pressure sores presentation
TRANSCRIPT
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Pressure SoresAndre Sookdar
Class of 2013
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Objectives
• Definition• Epidemiology• Pathogenesis• Risk Factors• Stage & Risk Assessment• Prevention• Management
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Definition
• A Pressure sore is a localized injury to the skin or underlying tissue as a result of unrelieved pressure.
• Decubitus Ulcer, bedsore
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Epidemiology
• Between 1-3 million US affected• 11 - 18% nursing home residents (2004)• 9 - 60% hospital• 3 - 18% home• Health care expenditure $5 Billion US/year• 1.4 – 2.1 Billion pounds (UK)/year• More than 17,000 lawsuits annually• The longer the patient stays in a hospital
or nursing home the greater the risk
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Pathogenesis
Traditional Theory• Prolonged Pressure• Friction• Shearing Forces• Moisture
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Pathogenesis
• Pressure: When external pressures are greater than capillary pressure (12-32mmHg) ischaemia results
• Intermittent pressure relief helps prevent ulcer formation
• Friction: Compromise of the protective stratum corneum decreases the pressure required for ischaemia
• The loss of the skin’s ability to act as a barrier further enhances ulcer formation
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Pathogenesis
• Shearing Forces: When the patient is raised at an angle > 30˚, shearing forces occur between the deep fascia and the outer skin
• Moisture: Chronic moist environment (incontinence, perspiration) leads to tissue damage and ulcer formation
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Pathogenesis
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Pathogenesis
• Impairment in lymphathic flow increase in metabolic waste products
• Reperfusion injury• Deformation of tissue cells
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Common Sites
• Commonly occurs at bony prominences, e.g. sacrum, greater trochanters, heels, ischial tuberosities
• 95% occur on the caudal aspect of the body; 65% in the pelvic area, 30% on the lower limbs
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Intrinsic Risk Factors
Limited Mobility• Spinal cord injury• CVA• Parkinson Disease• Alzheimer Disease• Pain• Fractures• Postsurgical
• Coma or sedation• Arthropathy
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Intrinsic Risk Factors
Poor Nutrition• Anorexia• Dehydration• Poor dentition• Poverty or lack of access to food• Dietary Restriction
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Intrinsic Risk Factors
Co-morbidities• Diabetes• Depression• Peripheral Vascular
Disease• Decreased pain
sensation• Immunodeficiency• Corticosteroid
effects• Congestive heart
failure• Malignancies• Renal Disease• COPD• Dementia
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Intrinsic Risk Factors
Aging skin• Loss of elasticity• Decreased cutaneous blood flow• Changes in dermal pH• Flattening of rete ridges• Loss of subcutaneous fat• Decreased dermal-epidermal blood
flow
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Extrinsic Risk Factors
• Pressure from external surface e.g. bed, chair
• Friction from being unable to move well• Shear forces form involuntary
movement• Moisture – bowel or bladder
incontinence, perspiration, wound drainage
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National Pressure Ulcer Advisory Panel Pressure Ulcer Staging Classification
• Stage 1 – Intact skin with non-blanchable redness of a localized area, usually over a bony prominence. The area may be painful, firm, soft, warmer or cooler than adjacent tissue.
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National Pressure Ulcer Advisory Panel Pressure Ulcer Staging Classification
• Stage 2 – Partial thickness skin loss, presenting as a shallow open ulcer with a red-pink wound bed without slough. May also present as an intact or open serum-filled blister. Includes tears, tape burns, maceration or excoriation
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National Pressure Ulcer Advisory Panel Pressure Ulcer Staging Classification
• Stage 3 – Full thickness skin loss. Fat may be visible but bone, tendon or muscle tissue are not. Slough may be present.
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National Pressure Ulcer Advisory Panel Pressure Ulcer Staging Classification
• Stage 4 – Full-thickness tissue loss with exposed bone, tendon or muscle. Slough or eschar may be present.
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National Pressure Ulcer Advisory Panel Pressure Ulcer Staging Classification
• Unstageable – Full thickness tissue loss in which the base of the ulcer is covered by slough or eschar. Until enough of the base is exposed, the true depth and stage cannot be determined.
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National Pressure Ulcer Advisory Panel Pressure Ulcer Staging Classification
• Suspected Deep Tissue Injury – Purple or maroon discoloured intact skin or blood-filled blister due to damaged underlying soft tissue from pressure or shearing forces. The area may be painful, firm, mushy, boggy, wormer or cooler than surrounding tissue
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Risk Assessment and Evaluation
• Braden Scale• Push Tool
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Braden Scale
• Sensory Perception 1-4• Moisture 1-4• Physical Activity 1-4• Mobility 1-4• Nutrition 1-4• Friction & Shear 1-3• Score 18+ Low risk• 15-18 Mild risk, 13-14 Moderate risk, 10-
12 High risk, below 10 Very High Risk
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PUSH Tool
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Prevention
Aims• Reduce Pressure and Shearing effects• Reduce Moisture• General Skin Care• Nutrition• Co-morbidities• Involve patient, family, caregivers
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Prevention
• Daily skin inspection• Bathing and skin cleaning frequency• Moisturize skin; avoid hot water or harsh
solutions• Assess and treat incontinence; use topical
barriers or absorbent padding when needed• Proper re-positioning frequently; q2hrly for
those bed-bound, q1hrly for those in wheelchairs; self re-positioning every 15 minutes for those in wheelchairs
• Avoid manipulating bony prominences
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Prevention
• Practice proper positioning, transferring and turning techniques to avoid friction and shearing forces; lift don’t shift
• Use dry lubricants (cornstarch) or protective coverings to reduce friction injury
• Institute a rehabilitation program to maintain or improve mobility/activity status
• Consider nutritional supplementation/support for nutritionally compromised persons
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Prevention
• Use adjunct devices (air mattresses, limb padding) where necessary
• Use pillows or padding to avoid bony prominences such as knees from having direct contact
• Elevate the head of the bed no more than 30˚ unless absolutely necessary
• Monitor and document interventions and outcomes
• Have a fixed repositioning schedule
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How might the Leg Ulcer and Thumb Bruises be related?
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Management
• Based on Staging and Investigation• Wound swabs and cultures usually show
mixed growth• Blood – CBC, CRP, ESR, Serum
Protein/Albumin• MRI• X-Rays• Ultrasound• Tissue Biopsy – suspect malignancy
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Management - AAFP
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Dressings
Dressing Type Description Indication Brand Names
Transparent Film Adhesive, semi-permeable, allows vaporization
Stage I and II with light or no exudates
Opsite, Tegaderm
Hydrogel Water/Glycerin based gels on gauze or dressings
Stage II, III, IV; deep ulcers; necrosis & slough
Acryderm, Flexigel, Intrasite
Alginate From Seaweed Stage III, IV with moderate to heavy exudate
Algicell, Algisite, Tegagen
Foam Moist, thermal Insulation
Stage II to IV with varying drainage
Hydrocell, Polyderm
Hydrocolloid Occlusive or semiocclusive; gelatin and pectin
Stage II to IV with sough and necrosis
Dermafilm, Tegaderm
Moistened Gauze Gauze in saline Stage III to IV
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Management
• Clean, barrier• Antibiotic where appropriate• Debride necrotic tissue
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Complications
• Sepsis, cellulitis, endocarditis, meningitis• Fistula formation• Osteomyelitis, septic arthritis• Sinus tracts• Squamous Cell Carcinoma (Marjolin’s
ulcer)• Amyloidosis• Drug resistant bacteria• Maggot infestation
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Conclusion
• Risk • Prevention• Identify early• Manage
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The End
Thank You
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References
• www.aafp.org• Sussman C, Bates-jensen B. Wound Care:
A Collaborative Practice Manual for Health Professionals 4th Ed Lippincott Williams & Wilkins 2012
• Falabella A, Kirsner R.S. Wound Healing Taylor & Francis Group 2005
• Ruiz J.G. Pressure Ulcers University of Miami Grand Rounds Presentation