neurovascular cases in nsv

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    NEUROVASCULAR CASES

    IN NSVProf.Subbiah MCh

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    CASE 1

    Harikrishnan 26 M

    C/O Benumbed sensation of entire Rt

    UL for 1-2 mts and recovery . 1-2episodes a week in last 3 months.

    O/E HMF,Cr Nerves,SMS,Cerebellum-

    Normal Pt came with CT Angio which shows

    AVM 1.8X1 cm Lt Frontoparietal

    region.Feeding from MCA.Drains intoSu r sa ittal sinus.

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    CASE 1

    Spletzer Martin grading:

    Size:1 Eloquency:1 Draining

    Vein:0Score : 2

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    CASE 1

    Preop pictureshowing the

    Ltfrontoparietalregion AVM

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    CASE 1

    Preop picture showing the Ltfrontoparietal region AVM

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    CASE 1

    Pt undergone Lt frontoparietalcraniotomy and excision of AVM.

    Postoperatively pt had Dysphasiaand Rt UL monoparesis whichrecovered gradually.

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    CASE 1

    Postop picture showing theexcision of the AVM

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    CASE 2

    Surya 10 MCh

    C/O Severe headache and vomiting

    for which he was admitted in a pvthospital.Pt referred to GGH with CTBrain.

    O/E HMF,Cr Nerves,SMS,Cerebellum-Normal

    CT Brain shows ICH in Rt Temporal

    region with thin SDH with mass effectand minimal midline shift

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    CASE 2

    Pt initially managed conservativelyfor ICH and evaluated.

    CT Angio shows 1X1 cm AVM in RtTemporal lobe withhemorrhage.Feeding Artery RtMCA.Draining vein Superficial

    cortical vein. Spletzer Martin grading:

    Size:1 Eloquency:1 DrainingVein:0

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    CASE 2

    Preop picture showingthe temporal ICH

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    CASE 2

    Preop picture showingthe Rt temporal AVM

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    CASE 2

    Pt undergone Rt temporalcraniotomy and excision of AVM.

    Postoperatively pt had noneurological deficit but had CSFcollection underneath the flap whichwas managed by LP drain.

    Pt improved and discharged.

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    CASE 2

    Postop picture showing theexcision of Rt temporal AVM

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    CASE 3

    Dhanalakshmi 54 F

    C/O Giddiness and fall followed by

    severe headache, vomiting andaltered sensorium.

    Not a known HTN,DM or IHD.

    O/E Drowsy,obeys simplecommands,Lt 6th nervepalsy,PERL,no weakness of limbs.

    CT shows SAH in both the sylvian

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    CASE 3

    Pt conservatively managed for SAHand evaluated.

    CT Angio shows Acom Aneurysm

    Spletzer Martin grading:

    Size:1 Eloquency:1 Draining

    Vein:0Score : 2

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    CASE 3

    Preop picture showing theSAH

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    CASE 3

    Preop picture showing the RtAcom Aneurysm

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    CASE 3

    Preop picture showing the RtAcom Aneurysm

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    CASE 3

    Pt undergone Rt pterional craniotomyand clipping of Acom aneurysm.

    Postoperatively pt had no significantneurological deficit.

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    CASE 3

    Postop picture after theclipping of Rt Acom Aneurysm

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    CASE 4

    Masthan 23 M

    Alleged H/O RTA 2 wheeler vs 2wheeler and patient admitted in

    Trauma ward.

    O/E E3V2M5 PERL no weakness oflimbs.

    CT shows Lt temporoparietal ICH withmass effect . Rt temporal small EDH.

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    CASE 4

    Pt undergone Emergency LtTemporoparietal craniotomy andevacuation of ICH.

    Pt improved postoperatively andevaluated for ICH.

    CT angio shows posttraumaticaneurysm of size 6.3X8 mm from Rtmiddle meningeal artery.

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    CASE 4

    Preop picture showing Lttemporal and parietal ICH

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    CASE 4

    Postop picture showingevacuation of ICH

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    CASE 4

    Picture showing Rt temporalMiddle meningeal arteryaneurysm

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    CASE 4

    Picture showing Rt temporalMiddle meningeal arteryaneurysm

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    CASE 4

    Picture showing Rt temporalMiddle meningeal arteryaneurysm

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    CASE 4

    Pt undergone Rt temporalcraniotomy and clipping andcauterization of aneurysm.

    Postoperatively pt had no significantneurological deficit.

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    DISCUSSION

    Traumatic intracranial aneurysmsrarely occur and can develop as theresult of either blunt or penetrating

    head trauma. Traumatic intracranialaneurysms can present in a varietyof ways such as subarachnoid

    hemorrhage, intracranialhemorrhage, and subduralhematoma.

    The mortality rate for patients with

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    DISCUSSION

    Traumatic aneurysms comprise lessthan 1% of all intracranialaneurysms. Most of these aneurysms

    are actually false aneurysms,orpseudoaneurysms, which are causedby the rupture of entire vessel wall

    layers, with the wall of the aneurysmbeing formed by thesurroundingcerebral structures.

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    DISCUSSION

    The natural history of traumaticaneurysms is not well known, butprogressive growth of traumatic

    aneurysms has been demonstratedon repeated angiograms .

    Traumatic MMA may regress,thrombose, enlarge, or rupture.

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    DISCUSSION

    Therefore, preventive therapy isrequired for this vascular lesions .

    Because rupture of a

    pseudoaneurysm of the middlemeningeal artery can be lethal, likethis case, we emphasize earlydiagnosis and early preventivetreatment.

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    THANK YOU