ocular emergency
TRANSCRIPT
Ocular Emergencies
Pisit Preechawat, MD
Department of Ophthalmology, Ramathibodi Hospital
Ocular AnatomyOcular Anatomy
1. Frontal bone
2. Zygomatic bone
3. Maxillary bone
4. Sphenoid bone
5. Ethmoid bone
6. Lacrimal bone
7. Palatine bone
1
2 3
4 5
67
Bony Components of OrbitBony Components of Orbit
Size 30 x 40 x 45 mm Size 30 x 40 x 45 mm
Paranasal SinusParanasal Sinus
Ocular AnatomyOcular Anatomy
Orbicularis Oculi
Ocular AnatomyOcular Anatomy
Ocular AnatomyOcular Anatomy
Ocular AnatomyOcular Anatomy
Extraocular MusclesExtraocular Muscles
Optic NerveOptic Nerve
Venous SystemVenous System
Ocular EmergenciesOcular Emergencies
TraumaTrauma
Non - traumaNon - trauma
Blunt traumaBlunt trauma
Penetrating traumaPenetrating trauma
Retinal arterial Perforation Orbital cellulitis occlusion Ruptured Orbital injury Chemical burns Acute glaucoma Corneal ulcer
Sudden congestion Corneal abrasion proptosis Hyphema
Intraocular FB Retinal detachment Macular edema
( Immediately ) ( Within a few hours ) ( Within one day )
Acute Eye ConditionsAcute Eye Conditions
EmergencyEmergency Very UrgentVery Urgent UrgentUrgent
Ocular condiitons requiring immediate
treatment
Acute Angle-Closure Glaucoma
Central Retinal Artery Occlusion
Orbital Cellulitis
Cavernous Sinus Thrombosis
Endophthalmitis
Retinal Detachment
Toxic Causes of blindness
Nontraumatic Ocular Emergencies
Acute Dacryocystitis
Acute Dacryoadenitis
Acute Hordeolum
Preseptal cellulitis
Spontaneous subconjunctival hemorrhage
Conjunctivitis
Bacterial corneal ulcer
Viral keratoconjunctivitis
Acute hydrops of the cornea
Hyphema
Uveitis ( iritis & iridocyclitis )
Vitreous hemorrhage
Retinal hemorrhage
Central retinal vein occlusion
Optic neuritis
Ocular EmergenciesOcular Emergencies
Ocular burns and trauma
Ocular Burn
Alkali Burns
Acid Burns
Thermal Burns
Burns Due to Ultraviolet Radiation
Mechanical Trauma to the Eye
Penetrating or Perforating injuries
Blunt Trauma to the Eye, Adnexa,& Orbit
1. Ecchymosis of the Eyelids
2. Lacerations of the Eyelids
3. Orbital hemorrhage
4. Fracture of the Ethmoid bone
5. Blowout Fractures of the Floor of the Orbit
6. Corneal Abrasions
7. Corneal & Conjunctival Foreign Bodies
Ocular EmergenciesOcular Emergencies
Eye ExaminationEye Examination
Visual acuity Visual acuity
External Eye : orbit, periorbital skin, eyelidsExternal Eye : orbit, periorbital skin, eyelids
Confrontation visual fieldsConfrontation visual fields
Ocular motilityOcular motility
Anterior SegmentAnterior Segment
ConjunctivaConjunctiva
CorneaCornea
Anterior chamberAnterior chamber
Iris Iris
LensLens
Pupils : RAPDPupils : RAPD
Eye ExaminationEye Examination
A dilated pupil makes it easier to see the optic A dilated pupil makes it easier to see the optic nerve, macula, and retinanerve, macula, and retina
- 1% tropicamide ( Mydriacyl )- 1% tropicamide ( Mydriacyl )
- 2.5% phenylephrine ( Neo-Synephrine )- 2.5% phenylephrine ( Neo-Synephrine )
PanOptic Ophthalmoscope
Indirect Ophthalmoscope
Fundus ExaminationFundus Examination
Digital palpationDigital palpation
Schiotz tonometerSchiotz tonometer
Intraocular Pressure MeasurementIntraocular Pressure Measurement
Ocular TraumaOcular Trauma
Closed Globe Open Globe
Burn
Contusion
Laceration Laceration
Penetrating Perforating
Rupture
CausesCauses
• Trauma, HypertensionTrauma, Hypertension
• Valsava pressure spikesValsava pressure spikes
• SpontaneousSpontaneous
No treatmentNo treatment
Resolve within 2 weeksResolve within 2 weeks
Subconjunctival HemorrhageSubconjunctival Hemorrhage
Pain , photophobia , FB sensation, tearing
Conjunctival injection, swollen eyelid
Epithelial staining defect with fluorescein
Corneal AbrasionCorneal Abrasion
Topical cycloplegia, ATB ointment Pressure patching for 24 hours
Searching for conjunctival foreign body
Don’t apply PP if there is a significant risk of infection.
Corneal AbrasionCorneal Abrasion : Management: Management
Corneal UlcerCorneal Ulcer
Hypopyon
Eye Shield
No patching
Topical antibiotics
Ophthalmologist referral
Conjunctival Foreign BodiesConjunctival Foreign Bodies
Corneal foreign body with rust ring
Rust ring
Corneal Foreign BodiesCorneal Foreign Bodies
Remove the FB under the best magnification
Evert the eyelid to rule out additional FB
Treat resulting corneal abrasion
Referral to ophthalmologist, next day
Residual rust ring
Corneal Foreign BodiesCorneal Foreign Bodies
Corneal Foreign Body RemovalCorneal Foreign Body Removal
Disruption of blood vessels in the iris or ciliary body Blood in anterior chamber
Traumatic HyphemaTraumatic Hyphema
Grade Grade Size of HyphemaSize of Hyphema
00 No layered bloodNo layered bloodcirculating red blood cells onlycirculating red blood cells only
I I Less thanLess than 1/3 1/3
II II 1/31/3 to to 1/21/2
III III 1/21/2 to less than total to less than total
IV IV Total Total
Traumatic Hyphema : ClassificationTraumatic Hyphema : Classification
Traumatic HyphemaTraumatic Hyphema
Elevate the patient’s head
Bed rest
1% atropine one drop 3-4 times daily
1% prednisolone acetate one drop 3-4 times daily
If the globe is intact, measure IOP
Reduce IOP
Ophthalmology consult
Traumatic Hyphema : ManagementTraumatic Hyphema : Management
Rebleeding can occur 3 to 5 days later in 30%
Uncontrolled glaucoma or blood stained cornea
requires anterior chamber “wash out”
Traumatic Hyphema : ManagementTraumatic Hyphema : Management
Sharp or blunt traumaSharp or blunt trauma
R/O associated ocular injuryR/O associated ocular injury
Remove superficial FBRemove superficial FB
Rule out deeper FBRule out deeper FB
Give tetanus prophylaxis Give tetanus prophylaxis
Lid LacerationsLid Lacerations
Tear lid margin
Full Thickness Lid LacerationsFull Thickness Lid Lacerations
- Gray line
- Lash line
- Mucocutaneous junction
Laceration of lower eyelid margin Post-operative result following a primary repair
Lid Margin RepairLid Margin Repair
Refer to ophthalmologist if there are Refer to ophthalmologist if there are associated ocular injuriesassociated ocular injuries
Lid LacerationsLid Lacerations
Ruptured globeRuptured globe
Lacrimal drainage systemLacrimal drainage system
Levator aponeurosisLevator aponeurosis
Medial canthal tendonMedial canthal tendon
Tissue loss ( > 1/3 )Tissue loss ( > 1/3 )
Lid LacerationsLid Lacerations with tear canaliculiwith tear canaliculi
Canalicular RepairCanalicular Repair
Tear Canthal TendonTear Canthal Tendon
Woman with tearing and medial canthal Woman with tearing and medial canthal asymmetry after the repair of a laceration asymmetry after the repair of a laceration sustained during a domestic assaultsustained during a domestic assault
Penetrating / Ruptured Globe Penetrating / Ruptured Globe
Corneal or scleral lacerationsCorneal or scleral lacerations
Hypotony (not always present)Hypotony (not always present)
Severe chemosis & hemorrhageSevere chemosis & hemorrhage
Intraocular contents may be outside the globeIntraocular contents may be outside the globe
Limitation of extraocular motilityLimitation of extraocular motility
Shallow anterior chamberShallow anterior chamber
Irregular pupilIrregular pupil
Irregular pupilIrregular pupil
Penetrating / Ruptured Globe Penetrating / Ruptured Globe
Ruptured globe caused by golf ball
Penetrating / Ruptured Globe Penetrating / Ruptured Globe
Penetrating / Ruptured Globe : Management Penetrating / Ruptured Globe : Management
Stop examinationStop examination
Shield the eye (do not patch)Shield the eye (do not patch)
Give tetanus prophylaxisGive tetanus prophylaxis
NPO and systemic antibioticsNPO and systemic antibiotics
Do not apply eye ointment or eye dropDo not apply eye ointment or eye drop
Film orbit if IOFB can’t be R/OFilm orbit if IOFB can’t be R/O
Refer immediately to ophthalmologistRefer immediately to ophthalmologist
Intraocular or Intraorbital Foreign BodiesIntraocular or Intraorbital Foreign Bodies
Ocular TraumaOcular Trauma
Traumatic cataract
Traumatic mydriasis Traumatic lens subluxation
Traumatic lens subluxation
True ocular emergency
Both acid and alkali burns can be blinding
- Acid burns tend to coagulate proteins, limiting
the depth of penetration.
- Alkali burns can rapidly penetrate the cornea,
causing damage to intraocular structures.
Chemical Ocular InjuryChemical Ocular Injury
Immediate copious irrigation with a minimum of
1-2 L of saline or until pH is normalized ( 7.3-7.7 )
- Instill a topical anesthetic
- Use eyelid retractor
- Double eversion of the eyelids
Chemical Ocular Injury : ManagementChemical Ocular Injury : Management
Irrigation in case of chemical injuryIrrigation in case of chemical injury
Immediate copious irrigation with a minimum of
1-2 L of saline or until pH is normalized ( 7.3-7.7 )
- Instill a topical anesthetic
- Use eyelid retractor
- Double eversion of the eyelids
Chemical Ocular Injury : ManagementChemical Ocular Injury : Management
Ophthalmologists Referral
No corneal involvement
- ATB + steroid eye drop
Chemical Ocular Injury : Classification Chemical Ocular Injury : Classification
Grade I Grade II
Grade III Grade IV
Chemical Ocular Injury : Management Chemical Ocular Injury : Management
Preservative-free artificial tears
Topical non-preserved steroid
Topical cycloplegic
Topical antibiotics
Oral analgesics
Pressure patch or bandage CL
Antiglaucoma +
Bilateral Alkali Injuries
Chemical Ocular Injury Chemical Ocular Injury
Chemical Ocular Injury : Management Chemical Ocular Injury : Management
Corneal Transplantation
Keratoprosthesis
Accidental into the eye can cause the lids to
adhere and adhesive clumps to form on the cornea
Not permanently harmful to the eye
Cyanoacrylates are used occasionally directly on the
cornea to seal corneal perforations.
Cyanoacrylate GlueCyanoacrylate Glue
Moisten the glue with eye ointment, and remove
as much as can be removed easily without causing
damage to underlying tissue
The glue will loosen and become easier to remove
in a few days.
Cyanoacrylate GlueCyanoacrylate Glue
Non-traumatic Ocular EmergenciesNon-traumatic Ocular Emergencies
The woman suddenly experienced nausea, vomiting, and extreme The woman suddenly experienced nausea, vomiting, and extreme pain in the left eye while in a movie theater. Her vision has pain in the left eye while in a movie theater. Her vision has worsened since that time and the eye has become very red. worsened since that time and the eye has become very red.
A 55-year-old woman with a red eye, blurred A 55-year-old woman with a red eye, blurred vision with halos, nausea, and vomitingvision with halos, nausea, and vomiting
VA - HMVA - HM
Conjunctival injectionConjunctival injection
Hazy corneaHazy cornea
Shallow anterior chamberShallow anterior chamber
Fixed mid-dilated pupil Fixed mid-dilated pupil
A 55-year-old woman with a red eye, blurred A 55-year-old woman with a red eye, blurred vision with halos, nausea, and vomitingvision with halos, nausea, and vomiting
Acute Angle Closure GlaucomaAcute Angle Closure Glaucoma
IOP 56 mmHgIOP 56 mmHg
Anterior Chamber DepthAnterior Chamber Depth
Reduce the intraocular pressureReduce the intraocular pressure
O.5% Timolol 1 drop O.5% Timolol 1 drop
2-4 % Pilocarpine 1 drop every 15 minutes2-4 % Pilocarpine 1 drop every 15 minutes
20% Mannitol 250-500 ml IV drip20% Mannitol 250-500 ml IV drip
Acetazolamide 500 mg oral Acetazolamide 500 mg oral
100% Glycerin 1 cc/kg 100% Glycerin 1 cc/kg
Consult ophthalmologistConsult ophthalmologist
Acute Angle Closure GlaucomaAcute Angle Closure Glaucoma
A 60-year-old woman with acute, painless loss A 60-year-old woman with acute, painless loss of vision in the right eyeof vision in the right eye
Visual acuity CF – LPVisual acuity CF – LP in 90% of casesin 90% of cases
Opaque white retina and attenuated vesselsOpaque white retina and attenuated vessels
Central Retinal Artery OcclusionCentral Retinal Artery Occlusion
Treatment must be initiated immediately.Treatment must be initiated immediately.
Ocular massageOcular massage
Inhaled carbogen ( 95% O2 and 5% CO2 )Inhaled carbogen ( 95% O2 and 5% CO2 )
Reduced intraocular pressureReduced intraocular pressure
Central Retinal Artery OcclusionCentral Retinal Artery Occlusion
Consult ophthalmologist immediately Consult ophthalmologist immediately
Anterior chamber paracentesisAnterior chamber paracentesis
Direct infusion of t-PA or urokinase in the Direct infusion of t-PA or urokinase in the
ophthalmic artery ophthalmic artery
A 40-year-old man with left eyelid edema and painA 40-year-old man with left eyelid edema and pain ( worse on eye movement )( worse on eye movement )
A 40-year-old man with left eyelid edema and painA 40-year-old man with left eyelid edema and pain ( worse on eye movement )( worse on eye movement )
Periorbital erythema and edema
Proptosis
Restricted extraocular motility
Decreased visual acuity
Chemosis
Fever
Orbital CellulitisOrbital Cellulitis
Broad spectrum intravenous antibioticsBroad spectrum intravenous antibiotics
CT scan orbitCT scan orbit
Ophthalmology & ENT consultation Ophthalmology & ENT consultation
Orbital CellulitisOrbital Cellulitis
Subperiosteal abscess
Preseptal CellulitisPreseptal Cellulitis
EndophthalmitisEndophthalmitis
Urgent Neuro-ophthalmologyUrgent Neuro-ophthalmology
A 36-year-old-woman with subacute visual loss in right eye and pain on eye movement
VA 20/200, 20/25 RAPD +ve OD
VF central scotoma OD
Retrobulbar optic neuritis
A 55-year-old man with HT and acute visual loss in RE
VA 20/100, 20/20 RAPD +ve RE
Nonarteritic anterior ischemic optic neuropathy
ESR 10 mm/hr
A 73-year-old woman with acute visual loss of right eye, headache, anorexia and weight loss
VA 10/200, 20/25 RAPD + ve RE
ESR 94 mm/hr, high level of C - reactive protein
Arteritic anterior ischemic optic neuropathy
Pathology : Giant Cell ( Temporal ) Arteritis
A 35-year-old man with left painful third nerve palsy
VA 20/25, 20/30
Dilated, nonreactive pupil LE
A 35-year-old man with a suspicious of aneurysmal third nerve palsy
Conventional CT scan or MRI are not the procedure of choice
High false negative rate 12 – 40 %
Magnetic resonance angiography (MRA)
Computed tomography angiography (CTA)
Overall sensitivity up to 97 %
A 35-year-old man with a suspicious of aneurysmal third nerve palsy
A 40-year-old woman with sudden onset of left third nerve palsy, visual loss and severe headache
What is the diagnosis?
VA 20/30, LP +ve RAPD LE
Pituitary Apoplexy
Characterized by sudden visual loss, headache, and ophthalmoplegia secondary to rapid expansion of pituitary macroadenoma into the suprasellar space and/or cavernous sinus
Commonly results from hemorrhage into a pre-existing pituitary mass
A 17-year-old man with right blured vision after minor blunt trauma.
VA 20/32, 20/20 + ve RAPD RE
Normal fundi
RELE
A 16-year-old man with head injury and left blured vision after falls from height
VA 20/30, LP + ve RAPD LE
Normal fundi
Traumatic Optic Neuropathy :
Classification and Mechanisms
Direct injury
- Penetrating injury from knife, projectile
- Injury from fractured bone
- Avulsion, transection
Indirect injury
- Contusion with transmission of force through bone
- Compression secondary to orbital hemorrhage or
intrasheath hemorrhage
Clinical Features of Traumatic Optic Neuropathy
Most commonly unilateral
May be overlooked in setting of significant
globe or maxillofacial trauma
Reduced visual acuity ( NLP to 20/20 )
Visual field defect : No pathognomonic defect
Normal optic disc with development of optic atrophy
Medical Management Options
Steroids : Controversial
- Thought to limit free-radical amplification
of the injury response
- Dosages ( low, high, mega)
- May be harmful
Observation : 57% of untreated patients shown to have 3 lines or more acuity improvement
Surgical Management Options
Lateral canthotomy and cantholysis for orbital hemorrhage
Surgical decompression of the optic nerve within its canal
There is no defined standard protocol of
treatment for indirect optic nerve injury .