

02.16 Dünndarm-Meridian 2 min.

02.17 Blasen-Meridian 2 min.

02.19 Leber-Meridian 2 min.

02.22 Gallenblasen-Meridian 2 min.

31.38 ATP-Produktion Haut These ATP programs are to be taken into consideration for the

illness.

5 min.

31.40 ATP-Produktion Muskeln 5 min.

04.00 Elektrosmog gesamt By means of the named programs, the cause-orientated treat-


5 min.

05.00 Geopathic disorders, complete 5 min.

06.00 Säure-Basen-Haushalt gesamt 5 min.

07.00 Vitalstoffe gesamt 5 min.

08.00 Schadstoffe gesamt 5 min.

32.20 Leukozyten gesamt In the case of auto-immune diseases such as fibromyalgia, the

unspecific and the specific defence needs to be strengthened by

means of the white blood corpuscles.

5 min.

33.60 Sauerstoffversorgung / Verbesserung

der Utilisation

In fibromyalgia, pain outside of the joints in the entire connec-

tive tissue occurs. Oxygen supply in the entire locomotor system

needs to be boosted.

5 min.

34.00 Immunsystem Physiologie gesamt The immune system is changed in auto-immune diseases, and

needs to be supported.

5 min.

35.10 Steigerung der Abwehrleistung,

Grundprogramm

In auto-immune diseases, the unspecific and specific defence

needs to be boosted.

5 min.

35.20 Allergie gesamt With auto-immune diseases, defence reactions against the

body’s own organ structures which can manifest themselves in

various types of allergy, occur.

5 min.

36.00 Lymphatisches System Physiologie

gesamt

With fibromyalgia, reactions in the lymphatic organs can occur,

which can manifest themselves as inflammation symptoms, for

instance.

5 min.

46.00 Digestive system, physiology complete With fibromyalgia, irritable bowel symptoms which manifest

themselves as pain and frequent laxation can occur.

5 min.

52.00 Musculoskeletal system, physiology

complete

The locomotor system, with its entire connective tissue, the mus-

cles, the ligaments and tendons can be affected by fibromyalgia.

5 min.

146


53.23 Tense muscles Tense muscles are caused by the chronic pain in the soft parts. 5 min.

53.25 Inflammation of the muscle Inflammatory changes in the entire connective tissue, especially

in the musculature, can occur.

5 min.

53.28 Syndesmitis / tendosynovitis With fibromyalgia, the auto-immune processes can also result in

inflammation of the ligamentous apparatus and tendon sheaths.

5 min.

53.62 Bursitis The bursae on the particularly stressed joints such as the shoulder

joint and the knee joint can experience inflammatory changes.

5 min.

53.84 Fibromyalgia Fibromyalgia (pain syndrome with chronic soft part symptoms) 5 min.

55.55 Headaches Tension headaches can be accompanied by hormonal influences

in the case of fibromyalgia.

5 min.

62.13 Hypodermis The hypodermis is also affected in the case of fibromyalgia. 5 min.

62.14 Fatty tissue The fatty tissue is also affected in the case of fibromyalgia. 5 min.

64.10 Hypothalamus In the first hormone level with CRH, the hypothalamus is respon-

sible for the formation of cortisol. This stimulates the anterior

lobes of the pituitary gland, which in turn stimulates the cortisol

release in the adrenal cortex.

5 min.

64.20 Pituitary gland The anterior lobes of the pituitary gland form ACTH, which

stimulates the adrenal cortex to form cortisol.

5 min.

64.55 Adrenal cortex The adrenal cortex produces cortisol which, in the case of in-

flammation and auto-immune diseases, throttles the reaction

of the immune system and, hence, has an immunosuppressive

effect.

5 min.

64.80 Ovaries Female hormonal factors can encourage fibromyalgia, since it is

mainly women who are affected.

5 min.

65.10 Female hormone balance basic regu-

lation

Disruptions to / imbalance in the female hormone balance can

have an increasing effect.

5 min.

72.00 Psyche Fibromyalgia can be encouraged and increased by emotional

factors.

5 min.

75.10 Stress reduction Stress factors can have a triggering or even worsening effect. 5 min.

75.18 Meteorosensitiveness / sferics Meteorosensitiveness can encourage the pain syndrome. 5 min.

75.20 Emotional stress Emotional stress, particularly continuous stress, can trigger and

worsen fibromyalgia.

5 min.



5 min.







147


21.96 Tuberculin burnetii From an energetic point of view, these pathogens require parti-


5 min.

















51.11 Prions 5 min.



5 min.

31.53 Detoxification acidosis 5 min.

31.64 Detoxification female / female-speci-

fic

5 min.




2 min.

Own notice of 53.84 Fibromyalgia

148

55.30 Alzheimer’s diseaseProgram No. / description Explanation Time




tensor.

0 min.


02.15 Heart meridian Meridians associated with the target illness. 2 min.





31.35 ATP production cerebrum These ATP programs are to be taken into consideration for the

illness.

5 min.



5 min.





34.00 Immune system physiology, total The immune system needs to be aided in the case of a degene-

rative brain disease.

5 min.

38.10 Arteries The arterial vascular system ensures the supply of oxygen in the

entire body, supporting supply of the brain.

5 min.

39.10 Circulatory problem, arterial Arterial circulatory problems intensify the demential break-down

of the cerebral cortex in the presence of Alzheimer’s.

5 min.

50.10 Protein metabolism With Alzheimer’s, protein metabolism disruptions to the nerve

cells followed by degeneration, occur.

5 min.

54.00 Nervous system physiology, total The degenerative break-down of the cerebral cortex occurs, and

this can be followed by further areas of the brain.

5 min.

55.30 Alzheimer’s disease Alzheimer-type dementia. This program serves the purpose of

completing diagnostics, and is important for the early, preventa-

tive aid of the course of the disease.

5 min.

55.42 Nerve degeneration The degeneration of nerve cells is the main aspect of the disease. 5 min.

72.00 Psyche Fluctuating moods and depression are symptoms of Alzheimer’s

disease.

5 min.

75.10 Stress reduction Stress factors increase the symptoms of the disease. 5 min.

75.20 Emotional stress Emotional stress can increase the symptoms of the disease. 5 min.



5 min.




149

55.30 Alzheimer’s diseaseProgram No. / description Explanation Time

22.12 Cytomegalovirus (CMV) From an energetic point of view, these pathogens require parti-


5 min.







23.11 Borna viruses 5 min.





mite)

5 min.






5 min.


31.55 Detoxification intra-cellular 5 min.




2 min.

Own notice of 55.30 Alzheimer’s disease

150

55.31 Parkinson’s diseaseProgram No. / description Explanation Time




tensor.

0 min.







31.10 ATP production overall These ATP programs are to be taken into consideration for the

illness.

5 min.

31.34 ATP production cerebellum 5 min.

31.35 ATP production cerebrum 5 min.



5 min.





31.70 Degeneration cellular tissue A degeneration of cell tissue / a tumour in the brain can be a

cause of Parkinson’s.

5 min.

34.00 Immune system physiology, total The immune system needs to be aided in the case of a degene-

rative brain disease.

5 min.

35.10 Increased defences, basic program The entire defence should be strengthened, since Parkinson’s can

also have infectious causes.

5 min.

38.10 Arteries The arterial vascular system ensures the supply of oxygen in the

entire body, supporting supply of blood to the brain.

5 min.

54.00 Nervous system physiology, total A degenerative break-down of dopamine-forming nerve cells in

the midbrain occurs: other regions of the brain are consequently

also involved.

5 min.

55.31 Parkinson’s disease Parkinson’s syndrome. This program serves the purpose of com-

pleting diagnostics, and is important for the early, preventative

aid of the course of the disease.

5 min.

62.10 Skin, total With Parkinson’s, the skin metabolism undergoes changes, with a

shiny, ointment-like skin consistency (hatchet face).

5 min.

62.21 Sebaceous gland Overproduction of the skin’s sebaceous glands occurs. 5 min.

72.00 Psyche During the disease, emotional symptoms such as lability and de-

pression also occur.

5 min.

151

55.31 Parkinson’s diseaseProgram No. / description Explanation Time



85.13 Aluminium (Al) Aluminium exposure or deposits can cause the disease to worsen. 5 min.

85.25 Manganese (Mn) Manganese exposure or deposits can cause the disease to wor-

sen.

5 min.

85.27 Cobalt (Co) Cobalt exposure or deposits can cause the disease to worsen. 5 min.



5 min.
















mite)

5 min.






5 min.



31.55 Detoxification intra-cellular 5 min.


requires energy. For this reason, the vitalisation program is

always used once more during the harmonisation process.

2 min.

152

Own notice of 55.31 Parkinson’s disease

153

55.60 MigrainesProgram No. / description Explanation Time




tensor.

0 min.



02.13 Stomach meridian 2 min.












illness.

5 min.



5 min.






utilisation

Oxygen supply needs to be aided. 5 min.

38.10 Arteries Arteries have the job of supplying organs and tissue with oxygen.

Lack of oxygen supply results in pain and break-down of the

tissue in the body.

5 min.

39.10 Circulatory problem, arterial Arterial circulatory problems result in a lack of oxygen supply,

which can be a frequent cause of headaches and migraines.

5 min.

39.40 Degeneration of the blood vessels Deposits and hardening of the arteries result in changes to the

vascular layers of the arteries, which encourage circulatory

problems and high blood pressure.

5 min.

39.60 High blood pressure (hypertension) Over the long term, high blood pressure results in the undersup-

ply of oxygen, especially to the organs, which are supplied by the

small arteries.

5 min.

154


44.10 Niere gesamt The kidneys produce the enzyme renin, which increases blood

pressure. Kidney diseases can result in high blood pressure, renal

hypertension, which in turn result in circulatory problems and

undersupply of oxygen.

5 min.

46.11 Mundhöhle Diseases such as inflammation of the oral cavity and of the teeth

can be the cause of headaches and migraines.

5 min.

52.30 Wirbelsäule gesamt Diseases of the spinal column can cause headaches and mi-

graines, since they can result in reduced circulation of the spinal

column arteries and of the head and neck area.

5 min.

52.31 Halswirbelsäule (C1 – C7) Diseases of the cervical spine in particular frequently result in

circulatory problems and, consequently, to the undersupply of

oxygen to the head.

5 min.

54.10 Zentralnervensystem gesamt Disorders of and diseases in various parts of the brain can be

causes of headaches and migraines.

5 min.

54.25 V. Hirnnerv (N. trigeminus) Irritation / inflammation of the trigeminal nerve of the 5th ce-

rebral nerve which, with its 3 branches each arranged in pairs,

mainly supplies the forehead, the eyes, the upper jaw, the lower

jaw and the teeth, can result in extremely painful headaches and

migraines, as well as to trigeminal facial neuralgia.

5 min.

55.55 Kopfschmerzen Headaches 5 min.

55.60 Migräne Migraines 5 min.

56.00 Organ of vision, physiology complete Visual defects and diseases of the eye can be the causes behind

pain in the head and need to be clarified.

5 min.

57.30 Grüner Star Glaucoma is accompanied by increased inner pressure in the eye,

and can result in headaches.

5 min.

58.00 Hörorgan / Gleichgewichtsorgan

Physiologie gesamt

Ear diseases can encourage pains in the head, and need to be

clarified.

5 min.

64.00 Hormonsystem Physiologie gesamt Functional disorders of hormone glands can cause headaches. 5 min.

65.10 Weiblicher Hormonhaushalt Grundre-

gulation

Changes to or disorders of the female hormone balance are fre-

quently accompanied by pains in the head.

5 min.

65.50 Menstruationsprogramme gesamt Menstrual disorders are frequently accompanied by pains in the

head.

5 min.

66.00 Female sexual organs, physiology

complete

Diseases of the female sexual organs can be the cause of pains

in the head and need to be clarified.

5 min.

72.00 Psyche Emotional strain / illnesses can cause headaches and migraines. 5 min.

75.00 Stress Stress is a frequently furthering factor for causing pains in the

head.

5 min.

76.00 Dental physiology, total Inflammation or diseases of the teeth are possible causes for

pains in the head and always need to be clarified.

5 min.

155




5 min.
















mite)

5 min.






5 min.


fic

5 min.




2 min.

Own notice of 55.60 Migraines

156

Own notice of 55.60 Migraines

157

57.40 Humid maculadegenerationProgram No. / description Explanation Time




tensor.

0 min.




31.31 ATP production eyes These ATP programs are to be taken into consideration for the

illness.

5 min.



5 min.






utilisation

Maculadegeneration is a disease of the retina, which has very

fine blood circulation. Increasing the oxygen supply is an impor-

tant objective of the therapy.

5 min.

34.00 Immune system physiology, total The immune system should be aided. 5 min.

38.10 Arteries Arteries have the job of supplying organs and tissue with oxy-

gen-rich blood. Undersupply of oxygen results in the break-down

of tissue and dysfunctions.

5 min.

39.10 Circulatory problem, arterial Arterial circulatory problems, especially on the retina, can result

in extremely degenerative changes and progression of the loss

of sight.

5 min.

54.22 II. Cerebral nerve (optic nerve) Disease of the retina can result in impairment of the visual nerve

(optic nerve).

5 min.

56.34 Retina The retina and the yellow spot, the keenest visual area, are af-

fected.

5 min.

56.61 Optical nerve The optic nerve can be impaired. 5 min.

56.62 Yellow spot The yellow spot, the keenest visual area, is affected. 5 min.

57.40 Humid maculadegeneration With humid maculadegeneration, extreme sight loss occurs in

the event of serious withdrawal of the retina and pigmentary

epithelium.

5 min.

72.00 Psyche Emotionally trying situations can intensify the symptoms of the

illness.

5 min.



5 min.



158

57.40 Humid maculadegenerationProgram No. / description Explanation Time

21.88 Rickettsias From an energetic point of view, these pathogens require parti-


5 min.















5 min.




2 min.

Own notice of 57.40 Humid maculadegeneration

159

57.41 Dry maculadegenerationProgram No. / description Explanation Time




tensor.

0 min.




31.31 ATP production eyes This ATP program is to be taken into consideration for the illness. 5 min.



5 min.






utilisation

Maculadegeneration is a disease of the retina, which has very

fine blood circulation. Increasing the oxygen supply is an impor-

tant objective of the therapy.

5 min.

34.00 Immune system physiology, total The immune system should be aided. 5 min.

38.10 Arteries Arteries have the job of supplying organs and tissue with oxy-

gen-rich blood. Undersupply of oxygen results in the break-down

of tissue and dysfunctions.

5 min.

39.10 Circulatory problem, arterial Arterial circulatory problems, especially on the retina, can result

in extremely degenerative changes and progression of the loss

of sight.

5 min.

54.22 II. Cerebral nerve (optic nerve) Disease of the retina can result in impairment of the visual nerve

(optic nerve).

5 min.

56.34 Retina The retina and the yellow spot, the keenest visual area, are af-

fected.

5 min.

56.61 Optical nerve The optic nerve can be impaired. 5 min.

56.62 Yellow spot The yellow spot, the keenest visual area, is affected. 5 min.

57.41 Dry maculadegeneration With dry maculadegeneration, medium loss of visual acuity oc-

curs during the degeneration of the retina epithelium.

5 min.

72.00 Psyche Emotionally trying situations can intensify the symptoms of the

illness.

5 min.



5 min.






160

57.41 Dry maculadegenerationProgram No. / description Explanation Time

22.15 Herpes simplex From an energetic point of view, these pathogens require parti-


5 min.












5 min.




2 min.

Own notice of 57.41 Dry maculadegeneration

161

59.10 TinnitusProgram No. / description Explanation Time




tensor.

0 min.







illness.

5 min.



5 min.





38.10 Arteries Arteries have the job of supplying organs and tissue with oxygen.

Undersupply of oxygen can result in dysfunctions and organic

diseases.

5 min.

39.10 Circulatory problem, arterial Arterial circulatory problems result in an undersupply of oxygen

which manifests itself particularly in organs and tissues supplied

by small arteries.

5 min.

39.60 High blood pressure (hypertension) Over the long term, high blood pressure results in the undersup-

ply of oxygen, especially to the organs, which are supplied by the

small arteries.

5 min.

58.30 Middle ear, total Damage to or diseases of the middle ear can result in tinnitus. 5 min.

58.40 Inner ear, total Circulatory problems or diseases to the inner ear can result in

tinnitus.

5 min.

59.10 Tinnitus Tinnitus 5 min.

72.00 Psyche Emotional strain / illnesses can cause or worsen tinnitus. 5 min.

72.10 Depression Depression can worsen tinnitus or even be triggered by tinnitus. 5 min.

75.10 Stress reduction Stress factors can have a triggering or even worsening effect on

tinnitus.

5 min.


worsen tinnitus.

5 min.

162

59.10 TinnitusProgram No. / description Explanation Time



5 min.















5 min.






2 min.

Own notice of 59.10 Tinnitus

163

63.20 NeurodermatitisProgram No. / description Explanation Time




tensor.

0 min.








illness.

5 min.





5 min.





32.20 Leucocytes total Leucocytes (white blood corpuscles) are responsible for unspeci-

fic and specific defence which is usually impaired by immunolo-

gical regulatory disorders in the case of neurodermatitis. Allergic

reactions are a frequent occurrence.

5 min.

34.00 Immune system physiology, total In the presence of neurodermatitis, the immune system is wea-

kened and, hence, needs to be taken into consideration during

the testing and harmonisation processes.

5 min.

35.20 Allergy, total Patients with neurodermatitis usually have a tendency towards

allergic reactions. In the presence of a linear motion on the

Rayotensor, please carry out differentiated further testing using

an allergen test kit.

5 min.

46.40 Small intestine, total In the presence of neurodermatitis, the flora of the small intestine

is usually imbalanced, and needs strengthening.

5 min.

46.50 Colon, total In the presence of neurodermatitis, the flora of the colon is usu-

ally imbalanced, and needs strengthening.

5 min.

62.10 Skin, total The skin is very dry in the presence of neurodermatitis, and ecze-

ma occurs on various parts of the body.

5 min.

62.20 Skin glands, total Hypofunction of skin glands, the sebaceous and sweat glands,

occurs in the presence of neurodermatitis.

5 min.

62.50 Hair Hair, especially hair on the head, is affected in the presence of

neurodermatitis, and is very dry.

5 min.

164


63.20 Neurodermatitis Neurodermatitis 5 min.

72.00 Psyche Emotional ordeals can intensify and worsen neurodermatitis. 5 min.

75.10 Stress reduction Stress factors can have a triggering or even worsening effect on

neurodermatitis.

5 min.


worsen neurodermatitis.

5 min.



5 min.

20.13 Eikenella corrodens 5 min.







20.42 Actinomyces israelii 5 min.

20.46 Bacillus cereus 5 min.

20.47 Bacteroides fragilis 5 min.


20.70 Lactobacillus acidophilus 5 min.

20.81 Propionibacterium acnes 5 min.

21.12 Erwinia amylovora 5 min.

21.13 Erwinia carotovora 5 min.



21.22 Serratia marcescens 5 min.





22.82 Tobacco mosaic virus 5 min.

23.70 Warts, total 5 min.




mite)

5 min.

25.67 Ornithonyssus (bird mite) 5 min.

25.68 Sarcoptes scabiei (scabies) 5 min.

25.84 Troglodytella abrasseri 5 min.

165


26.05 Fungi I, total From an energetic point of view, these pathogens require parti-


5 min.

27.05 Fungi II, total 5 min.

31.61 Detoxification intestines The detoxification programs opposite should be taken into con-


5 min.


31.63 Detoxification bladder 5 min.

31.65 Detoxification skin 5 min.




2 min.

Own notice of 63.20 Neurodermatitis

166

65.60 Menopause symptomsProgram No. / description Explanation Time




tensor.

0 min.







illness.

5 min.

31.20 ATP production uterus 5 min.

31.22 ATP production ovaries 5 min.



5 min.





52.10 Skeleton, total During the menopause, osteoporosis arises during the course of

the changes to the hormone balance.

5 min.

55.10 Difficulty in falling asleep (21h-23h)

– frequently hormone disorders

Hormone-related difficulties falling asleep are frequent

occurrences.

5 min.

55.20 Difficulty in staying asleep (23-01h

premature wakening)

Various problems with sleeping through the night are further

symptoms of the menopause.

5 min.


premature wakening)



5 min.


premature wakening)



5 min.

64.10 Hypothalamus The hypothalamus regulates, at the first hormone level, the for-

mation of sex hormones, and affects the pituitary gland.

5 min.

64.20 Pituitary gland The anterior lobes of the pituitary gland form hormones which

affect the ovaries.

5 min.

64.80 Ovaries The ovaries produce hormones, oestrogen and progestin (proge-

sterone).

5 min.


lation

Aid of the female hormone balance is important due to the

changes experienced during the menopause.

5 min.

65.30 Hypothalamus The hypothalamus affects the pituitary gland at first hormone

level and affects the production of the sex hormones.

5 min.

65.31 Pituitary gland anterior lobes The anterior lobes of the pituitary glands, on the other hand,

affect the production of hormones in the ovaries.

5 min.

65.60 Menopause symptoms Menopause, menopause syndrome 5 min.

167

65.60 Menopause symptomsProgram No. / description Explanation Time

66.00 Female sexual organs physiology,

total

Due to the change of the hormone balance, the female sexual

organs need to be aided.

5 min.

72.00 Psyche Emotional ordeals worsen the symptoms and, due to the hor-

monal changes in the body, psycho-nervous symptoms such as

irritability, lethargy and sleep disorders frequently occur.

5 min.

75.10 Stress reduction Stress factors increase the symptoms. 5 min.

75.15 Weight reduction Being overweight / adiposity are frequent accompanying

symptoms during the menopause.

5 min.

75.18 Meteorosensitiveness / sferics Increased meteorosensitiveness caused by the hormonal imba-

lance can also occur.

5 min.

75.20 Emotional stress Emotional stress usually worsens the symptoms. 5 min.



5 min.










22.18 Human Papilloma Virus (HPV) 5 min.



31.51 Detoxification blood system The detoxification programs opposite should be taken into con-


5 min.

31.64 Detoxification female / female-

specific

5 min.




2 min.

Own notice of 65.60 Menopause symptoms

168

Own notice of 65.60 Menopause symptoms

169

67.30 EndometriosisProgram No. / description Explanation Time




tensor.

0 min.






31.20 ATP production uterus These ATP programs are to be taken into consideration for the

illness.

5 min.

31.22 ATP production ovaries 5 min.



5 min.





31.81 Scarring Especially in the case of chronic endometriosis, distortion and

scarring can occur.

5 min.

34.00 Immune system physiology, total The immune system should be aided and strengthened. 5 min.

36.10 Lymph vessels The uterus mucous membrane can also enter other organs via

the lymph vessels.

5 min.

52.34 Sacrum / coccyx Pain, especially in the sacrum, can occur. 5 min.

53.73 Backache, lumbar vertebrae Pain in the back extends as far as the lumbar vertebrae. 5 min.

64.80 Ovaries In the presence of endometriosis, during menstruation, mucous

membrane tissue divulses from the uterus and, via the Fallopian

tubes, enters the abdominal cavity.

5 min.


lation

The female hormone balance needs to be balanced. 5 min.

65.30 Hypothalamus Hypothalamus regulates, at first level, the release of hormones

stimulated by the ovaries.

5 min.

65.31 Pituitary gland anterior lobes The anterior lobes of the pituitary glands affect the production

of hormones in the ovaries.

5 min.

65.50 Menstruation programs, total Menstrual disorders can occur. 5 min.

66.00 Female sexual organs physiology,

total

Since a divulsion of the mucous membrane of the uterus occurs

in the abdominal cavity or other organs, the female sexual or-

gans should be aided.

5 min.

67.30 Endometriosis Endometriosis 5 min.

72.00 Psyche Emotional ordeals can have a worsening effect. 5 min.

170

67.30 EndometriosisProgram No. / description Explanation Time





5 min.










22.18 Human Papilloma Virus (HPV) 5 min.



31.51 Detoxification blood system The detoxification programs opposite should be taken into con-


5 min.



fic

5 min.




2 min.

Own notice of 67.30 Endometriosis

171

Notes

172

Notes

173

Notes

174

Notes

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special edition impuls magazine

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