lecture 4 (part 2) discoloration of teeth (slide)
TRANSCRIPT
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8/3/2019 lecture 4 (part 2) Discoloration of Teeth (Slide)
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Discoloration of TeethDr. Rima Safadi
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Causes of Discoloration
Extrinsic staining
Changes in tooth structure
Diffusion of pigments after tooth formation
during tooth formation
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Extrinsic staining Adsorption on tooth surface
Food, drinks, tobacco, mouth rinses
Bacteria: green and black pigments
Chromogenic bacteria
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Changes in tooth structure Amelogenesis and dentinogenesis
imperfecta
White spot caries
Enamel hypoplasia
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Dentinogenesis imperfecta
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Diffusion of pigments after tooth
formation
From food and tobacco into dentinexposed by caries or tooth wear
From restorative and root filling materialand corrosion products
Pulp necrosis: lysis of necrotic tissuediffuses in dentine
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Pink Tooth
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Incorporation of pigments duringtooth formation
In congenital disorders:Neonatal jaundice (congenital
hyperbilirubineamia)
Deposition of bile pigments in calcifyingenamel and dentine
Mainly in dentine
Green to yellow brown
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Heamolytic anemia teeth
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Congenital Porphyria:
AR error in porphyrin metabolism
Escretion of porphyrin pigments (red) in urineand blood
Pink brown discoloration
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Porpheria teeth
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Extrinsic stain
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Tetracycline pigmentation
Deposition in dental hard tissue (enamel,dentine, cementum) and bone More in dentine
Yellow bands Flouresce bright yellow under UV light
Yellow then darken with light
Severity depends on dose, age at time of
adminstration Cross placenta
Should not be given from 29 weeks-fullterm
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Tetracyclin stain
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Transplantation and Reimplantation
of Teeth
Transplanatation:
From one site to another
Extraction site or surgically prepared socket
Autotransplantation vs allografting(between individuals)
Autotransplant: no immune response
Traumatic severness of blood supply
In open apex: revascularization occur (>1mm foramenwidth)
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Root resorption is most commoncomplication
Rapid or slow (10-15 years beforeexfoliation)
Pain is not a feature
Early acute inflammation leading to root
resorption then chronic inflammation RL bone area
Bony infilling: long term replacement resorption
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Prevention of root resorption: RCT within 4weeks
Reimplanted teeth: worse prognosis than
transplanted
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Root Fracture
Prognosis depends on presence and absence ofinfection Pulp vitality
Position of fragments
Mobility of coronal fragment 3 healing patterns if the fracture is sterile:
1. united totally by a tissue resembling bone or cementum
2. May be rounded off by cementum but not united bycalcified tissue
3. Rounded and coated by cementum but fragments arewidely seperated
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Age changes:
Enamel: brittle, less permeable and darker
Dentine: Formation of secondary dentine:reduced or obliterated pulp chamber
Associated with caries and tooth waer
Cementum: hypercementosis
Compensate for tooth substance loss