sensory examination sensory lesions

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15/04/2021 1 Neurophysiology I Module Physiology Lab 1 Presented by: Dr.Shaimaa Nasr Amin Associate Professor of Medical Physiology Sensory Examination & Sensory Lesions Sensory Examination 1 2

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Page 1: Sensory Examination Sensory Lesions

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Neurophysiology I ModulePhysiology Lab 1

Presented by:

Dr.Shaimaa Nasr Amin

Associate Professor of Medical Physiology

Sensory Examination&

Sensory Lesions

Sensory Examination

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Dr.Shaimaa N.Amin, CNS 2021

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Positive Romberg’s sign

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EXAMINATION OF SPINOTHALAMICSENSATIONS

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MAJOR SENSORY ABNORMALITIES:

Hyposthesia, Anaesthesia: reduced or absent touch sensation.

Hyperesthesia: lowered threshold to tactile stimuli.

Parasthesia: tingling or numbness sensation (pins and needles).

Hypo`algesia, Analgesia: reduced or absent pain sensation.

Hyperalgesia: exaggerated response to painful stimuli.

a- Primary hyperalgesia: occurs in the injured area and is caused by

sensitization of local nociceptors by inflammatory mediators, e.g. in sunburned

skin.

b- Secondary hyperalgesia: occurs in uninjured area due to facilitation of sensory

transmission, e.g. in thalamic syndrome.

Allodynia: exaggerated pain response to non-painful stimuli, e.g. light touch.

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SENSORY LESIONS

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Peripheral nerve lesions

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Spinal cord lesions

Herpes zoster

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Herpes Zoster:

-It is a viral infection, in which the herpes virus attacks a dorsal root ganglion (DRG) in patients with history of varicella (chicken pox) infection.

-Virus starts to reproduce causing irritation of pain afferents in the DRG leading to severe pain felt in the dermatomal segment supplied by the infected ganglion.

-The virus also migrates with neuronal cytoplasmic flow towards the peripheral axons to their cutaneous terminals, where it reproduces leading to painful skin rash and vesicular formation.

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Syringomyelia

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Tabes dorsalis

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Sub-acute combined degeneration

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Thank You

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