indian psychiatric society western zonal branch indian psychiatry · 2012. 6. 15. · dattatrey...
TRANSCRIPT
Editorial
5HTT Gene-environment interactions in human behaviour
Chittaranjan Andade 4
Review Article
Night Eating Syndome Ameya Amritwar, Nilesh Shah
5
Developmental Psychopathology an d its relevance to Child Psychiatry – a Clinical Perspective
Avinash deSouza8
Original Articles
16
Traditional Healing practices inPsychiatric Out Patients
Rahul ShidhayeG .K .Vankar
28
Newspaper Portrayal of Psychiatry Laxeshkumar PatelG .K .Vankar
31
E! cacy of Social Skills Training as Intervention for Stuttering for adolescents v /s adults
Sadhana DeshmukhAnuradha Sovani
37
Stress and Coping among Resident Doctors Sohang BhadaniaMinakshi Parikh, G .K .Vankar
44
Who cares for caregivers?: A ReviewPooja DangarParag Shah
50
Case Reports
Familial Hypokalemic Periodic Paralysis : A Case Report
Rajat Oswal,Devendra Chaudhari,Ritambhara Mehta
61
Quiz Bhavesh Lakdawala 64
Archives ofIndian Psychiatry
O! cial Publication ofIndian Psychiatric SocietyWestern Zonal BranchVolume 13, No.2, October 2011
Contents
O! cial Publication of Indian Psychiatric Society, Western Zonal BranchVolume 14, No.1, April 2012
Kaushik GuptePresident
Vivek KirpekarHon. Secretary
Vivek Kirpekar Vice President
Jay ShastriTreasurer
Bansi SuwalkaImm. Past President
Govind BangKishor GujarRepresentatives of
Central Council
Kausar AbbasiNeena SawantKailash JhalaniP.M.ChuguleRajendra HegdeRavindra KamathExecutive Council Mambers
Archives ofIndian Psychiatry
Chairpersons Subcommittees
Dattatrey DhavleAwards Committee
Sudhir BhaveCME
Mukesh JaggiwalaConference
S.M.AminConstitution
Sandip DeshpandeLegal
Deepak RathodMembership
Sandesh Patil Mental Health Awareness
Kamlesh Dave Psychiatric Education
Malay Dave Quiz
Rahul Tadke Accreditation
Abhishek Somani Website
Archives ofIndian Psychiatry
O! cial Publication of Indian Psychiatric Society, Western Zonal Branch
Volume 13, No.2, October 2011
Honorary Editor
G. K. VankarProfessor and HeadDept. of PsychiatryB.J. Medical College andCivil Hospital,Ahmedabad-380016.Cell. : 09904160338
Rahul ShidhayeStatistics ConsultantRajat OswalWebsite Development
Editorial O! ce
Dept. of Psychiatry,OPDBuilding,Wing 1I FloorCivil Hospital,Ahmedabd-380 016.E-mail : [email protected]: http://archivesindianpsychiatry.wordpress.comhttp://www.archindianpsychiatry.org
Phone : 079-65542770Fax : 079-65542770
Type SettingPankit Patel Karnavati Computer
Editorial Board
R. Srinivasa MurthyE. MohhandasVikram PatelNimesh DesaiK. S. JacobNilesh ShahChittaranjan AndradeBharat PanchalShekhar SheshadriAshok Nagpal
Board of Directors Laxman DuttJitendra NanawalaI. R. RajkumarV. G. VatweMukesh JagiwalaRajendra HegdeK. S. AyyarHemangee DhavleGovind BangShubhangi ParkarKaushik Gupte
Corresponding Members
Dinesh BhugraStuart MontgomaryAfzal JavedJoseph JoharPrakash MasandAndre JoubertManoj Shah
Journal Committee
Rashmin CholeraParag ShahRitambhara MehtaKhyati MehtaliaAnuradha SovaniCharles PintoYusuf MatchewalaRjaesh DhumeD.M. DhavleShivarathnammaVivek Kirpekar
Archieves of Indian Psychiatry is the o! cial journal of Indian Psychiatric Society, Western Zonal Branch published twice in a yearSubscitpion :Annual subscription rates are Rs. 700/- for individuals andRs. 1000/- for institutions. Please send DD in favour of Editor, Archives of Indian Psychiatry Payable at Ahmedabad.
Correspondence related to advertisements should be addressed to the editorial o! ce.
Copyright : Indian Psychiatric Society Western Zonal Branch
O cial Publication of Indian Psychiatric Society, Western Zonal BranchVolume 13, No.2, October 2011
Archives ofIndian Psychiatry
Bansi SuwalkaPresident
Vivek KirpekarHon. Secretary
Vivek Kirpekar Vice President
Jay ShastriTreasurer
G.K.Vankar Editor
Shrikant DeshmukhImm. Past President
Mukesh JagiwalaKishor GujarRepresentatives of
Central Council
Lalit VayaMangalam RathodLalit J.ShahPramod " akreDeepak RathodRavindra KamathExecutive Council Mambers
Chairpersons Subcommittees
Dattatrey DhavleAwards Committee
Neena SawantCME
Mukesh JagiwalaConference
Govind BangConstitution
Sanjeev SaojiLegal
Jayita DeodharMembership
Rajesh KumarMental Health Awareness
Henal Shah Psychiatric Education
Kousar AbbasiQuiz
Archives ofIndian Psychiatry
O! cial Publication of Indian Psychiatric Society, Western Zonal Branch
Volume 13, No.2, October 2011
Honorary Editor
G. K. VankarProfessor and HeadDept. of PsychiatryB.J. Medical College andCivil Hospital,Ahmedabad-380016.Cell. : 09904160338
Rahul ShidhayeStatistics ConsultantRajat OswalWebsite Development
Editorial O! ce
Dept. of Psychiatry,OPDBuilding,Wing 1I FloorCivil Hospital,Ahmedabd-380 016.E-mail : [email protected]: http://archivesindianpsychiatry.wordpress.comhttp://www.archindianpsychiatry.org
Phone : 079-65542770Fax : 079-65542770
Editorial Board
R. Srinivasa MurthyE. MohhandasVikram PatelNimesh DesaiK. S. JacobNilesh ShahChittaranjan AndradeBharat PanchalShekhar SheshadriAshok Nagpal
Board of Directors Laxman DuttJitendra NanawalaI. R. RajkumarV. G. VatweMukesh JagiwalaRajendra HegdeK. S. AyyarHemangee DhavleGovind BangShubhangi ParkarKaushik Gupte
Corresponding Members
Dinesh BhugraStuart MontgomaryAfzal JavedJoseph JoharPrakash MasandAndre JoubertManoj Shah
Journal Committee
Rashmin CholeraParag ShahRitambhara MehtaKhyati MehtaliaAnuradha SovaniCharles PintoYusuf MatchewalaRjaesh DhumeD.M. DhavleDevavrat KumarVivek Kirpekar
Archieves of Indian Psychiatry is the o! cial journal of Indian Psychiatric Society, Western Zonal Branch published twice in a yearSubscitpion :Annual subscription rates are Rs. 700/- for individuals andRs. 1000/- for institutions. Please send DD in favour of Editor, Archives of Indian Psychiatry Payable at Ahmedabad.
Correspondence related to advertisements should be addressed to the editorial o! ce.
Copyright : Indian Psychiatric Society Western Zonal Branch
Editorial
5HTT Gene-Environment Interactions In Human Behavior
Chittaranjan Andrade
Serotonin is an important neurotransmitter in psychiatry. Serotonin regulates a number of behaviors, including sleep, appetite, sexual functioning, gastrointestinal functioning, and others. Disturbances of serotonergic neurotransmission have been implicated in diverse behavioral symptoms and disorders, including anxiety, obsessive-compulsive disorder, aggression, depression, suicidality, and others.
! e serotonin transporter protein (5HTT)! e serotonin transporter protein (5HTT) is responsible for the reuptake of serotonin from the synaptic cle" . ! is protein, therefore, in# uences the intensity and duration of 5-HT signalling in the brain. Genetic variations in 5HTT in# uence diverse aspects of mental health. For example, the 5HTT gene, which is located on the short arm of chromosome 17, has been implicated in violent suicidal behavior (Courtet et al, 2001), in anxiety-related personality traits in women (Melke et al, 2001), and in depression subtypes (Willeit et al, 2003) and personality (! ierry et al, 2004) in patients with seasonal a$ ective disorder. A rare mutation of this gene may result in OCD or obsessive-compulsive personality disorder (Ozaki et al, 2003).
! e s/l 5HTT polymorphism
A functional polymorphism in the promoter region of the 5HTT gene has been described. ! is polymorphism is expressed as a short (s) or long (l) allele. ! e short allele is associated with a decrease in the level and activity of the 5HTT protein.
! e pattern of the s/l alleles in the genotype of an individual has been shown to in# uence the individual's behavior. For example, in separate meta-analyses, Lin and Tsai (2004) found that, in psychiatric subjects, the short allele was associated with an increased risk of violent suicide; alcoholic persons were particularly at risk. In a later meta-analysis, Li and He (2007) found that the presence of the ll genotype was associated
with a 22% reduction in the risk of suicide; however, there were gender and ethnic di$ erences in the results.
5HTT gene-environment interaction and
depression
Very interestingly, the pattern of the 5HTT s/l alleles present in the genotype of an individual has also been shown to modulate the in# uence of the environment on mental health. In a pathbreaking study, Caspi et al (2003) showed that the risk of depression in response to environmental stress was directly proportionate to the degree of homozygosity for the short allele of the gene (l/l vs s/l vs s/s). ! at is, persons with the s/s genotype were at highest risk of stress-related depression; those with the s/l genotype were at intermediate risk, and those with the l/l genotype were at lowest risk.
If the s/s genotype is associated with stress-related vulnerability to depression, is the individual doomed to stress-related depression or can the risk be modi& ed? ! e latter appears to be the case: in another important study, Kaufman et al (2004) showed that adequate social support protected maltreated children from the depressogenic e$ ect of the short allele of the gene.
5HTT gene-environment interaction and
substance abuse
In the most recent study on the 5HTT gene-environment interaction, Brody et al (2009) studied 253 African-American children and adolescents in rural Georgia, USA. ! e mean age of these subjects was nearly 12 years. Use of cigarettes, alcohol, cannabis, and other substances was ascertained from the participants through self-report. Mothers provided data on parenting practices.
Given the young age of the sample at recruitment, substance use was expectedly very low at baseline. By age 14, however, 21% of subjects had smoked cigarettes, 42% had used alcohol, 5% reported occasions of heavy
Archives of Indian Psychiatry 13(2) October 20114
drinking, and 5% had used cannabis. ! is increase in substance use was examined in the context of 5HTT polymorphisms and the quality of parenting.
! ere were two important " ndings:
1. Among children who received low levels of involved-supportive parenting, substance use rose three-fold in those with one or two short alleles relative to those with two long alleles.
2. Among children who received high levels of involved-supportive parenting, presence of the short allele did not increase the risk of substance use.
! e study therefore shows that the short allele of the serotonin transporter gene is associated with an increased risk of development of substance use in children and adolescents. Importantly, this risk is neutralized by involved-supportive parenting.
Take-home message
! is series of studies tells us that adverse environmental in# uences are depressogenic or increase the risk of substance abuse in persons with the s allele of the 5-HT transporter gene. However, supportive environmental in# uences bu$ er the risk. ! ese data provide strong support for the importance of social and environmental e$ orts to mitigate genetic risk factors in psychiatry.
Parting note
! e 5HTT gene polymorphism is a good example not only of how a gene may in# uence the experience of certain symptoms but, more dramatically, also of how a gene may in# uence the expression of a speci" c behavior. If the output of a computer is a function of the so& ware which is running, so too is the behavior of a human being a function of the DNA which is expressed. However, the in# uence of the DNA is not invariable, but is modi" ed by constitutional and
environmental factors.
References
Brody GH, Beach SRH, Philibert RA, Chen Y-f, Lei M-K,
Murry VM. Parenting moderates a genetic vulnerability factor
in longitudinal increases in youths' substance use. J Consult Clin
Psychol 2009; 77: 1-11.
Caspi A, Sugden K, Moffi tt TE, Taylor A, Craig IW, Harrington
H et al. Infl uence of life stress on depression: moderation by a
polymorphism in the 5-HTT gene. Science 2003; 301: 386-389.
Courtet P, Baud P, Abbar M, Boulenger JP, Castelnau D,
Mouthon D et al. Association between violent suicidal behavior
and the low activity allele of the serotonin transporter gene. Mol
Psychiatry 2001; 6: 338-341.
Kaufman J, Yang B-Z, Douglas-Palumberi H, Houshyar S,
Lipschitz D, Krystal JH et al. Social supports and serotonin
transporter gene moderate depression in maltreated children.
PNAS 2004; 49: 17316-17321.
Li D, He, L. Meta-analysis supports association between
serotonin transporter (5-HTT) and suicidal behavior. Mol
Psychiatry 2007; 12: 47-54.
Lin P-Y, Tsai G. Association between serotonin transporter gene
promoter polymorphism and suicide: results of a meta-analysis.
Biol Psychiatry 2004; 55: 1023-1030.
Melke J, Landen M, Baghei F, Rosmond R, Holm G, Bjorntorp
P et al. Serotonin transporter gene polymorphisms are associated
with anxiety-related personality traits in women. Am J Med
Genet 2001; 105: 458-463.
Ozaki N, Goldman D, Kaye WH, Plotnicov K, Greenberg BD,
Lappalainen J et al. Serotonin transporter missense mutation
associated with a complex neuropsychiatric phenotype. Mol
Psychiatry 2003; 8: 933-936.
Thierry N, Willeit M, Praschak-Rieder N, Zill P, Hornik K,
Neumeister A et al. Serotonin transporter promoter gene
polymorphic region (5-HTTLPR) and personality in female
patients with seasonal affective disorder and in healthy controls.
Eur Neuropsychopharmacol 2004; 14: 53-58.
Willeit M, Praschak-Rieder N, Neumeister A, Zill P, Leisch F,
Stastny J et al. A polymorphism (5-HTTLPR) in the serotonin
transporter promoter gene is associated with DSM-IV depression
subtypes in seasonal affective disorder. Mol Psychiatry 2003; 8:
942-946.
Chittaranjan Andrade, M.D.Professor and Head, Department of PsychopharmacologyNational Institute of Mental Health and NeurosciencesBangalore 560 029, Indiae-mail: [email protected]; [email protected]
Archives of Indian Psychiatry 13(2) October 2011 5
Overview
Night Eating Syndrome: an overview
Ameya Amritwar
Nilesh Shah
Abstract
A less known eating disorder “Night Eating syndrome” (NES) characterised by morning aphagia,
evening hyperphagia and sleep disturbances for a period of at least three months has been
described. Exact pathophysiology of this disorder remains elusive in spite of behavioural and
Neuro-endocrinological studies and considered to variously to be a sleep disorder, eating disorder
or disorder of circadian rhythm. This disorder is associated with stress reaction, abstinence from
substance or smoking and many other psychiatric manifestations; apart from signifi cant weight gain
and associated metabolic problems. This disorder is thought to exist in signifi cant number of general
population, more than that of some more widely discussed eating disorders like anorexia. A multiple
reported studies have shown high prevalence of this disorder in obese patients and psychiatric patients
in western countries. No such data exists for Indian population. This overview attempts to present
‘Night Eating Syndrome’ and recent developments in it.
Key words : Night eating disorder, etiology. clinical features, diagnosis, management
Introduction
Night Eating Disorder (NES) characterised by lack
of appetite in the morning, overeating at night with
agitation, sleep disturbances (waking up at least
once on most nights of the week to consume food)
lasting for at least for a period of three months
was fi rst reported by Stunkard, in 1955[1,2,3].
Considered variously as sleep disorder, eating
disorder or disorder of circadian rhythm, the exact
pathophysiology remains to be understood. There
may also be associated dysphoria, specially in
evening hours [1,2,3]. This 28 and 29-38years,
respectively. No preponderance of males or
females, any particular diet type, socioeconomic
status, education, type of work or shift duty
timings etc. was noted, though 4 female patients
(50% of patients) were homemakers. No obvious
Mrs. A, a 47 year old patient was diagnosed as Major Depressive Disorder about 5 years back, when
she presented with complaints of pervasive sadness of mood, lethargy, not being able to work, not en-
joying activities she enjoyed earlier; following a stressor at home. Patient weighed about 80 kilograms
with a height of 154 centimetres, some 10-16 kilograms overweight. Patient also had blood pressure
on higher side for which she was advised small dose antihypertensive medication by a physician along
with strict instruction for weight reduction. Over a period of time patient was tried on various antide-
pressants with best control of her symptoms achieved with combination of Mirtazepine (30 m.g.) and
amitriptylene (125 m.g.), on which she is maintained for more than a year.
One major problem remained though, or even worsened; and it was patient’s ever increasing weight.
Within a span of 5 years patient had gained considerably to 112 kilograms. Her antihypertensive medi-
cation increased in similar proportions. All the investigations such as thyroid profi le, gynaecological
investigations turned out normal. No amount of counselling, dietary restrictions would prove to be of
any use in bringing her weight down. To add to it all, patient always claimed that she barely ate any-
thing in the day.
Finally, out of pure frustration; patient was asked whether she ate anything in the night. We were taken
by surprise, when patient accepted having multiple
sessions of consuming food in the night, at least 4-5 days per week. Her reported intake in the night
was much more than what she ate in the day, mostly in the form of sweets and milk products. She was
having these symptoms for more than 2-3 years. She was our fi rst case of NES.
Archives of Indian Psychiatry 13(2) October 2011 6
relationship between psychiatric illness, duration
of illness or treatment could be observed. Three
patients were obese (BMI > 30), two overweight
(BMI 25-30), and rest three were normal weight [36,37,38]. None of diagnosed patients reported
any family history, but two of the 142 screened
patients who did not have NES reported that one
or more of their family members had symptoms
fulfi lling criterion of NES, giving overall
prevalence of NES reported in family members to
be 1.5%.
Aetiology
Though various behavioural and neuroendocrine
studies have been carried out, the exact
pathophysiology remains elusive. Considered
variously as sleep disorder, eating disorder or
disorder of circadian rhythm; strongest evidence
supporting the latter comes from neuroendocrine
studies. The neuroendocrine characteristics have
been described as changes in the circadian rhythm
by attenuation in the nocturnal rise of the plasma
concentrations of melatonin (the sleep inducing
hormone) and leptin (satiety producing hormone)
and an increased circadian secretion of cortisol
(stress related hormone). The night eaters also
have an over expressed hypothalamic-pituitary-
adrenal axis with an attenuated response to stress,
as expressed by an attenuated ACTH and cortisol
response. In conclusion the mechanisms behind
the increased CRH stimulation may involve
alterations in the neurotransmitter systems, causing
increased nocturnal appetite and disruption in the
sleep pattern. This may, to some extent, explain
the disturbances in the circadian secretions
of melatonin and leptin and the behavioural
characteristics of the night eating syndrome. Some
disturbances regarding blood glucose levels and
insulin levels have also been reported, causing
their rise in the night hours [4].
Recently SPECT study has shown signifi cant
elevation in the serotonin transporters in midbrain
of night eaters. This elevation may result as the
established heritability of NES; which may be
triggered by the stress that night eaters report.
Elevation in the serotonin transporter levels lead
to decreased post synaptic serotonin transmission
which is thought to impair circadian rhythm and
satiety [11].
Psychological and emotional components may be
important in precipitation but not well understood.
Some of the factors commonly associated with
onset of NES are acute stress reaction, abstinence
from alcohol or opiates/cocaine abuse and
cessation of cigarette smoking.
Various sleep laboratory studies have also been
carried out in patients with NES, which is been
considered a ‘parasomnia’ and sometimes even
a rare variety of sleep walking. NES is often
accompanied by or confused with sleep-related
eating disorder, which is primarily a sleep disorder
rather than an eating disorder, in which people
are unaware of having eaten while asleep. Whilst
there is a debate whether night eating disorders are
eating disorders or sleep disorders it has become
clear that investigating the sleep characteristics
is worthwhile. Various sleep disorders found in
individuals who have been investigated in a sleep
laboratory or sleep disorders centre are Sleep
walking, Periodic Limb Movement (PLM),
Obstructive Sleep Apnoea (OSA), Irregular
sleep/wake pattern disorder, Familial restless
legs syndrome, as reported in various studies by
Manni et al., 1997; Schenck et al., 1993; Schenck
and Mahowald, 1994; Schenck and Mahowald,
2000; Winkelman, 1998; Winkelman et al., 1999 [17,18,19].
In one of the studies, a strong familial component
is also reported [35].
Some studies mention possible relationship with
antipsychotic medication, specially atypical
antipsychotics [12,2].
Clinical Features
The patient suffering from NES has little or no
appetite for breakfast. Proverbial term ‘Refrigerator
Raids’ a common phenomenon proverbially
talked about with reference to such night eating
syndrome in people who diet during the day and
overeat at night, though it was never thought of
as a psychiatric illness. Patient delays fi rst meal
for several hours after waking up. The patient is
not hungry or is upset about how much was eaten
the night before. The patient eats more food after
dinner than during that meal and consumes more
than half of daily food intake after dinner but
before breakfast the next day. The patient may
Archives of Indian Psychiatry 13(2) October 20117
leave the bed to snack at night several times. This
pattern must persist for at least two to three months.
Person feels tense, anxious, upset, or guilty while
eating. NES is thought to be stress related and is
often accompanied by depression. Especially at
night the person may be moody, tense, anxious,
nervous, agitated, etc. Patient has trouble falling
asleep or staying asleep. Patient has to Wake up
frequently and then often eat to fall back to sound
sleep. Foods ingested are often carbohydrates:
sugary and starch. Behaviour is not like binge
eating which is done in relatively short episodes.
Night-eating syndrome involves continual eating
throughout evening hours. This eating produces
guilt and shame, not enjoyment. This disorder can
start at any age. Psychological components are
thought to be important in precipitation, but are
not absolutely necessary features of NES. Though
commonly seen in overweight or obese individuals,
this is not necessary criteria for diagnosis as up
to half the patients suffering from NES may be of
normal weight [6,13].
Our study corroborated the reported clinical
features from western studies. Almost all patients
had no appetite in the morning, had delayed their
fi rst food consumption for the day beyond 12:00
noon but consumed food before 3:00 pm. As
evening passed by, their consumption of food went
on increasing in quantity and frequency; continuing
well in to the night. Patients had severe craving
for food even following dinner, and all of them
consumed some or the other snacks, and sometimes
the same food cooked for the supper before retiring
to the bed. All experienced diffi culty in initiating
sleep, which was characterised by awakenings at
least 1-2 times/night(avg. 2 awakenings/night.
ranging from 1-4).This would happen at least
3-4 times/week(avg. 3.4). All patients required to
consume food to get back to sound sleep. Time
most frequently reported for such awakening was
between 12:01 am to 2:00 am, followed by 10:00
pm -12:00 am. This is not consistent with binge
pattern, as consumption is spread over few hours.
All patients were completely aware of their night
eating. One patient experienced amelioration of
symptoms on stopping olanzapine, where as other
patient had relief when fl uvoxetine was added to
the medications. Most of these patients reported
dysphoric mood though not fulfi lling criterion for
MDD. One patient claimed to have started with
the symptoms on adding clozapin to medications.
Three of the eight patients were receiving clozapin
for treatment. One patient had NES symptoms
well before starting any psychiatric treatment and
has been experiencing these symptoms for about
a decade. Most other patients (5) are experiencing
the symptoms for past 1 ½ to 2 years.
All the patients preferentially consumed sweet
food, such as 3-4 pieces of Mava-mithai, Falooda
and rice mixed with sweet milk and sometimes
up to a dozen of bananas as reported by a patient.
Others consumed 2-3 packs Glucose biscuits and
2-3 packs of potato chips which they kept stored in
house to avoid any discomfort in the night just like
their western counterparts, as reported. None of
our patients reported any form of sleep disorders
or benzodiazepine abuse which were found to
be commonly associated with NES by Manni,
Schenck , Mahowald, Winkelman in various sleep
laboratory studies[17,18,19]
Diagnosis
NES has not been formally included in the DSM
or ICD as a separate disorder and diagnostic
criterion have somewhat changed since its original
description by Dr. Albert Stunkard. Though the
standard criterion for diagnosis require symptoms
in form of 1) Morning anorexia 2) Evening
hyperphagia 3) Multiple sleep awakenings
associated with craving for food and food
consumption on at least three nights per week; the
total proportion of daily calorie intake to be taken
after dinner to qualify as NES has been variously
quoted as 25% in original paper presented by Dr.
Albert Stunkard in 1955 [1] to about 1/3 according
to some references, and now it is accepted by
many researchers that at least 50% of daily calorie
intake should be following dinner to qualify for
NES[4,8,9,10,12,13]. There are differences among
researchers as whether to count desserts after
dinner for calorie intake for NES or no [8,9,10].
This has resulted in some diagnostic variations
by various researchers. Primary tool used for
identifying NES patients by most researchers
has been Night eating Syndrome Questionnaire [8,19], devised by Centre for Weight and Eating
Disorder, Department of psychiatry, University of
Pennsylvania, school of medicine; the pioneering
institute in research on NES. This questioner
Archives of Indian Psychiatry 13(2) October 2011 8
has sixteen questions regarding symptoms and
phenomenological aspects of NES. A total score
of twenty or more is considered diagnostic of NES [8].
Before a patient is considered for diagnosis of
NES, other disorders which may intermittently
produce picture similar to NES, for example D.M.
which may lead to nocturnal hunger and food
consumption should be ruled out. Other sleep
disorder such as sleep related eating disorder,
where patient doesn’t remember about his/her
night consumption or obstructive sleep apnoea,
where patient may have multiple awakenings
which may or may not be associated with food or
water consumption should also be ruled out with
appropriate history. Some times even occupational
factors such as shift duties may have adverse
effect on sleep-wake cycle and may produce
clinical picture which may be confused with NES [17,18,19,20].
In our study we utilised NES questionnaire for
screening and diagnosis. To calculate total amount
screening and diagnosis. To calculate total amount
of consumed calories and their proportion after
dinner, we used standard calorifi c value for Indian
food items [42,43,44]. BMI was calculated using
standard formulae [38,39,40,41]
Management
Treatment for this condition is tricky [33].
Researchers have variously used Cognitive
Behavioural Therapy [35,36], dietary modifi cations,
various pharmacotherapy or combination of both
for treatment [35]. Pharmacotherapy includes
melatonin, leptins given therapeutically as
suggested by their defi ciency in the evening in these
patients [4,9,10]. According to J.A.M.A. (Journal
of American Medical Association), carbohydrate
containing foods tend to increase serotonin, and
they are preferentially consumed by NES patients [10]. Stunkard even has suggested diet rich in
tryptophan, such as pea-nut butter which helps in
increasing serotonin in body to be consumed by the
NES patients [3,4,7,10]. Sleep inducing effect of
these foods theoretically implied that SSRI; which
increase serotonin concentration in the brain might
be useful in this condition, assumption which was
also supported by successful use of Sertraline in
double blind trials and recent demonstration of
elevated serotonin transporters in midbrain of
these patients, causing serotonin defi ciency in
synaptic clefts [11]. There have been reports of
other two SSRI’s namely paroxetine and fl uoxetine
being equally effective. These trials have reported
a remission rate in excess of 75% [32,33,34].The
combination of pharmacotherapy with cognitive
behavioural therapy seems to produce better and
sustained improvement [35]. Reports of such
specifi c and effective treatment for NES have
further strengthened the claims for identifi cation
and inclusion of NES as distinct disorder in DSM [11].
References
1. Stunkard AJ, Grace WJ, Wolf HG: The night-
eating syndrome. American Journal of Medi-
cine. 1955; 19:78–86
2. Leder Mh, Cardinalli DP, Pand-parumal
SR(eds).Sleep & sleep disorder: A neurophysi-
ological approach: Springer; 2006.Birketvedt
GS, fl orholmen JR.28:251.
3. O’Reardon JP, Ringel BL, Dinges DF, Al-
lison KC, Rogers NS, Martino NS, Stunkard
AJ: Circadian eating and sleeping patterns in
the night eating syndrome. Obesity Research
2004; 12:1789–1796
4. Allison KC, Ahima RS, O’Reardon JP, Dinges
DF, Sharma V, Cummings DE, Heo M, Mar-
tino NS, Stunkard AJ. Neuroendocrine profi les
associated with energy intake, sleep and stress
in NES. Journal of clinical endocrinology and
metabolism.nov 2005; 90(11): 6214-17
5. Rand CSW, Macgregor MD, Stunkard AJ: The
night eating syndrome in the general popula-
tion and amongst post-operative obesity sur-
gery patients. Int J Eat Disord 1997; 22:65–69
6. Marshall HM, Allison KC, O’Reardon JP,
Birketvedt G, Stunkard AJ: Night eating syn-
drome among nonobese persons. International
Journal of Eating Disorders 2004; 35:217-22
7. Stunkard AJ, Berkowitz R, Wadden T, Tanri-
kut C, Reiss E, Young L: Binge eating disor-
der and the night-eating syndrome. Int J Obes
1996; 20:1–6
8. Centre for Weight and Eating Disorder, De-
partment of psychiatry, University of Pennsyl-
vania, school of medicine. Night eating Syn-
drome Questionnaire. Available at www.med.
upenn.edu/weight/night eating.shtml-5k
9. Fairburn C, Brown KD. Eating disorders and
Archives of Indian Psychiatry 13(2) October 20119
obesity-a comprehensive hand book. 2nd edi-
tion. Guilford Press:USA;2002
10. Birketvedt GS, fl orholmen JR, Sundsford J,
Osterud B, Dinges D, Bilker W, Stunkard A.
Behavioural and neuroendocrine characteris-
tics of NES. Journal of American Med. Asso-
ciation (J.A.M.A).1999.Aug 18;282: 657-663
11. Allison KC, Wintering N, Stunkard AJ, Lund-
gren JD. Issues for DSM V: Night Eating Syn-
drome. Am J psychiatry; April 2008, 165: 4
12. Allison KC, Carlos MG, Masheb MR, Stunk-
ard AJ. High self reported rates of neglect and
self abuse by persons with binge eating disor-
ders and night eating syndrome. Obesity (sil-
ver spring).2007 May; 15(5):1287-93
13. Anderson GS, Stunkard AJ, Sorenson TI, Pe-
terson L, Heitmann TL. Night Eating and
weight changes in middle aged men and wom-
en. Int J Obes relat Metab disorders 2004;28:
1338-1343
14. Lundgren JD, Allison KC, Crow S, Odeurdon
JP, Berg KC, Galbraith J, Martino NS, Stunk-
ard AJ. Prevalence of NES among people with
psychiatric conditions. American Journal of
psychiatry (A.J.P).2006;163:153-156
15. Hoek HW: The distribution of eating disor-
ders, in Eating Disorders and Obesity: A Com-
prehensive Handbook. Edited by Brownell
KD, Fairburn CG. New York, Guilford, 1995,
pp 207-211
16. Bulik CM, Sullivan PF, Carter FA, Joyce PR:
Lifetime comorbidity of alcohol dependence
in women with bulimia nervosa. Addiction and
Behaviour 1997; 22:437–446
17. Adami j. Night eating in obesity: a descriptive
study. Nutrition , Volume 18 , Issue 8 , 587 -
589 G
18. Manni, R., Ratti, M.T., and Tartara, A. (1997)
Nocturnal eating: prevalence and features in
120 insomniac referrals. Sleep 20, 734-738.
19. Schenck, C.H., Hurwitz, T.D., O’Connor,
K.A., and Mahowald, M.W. (1993) Additional
categories of sleep-related eating disorders and
the current status of treatment. Sleep 16, 457-
466.
20. Schenck, C.H. and Mahowald, M.W. (1994)
Review of nocturnal sleep-related eating dis-
orders. Int J Eat Disord 15, 343-356.
21. Winkelman, J.W. (1998) Clinical and poly-
somnographic features of sleep-related eating
disorder. J Clin Psychiatry 59, 14-19.
22. Jillon S. Vander Wal, a, , , Sandia M. Wallerb,
David M. Klurfeldb, Michael I. McBurneyc
and Nikhil V. Dhurandhar. Night eating syn-
drome: Evaluation of two screening instru-
ments. Eating Behaviours, vol. 6, issue 1, Jan.
2005,63-67
23. Debases Bagchi, Harry G. Obesity : epidemi-
ology, pathophysiology and prevention. CRC
press. Tyler and Fransis. Review available at
24. http://books.google.co.in/books
25. Heather Mudget, write s group. Night eating
Syndrome and nocturnal sleep eating disor-
ders. Available at http://www.suite101.com/
article.cfm/eating_disorders/89879
26. Wadden T, Stunkard J. Hand book of Obesity
treatment. Guilford press:USA;2002
27. Diagnose Me.com. Night Eating Syndrome.
Available at http://www.diagnose_me.com/
cond/c303927.html
28. Anorexia Nervosa and related Eating disorders
(ANRED), Inc©2005. Available at
29. http://www.anred.com/nes.html
30. Stunkard A, Berkowthz R, Wadden T, Tantri-
cut C, Reiss E, Young L Binge eating and night
eating syndrome. International Journal of Obe-
sity and Related Metabolic disorders.1996.
Jan;20(1):1-6
31. Sleep doctor. Night Eating Syndrome. Avail-
able..at.”sleepdoctor.blogspot.com/2006/01/
night-eating-syndrome-nes.html”
32. Selfgrowth.com©1996-2009.Night Eating
Syndrome-a case report. Available at:”www.
Selfgrowth.com/article/Night-Eating-Syn-
drome.html.”
33. Wikipedia, the free encyclopaedia. Night Eat-
ing Syndrome. Available at http://en.wikipedia.
org/wiki/night-eating-syndrome
34. APA guidelines for treatment-2006 “ sertralin
may be effective in NES”
35. Barkley L, Vega C. Sertralin may be effective
for night eating syndrome. American journal
of psychiatry.2006;163:893-898
36. O’Reardon JP, Allison KC, Martino NS, Lun-
dgren JD, Heo M, Stunkard AJ: A randomized
placebo-controlled trial of sertraline in the
treatment of the night eating syndrome. Amer-
ican Journal of Psychiatry (in press)
37. Allison KC, Stunkard AJ, Thair SL. Overcom-
ing NES-a step by step guide in breaking the
cycle. New Harbinger Publication; 1st edi-
tion: May 2004
Archives of Indian Psychiatry 13(2) October 2011 10
38. Hilt E. Relaxation may help prevent NES. In-
ternational journal of obesity and related meta-
bolic disorder.2002:vol 21;342-356
39. Allison KS, Lundgren j, Stunkard AJ. Familial
aggregation in NES. International Journal of
Eating Disorders,2006:vol39;516-518
40. http://www.metlife.com/Lifeadvice/Tools/
Heightnweight/Dcmen.html
41. Harrison GG.Height-weight tables. Ann Intern
Med 1985;103:489-94
42. Pai MP, Paloucek FP. The origin of the “Ideal”
body weight equations. Annual Pharmacol-
ogy.2000; 34:1066-69
43. Wikepedia, the free encyclopaedia. Body
Mass Index.
44. weightlossresourses.co.uk.Indian food calorie
content. Available at “http://www.weightloss-
resoursec.co.uk/calories/clorie-content/Indi-
an-food.htm”
45. Kellow j. Eating out on a diet Indian food.
New harbinger publication: 2002;123-134
46. Bawarchi,Health and nutrition .Indian food
calorie content available at:“www.bawarchi.
com/healthquerries.co.h
Dr.Ameya Amrritwar, Senior Resident
Dr. Nilesh Shah, Professor and Head
Dept. of Psychiatry,
OPD 21, College Building,
LTMMC, Sion Hospital,
Mumbai 400022.
e-mail : [email protected]
Tel: 022 24011984
Archives of Indian Psychiatry 13(2) October 2011 11
Review Article
Developmental Psychopathology and its relevance to
Child Psychiatry – a Clinical Perspective
Avinash De Sousa
Abstract
Today more so than ever child psychiatrists worldwide are looking at developmental factors to provide
answers to causative factors in psychiatric disorders of children and adolescents as well as adults. This
review looks at developmental psychopathology and its relevance to modern day clinical child psychia-
try. It probes whether a developmental approach is an appropriate one while examining the infl uences of
genes, environment and social factors in the genesis of child psychiatric disorders. It is looks at research
new and old and proposes that a multi-modal multi-pronged approach is needed when examining causa-
tive factors in various child psychiatric disorders. An approach combining psychology, development,
neurosciences and genetics along with biology is a sound one. This paper also looks at the pitfalls of
developmental psychopathology research so far and questions that remain to be answered along with
research challenges that lie ahead
(Key words: Developmental Psychopathology, Child Psychiatry)
Introduction
We have always been taught the importance
of a good developmental history when it
comes to determining possible causative
factors in adult psychiatric disorders. The
history of developmental psychopathology
as a valuable and firm construct really began
with the work of Achenbach. He argued
that developmental dimensions should
constitute the primary basis for the study
of childhood and adult psychopathology
and that it is not accept able to view it as
no more than a downward extension of
adult mental dis order. However, in making
that point, he emphasized the crucial
importance of scientific strategies to test
developmental concepts, and noted the
inadequacy of all the prevailing theories
and concepts that paid little attention to
childhood development.
Developmental psychopathology has
been defined by Achenbach as the study
of various psychiatric disorders keeping
in mind developmental factors that have
contributed towards psychopathology and
that affect the course of the disorder while
studying the disorder from a life-span
perspective[ 1 ] .
Many researchers later seconded his view.
They had a lot in common, but with critical
differences in opinion. They aimed for a
bringing together of research into child
development and into child psychi atry but
with the need to do so in diverse ways[ 2 - 3 ] .
Traditionally developmental psychology
has focused on developmental universals
over time, whereas it is needed that it focuses
on individual differences, lifeevents, role
models, parents, parenting, peer influences
and on the modifications and changes that
occur with altered circumstances. Child
psychiatry had tended to concentrate on the
causes and course of individual diagnostic
conditions and criteria for better diagnosis
along with diagnostic stability over time.
Developmental psychology accepted this
but argued that a developmental perspective
answers many more questions[ 4 ] .
Some of the questions that developmental
psychology addresses are based as to what
extent are there age-related variations in
susceptibility to stress. Another is whether
there are points in development when
psychological qualities became relatively
stabilized so that, although behavior might
change, it was no longer possible for
Archives of Indian Psychiatry 13(2) October 2011 12
functioning to be to tally transformed. For
example we know that personality traits
are modifiable in the first 20-30 years of
life but this often becomes difficult as
life moves on[ 5 ] . We also know of all
the emphasis that has been laid on early
intervention and treatment and how delaying
treatment may have deleterious effects on
prognosis of childhood disorders. Another
issue is the reasons for developmental of
psychiatric disorders across age groups
with some peaking in childhood and
some in adolescence[ 6 - 7 ] . Disorders like
enuresis, separation anxiety often start in
childhood compared to adolescence[ 8 ] .
Dissociative disorders are commoner in
adolescence and rare in childhood[ 9 ] .
Childhood onset schizophrenia differs
markedly in symptomatology than one
with an adolescent onset[ 1 0 ] . It is well
known that attentionIt became clear that
experiences had biological effects that
played a part in bringing the environment
inside biology, thereby provid ing possible
mechanisms for long-term effects. There
was also research demonstrating the
relevance of birth trauma, perinatal events,
maternal responses to pregnancy and child
birth, obstetrical complications and early
life insults in the development of child
psychiatric disorders[ 3 4 ] .
Importance Of Early Life Experiences
In Psychiatric Disorders
Today, more than ever psychiatrists have
accepted worldwide that a developmental
perspective is essential for studying
and understanding psychiatric disorders
in adult life[ 3 5 ] . This has placed the
neuro-developmental hypothesis for most
psychiatric illness in mainstream thinking.
This has already been accepted for cases
of schizophrenia[ 3 6 ] . Depression in
young people is associated with a high
risk of major depression in adult life, and
attention deficit hyperactivity disorder
(ADHD) is followed by a substantial
increase in psychiatric illnesses like adult
ADHD, conduct problems, antisocial
behavior, substance abuse and bipolar
disorder in adult hood[ 3 7 - 3 8 ] . The
childhood psychiatric illness carrying the
highest risk for adult psychopathology
is conduct disorder and ADHD and must
be treated appropriately at the right time.
Child physical and sexual abuse long with
childhood neglect is another important risk
factor. Parenting styles, teacher influences,
bullying and school atmosphere along with
peers have also been shown to involve
long-term risks[ 3 9 - 4 1 ] .
Early Research In
Developmental Psychopathology
Much early research in developmental
psychopathology was preoccupied with
identifying the effect of some risk or
protective factor in isolation from all
others. Some researchers challenged this
concept. They did not deny that effects
could be independent, but they noted the
frequency of two-way interplay in what
they termed transactional effects[ 4 2 ] .
In an influential paper published in 1968,
the author argued that some of the supposed
effects of social ization experiences might
derive from children’s effects on their
rearing en vironments, rather than the other
way round[ 4 3 ] . Thomas and Chess, in
their research on children’s temperamental
styles, made much the same point[ 4 4 ] .
Researchers also showed the importance
of parent-child gene-environment
correlations. Parental behavior had
a passive influence on the rearing
environment because parents transmitted
both risky genes and risky environments.
Child often have various risky genes that
they inherit but environmental factors and
life event triggers determine whether they
manifest with the disorder. Child behavior
had active effects because genetically
influenced behaviors played a part in
shaping and selectingenvironments and
in evoking particular responses in other
people[ 4 5 ] .
Emergence Of Resilience As A Factor In
Child Psychiatric DisordersResilience, had its origins in the observation
Archives of Indian Psychiatry 13(2) October 201113
that people varied greatly in their re sponse
to stress and adversity. Some become
severely impaired, some survive relatively
unscathed, and a few appear even to be
strengthened through cop ing successfully
with the hazards they face. It has to be said
that the potential of the resilience concept
still requires much better empir ical study,
but it constitutes a prime example of the
developmental psychopathology focus on
individual differences in developmental
functioning[ 4 6 ] . The study of resilience
comprises a growing body of work that has
examined coping, hardiness, protective
factors, positive youth development,
personal and community assets and
thriving. It is also related to the work done
in positive psychology, strengths based
social work and the study of salutogenesis
in medicine[ 4 7 ] . Combined, these multiple
threads have provided a theoretically
sound set of principles that detail how
children that face significant amounts of
adversity manage not only to survive, but
also thrive[ 4 8 ] .
The Effect Of Age On
Child Psychiatric Disorders
Despite its centrality in developmental
psychopathology concerns, age differ ences
in susceptibility have been investigated
only to a very limited extent. The nature
and degree of brain plasticity does change
with age but we know surprisingly little
about the mechanisms. It is known that
myelination starts at 5-6 months of age and
completes by 30-35 years of age. Changes
in neuronal number, synaptic density
and effects of hormones too vary across
various age groups in different parts of the
brain[ 4 9 - 5 0 ] .
Antisocial behav ior may exist in two
forms i.e. life course-persistent antisocial
behavior (which typically begins early and
is associated with neuro-developmental
impairment) and an adolescence-limited
variety, which is less strongly associated
with risk factors both biological and
experiential and is determined more by
environment and life events[ 5 1 ] . The
validity of this differentiation has been
mainly supported by other research, but it
is clear that an onset of conduct disorder
in childhood does not usu ally lead to life-
long persistent problems[ 5 2 ] .
The Role Of Genetic Factors
Early claims in the field of behavioral
genetics tended to use terms such as
the “gene for” schizophrenia or bipolar
disorder, with the misleading implication
of asomewhat direct deterministic effect.
In many circumstances, there is co-action
between genes and environments, and it is
misleading to conceptualize ef fects as due
to either nature or nurture[ 5 3 ] . Today we
are sure that most psychiatric disorders are
polygenic in nature and it is very difficult
to blame genes alone for causation. Many
gene loci that have been determined
have not been replicated across various
populations and few genetic causes like
that for disorders like mental retardation as
well as Rett’s syndrome have been firmly
established[ 5 4 ] .
For many years, few psychosocial
researchers showed much interest in the
varied mechanisms by which environments
might have long-term effects[ 5 5 ] . Cross-
foster ing studies have shown that the effects
are environmentally mediated, and neuro-
chemical investigations have indicated
that the mediation conies about through
effects on gene expression. Environments
cannot change gene se quences, but they
can change gene expression, which is
a necessary process for genes to have
effects[ 5 6 - 5 7 ] .
Evidence from both human and animal studies
suggests that there are biological programming
effects from environmental infl uences dur-
ing sensitive periods of development. A human
example is provided by the long-term effects of
institutional deprivation, which have been found
to be surprisingly persistent and highly sensitive
to specifi c developmental phases.
There is increasing evidence on the neuro-
Archives of Indian Psychiatry 13(2) October 2011 14
endocrine ef fects of acute and chronic
stress experiences[ 5 8 - 5 9 ] . At present, we
lack evidence on the role of the neuro-
endocrine changes on psychological
sequelae, and that stands out as an important
research challenge. It will also have to
be seen whether diverse transcultural
factors influence neurobiology and neuro-
endocrinology[ 6 0 ] .
Neuro-Imaging And Developmental
Psychopathology
The development of structural and
functional brain imaging initially positron
emission tomography (PET), magnetic
resonance spectroscopy (MRS) and then
magnetic resonance im aging (MRI)—
has opened up new possibilities to
investigate brain-mind interconnections.
Thus, studies of individuals with autism
have produced evidence of differences in
interconnectivity of brain functioning[ 6 1 ] .
When combined with longitu dinal studies,
brain imaging may also throw light on the
brain changes as sociated with both neural
development[ 6 2 ] and the development of
psychopathology[ 6 3 - 6 4 ] . Serial studies
in the same population across various
ages may help determine neurobiological
factors that determine psychopathology
and its variations across the life span.
The New Epigenetic Concept Of
Mental Illness
The development of the idea and the first
use of the term ‘epigenotype’ occurred as
far back as 1942. Conrad H. Waddington
suggested the existence of epigenetic
mechanisms to explain the control of
gene expression programmes during
development[ 6 5 ] . Implicit in the term
epigenetics is that these mechanisms are
labile and are erased and reset. Over the
years the definition of epigenetics has
shifted a little, such that these mechanisms
are not limited to development only,
although there is still an understanding
that epigenetic marks controlling gene
expression are stably transmitted through
cell divisions. For the purposes of this
review, epigenetics can be loosely defined
as the transmission and perpetuation of
coding information that is not based on
alteration of the DNA sequence. These
coding changes may be mediated via
chemical marking of the DNA sequence
itself (DNA methylation) and/or chemical
tagging of histone proteins that bind DNA
and are molecular tools by which gene
expression levels are controlled.
Today research has shown the contribution
of epigenetic mechanisms to brain
function, and consequently dysfunction
in the form of neuropsychiatric disorders.
In the classical sense epigenetics describe
the machinery and process involved in
regulating gene expression, particularly
during development. In this respect we
have outlined a sub-group of genes, namely
those subject to genomic imprinting,
for which the epigenetic control is very
important and when this goes wrong can
result in clinical conditions. Although it
is not entirely clear at present, work from
animal studies suggests that imprinted
genes play an important role in the
brain and may well contribute more
generally to neuropsychiatric disorder in
humans[ 6 6 - 6 7 ] .
In addition to pre-programmed gene
regulation, there is a growing body of
evidence suggesting that epigenetic
mechanisms may also provide a molecular
memory of environmental experiences.
This has been clearly shown in animal
models[ 6 8 ] and indicated in studies of
human twins[ 6 9 ] . In some circumstances
the epigenetic changes simply reflect long-
term changes in gene expression levels.
However, alterations to the epigenetic
code may not result in gross changes in
gene expression per se, but provide an
additional level of molecular information.
A good example of this has been provided
by studies of acute and chronic cocaine
Archives of Indian Psychiatry 13(2) October 201115
use in rats. These drug regimens give rise
to subtly different histone modifications
around the promoter of the FosB gene[ 7 0 ] ,
but not differences in expression per se.
Instead, it is thought that this may underlie
the fact that expression of the FosB gene
variant, DFosB only partially desensitises
to chronic cocaine treatment.
Although clearly epigenetic mechanisms
are of potential clinical relevance, a key
question is whetherthey are accessible to
pharmacological intervention. There are
a number of histone deacetylase (HDAC)
inhibitors and DNA methyltransferase
inhibitors that are currently being used
and/or tested as anticancer drugs[ 7 1 ] .
However, how applicable these are to
treating neuropsychiatric disorders is
not clear—although valproate has HDAC
inhibitor properties and may, in part, exert
its action through epigenetic effects on the
schizophrenia candidate gene Reelin[ 7 2 ] .
Currently a problem exists between the
specificity of the epigenetic changes that
may occur (i.e. specific modifications, key
time points and discrete brain regions)[ 7 3 -
7 4 ] , and the general influence of HDAC
and DNA methyltransferase inhibitors
on gene expression. It is hoped that the
ongoing epigenome project[ 7 5 ] and the
development of more specific drugs may
address these issues.
The Need Of The Hour
In keeping with the whole of science,
but especially biomedical science,
interdisciplinary collaboration has played
a major role in the discoveries, and that
will certainly continue to be the case. The
particular challenge for clinical scientists
is the need to develop exper imental
medicine in which there can be a two-
way interplay between clinical advances
and scientific advances, and not just the
mechanized application at the bedside of
findings deriving from basic science[ 7 6 ] .
The big questions that demand our attention
involve fi ve key issues.
1. Given the evidence on the importance
of multiphase causal pathways, what are the
mechanisms involved in phase transitions such
as those from the precursors and prodromata of
schizophrenia to overt psycho sis? Or from early
physical aggression or disruptive behavior or
inattention / overactivity to life course-persistent
antisocial behavior and antisocial personality
disorder.
2. What are the causal processes involved in the
direct and indirect pathways from the presence of
a susceptibility gene, on through gene expression,
to manifestations of particular phenotypes ? What
is the im portance of gene actions through gene-
environment correlations and interactions ? How
do genes and environments come together in
causal pathways in either normal development or
psychopathology ?
3. How do the neural changes involved in
brain development relate to the alterations in the
workings of the mind as they apply across both the
span of development and psychopathology.
4. What are the causal mechanisms involved in
individual differences in responses to stress and
adversity ? What is the role of personal agency in
successful coping with stress and adver sity? The
development of positive, rather than negative,
mental sets or internal working models seems
likely to be important, but how do such sets
develop and how do they operate ?
5. What underlies the age differences we see in
rates and patterns of psy chopathology and in styles
of response to psychosocial hazards or to physical
substances? A central tenet of developmental
psychopathology is that children are not just little
adults, but what are the processes in volved in age-
indexed variations ?
6. We need to look at newer diagnoses that
could be incorporated in DSM-V keeping
developmental psychopathology in mind.
Developmental Trauma Disorder as
suggested by some researchers to encompass
the multiple traumatic events a subject may
undergo during the developmental phase is
one such example[ 7 7 ] .
7. We also need to focus on complex
comorbidities (more than 5 child psychiatric
diagnoses existing together) that exist
in child psychiatry and find integrative
treatments that focus on this disorders as
a spectrum rather than looking at them as
individual disorders[ 7 8 ] .
8. We also need to integrate the fi ndings of
Archives of Indian Psychiatry 13(2) October 2011 16
neurobiology and psychological factors along with
a developmental psychopathological perspective
to gain fresh insight into issues like the links
between ADHD and mood disorders, ADHD
and antisocial personality disorders, causation of
autism spectrum disorders and the role of resilience
and temperamental adversity and ADHD.
Of course, clinical practice in psychiatry or
psychology or pediatrics raises a host of Issues
beyond these, but the importance of developmental
psychopathology lies in its role in making us frame
particular sorts of ques tions about development
and about psychopathology, and in suggesting
what might be useful research strategies to pursue.
Developmental psychopathology is not, and should
not become, a “big” theory or ideology Also, it
con stitutes a central feature in the whole of biology
and of medicine, and we need to ensure that we
do not cut ourselves off from these broader fi elds.
The pioneers who paved the way to the growth
of developmental psychopathol ogy tended to be
iconoclasts who challenged set ways of thinking
and who re fused to accept the given wisdom just
because it came from senior fi gures who held
positions of power. We should follow their model
and do the same in pursuing our interest in taking
developmental psychopathology forward.
References
1. Achenbach TM. Developmental
Psychopathology. New York : Wiley ; 1974.
2. Rutter M, Garmezy N. Developmental
Psychopathology. In : Mussen PH,
Hetherington EM, editors. Handbook
of Child Psychology, 4t h edition, Vol 4
: Socialization, Personality and Social
Development. New York : Wiley ; 1983.
pp 775-911.
3. roufe A, Rutter M. The domain of
developmental psychopathology. Child Dev
1984 ; 55 : 17-29.
4. Rutter M. Multiple meanings of a developmental
perspective on psychopathology. Eur J Dev
Psychol 2005 ; 2 : 221-252.
5. Goldberg LR. The structure of phenotypic
personality traits. Amer Psychol 1993 ; 48(1)
: 26-34.
6. Rutter M. Changing Youth in a Changing
Society : Patterns of Adolescent. Development
and Disorder. London : Nuffi eld Provincial
Hospitals Trust ; 1979.
7. Rutter M. Proceeding from observed
correlations to causal inference : the use
of natural experiments. Perspectives on
Psychological Science 2007 ; 2 : 377-395.
8. Robson WLM. The evaluation and management
of enuresis. N Engl J Med 2009 ; 360 : 1429-
1436.
9. Putnam FW. Dissociative disorders in children
: behavioral profi le and problems. Child Abuse
Neglect 1993 ; 17(1) : 39-45.
10. Jacobsen LK, Rapoport JL. Childhood onset
schizophrenia : implications of clinical and
neurobiological research. J Child Psychol
Psychiatry 1998 ; 39(1) : 101-113.
11. Wahlstedt C, Thorell LB, Bohlin G.
Heterogeneity in ADHD : neurophysiologic
pathways, comorbidity and symptom domains.
J Abn Child Psychol 2009 ; 37(4) : 551-564.
12. Kearney CA. School absenteeism and school
refusal behavior in youth : a contemporary
review. Clin Psychol Rev 2008 ; 28 : 451-471.
13. Drabick DAG. Can a developmental
psychopathology perspective face a paradigm
shift towards a mixed categorical dimensional
classifi cation system. Clin Psychol Rev 2009 ;
16(1): 41-49.
14. Cichetti D. The emergence of developmental
psychopathology. Child Dev 1984 ; 55 : 1-7.
15. Paykel ES. Contributions of life events to the
causation of psychiatric illness. Psychol Med
1978; 8 : 245-253.
16. Rutter M. Age as an ambiguous variable in
developmental research : some epidemiological
considerations from developmental
psychopathology. Int J Behav Dev 1989 ; 12
: 1-34.
17. Arnsten AFT. Stress signaling and pathways
that impair prefrontal cortex structure and
function. Nat Neurosci Rev 2009 ; 10 : 410-
422.
18. Achenbach TM, Edelbrock CS. The
classifi cation of child psychopathology :
a review and analysis of empirical efforts.
Psychol Bull 1978 ; 85 : 1275-1301.
19. Cichetti D, Cohen DJ. Developmental
Psychopathology, 2n d edition. Hoboken
NJ : Wiley ; 2006.
20. Lewis M, Miller SM. Handbook of
Developmental Psychopathology. New York :
Archives of Indian Psychiatry 13(2) October 201117
Plenum Press ; 1990.
21. Rutter M. Developmental Psychopathology
: concepts and prospects. In : Lenzenweger
MF, Haugaard JJ, editors. Frontiers of
Developmental Psychopathology. New York :
Oxford University Press ; 1996.
22. Rutter M, Sroufe LA. Developmental
Psychopathology : concepts and challenges.
Dev Psychopathol 2000 ; 12 : 265-296.
23. Cummings EM, Davies PT, Campbell SB.
Developmental Psychopathology and Family
Process : Theory, Research and Implications.
New York : Guilford ; 2000.
24. Rutter M. Gene-environment interplay and
dGevelopmental psychopathology across
adolescence. In : Masten A, Mahwah NJ,
editors. Multilevel Dynamics in Developmental
Psychopathology. NJ : Erlbaum ; 2007.
25. Caspi A, Moffi tt TE. Gene environment
interactions in psychiatry : joining forces with
neuroscience. Nat Rev Neurosci 2006 ; 7 :
583-590.
26. Rutter M. Psychopathological development
across adolescence. J Youth Adolesc 2007 ; 36
: 101-110.
27. Grossman KE, Grossman K, Waters E.
Attachment from Infancy to Adulthood : The
Major Longitudinal Studies. London : Guilford
; 2005.
28. Plomin R, Bergeman CS. The nature of nurture :
genetic infl uences on environmental measures.
Behav Brain Sci 1987 ; 14 : 373-427.
29. Kendler KS, Prescott CA. Genes, Environment
and Psychopathology : Understanding the
causes of Psychiatric and Substance Use
Disorders. New York : Guilford ; 2006.
30. Hedebrand J, Scherag A, Schimmelman BG,
Hiney A. Child and adolescent psychiatric
genetics. Eur Child Adolesc Psychiatry 2009 ;
19(3) : 259-279.
31. Bowlby J. Maternal Care and Mental
Health. Geneva, Switzerland, World Health
Organization ; 1951.
32. Harlow HF. The nature of love. Am Psychol
1958 ; 13 : 673-685.
33. Blum D. Love at Goon Park : Harry Harlow
and the Science of Affection. Cambridge MA :
Porseus Publishing ; 2002.
34. Streissguth AP, Barr HM, Bookstein FL. The
long term neurocognitive consequences of
prenatal alcohol exposure. Psychol Sci 1999 ;
10 : 186-190.
35. Rutter M. Genes and Behavior : Nature Nurture
Interplay Explained. Malden MA : Blackwell
Publishing ; 2006.
36. Fatemi SH, Folsom TD. Neurodevelopmental
hypothesis of schizophrenia revisited.
Schizophr Bull 2009 ; 35(3) : 528-548.
37. nyder J, Bullard L, Wagener A, Leong PK,
Snyder j, Jenkins M. Childhood anxiety and
depression symptomatology : trajectories,
relationships and associations with substance
dependence. J Clin Child Adolesc Psychol
2009 ; 38(6) : 837-849.
38. 38. Klassen LJ, Katzman MA, Chokka P. Adult
ADHD and its comorbidity with a focus on
bipolar disorder. J Affect Disord 2010 ; 124(1)
: 1-8.
39. Shipman K, Taussig H. Mental health treatment
of child abuse and neglect : the promise of
evidence based practice. Pediatr Clin North
Am 2009 ; 56(2) : 417-428.
40. Wang J, Ianotti RJ, Nansel TR. School bullying
among adolescnets in the US : physical, verbal,
relational and cyber. J Adolesc Health 2009 ;
45(4) : 323-325.
41. Pluess M, Belsky J. Differential susceptibility
to parenting and quality child care. Dev
Psychol 2010 ; 46(2) : 379-380.
42. Costello EJ, Angold A. Developmental
Psychopathology. In : Cairns RB, Elder GH,
Costello EJ, editors. Developmental Science.
New York : Cambridge University Press ;
1996. pp 168-189.
43. Bell RQ. A reinterpretation of the direction of
effects in studies of socialization. Psychol Rev
1968 ; 75 : 81-95.
44. Thomas A, Chess S, Birch HG. Temperament
and Behavior Disorders. New York : New York
University Press ; 1968.
45. Plomin R, Bergeman CS. The nature of nurture :
genetic infl uences on environmental measures.
Behav Brain Sci 1987 ; 14 : 373-427.
46. Charney KDS. Psychobiological mechanisms
of resilience and vulnerability for successful
adaptation to extreme stress. Am J Psychiatry
2004; 161 : 195-216.
47. Masten AS. Registering progress and risk –
resilience in adolescent development. Ann NY
Acad Sci 2004 ; 1021 : 310-319.
48. Walsh F. Family resilience : a framework for
clinical practice. Fam Process 2003 ; 42(1) :
Archives of Indian Psychiatry 13(2) October 201118
1-18.
49. Calabrese F, Molteni R, Racagni G, Riva MA.
Neuronal plasticity : a link between stress and
mood disorders. Psychoneuroendocrinol 2009
; 34(suppl 1) : S208-S216.
50. Berluchi G, Buchtel HA. Neuronal plasticity :
historical roots and the evolution of meaning.
Exp Brain Res 2009 ; 192(3) : 307-319.
51. Moffi tt TE. Adolescence limited and life
course persistent antisocial behavior : a devel-
opmental taxonomy. Psychol Rev 1993 ; 100
: 674-701.
52. 52. Odgers CL, Caspi A, Broadbent JM. Pre-
diction of differential adult health burden by
conduct problem subtypes in males. Arch Gen
Psychiatry 2007 ; 64 : 476-484.
53. 53. Mann JJ, Haghighi F. Genes and environ-
ment : multiple pathways to psychopathology.
Biol Psychiatry 2010 ; 68(5) : 403-404.
54. Zoghbi HY. Rett syndrome : what do we
know for sure. Nat Neurosci 2009 ; 12 : 239-
240.
55. Rutter M. Pathways from childhood to adult
life. J Child Psychol Psychiatry 1989 ; 30 :
23-51.
56. Rutter M. Implications of resilience concepts
for scientifi c understanding. Ann NY Acad
Sci 2006 ; 1094 : 1-12.
57. Weaver ICG, Cervoni N, Champagne NA.
Epigenetic programming by maternal behav-
ior. Nat Neurosci 2004 ; 7 : 847-854.
58. McEwen B, Lasley EN. The End of Stress as
We Know It. Washington DC : Joseph Henry
Press ; 2002.
59. Gunnar M, Quevedo K. The neurobiology of
stress and development. Annu Rev Psychol
2007 ; 58 : 145-173.
60. Kartmann F. Transcultural Psychiatry : from
practice to theory. Transcult Psychiatry 2010 ;
47(2) : 203-223.
61. Frith U. Autism : explaining the enigma. Ox-
ford : UK Blackwell ; 2003.
Dr. Avinash De Sousa
MD, DPM, MS, MBA, DPH, DHA
Consultant Psychiatrist
Carmel, 18 St. Francis Road, Off SV Road,
Santacruz (W), Mumbai-54.
Phone : 91-22-26460002
e-mail: [email protected]
62. Sroufe A. Psychopathology as an outcome of
development. Dev Psychopathol 1997 ; 9 :
251-268.
63. Rapoport JL, Addington AM, Frangou S. The
neurodevelopmental model of schizophrenia :
update 2005. Mol Psychiatry 2005 ; 10 : 1-16.
64. Suomi SJ. Aggression and social behavior in
rhesus monkeys. Novartis Foundn Symp 2005
; 268 : 216-226.
65. Waddington CH. The epigenotype. Endeavour
1942; 1 : 18-20.
66. Isles AR, Wilkinson LS. Imprinted genes,
cognition and behavior. Trends Cogn Sci 2000
; 4 : 309-318.
67. 67. Isles AR, Davies W, Wilkinson LS.
Genomic imprinting and the social brain.
Philos Trans R Soc Lond B Biol Sci 2006 ; 361
: 2229-2237.
68. Jirtle RL, Skinner MK. Environmental
epigenomics and disease susceptibility. Nat
Rev Genet 2007 ; 8 : 253-262.
69. Zoghbi HY. Postnatal neurodevelopmental
disorders : meeting at the synapse. Science
2003 ; 302 : 826-830.
70. Richards EJ. Inherited epigenetic variation
: revisiting soft inheritance. Nat Rev Genet
2006 ; 7 : 395-401.
71. Laird PW. Cancer epigenetics. Hum Mol Genet
2005 ; 14(1) : R65-R76.
72. Dong E, Agis-Balboa RC, Simonini MV,
Grayson DR, Costa E, Guidotti A. Reelin
and glutamic acid decarboxylase 67 promoter
remodeling in an epigenetic methionine
nduced mouse model of schizophrenia. Proc
Natl Acad Sci USA 2005 ; 102 : 12578-12583.
73. Vu TH, Hoffmann AR. Imprinting of the
Angelman syndrome gene UBE3A is restricted
to the brain. Nat Genet 1997 ; 17 : 12-13.
74. Champagne FA, Curley JP. How social
experiences change the brain. Curr Opin
Neurobiol 2005 ; 15 : 704-709.
Sources of support : None
Confl ict of Interest : None
Archives of Indian Psychiatry 13(2) October 2011 19
Original Article
Prevalence of Traditional Healing practices in Psychiatric Outpatients
Rahul Shidhaye G.K.Vankar
AbstractTraditional healing is widely practiced by psychiatric patients. This study explored traditional
healing practices in 201 psychiatric patients attending Psychiatry OPD in a tertiary care center
in Western India. 54.7% patients in this study irrespective of diagnosis consulted a traditional
healer. Sixteen of the 110 patients had to pay an amount greater than Rs 2500. The view that
traditional healing is effective for mental health problems was supported by 22.7% of the
patients and 20.9% would recommend going to a traditional healer. Implications of these for
psychiatric practice are discussed
Key words: Traditional healing, psychiatry, India
Introduction
Traditional Healing practices are commonly
used especially in the developing world.
WHO defi nes “Traditional medicine” as
a comprehensive term used to refer both,
to Traditional Medicine systems such as
traditional Chinese medicine, Indian ayurveda
and Arabic unani medicine, and to various
forms of indigenous medicine (1). Aboriginal
concepts of health and wellness involve not
only the physical but also the mental, spiritual
and emotional aspects of health. This holistic
approach to health is widely accepted and
practiced in aboriginal communities, through
traditional medicine and healing practices and
rituals conducted by the traditional healers (2).
Historically, individuals known as “traditional
healers” were known to be the experts on
holistic health practices. They were sought
extensively by community members, as they
were considered to have different gifts to assist
individuals in maintaining a balance in
health (2) .
The opinion about traditional healers widely
varies from being uncritically enthusiastic to
being highly skeptic without much information.
Despite historical efforts by mainstream
society to obliterate various traditional healing
practices and replace them with Western
medical concepts of health, there is a resurgence
in the use of traditional holistic concepts of
health and healing practices all over the world
(2). In Africa up to 80% of the population uses
TM to help meet their health care needs. In Asia
and Latin America, populations continue to use
TM as a result of historical circumstances and
cultural beliefs (1). In many parts of the world
expenditure on traditional medicine (TM)
/ complementary and alternative medicine
(CAM) is not only signifi cant, but growing
rapidly (1). In most African countries primary
care is provided by traditional healers as well
as by biomedical practitioners, the former
often being more accessible (3-5).
In India, traditional healing systems co-exist
with modern mental health care, and they are
seen as providers of curative, rehabilitative
and restorative benefi ts. Traditional healing
systems and practices play an important but
controversial role in mental health service
delivery (6). In India, the percentage of patients
seeking traditional faith healing in various
studies have been like follows: 85.6% (7),
Archives of Indian Psychiatry 13(2) October 201120
57.5% (8), 33.3% (9) and 74.7% (10). It seems
that over a period of years the percentage of
patients consulting traditional faith healer is
increasing (10). Shamans and mystics play a
very important role in mental health disorders
(11).
Knowledge of traditional healing practice
and of the use of these services by people
with common mental disorders is limited and
impressionistic (12). Westernized studies
on traditional healing systems tend to be
derogatory, exposing only what are seen as
“abusive” in such methods (6). There is a
tendency to disregard such realities, which
is characteristic of medical practitioners and
other medical care professionals in Western
countries (13). In India many people troubled
by emotional distress or more serious mental
illnesses go to Hindu, Muslim, Christian, and
other religious centers (14). The healing power
identifi ed with these institutions may reside in
the site itself, rather than in the religious leader
or any medicines provided at the site. Studies
of these healing sites have focused primarily
on ethnographic accounts (14). Research has
not systematically examined the psychiatric
status of the people coming for help at these
religious centers or the clinical impact of
healing. It has focused primarily on possession
and non-psychotic disorders, rather than
serious psychotic illnesses (14).
Very few studies have been conducted in
India which have focused on traditional
healing practices in psychiatric patients. So
we designed this study in order to address this
research gap.
Aims and objectives
1. To study the prevalence of traditional
healing consultation in patients coming to the
psychiatry OPD.
2. To study the socio-demographic and
disease related characteristics associated with
traditional healing consultation.
Methods
This is a cross-sectional study which was
carried out in psychiatry department of a
general hospital. Consecutive 201 OPD
patients irrespective of their diagnosis were
selected for the study. Either the patient or
the relative accompanying the patient were
interviewed using the questionnaire.
Following information was collected:
Socio-demographic characteristics: This
included information related to age, sex,
education, religion, family type, domicile and
monthly income.
Disease related characteristics: The DSM-
IV diagnosis of the patients was based on
clinical interview by a trained psychiatrist. The
duration of the illness was noted.
Traditional Healer Consultation: Every
patient/relative was asked whether they had
consulted a traditional healer before coming to
the hospital. The response was noted as either
‘yes’ or ‘no’. Information regarding number of
visits, expenses incurred intervention by the
traditional healer and whether they think that it
was effective or not, was also recorded.
Data analysis was done using SAS version
9.2 (for windows) (15). Continuous variables
age, monthly income and duration of illness
are analyzed using t-test. This analysis is
done in SAS ver. 9.2 using proc t test and
proc univariate. Categorical variables sex,
education, family type, domicile and diagnosis
are analyzed using chi-square test. This analysis
is done in SAS ver. 9.2 using proc freq.
Results
The study sample consisted of 201 patients.
The mean age of the sample was 35.75 years
and it ranged from 8 years to 72 years. Males
were 64.18% and females were 35.82%.
Ninety-two percent of the patients were Hindus
and 27 % of the total sample belonged to rural
areas. Education up to SSC or beyond it was
completed by 39.3% of the patients. Nearly
one-third (38 %) of the patients were from
Archives of Indian Psychiatry 13(2) October 201121
Table 2 : Pattern of disorder and traditional healing
Variable Patients who visited Tradi-tional Healer
Patients who did not visit Traditional Healer
t-value chi-square value
degree of freedom
p-value
Diagnosis [No. (%)]$ 110(54.7) 91(45.3) -2.69 NA
Schizophrenia 30(83.3) 6(16.7) NA 14.49 1 <0.001*
Major Depression 40(44.0) 51(56.0) NA 7.78 1 0.005*
Bipolar Disorder 5(38.5) 8(61.5) NA 1.48 1 0.22
Epilepsy 7(53.8) 6(46.2) NA 0.004 1 0.22
Mental Retardation 7(70.0) 3(30.0) NA 0.991 1 0.32
Substance Use 2(40.0) 3(60.0) NA 0.449 1 0.50
Possession Disorder 22(100) 0 NA 1 <0.001*
Duration (Mean, SD) 67.4(78) 65.0(96.1) NA NA 199 0.84
Archives of Indian Psychiatry 13(2) October 2011 22
Table 1: Comparison between patients who visited Traditional Healer and those who did not visit Tradi-
tional Healer
Variable
Patients
who visited
Traditional
Healer
Patients who
did not visit
Traditional
Healer
t-value chi-square
value
degree
of free-
dom
p-value
No. of patients (%) 110(54.7) 91(45.3) -2.69 NA 0.007*
Socio-demographic characteristics
Age [Mean( SD)] 34.32 (12.2) 37.5(14.3) 1.69 NA 199 0.09
Sex [No. (%)]
Male
Female
70(63.6)
40(36.4)
59(64.9)
32(35.1) NA 0.03 1 0.86
Education[No. (%)]
Less than 10th grade
10th grade
Beyond 10th grade
63(57.0)
34(31.0)
13(12.0)
59(64.8)
30(33.0)
2(2.2)
NA 6.7
2
0.03*
Monthly income (Mean,
SD)
2225(2073) 2166(1619) -0.2 NA 150 0.85
Family Type [No. (%)]
Nuclear
Joint
36(32.7)
74(67.3)
41(45.1)
50(54.9) NA 3.20 1 0.07
Domicile [No. (%)]
Urban
Rural
80(73.6)
30(26.4)
67(72.7)
24(27.3)
NA 0.021 1 0.89
Major depression was the predominant
diagnosis, with 45% of the patients suffering
from it. Eighteen percent of the patients had
schizophrenia and 6.5% had bipolar disorder
and epilepsy. Possession syndrome was present
in 10.5% of the cases. The mean duration of
illness was 5.5 years. Patients with up to one
year of illness were 32.34%, one to fi ve years
of illness were 35.32%, fi ve to ten years were
18.41% and beyond ten years were 14%.
Table 3 :
Patients who visited traditional healer (N=110)%
Number of Visits
One Two-three More than ! ree
233641
Number of traditional
healers consulted
One Two-three More than three
493120
Setup from where services
were o! ered
Temple Dargah Home other
2519479
Physical abuse
Yes (beating, cut ting hear) No
10.889.2
Expenses incurred
upto Rs. 500 Rs.500-Rs.2500 more than Rs.2500
601822
Belief that traditional
healing is e! ective
Yes No
22.777.3
Hundred and ten out of the total two
hundred and one (54.7%) of the patients had
consulted traditional healer before coming
to the psychiatric hospital. Table 1 gives the
comparison between patients who had attended
traditional healers and those who had not, as
regards socio-demographic and disease-related
characteristics. Various characteristics related
to traditional healer consultation are described
in table 2. Of the patients who had consulted
traditional healers, 49% had visited only 1
healer while 31% had gone to 2-3 healers.
Thirty-six percent patients visited 2-3 times
and 41% went for more than three times. Most
of traditional healers offered services from their
home (47%) while 25% from a temple set up.
Physical abuse in the form of being beaten up
or hair being cut was experienced by 10.8% of
the patients who had visited traditional healer.
A quarter of the patients only paid up to Rs 5
to the healer while half of them paid an amount
up to Rs 300. Sixteen of the 110 patients had to
pay an amount greater than Rs 2500. The view
that traditional healing is effective for mental
health problem was supported by 22.7% of the
patients and 20.9% would recommend going to
a traditional healer.
Discussion
In our study we found that 54.7% of the
patients who had some kind of mental illness
had consulted traditional healer before coming
to the psychiatric hospital. This means that
every second patient whom we see in the
hospital setting has already consulted someone
from the indigenous and culturally acceptable
system of practice/cure. These fi gures are
very high and similar to the fi gures quoted in
WHO report on traditional medicine (1). This
report mentions that in developing countries
40 to 80% of the population seeks traditional
medical systems (1). In this study, ‘traditional
healer’ refers to individuals who offered faith-
based services either from Hindu temples,
Muslim dargahs or similar kind of intervention
from their homes. We did not study the Indian
systems of medicine like Ayurveda, Unani or
Siddha medicinal systems. Inclusion of these
systems might have increased the prevalence
of ‘non-psychiatric’ setting consultation.
There was no signifi cant difference between
Archives of Indian Psychiatry 13(2) October 201123
patients who visited traditional healer and those
who did not as regards socio-demographic
characteristics. Age, sex, family type, domicile,
monthly income, were not associated with
traditional healer consultation. No gender
disparities were associated with traditional
healing consultation, even though it has been
well documented that males get preference in
conventional/alternative mental health services
utilization. Higher education was associated
with visiting traditional healer. Out of the 15
patients who had studied beyond S.S.C. (10 th
grade), 13 had gone to the traditional healer
(p=0.05). This result needs to be very carefully
interpreted. People with higher education
may be more motivated to relieve their stress
and hence they may have more consultations
even with traditional healers also. It is very
diffi cult to generalize from this fi nding that
higher education leads to help seeking from
traditional healers. Of all the diagnoses,
possession syndrome, and schizophrenia were
signifi cantly associated with traditional healing
consultation.
All 22 patients who had possession syndrome
were fi rst taken to the traditional healer and it is
easy to understand this as possession syndrome
is a culture-bound disorder and traditional
healer is largely viewed as the best resource
for such kind of complaints. Schizophrenia
and mental retardation are chronic illnesses
and our fi nding of signifi cant association with
traditional healing is consistent with the one
reported by Ngoma et.al (12). They reported that
traditional healers’ patients had long-standing
complaints and the primary care patients had
more acute complaints. Although caution is
needed when making dynamic inferences from
cross-sectional data, traditional healers would
seem often to be a last resort for patients with
persistent problems who were presumably
dissatisfi ed with the outcome of previous
consultations with biomedical providers (12).
In case of patients suffering from chronic
mental health problems, higher referrals to
traditional healers may be because of caregiver
burnout.
In the light of this information it is very
important to identify the reasons for such
widespread use of traditional healers for
mental health problems. In the developed
world, common mental disorders are costly
and disabling disorders, which present often in
primary care but are rarely recognised or treated
(16). In developing countries, broad use of
traditional medicine is often attributable to its
accessibility and affordability. In Uganda, for
instance, the ratio of traditional practitioners
to population is between 1:200 and 1:400.
This contrasts starkly with the availability of
allopathic practitioners, for which the ratio is
typically 1:20 000 or less (1). In rural Tanzania
the ratio of doctors to population is 1:20000
whereas that of the traditional healers is 1:25
(4). Moreover, distribution of such personnel
may be uneven, with most being found in cities
or other urban areas, and therefore diffi cult for
rural population to access (1). Corresponding
fi gures for India are not available, but it may
not vary much.
Psychiatric consultations are free in general
hospitals, but consultation time is short, with
little opportunity to discuss symptoms or
receive explanations about health problems.
Mental disorders are often perceived as a
source of misfortune; ancestors and witches
are believed to have a crucial role in bringing
them about. Such disorders may be viewed in
terms of magical, social, physical and religious
causes, but rarely as diseases within the Western
biomedical paradigm (17). Traditional healers’
rituals are linked to these belief systems and
patients may think that this can result in over-
all well-being.
Limitations
The sample for this study comes from the
hospital setting, which has resulted in an
important problem related to external validity
of the study. It is diffi cult to generalize the
results of this study to the community as the
patients coming to the psychiatric hospital
may be essentially different than the patients
in the community who have not come to the
hospital. The prevalence for seeking help from
traditional healer may be higher in community
Archives of Indian Psychiatry 13(2) October 2011 24
References
1. WHO Traditional Medicine Strategy 2002–
2005. Available at http://whqlibdoc.who.int/
hq/2002/WHO_EDM_TRM_2002.1.pdf
and accessed on September 29, 2008
2. Ontario Aboriginal Health Advocacy
Initiative, Manual on traditional
healers-1999.
3. Ben-Tovim DI. Therapy managing in
Botswana. The Australian and New Zealand
journal of psychiatry. 1985 Mar;19(1):88-
91.
4. Swantz L. The Medicine Man Among the
Zaramo of Dar-es-Salaam. Dar-es-Salaam:
University Press. 1990.
5. Patel V, Mann A. Etic and emic criteria for
non-psychotic mental disorder: a study of
the CISR and care provider assessment in
Harare. Social psychiatry and psychiatric
epidemiology. 1997 Feb;32(2):84-9.
6. Centre for Advocacy in Mental Health
(CAMH). Health and healing in western
Maharashtra- the role of traditional healing
centers in mental health service delivery.
2006.
7. Gandhi P, Bhatt S, Tilwani M, Vankar GK.
Religious Beliefs and Traditional Healing
Practices among Psychiatric Patients Paper
presented at the 50th Annual Conference of
Indian Psychiatric Society at Jaipur. 1998.
8. Kulhara A. Psychopathology. 2000;33:62-
9. Sethi BB, Trivedi JK, Shitholey P.
Traditional healing practices in Psychiatry.
Indian Journal of Psychiatry. 1977;13:5-13.
10. Johri N. Religious beliefs, practices
experiences among psychiatric patients
Unpublished MD (Psychiatry) Dissertation,
Gujarat University. 2003.
11. Kakar S. Shamans, Mystics and Doctors.
A Psychological Inquiry into India and its
Healing Traditions. 1982.
12. Ngoma MC, Prince M, Mann A. Common
mental disorders among those attending
primary health clinics and traditional
healers in urban Tanzania. Br J Psychiatry.
2003 Oct;183:349-55.
setting for the individuals suffering from
mental health disorders. The other limitation of
this study is the sample size. With 201 patients
this study did not have enough power to
detect socio-demographic and disease related
differences between patients who had visited
traditional healers and those who had not.
Implications
Many traditional healers seek continued or
increased recognition and support for their fi eld.
At the same time many allopathic medicine
professionals, even those in countries with a
strong history of traditional medicine, express
strong reservations and often frank disbelief
about the purported benefi ts of traditional
medicine (1). Communities consistently and
predictably choose traditional healers, often as
fi rst choice of help seeking. The community
logic of this choice has to be understood before
evaluation of the choice (6). Patients with
common mental disorder constitute a large part
of the workload of both psychiatric hospitals
and traditional healers. There is little time,
particularly in hospital setting, to explore the
possible psychological basis of the complaints
related to common mental disorders, or to
investigate the wider family and social context
of the disorder (12). Traditional healers may be
better placed in this respect; it would certainly
be interesting to know more of their approach to
these cases in terms of their formulation, their
management and the treated outcome. With the
support of the formal health system, indigenous
practitioners might become important agents
in organising efforts to improve the mental
health of the community. Traditional healers
may have much to offer, and could usefully
participate in joint training programmes in
medical schools (12). Further studies especially
with qualitative/ethnographic design should be
undertaken to address the questions related to
role of traditional healers in providing mental
health services and what are the community
perceptions regarding traditional healing vis-à-
vis modern psychiatric practice.
Archives of Indian Psychiatry 13(2) October 201125
16. Ormel J, VonKorff M, Ustun TB, Pini S,
Korten A, Oldehinkel T. Common mental
disorders and disability across cultures.
Results from the WHO Collaborative
Study on Psychological Problems in
General Health Care. Jama. 1994 Dec
14;272(22):1741-8.
17. Ndetei DM, Muhangi J. The prevalence and
clinical presentation of psychiatric illness
in a rural setting in Kenya. Br J Psychiatry.
1979 Sep;135:269-72.
13. Salan R, Maretzki T. Mental health services
and traditional healing in Indonesia: are the
roles compatible? Culture, medicine and
psychiatry. 1983 Dec;7(4):377-412.
14. Raguram R, Venkateswaran A, Ramakrishna
J, Weiss MG. Traditional community
resources for mental health: a report of
temple healing from India. BMJ (Clinical
research ed. 2002 Jul 6;325(7354):38-40.
15. SAS Version 9.2. SAS Institute, Cary, NC.
2008.
Dr.Rahul R. Shidhaye M.D.(Psy) M.H.S.(Mental Health)*
Faculty
Public Health Foundation of India
Indian Institute of Public Health,
Opp. Nalanda College,
Vengal Rao Nagar,
Hyderabad. 500038.
e-mail:[email protected]
cell:: +919848520340
Dr.G.K.Vankar MD,DPM
Professor and Head
Dept. of Psychiatry
B.J. Medical College and Civil Hospital,
Ahmedabad 380016
*Correspondence
Archives of Indian Psychiatry 13(2) October 201126
Original Article
Newspaper Portrayal of Psychiatry
Laxeshkumar Patel
G. K. Vankar
AbstractMedia reporting have a direct impact on both community attitudes towards mental illness and suicide attempts. ! is study examined how suicide and mental health are portrayed in the me-dia in Baroda.In this cross sectional study of " ve daily newspapers, total 350 cuttings were analyzed, about 2/3 had suicide theme and 1/3 had non-suicide theme.In portrayal of suicide theme they dramatized/sensationalized the headline in about half cut-tings 109(47.2%), used outdated, negative or inappropriate language in 38(16.5%) cuttings, did not mentioned about the helpline in 230(99.6%) cuttings, detailed discussion of the method used was given in 89(38.5%), oversimpli" cation of reason of suicide done in 104(45%) cuttings and in 217(93.9%) cuttings the bereaved were not interviewed.In portrayal of non-suicide theme, inaccurate headline were in 26(21.8%) cuttings, headline or content was dramatic or sensational in 51(42.9%) cuttings, outdated or negative language used in 18(15.1%) cuttings. 73(61.3%) cuttings have disclosed that a particular individual has a mental illness or identi" ed the person by name. About a quarter reinforced negative stereotype about mental illness. ! ere were 49(41.2%) cuttings that emphasis the illness rather than per-son.Key words: media portrayal, suicide, mental health
Introduction
! e expansion and implementation of di# erent types of media-derived information transmission have driven far-reaching changes for both individuals and society, with particular implications for children and adolescents.Most studies suggest that the media’s depiction of mental illness and suicide is mostly negative, inaccurate and unhelpful2-14. ! is is an important " nding as media portrayal may be an important in$ uence on community attitudes towards mental health issues 15. However, not all results have been negative. Two recent studies of non-" ction media found that the vast majority of items were of good quality, the only failing being the absence of information on appropriate services 16, 17. Methodological problems with previous research include the representativeness of the samples, retrospective
collection of data, and the use of secondary data, which meant that researchers could not independently evaluate the importance or de" nition of mental illness to the item14. A related problem is that few studies have used standardized instruments with explicit criteria 16. Finally, although most have been restricted to non" ction media, some have been restricted to print media, while others have included electronic media as well. ! ese di# erences make it di& cult to compare studies 3,4,16. ! ere is only one study from Canada on reporting of mental illness in the last decade and half, and this was restricted to a single newspaper in Calgary 16.
Although there have been studies of the e# ect of reporting on suicide, there had been little in the way of systematic collection of the extent, type and quality of reporting on the issue 4. ! e only Canadianstudy to examine
Archives of Indian Psychiatry 13(2) October 201127
the portrayal of suicide by the media were conducted 20 years ago 4: one was a survey of editor’s policies on the portrayal of suicide that showed that a story was most likely to printed if it involved a prominent person, occurred in a public place, or was done by an unusual method 18; the other compared the frequency of suicide items in Toronto news papers before and a! er suicides in ‘epidemic’ and ‘non-epidemic’ suicide years. Neither speci" cally examined the content of actual news items 19. In response, the Prevention Promotion and Advocacy (PPA) network committee and anti Stigma / Discrimination Working Group of the Department of Health in Nova-Scotia undertook a six month survey to monitor all non-" ction media items (newspaper, radio and television) that referred to suicide, mental health and illness.
Media professionals are constantly required to make judgment about what to publish and how to tell the story, but when we make decisions that could in# uence the behavior of others whom we do not know, it is incumbent upon us to ensure that our judgments are as informed as might reasonably be expected. If there is ex-pert advice available we have a responsibility to listen. $ e power and in# uence of the media on sui-cidal behavior has been a key subject of debate over many years. Public attitudes to suicide (de-criminalization of suicide and reduction of its taboo status) have become more empathic, and although suicide rates are falling, they remain high. Although psychiatrist should encourage open discussion of suicide among individuals and in a wider media context, there are speci" c concerns when " ctional and real suicidesare represented in the media. Schmidtke & Hafner (1989), in an extensive review with 131 refer-ences, examined the in# uence of the mass me-dia, predominately news media, on suicidol-ogy. $ e evidence for imitation (with regard to explicit details of method and celebrity sui-cides) proved conclusive, thus retrospectively justifying the American Academy of Medicine’s " rst proposal for press constraints in 1911 20.
Two subsequent studies have examined the re-lationships between print media and the choice of suicide method. Etzersdorfer et al 1992 21 showed how attempted and completed suicides on the Viennese underground railway have been reduced to single " gures a! er the media were given guidelines for reporting suicide.$ e occurrence of imitative suicides following media stories is largely known as the “Werther e% ect” derived from the impression that Goethe’s novel ‘$ e Sorrows of Young Werther’ in 1774 triggered an increase in suicides, lead-ing to its ban in many European states. Re-search on the “Werther e% ect” was advanced by the systematic work of Phillips 22-27, whose research consistently found a strong relation-ship between reports of suicide in newspapers or on television and subsequent increases in the suicide rate.
$ e magnitude of the increase in suicides fol-lowing a suicide story is proportional to the amount, duration, and prominence of media coverage. A “dose-response” relationship has recently been reported by Etzersdorfer, Vora-cek, and Sonneck 28 in an examination of the relationship between the regional distribution of a tabloid newspaper’s coverage of a celebrity suicide by " rearms in Austria and an increase in " rearm suicides. Nearly 40% of the variance in changes in suicide by " rearm was attribut-able to the di% erential distribution of the tab-loid.
$ is is consistent with the dose-response e% ect " rst reported by Phillips 24. In a quantitative analysis of 293 " ndings from 42 studies, Stack 29 found that studies assessing the e% ect of the suicide of an entertainer or political celebrity were 14.3 times more likely to " nd a “copycat” e% ect than studies that did not. Furthermore, studies based on real suicides in contrast to " ctional stories were 4.03 times more likely to " nd an imitation e% ect.
Aims and objectives
1. To investigate how the non-" ction media in Baroda portray both suicide and mental
Archives of Indian Psychiatry 13(2) October 2011 28
health.2. To know the attitude of media people
towards psychiatry.
Material and Methods
Sample
Five newspapers (three Gujarati newspapers and two English newspapers) over a period of 3 months were seen for the stories related to mental health or suicide news and such stories related to the mental health or suicides were collected in form of cuttings.Following newspapers were included: Gujarati( Gujarat Samachar, Sandesh, Divya Bhaskar) and English ( ! e Times of India and! e Indian Express).
Instrument
! e newspaper cuttings were analyzed with the checklists. Both checklists were derived from previous research in Australia, Achieving the balance: A resource kit for Australian media professional for the reporting and portrayal of suicide and mental illness 1.! ese checklists were also in" uenced by ‘WHO guidelines for reporting suicide’ and by ‘guidelines on coverage of suicide’ given by Hong Kong Journalist Association.! e checklists include 18 questions to determine reporting of suicide news and 9 questions to determine reporting of non-suicide news.
Screening:
Each question has three alternative answers: ‘yes’, ‘no’, ‘not applicable’. Response of ‘yes’ and ‘no’ indicate poor and good quality of portray-al respectively, except in the dimensions of the ‘helpline’, ‘suicide note included’ and ‘bereaved interviewed’, where the direction was reversed.
Media professionals were interviewed for the following questions:1.How do they portray suicide or mental health issues? Is there any quali# ed person or group to decide regarding the same?2.Do they have any speci# c guidelines for printing news regarding suicide or mental
health issues?3.How do they decide to print a photograph of suicide scene while reporting the news of suicide?4.What is the quali# cation of the reporter?5.What are the bases for the statistical records of suicide?
Analysis
! e statistical analysis of the collected data was done by using EPIINFO 6.0 so$ ware
Results and Discussion
1. Frequency of news
Five newspaper articles on themes of mental health were analyzed. We have received 350 newspaper cuttings. Out of which 116(33.1%) were from Gujarat Samachar, 92(26.3%) were from Sandesh, 98(28%) were from Divya Bhaskar, 19(5.4%) were from ! e Times of India and 25(7.1%) were from ! e Indian Express.Gujarat Samachar, Sandesh, Divya Bhaskar are the news papers in the regional language (Gujarati) which covers major parts of the cuttings, each comprise more than about 25%. While the rest two newspapers ! e Times of India and ! e Indian Express, which are in the English language covers small part of the total cuttings, each comprise less than about 8%.
Informative theme was found in 33(9.4%), ‘question-answer’ theme was found in 1(0.3%) cutting only. ‘Law and order’ theme was found in 19(5.4%), ‘substance’ theme was found in 32(9.1%) and 34(9.7%) cuttings fell into ‘others’ category.
3. Media portrayal of suicide theme:
About a half of the cuttings 109(47.2%) had headline or content unnecessarily dramatic or sensationalized (Table – 1).Examples:1.Love-birds of Vadodara went to jump from Saradar Bridge at Bharuch but…!!
Archives of Indian Psychiatry 13(2) October 201129
Table 1: Newspaper portrayal of Suicide
theme(%)
(N=)
Item Yes No No Answer
Headline inaccurate or inconsistent with the items focus? 10.0 90.0 0
Headline or content unnecessarily dramatic or sensationalized 47.2 52.8 0
Use of language that is outdated, negative or inappropriate? 16.5 83.5 0
Disclosure that a particular individual has a mental illness or identifying the person by name?
93.9 5.6 0.4
Medical terminology used inaccurately? (Not in correct con-text)
0.9 14.3 84.8
Reinforcement of negative stereotypes about mental illness? 7.4 68.5 76.2
Does the item emphasize the illness rather than the person? 5.6 67.5 26.8
Does the item imply that all mental illnesses are the same? 2.6 55.6 42.2
Is direct/indirect suggestion of help given? 0.4 99.6 0
Use of the word “suicide” in the headline? 80.1 19.9 0
Inappropriate language? (frequent use of the term committed suicide or that suicide was a desirable outcome)
23.4 76.6 0
Any reference to the fact that the person who died by suicide was a celebrity?
10.0 90.0 0
Is a photograph, diagram or footage of the suicide scene, pre-cise location or method used in the item?
17.3 82.7 0
Is there detailed discussion of the method used? 38.5 61.5 0
Suicide portrayed as ‘merely a social phenomenon’ as opposed to ‘being related to mental disorder’?
74.5 7.4 18.2
Are bereaved interviewed? 6.1 93.9 0
Over simpli! cation of the reason of suicide? 45.0 31.6 23.4
Is the suicide note included? 13.4 86.6 0
Table 1: Newspaper portrayal of Suicide theme
Item Yes No No Answe
r
Headline or content unnecessarily dramatic or sensational-ized
42.9 57.1 0
Use of language that is outdated, negative or inappropriate? 15.1 84.9 0
Disclosure that a particular individual has a mental illness or identifying the person by name?
61.3 34.5 4.2
Medical terminology used inaccurately? (Not in correct context)
10.1 48.7 41.2
Reinforcement of negative stereotypes about mental illness? 21.0 52.9 26.1
Does the item emphasize the illness rather than the person? 41.2 50.4 8.4
Does the item imply that all mental illnesses are the same? 12.6 74.8 12.6
Is direct/indirect suggestion of help given? 20.2 79.8 0
Archives of Indian Psychiatry 13(2) October 2011 30
2.Neeta with mehndi on her hands and heart-broken in love – Vipul could not live together so… ! ey died together…!!3.One female with her seven-year old son had jumped from the 20th # oor of a building but perhaps it was the warmth of mother’s lap that provided a shield and the child survived the mishap.! ousands of people crowded together at the wall of Kankaria Lake to see the corpses of this loving couple # oating in a romantic manner. ! e way they were together in the water of the lake was telltale sign of their deep love. Truly, this incident brought back memories of song which meant…. ‘Oh….!! A pair of swans got separated!!’We found 38(16.5%) cuttings had language that was outdated, negative or inappropriate (Table 1).Examples:1.Terror at the incident of mother wrapped in $ re along with daughter!!2.A dive of death from Ellis Bridge by a retired teacher.3.Terror prevails as they bid good-bye to this unjust world by jumping into the river.4.Unsuccessful attempt of suicide.
Disclosure that a particular individual has men-tal illness or identifying the person by name found in 217(93.9%) Direct or indirect sugges-tion of help was given only in one cutting out of 231 cuttings. It was not given in 230(99.6%) cuttings. Use of the word ‘suicide’ in the head-line were in 185(80.1%) cuttings and 54(23.4%) cuttings had frequently used the term commit-ted suicide or that suicide a desirable outcome. In 23(10%) cuttings they mentioned that the person who died by suicide was a celebrity. Photograph, Diagram or Footage of the suicide scene was given in 40(17.3%) cuttings.
89(38.5%) cuttings had reported the detailed discussion of the method used (Table – 1).Examples:1.Ravji had made noose of the sari twice. One more piece of the sari was found under-neath Ravji’s dead body. A loop was made at the end of this piece looking at which it seems
that Ravji had made noose before committing suicide, but since the sari got torn, he made a noose once again, hung it on the tree and com-mitted suicide.2.Today morning at 7:00 o’clock when Ramesh-bhai was brushing his teeth outside his house, as soon as she got a chance, his wife poured kerosene over the body of her daughter Dak-sha and lit it with a matchstick. A& er that she poured kerosene over her own body also and lit it with a matchstick. 3.A night, a suicide attempt was made by add-ing a poisonous insecticide powder, used to kill ants and insects into a bottle of the cold drinks- thumps up and then gulping down the drink.4.On 19th, at night she hung herself by tying a duppatta on the hook of the fan and committed suicide.
Suicide portrayed as ‘merely a social phenom-enon’ as oppose to ‘being related to mental dis-order’ in 172(74.5%) cuttings (Table – 1).Examples:1. Report of suicide committed by a 21 year married woman due to torture at her in law’s place.2.Young female jumped into the river as her lover ditched her…3.Depressed due to the worries of seven daugh-ter- father committed suicide…. “due to poor $ nancial condition, the daughters could not get married. Hence he has decided to hang himself to death.”4.A woman, who married second time, again became a widow- hence she poisoned herself.
Oversimpli$ cation of reason of suicide report-ed in 104(45%) cuttings (Table – 1).Examples:1.…due to a quarrel regarding eating pu* ed rice.2.…because he felt hurt on losing rupees 25.3.…as husband did not allow her to serve him food and took it by him.4.…as father scolded him regarding going for the marriage.5.… On not getting a deserving matched for marriage.6.… As the husband denied her request of
Archives of Indian Psychiatry 13(2) October 201131
visiting her parents.7.… As the mother refused to go outdoor for playing.In 217(93.9%) cuttings the bereaved were not interviewed, even though they are at height-ened risk of suicide themselves (Table – 1).Reporting was more generally appropriate across the other dimensions.
4. Media portrayal of non-suicide theme
Media items were of poor quality in seven out of nine dimensions measured as compared to the portrayal of suicide theme. ! e reporting was good in case of non suicide themes in dimensions of ‘disclosure that a particular individual has a mental illness or identifying the person by name’ and ‘direct or indirect suggestion of help given’ as compared to the suicide theme portrayal.Inaccurate or inconsistent headline were seen in 26(21.8%) (Table –1).Examples:1.Beautiful lady caught in a drunken state: chakchar.2.Black use of the dilutor of white in 3. Net chatting: an innocent prank or a little bit of betrayal?
Unnecessarily dramatic or sensationalized headline or content was given in 51(42.9%) cuttings (Table–2)
Examples:1.It is unfortunate for the society and a matter of shame that in Gandhiji’s Guajarat, little kids, innocent as " owers, are made to drink (pockets of) country liquor and being dragged into this business.2.In the cradle itself, he showed physical and mental signs of being a female and those signs have grown today from a tiny plant to a huge tree and yesterday’s guy has become a gal today.3.! is new technique, which can make hearts fall in love even across seven oceans sometimes, proves to be a double edged sword. Net chatting increased by leap and bounds and there a# er Rima started remaining clung to
the computer till late night! And then she goes so intoxicated in love that net chatting became her need just like food, water or alcohol.
18(15.1%) cuttings used inappropriate, negative or outdated language to portray non-suicide themes and 73(61.3%) cuttings disclosed that a particular individual had a mental illness or identi& ed the person by name. In 12(10.1%) cuttings had used medical terminology inaccurately to portray non-suicide themes. About a quarter reinforced negative stereotype about mental illness.
! ere were 49(41.2%) cuttings that emphasis the illness rather than person. 15(12.6%) cuttings implied that all mental illnesses are the same (Table – 2).
5. Comparison of suicide theme portrayal in
various newspapers
1.Only 2 reports of suicide have been made by ‘! e Times of India’ and 18 by ‘! e Indian Express’ as compare to 68, 66, 77 reports in ‘Gujarat Samachar’, ‘Sandesh’, ‘Divya Bhaskar’ respectively over a period of 3 months.2.Out of 18 cuttings, ! e Indian Express had not emphasized the illness in any of the articles and had emphasized the person in 16(88.9) cuttings.3Out of all cuttings in all the newspapers, ‘suggestion of help’ had given in only one cutting in Sandesh.4.In each newspaper, more than 74% of the cuttings contained the word ‘suicide’ in the headline.5.Indian Express had avoided the details of the method used to commit suicide in 83.3% of the cuttings as compare to 57.4%, 65.2%, 6.57.1% and 50% in Gujarat Samachar, Sandesh, Divya Bhaskar and ! e Times of India respectively.7.Out of all 5 & ve newspapers, ‘over simpli& cation of the reason of suicide’ had given maximally in Divya Bhaskar (51.9%).8.Sandesh had included suicide note maximally (10.4%).
Archives of Indian Psychiatry 13(2) October 2011 32
6.Comparison of non-suicide theme
portrayal in various newspapers:
1.Headline was inaccurate or inconsistent with the item focused in maximum number of the cuttings in ! e Times of India (29.4%).2.Outdated, negative and inappropriate language was used the most in Divya Bhaskar (23.8%).3.Medical terminology was used inaccurately in Divya Bhaskar the most (23.8%).4.Negative stereotypes of mental illness had reinforced in most of the cuttings of ! e Indian Express (42.9%) and Divya Bhaskar (38.1%).5.Emphasis the illness rather than the person were found in most of the cuttings of Gujarat Samachar (54.2%).6.Divya Bhaskar had depicted maximally that all mental illnesses are the same in 23.8% cuttings.7.Gujarat Samachar had given direct or indirect suggestion of help in 33.3%(maximum) cuttings while Divya Bhaskar had provided it in only 4.8%(minimum) cuttings.
Interview with members of newspaper
editor o! ce:
Newspaper-1
Newspaper-1 did not have any guideline to report mental health issues or suicide and some of their reporters did not have quali" cation of journalism. Newspaper-1 even did not feel the need for guideline or quali" ed reporters for the portrayal of mental health or suicide. Newspaper-1 was reporting news related to mental health or suicide in very professional way and Newspaper-1 was least concerned with impact by their reporting about mental health issues. ! ough Newspaper-1 told us that Newspaper-1 was planning to start one column regarding mental health awareness but there was no concrete planning as such. Reporting of newspaper-1, about mental health issues and suicide was mainly disappointing. Newspaper-1 had never given helpline and Newspaper-1 never tried to understand the reason for suicide, never interviewed the bereaved person. Newspaper-1 had emphasized on person rather the illness. Newspaper-1 had
also used inappropriate language in signi" cant number of cuttings.
Newspaper-2
Overall, Newspaper-2 emphasized more on his knowledge and his newspaper but quite disappointingly, Newspaper-2 seemed less concerned with the media portrayal of mental health and suicide issues. Newspaper-2 did have any speci" c guideline for reporting mental health or suicide. Newspaper 2 had reported mental health related issues and suicide with inappropriate language in signi" cant number of cuttings. Newspaper 2 had never given direct or indirect suggestion for the help. Most of the time, Newspaper 2 had not tried to understand the actual reason for suicide, had not interviewed the bereaved and had done over simpli" cation of the reason for suicide. Newspaper 2 had reported headline unnecessarily in dramatic or sensationalized manner.
Newspaper-3
Newspaper-3 had reported suicide cases very selectively, without over simpli" cation of reason for suicide, dramatization or inappropriate language. ! ough Newspaper-3 told us that they don’t print photograph of the suicide scene, it was given in half of the cuttings. Newspaper-3 emphasized person more than the illness. Newspaper-3 asserted that the concern for good reporting about the illness or suicide may propagate awareness in the society about mental health. Newspaper 3 also lack of guideline for reporting mental health issues or suicide. Newspaper-3 also had not given direct or indirect suggestion for help.
Discussion
! e media are an important source of information on both suicide and mental illness: importantly, reporting can have a direct impact on both community attitudes towards mental illness and suicide attempts, with the strongest evidence for non-" ctional media 4, 6. ! is study was therefore designed to examine how suicide
Archives of Indian Psychiatry 13(2) October 201133
and mental health and illness are portrayed in the media in a standardized way across Baroda. To our knowledge this is the ! rst time such kind of study considered in India.Our results on the portrayal of mental illness are very similar to those from Australia (instead of our 18 dimensions, they used 9 dimensions to check portrayal of suicide) and showed that media reporting on mental illness was predominately positive. " ere are in contrast to other research where media reporting of mental illness has mainly been negative 3, 5-14." e result for suicide reporting was less encouraging and this is consistent with the existing literature. Inappropriate, dramatic language is a central concern, as particular framing devices and inappropriate language may together contribute t the stigma associated with mental illness.
Another area of concern was the absence of information on where to get help in over 99% of suicide items: this is almost identical to the ! ndings from the Australian study, suggesting that this problem is not restricted to Nova Scotia 17.
" e nature of the reporting of these subjects varies considerably, as does the quality. In general, good items outnumber poorer items, particularly in the case of mental health and illness. However, there are still opportunities for improving the way in which the media report and portray suicide and mental health and illness.
Newspaper media have a choice in the way they frame stories about suicide, and mental health and illness. " e choice is not an issue of accuracy or objectively, but it does have serious rami! cations for the ways in which reader may interpret news and information about suicide, and mental health and illness. If the right choices are made, they can help to de-stigmatize mental illness in our community and improve the lives of many people with mental illnesses.
It is important to develop guidelines with the people who will use them; we should try
to involve editors and opinionmakers in the provincial media in their development 30. " ere is evidence from both Australia and Canada that local media may be easier to in# uence than national or international media 16." ere should be longer-term strategy to in# uence the education of journalism students. We should particularly focus on the inclusion of details of how to get help, and on suicide.Journalists would not tell a depressed relative or friend ways of committing suicide in graphic details. Do they not owe the same duty of care to their readers?
Strengths:
" e only study of its kind done in India.Reliability of study maximized by analyzing cuttings by one researcher.
Limitations:
1. " e comparatively low number of articles on suicide, and thus our sample size for items on mental illness was also much smaller than that of a similar study in Australia 1. However our numbers are comparable of most other studies that have considered media coverage of mental illness 5, 16.
2. " ough study conducted on newspapers, having maximum number of reader in the Baroda at the time of study, it did not include other magazines and electronic media in study so it might not represent the media as a whole.
Acknowledgement:
We acknowledge the support from Dr. P. R. Vesuna for his facilitation, Mrs. Asha Patel for assisting in computer work, Dr. Kunal Bhardwaj and Dr. Nilima Shah for their help in collecting newspaper cuttings. Without whose support our work could not get materialized.
References
1. American Heritage Dictionary, 2nd College Edition. New York, Dell, 1983.
2. Department of Health and Aged Care. Achieving the balance: A resource kit for
Archives of Indian Psychiatry 13(2) October 2011 34
Australian media professionals for the reporting and portrayal of suicide and mental illness. Canberra; Commonwealth Department of Health and Aged Care; 1999.
3. Department of Health and Ageing Reporting suicide and mental illness. Commonwealth Department of Health and Ageing; 2002.
4. Pirkis J. Wawick Blood R. Suicide and the media: a critical review. Canberra; Commonwealth Department of Health and Aged Care; 2001.
5. Francis C, Pirkis J, Dunt D, Wawick Blood R. Mental health and illness in the media: A review of the literature. Canberra; Commonwealth Department of Health and Aged Care; 2001.
6. Day DM, Page S. Portrayal of mental illness in Canadian newspapers. Can J Psychiatry, 1986; 31:813-817.
7. Matas M, el-Guebaly N et al. A mental illness and the media: part 2. Content analysis of press coverage ofmental health topics. Can J Psychiatry; 1986, 31:431-433.
8. Allen R, Nairn RG. Media depiction of mental illness: an analysis of the use of dangerousness. Aust N Z J Psychiatry, 1997, 31: 375-381.
9. Nairn R. Does the use of Psychiatrists as sources of information improved media depictions of mental illness? A pilot study. Aust N Z J Psychiatry 1999, 33:583-589.
10. Wilson C, Nairn R, et al. A. constructing mental illness as dangerous: A pilot study. Aust N Z J Psychiatry 1999, 33: 240-247.
11. ! ornton J, Wahl O. Impact of a newspaper article on attitudes toward mental illness. Journal of Community Psychology 1996; 24:17-25.
12. Steadman H, Cocozza J. Selective reporting and the public’s misconceptions of the criminally insane. Public Opinion Quarterly 1977, 41:523-533.
13. Angermeyer MC,Matschinger H.! e e" ect of violent attacks by schizophrenic persons on the attitude of the public towards the mentally ill.Soc Sci Med;1996,43:1721-1728.14
14. Coverdale J,Nairn R,Claasen D. Depictions of mental illness in press media: a prospective national sample.Aust N Z J Psychiatry:2002,36:697-700.
15. Hoggart, R. ! e Uses of Literacy London: Pelican books 1958.
16. Stuart H. Stigma and the daily news: evaluation of a newspaper intervention. Can J Psychiatry 2003, 48:651-656.
17. Francis C, Pirkis J Wawick Blood R et al. ! e portrayal of mental health and illness in Australian non-# ction media. Aust N Z J Psychiatry: 2004, 38:541-546.
18. Pell B and Watters D. Newspaper policies on suicide stories. Canada’s Mental Health, 1982; 30:8-9.
19. Littmann S K. Suicide epidemics and newspaper reporting. Suicide and Life ! reatening Behavior, 1985, 15:43-50.
20. Schmidtke A and Hafner H. Public attitudes towards and e" ects of the mass media on suicidal and deliberate self harm behavior. In Suicide and its Prevention 1989.
21. Etzersdorfer E, Sonneck G and Nagel-Kuess S. Newspaper reports and suicide. New England Journal of Medicine, 1992; 324,502-503.
22. Bollen K. A. and Phillips D. P. Suicidal motor vehicle fatalities in Detroit: A replication, American Journal of Sociology, 1981; 87,404-412.
23. Bollen K. A. and Phillips D. P. Imitative suicide: A national study of the e" ect of television news stories. American Sociological Review, 1982, 47,802-809.
24. Phillips D. ! e in$ uence of suggestion on suicide: Substantive and theoretical implications of the Werther e" ect. American Sociological Review, 1974, 39,340-354.
25. Phillips D. Suicide, motor vehicle fatalities, and the mass media: Evidence towards a theory of suggestion. American Journal of Sociology, 1979,84, 1150-1174.
26. Phillips D. and Carstensen L.L. Clustering of teenage suicide a% er television news stories about suicide. New England Journal of Medicine, 1986, 315,685-689.
27. Phillips D. and Carstensen L.L ! e e" ect
Archives of Indian Psychiatry 13(2) October 201135
of suicide stories on various demographic groups 1968-1985. In R. Maris (Ed.), understanding and preventing suicide: plenary papers of the ! rst cmbined meeting of the AAS and IASP 1988, (pp. 100-114). New York: Guilford.
28. Etzersdorfer E, Voracek, M, Sonneck G. A dose response relationship of imitational suicides with newspaper distribution, 2001.
29. Stack S. Media impacts on suicide: A quantitative review of 293 ! nding Social Science Quarterly, 2000, 81, 957-971.
30. Slaven J, Kisely S. " e Experience primary prevention of suicide project. Aus N Z J Psychiatry, 2002; 36:617-21.
Sources of support : None Con! icts of interest: None
Dr.Laxeshkumar Patel
Resident DoctorDept. of PsychiatryMedical College and S.S.G. Hospital,Baroda
Dr.G.K.Vankar
Professor and HeadDept. of PsychiatryB.J. Medical College and Civil Hospital,Ahmedabad 380016
Correspondence:
Dr. Laxeshkumar PatelF-6, Dream Plaza Flats,Camp Road, Shahibaug,Ahmedabad 380 004.Cell: 98258 47475E-mail: [email protected]
Archives of Indian Psychiatry 13(2) October 2011 36
Original Article
Effi cacy of Social Skills Training
as Intervention for Stuttering for adolescents v/s adults
Sadhana Deshmukh
Anuradha Sovani
Abstract
The purpose of the study was to examine the effi cacy of Social Skills training in the treatment of
stuttering. The sample consisted of 15 adolescents and 15 adults. The mean age of the adolescents
and adults was 17 years and 26.6 years, respectively.
Each of the participants were required to attend ten sessions of one hour each, on a weekly basis.
The therapy was conducted in accordance to the treatment module, which had a structured plan.
The dependant variables were situational anxiety, communication attitude, assertiveness, self-
esteem and fl uency. These were assessed using Speech Situational Checklist-(short form), the
S-24 Communication Attitude scale, Rathus Assertiveness Schedule, Rosenberg Self-Esteem
Scale and the Speech Severity Index, respectively.
Analysis of results indicated that adults showed more improvement than adolescents with respect
to most of the variables. Hence, it can be concluded that the social skills training approach proved
to be more effi cacious overall in the treatment of adults who stutter, as compared to adolescents,
especially when dealing with situational anxiety.
Key words: Stuttering, Social Skills Training, Adults, Adolescents
Introduction
The ability to interact with others in a competent
way is of great importance to all humans. Ease
in both verbal and non-verbal communication
is a vital factor in competent communication.
As a direct result of their impairment, persons
who stutter have a self-imposed restriction
to acquire interpersonal skills necessary for
skilled social functioning or the subtleties of
relationship exchange. There are resultant
avoidances of social exchanges, and inhibitions
in social settings, which further reduce learning
opportunities.
The persons who stutter tend to examine a
number of issues through the prism of their
speech defect. This fi xation on the speech
defect not only results in morbid suffering, but
further impacts communication - both verbal
and nonverbal. As a result of the vicious cycle
of tension, fear leading to helplessness in turn
leading to anxiety and avoidances, the persons
who stutter fi nd themselves depriving and
denying themselves the opportunity to observe
and practice ‘normal’ social interaction skills.
The term Social Skills was coined by a British
social psychologist, Argyle (1) when he
explored analogies between human-machine
and human-human interaction. L’ Abate and
Milan, (2). Libet and Lewinsohn (3) defi ned
social skills as “the complex ability both
to emit behaviours which are positively
or negatively reinforced and not to emit
behaviours that are punished or extinguished
by others.”
Bellack and Hersen (4) refer to social skills as
“an individual’s ability to express both positive
and negative feelings in the interpersonal
context without suffering consequent loss of
social reinforcement…. In a large varietyof
interpersonal context… (involving) ….the
coordinated delivery of appropriate verbal
and non-verbal response.”
Archives of Indian Psychiatry 13(2) October 201137
Birdwhistell (5) , one of the earliest authorities
in the fi eld of non-verbal processes in
communication, estimated that in atypical
dyadic encounter verbal components carry
about one-third of the social meaning of
the situation, while the non-verbal channel
conveys approximately two-thirds. Non-
verbal signals are more important than verbal
in expressing feelings and attitudes. Argyle et
al, (6) elaborate that the non-verbal channels
indicate how a message should be interpreted.
The term non-verbal includes all form of
human communication that is not controlled
by the spoken word. An overlap exists in the
paralinguistic aspects of speech i.e. voice, rate
and volume and other non-verbal information
like body movement, facial expressions and
appearance.
According to Dickson et al (7) there are six
main functions of non-verbal communication:
1. to replace speech
2. to complement the verbal message
3.to regulate and control the fl ow of
communication
4. to provide feedback
5. to defi ne relationships between people
6. to convey emotional states.
It is widely acknowledged that many persons
who stutter demonstrate defi cits in social
skills. These do not appear at the onset, but
become increasingly evident as the normal
process of social communication is disrupted
by the breakdown in fl uency. Awkwardness
experienced by such individuals in social
situations only adds to the social disadvantages
that already exist as a result of their
communication disorder.
There is evidence that interaction between
stuttering children and their families plays
an important role in the development
and maintenance of the problem.
Thereforeaccording to Rustin et al (8) simply
teaching parents of children who stutter, to
reevaluate and make small changes in their
social interaction skills has proved to be a
powerful tool in early remediation of stuttering.
There are many factors that have been shown
to be infl uential and these include observation
and listening skills, rate of speech and reducing
the complexities of interactions.
Sheehan (9) emphasized that stuttering
occurs in communicative situations where
others are present, “it takes two to stutter.”
He described stuttering to be a disorder of
interpersonal self-presentation, occurring via
social context. For the persons who stutter, the
establishment of social relationships within the
family and later outside is central to healthy,
adaptive functioning, according to Rustin
and Kuhr (10) . Prins (11) and Wingate(12)
concur that over the broad range of measures
(self-report, behavioural and observational)
children and adolescents who stutter do show
more diffi culties with social adjustments
than children who are fl uent. These include
poor interpersonal skills, avoidance of social
contact, withdrawal from social interaction and
low rates of initiating social contacts.
According to Bloodstein (13), there are many
factors that are infl uential in the remediation
of stuttering and these include observation and
listening skills, rate of speech, etc. Especially
for older children, where the stuttering has
become more fi rmly established, it begins to
affect their ability to deal with a wide range
of social situations. Repeated experience of
communication breakdown results in persons
who stutter having increasing diffi culties with
social adjustment.
Rustin and Kuhr (10) proposed a hierarchy of
seven distinct skills: observation, listening,
turn-taking, negotiation, relaxation, praise
and reinforcement and problem solving.
The researchers found them to be important
parameters of communication and what enabled
them to become more objective about their own
skills, particularly if used in conjunction with
video recordings. Interpersonal functioning or
defi cits in social skill repertoire of persons who
stutter has been shown to infl uence the ability
to maintain fl uency following therapy (Rustin
Archives of Indian Psychiatry 13(2) October 2011 38
(14) , Rustin and Kuhr (10).
A theoretical and methodical aspect of group
psychotherapy for treatment of stuttering
was developed after researching 200 patients
by Shklovskiy, Krol & Mikhailova (15).
According to them clients below 20 years of
age experience signifi cant problems of status
among peers and lack communicative skills of
older patients. The topics frequently featuring
in the group were: social opinion, professional
growth, relationship with superiors and
subordinates. They felt that persons who stutter
usually examined a number of topics through
the prism of their speech defect. Therefore
low level of awareness of their problems is
determined mainly by a fi xation on their speech
defect. The psychological effect of fi xation on
the speech defect is that it results not only in
morbid suffering and subjective exaggeration
of its manifestation, but alsoa tendency to
selectively and inadequately perceive, interpret
and construct any situation related to spoken
communication. Along with this, there is
also an overestimation of the verbal aspects
of communication. They observed that in the
process of group interaction those people who
stutter do not make much use of non-verbal
behaviour and are not inclined to give any
signifi cance to non-verbal behaviour of others.
In a stutterer, the non-verbal signals of
paralinguistic as well as extra linguistic type
are eclipsed. This gives the speaker, under
the effects of those techniques, a robot like
effect. (It could be either decoding weakness
traced back to reduced eye contact or it could
be encoding weakness–in that the person
who stutters does not produce the signals,
which indicate to his partner that it is now
his turn). Due to the peculiarities in the way
in which the “communicational personality”
of the person who stutters is formed it is
observed that there is not only an absence
of elementary communicative skills, (non-
verbal communication) but also a defi cit of
information necessary for interaction.
Therefore, it is important to deal with the
learning of new role behaviour in its fi ner
non-verbal aspects. Clients must practice
maintaining eye contact, practice to keep calm
in a group as well as to resist any form of time
related pressure.
According to Kraaimaat et al (16) the discomfort
level in social situations, which leads to high
levels of social and speech anxiety in persons
who stutter, calls for anxiety reduction as well
as social skills training. According to them
social skills training is highly recommended
because it includes exposure to social situations
and performing social responses.
Closely related to this is a novel concept by
Shklovskiy et al (15) of ‘communication
portrait’, which prepares and achieves qualitative
change in the client, a sort of psychological
correction. This change involves the extremely
important task of changing the patients’ image
of himself as the subject of communication and
of communication in general. Towards this it
is important to build a detailed communicative
portrait of every patient in the context of
group interaction. Creating communicating
portrait of every patient introduces many new
features into the ideas the group participants
have about their own peculiarities, these ideas
being corrected and expanded. In organizing
this process it is important to give attention
to “micro components” of speech that is;
details of behaviour at the level of a single
communicative act. An important place here
is occupied by various characteristics of non-
verbal communication. (Facial expression,
gestures, voice parameter, and eye contact).
The person who stutters, as he does not link
it with his major problem, does not take non-
verbal communication into consideration.
Communicative portrait is in principle (i.e.
allowing for alterations, additions, detailing)
a comprehensivedescription by the group
of individual features in a participant’s
communicative behaviour. In other words, the
communication portraits refl ect and consider
an essential quality of communication, namely
its wholesome and systematic character .; Krol,
(17).
Archives of Indian Psychiatry 13(2) October 201139
If so much of the information we are
transmitting and receiving is being conveyed
via the non-verbal channel then it becomes
essential to incorporate training of these skills.
Improvement in the non-verbal interaction
skills of the persons who stutter, despite the
defi cits in their verbal skills should increase
the effectiveness of their communication and
in turn maximize the social reinforcement they
receive from others. The consequent rise in the
self-esteem and confi dence levels should also
encourage an increase in their ability to initiate
and maintain conversation, skills which are
vital to successful communication.
Looking at the problem of persons who stutter
from this point of view, social skills training
might be a way of breaking this vicious cycle
as the repertoire of skills one has and uses ap-
propriately goes a long way in contributing to
a satisfying social interaction.
Materials and Method
Sample: Thirty clients who were self-referred
or referred from institutes, hospital and private
clinics and schools. Individuals, who had
been diagnosed as having stuttering, of any
severity, (mild, moderate, severe, profound)
with onset in childhood. They were more or
less equally distributed in the two groups in
terms of severity. The ages ranged from 13 to
40 years, both males and females. Exclusion
criteria were presence of intellectual challenge
or having psychiatric illness, as well as any
organic or neurological disorder.
Tools and defi nitions of variables
Southern Illinois Speech Situational
Checklist (SF-S.S.C) by Hanson, Gronhovd
and Rice (18) - shortened form. Situational
anxiety is a feeling of worry or uneasiness in
certain situations.
S-24 Communication Attitude Scale (S-24)
by Andrews and Cutler (19). Communication
attitude is the client’s self-perception as a
communicator.
Rathus Assertiveness Scale (R.A.S) by
Rathus (20). Assertiveness is an active, direct
and an honest approach, working for win-win
outcome.
Rosenberg Self-Esteem Scale (R.S.E) by
Rosenberg (21). Self Esteem is the value or
worth that one assigns to self.
Stuttering Severity Index (S.S.I) by Riley
(22). Fluency is the smooth fl ow of speech
without repetitions, prolongations or blocks.
Stuttering severity measures disruption of this
fl uency.
Procedure
Each of the clients was subjected to a
detailed intake interview. This standard
format gathered information on, presenting
complaints medical, family, social, academic
and psychological history and any other earlier
intervention. Subsequently, they responded to
the fi ve assessment tools as a part of the pre-
test procedure. The tools used were Speech
Situational Checklist, S-24 Communication
Attitude Scale, Rathus Assertiveness Scale,
Rosenberg Self-Esteem Scale, and Speech
Severity Index.
Speech Severity equivalence was ensured at
the time of allocation of clients to the two age
groups.
Instructions before starting therapy were:
“You will be part of a group treatment with
other clients who have the same problem as
yours i.e.: stuttering. By giving your consent
to participate in this you will be part of a study,
which will help to understand more about
interventions in the treatment of stuttering.” A
written consent was taken by having the clients
sign on a consent form. In case of children, the
consent of the parent or guardian was taken.
Very careful records were maintained through
the study, the client details recorded along with
the treatment module they were subjected to.
The pre and post intervention test scores of all
the subjects were also meticulously noted.
Archives of Indian Psychiatry 13(2) October 2011 40
TTable 1 Communication Skills
Skill Description
Communication Importance of both verbal and no- verbal aspects of communi-
cation.
Body Language The four non-verbal skills: eye contact, facial expressions, pos-
ture / gesture, and physical distance.
Voice Indices The paralinguistic features of voice: rate of speech, loudness /
volume, intonation, and tone voice.
Listening Importance of active listening, barriers in active listening and
skill steps of listening
Observation Importance of self and other observation in interaction, and the
skill steps involved.
Conversation Skill steps required in initiation, entering / maintaining, and
ending conversation included.
Expressing Feelings Importance of expressing feelings, and the skill steps involved.
Responding to Feelings Importance of responding to feelings, and the skill steps in-
volved.
Compliments Importance of giving and receiving compliments, and the skill
steps involved.
Complaints Importance of making and receiving a complaint, and the skill
steps involved.
Requests Importance of making and responding to requests, and the skill
steps involved.
Refusals Importance of making and accepting refusals, and the skill steps
involved.
Asking for Information Importance of asking for information, and the skill steps in-
volved.
Group Interaction Importance of group participation, and the skill steps
involved.
Table 2: t-test comparisons between D-scores of adolescents and adults
Dependent Variables
Adolescents Adults t Sign.
Mean S.D Mean S.D
Speech Situation Checklist
Communication Attitude Scale
Rathus Assertiveness Schedule
Rosenberg Self-Esteem Scale
Speech Severity Index
6.53
3.33
7.60
1.60
3.53
16.25
2.99
8.65
2.59
4.41
18.20
5.87
11.73
2.80
3.87
13.79
5.00
15.13
3.91
4.45
2.56
1.54
0.29
1.01
0.38
p < .05
NS
NS
NS
NS
Archives of Indian Psychiatry 13(2) October 201141
The treatment condition was structured in
accordance to a predetermined plan, session
by session, down to the details of activities
in each session. Each module had 10 sessions
of a given treatment condition of 60 minutes
each on a once a week basis. The fi rst session
began with self-introductions by the researcher
and the group members. Thereafter the rules to
follow, as a result of group membership were
made known.
The Skills Training module focused on inputs
for communication skills, which are essential
for interpersonal interaction. Handouts
consisting of skills steps were given to all
the clients in this module. The importance of
verbal and non-verbal communication was
emphasized.
These inputs for skill development were given
over ten sessions of one hour each. The skills
covered were (Table 1): body language, voice
indices, listening, observation, conversation,
expressing and responding to feelings,
compliments and complaints, requests and
refusal, and asking for information.
In the sessions there was at fi rst information
giving by researcher, this was followed by
viewing of a model video recording of the skill
in question, followed subsequently by a
discussion amongst members. Appropriate
exercises were done to practice the skill in the
safe environment of the group, after which
the feedback from the researcher and the
participants was given. The session ended with
home-work assignments for the week.
Table 2, shows that for SSC the mean scores
are higher for adults (18.20) as compared to
adolescents (6.53) and the difference between
the two groups is signifi cant (p< 0.05). The
same trend is seen over the remaining four
measures as well, viz the S-24, RAS, RSE, and
SSI, wherein the mean scores of adults tend to
be greater than those of adolescents, but not
signifi cantly so.
What is striking in the fi ndings with respect
to the variable of situational anxiety is that
the adults benefi ted signifi cantly more than
the adolescents. The adolescent who stutters
has been seen as a client who is diffi cult
and challenging to treat. Van Riper (23)
commented that adolescents are diffi cult cases
to treat and Daly, Simon and Burnett-Stolnack
(24) agreed that this age group was particularly
challenging. Some researchers reported similar
experiences. One such was a survey of 287
school-based clinicians by Brisk, Healey and
Hux (25), who reported they found that they
had fewer successes with adolescents who
stutter than any other student age group.
This would explain the signifi cant
improvement seen in the adults as compared
to the adolescents in the In order to critically
evaluate this study, it is necessary to consider
the limitations. The sample consisted of a
small group, hence the generalizations of
the results to the wider population of adults
and adolescents who stutter may be limited.
Also, the sample under investigation involved
persons who stutter who had been referred for
intervention may differ from a group who were
willing to receive support in a clinic, on own
accord.
To conclude then the fi ndings that have resulted
from this study highlight the importance of
behaviours secondary to stuttering, which are
often aberrant and attention getting nature and
the fact that they interfere with communication
all point to the need of incorporating these along
with the conventional forms of intervention of
such clients, so as to get global benefi ts.
References
1. Argyle, M. (1969). Social Interaction.
New York: Aldine-Atherton. In L.
Rustin & A. Kuhr, Social Skills and the
Speech Impaired (2nd. ed.). (p.5). Whurr
Publishers, London.
2. L’Abate, L., & Milan, M.A. (Eds.). (1985).
Handbook of Social Skills Training and
Research. New York: Wiley.
3. Libet, J. M., and Lewinsohn, P. M. (1973).
Archives of Indian Psychiatry 13(2) October 2011 42
Concept of social skills with special
reference to the behaviour of depressed
persons. Journal of Consulting and
Clinical Psychology, 40, 304-312. In L.
Rustin and A. Kuhr, Social Skills and the
Speech Impaired (2nd. ed.). (p.6). Whurr
Publishers, London.
4. Bellack, A. S., and Hersen, M. (Eds.).
(1979). Research and Practice in Social
Skills Training. New York: Plenum. In L.
L’Abate, & M. A. Milan (Eds.), Handbook
of Social Skills Training and Research
(p.4). New York: Wiley.
5. Birdwhistell, R. (1970). Kinetics and
Context. Philadelphia: University of
Pennsylvania Press. In L. Rustin, & A.
Kuhr, Social Skills and the Speech Impaired
(2nd. ed.). (p.18). Whurr Publishers, Ltd.
London.
6. Argyle, M., Furnham, A., & Graham, J.
(1981). Social Situations. Cambridge:
Cambridge University Press. In L. Rustin
& A. Kuhr, Social Skills and the Speech
Impaired (2nd. ed.). (p. 18). Whurr
Publishers, London.
7. Dickson, D. A., Hargie, O., & Morrow, N.
(1993). Communication Skills Training
for Health Professionals: An Instruction
Handbook. London: Chapman & Hall.
In L. Rustin and A. Kuhr, Social Skills
and the Speech Impaired (2nd. ed.). (p.18).
Whurr Publishers, London.
8. Rustin, L., Cook, F., & Spence, R.
(1995). The Management of Stuttering
in Adolescence: Communication Skills
Approach. Whurr Publishers.
9. Sheehan, J. G. (1970). Stuttering: Research
and Therapy. New York: Harper & Row. In
C. Van Riper, The Nature of Stuttering (pp.
279-322). Englewood Cliffs, NJ: Prentice-
Hall.
10. Rustin, L., and Kuhr, A (1999). Social
Skills and the Speech Impaired. Whurr
Publishers, London.
11. Prins, D. (1972). Personality, stuttering
severity, and age. Journal of Speech and
Hearing Research, 15, 148-154. In F. H.
Silverman, Stuttering and Other Fluency
Disorders (p.78). Englewood Cliffs, NJ:
Prentice-Hall.
12. Wingate, M. E. (1962). Personality
needs of stutterers. Logos, 5, 35-37. In
O. Bloodstein, A Handbook of Stuttering
(p.217). Singular Publishing.
13. Bloodstein, O. (1995). A Handbook on
Stuttering (5th. ed.). San Diego, CA:
Singular Publishing.
14. Rustin, L. (1984). Intensive treatment
models for adolescent stuttering: A
comparison of social skills training and
speech fl uency techniques. Unpublished
M. Phil Thesis: Leicester. In L. Rustin
and A. Kuhr, Social Skills and the Speech
Impaired. Whurr Publishers, London.
15. Shklovskiy, V. M., Krol, L. M.,
& Mikhailova, E. L. (1988). The
psychotherapy of stuttering: On the model
of stuttering patient’s psychotherapy
group. Soviet Journal of Psychiatry and
Psychology Today, 1, 130-141.
16. Kraaimaat, F., Vanryckeghem, M., &
Van-Dam-Baggen, R. (2002). Stuttering
and social anxiety. Journal of Fluency
Disorders, 27, 319-331.
17. Krol, L. M. (1979). Logo neurosis as
a model for studying the semiosis of
oral speech. Tartu, 164-184. In V. M.
Shklovskiy, L. M. Krol, E. L. Mikhailova,
The psychotherapy of stuttering: On the
model of stuttering patients psychotherapy
group. Soviet Journal of Psychiatry and
Psychology Today, 1, 130-141.
18. Hanson, B. R., Gronhovd, K. D., & Rice,
P. L. (1981). A shortened version of the
Southern Illinois University Speech
Situation Checklist for the identifi cation of
speech- related anxiety. Journal of Fluency
Disorders, 6, 351-360.
19. Andrews, G., & Cutler, J. (1974). Stuttering
therapy: The relation between changes in
symptom level and attitudes. Journal of
Speech and Hearing Disorders, 39, 312-
318.
20. Rathus, S. A. (1973). A 30-item schedule for
assessing assertive behaviour. Behaviour
Therapy, 4, 398-406.
21. Rosenberg, M. (1962). Self - esteem
scale. In Primary reference M. Rosenberg,
Archives of Indian Psychiatry 13(2) October 201143
(1979). Conceiving the Self. New York:
Basic Books.
22. Riley, G. D. (1972). A stuttering severity
instrument for children and adults. Journal
of Speech and Hearing Disorders, 37, 314-
321.
23. Van Riper, C. (1971). The Treatment of
Stuttering. Englewood Cliff NJ: Prentice-
Hall.
24. Daly, D., Simon, C., & Burnett-Stolnack,
M. (1995). Helping adolescents who stutter
focus on fl uency. Language, Speech, and
Hearing Services in Schools, 26, 162-168.
25. Brisk, D. J., Healey, E. C., & Hux, K. A.
(1997). Clinicians’ training and confi dence
associated with treating school-age children
who stutter: A national survey. Language,
Speech, and Hearing Services in Schools,
26, 162-168.
26. Cooper, E. B., & Cooper, C. S. (1985b).
Cooper Personalized Fluency Control
Therapy-Revised. Allen: DLM. In E. B.
Cooper, Treatment of dysfl uency: Future
trends. Journal of Fluency Disorders, 11,
317-327.
27. Shames, G., & Florence, C. (1980). Stutter
Free Speech: A Goal for Fluency. Merrill,
Columbus. In H. H. Gregory, Stuttering:
A Contemporary Perspective. Folia
Phoniatrica, 38, 89-120.
28. Craig, A., Franklin, J. A., & Andrews, G.
(1984). A scale to measure locus of control
behaviour. British Journal of Medical
Psychology, 57, 173-180. In L. F. De Nil,
& R. M. Kroll, The relationship between
locus of control and long-term stuttering
treatment outcome in adult stutterers.
Journal of Fluency Disorders, 20, 345-364.
29. Krause, R. (1976). [Problems in
psychological research on stutterers and
their treatment]. Zeitschrift fur klinische
Psychologie and Psychotheropie, 24(2):
128: 143.
30. Rustin, L., Purser, H. (1984). Intensive
Treatment Models for Adolescent
Stuttering: Social Skills versus Speech
Techniques. Proceeding of the XIX
Congress of the ILP. London: The College
of Speech Therapists. In L.Rustin, F.
Cook, & R.Spence, The Management of
Stuttering in Adolescence: Communication
Skills Approach. Whurr Publishers.
31. Rustin, L. (1987). Assessment and therapy
programme for dysfl uent children. Windsor:
NFER-Nelson. In L. Rustin, F. Cook, &
R. Spence, The Management of Stuttering
in Adolescence: A Communication Skills
Approach. Whurr Publishers.
32. Rustin, L., Botterill, G., & Kelman, E.
(1996). Assessment and Therapy for Young
Dysfl uent Children: Family Interaction.
London: Whurr. In P. Butcher, A. Elias
& R. Raven, Psychogenic Voice Disorders
and Cognitive Behaviour Therapy (p.38).
Whurr Publishers, London.
Dr. Sadhana Deshmukh
Clinical psychologist
Ali Yavar Jung National Institute for the
Hearing Handicapped (AYJNIHH),
Mumbai.
Dr. Anuradha Sovani*
Clinical psychologist
Reader, Department of Applied Psychology, University of
Mumbai.
Consultant and Trustee, Institute for Psychological Health,
Thane, Maharashtra.
e-mail: [email protected]
*Correspondence
Archives of Indian Psychiatry 13(2) October 2011 44
Original Paper
Stress and Coping among Resident Doctors
Sohang Bhadania
Minakshi Parikh
G.K.Vankar
Abstract
Background: Studies have shown medical school training to be a source of signifi cant stress. Despite
the critical importance of stress and well-being during residency training, only a few Indian studies
have examined stress in residency.
Aim: We investigated the psychiatric morbidity, stressors and coping in residents.
Methods: We conducted a cross-sectional descriptive teaching hospital based study involving
145(40%) residents voluntarily at a tertiary care medical college hospital.. Level of stress was meas-
ured by General Health Questionnaire-30(GHQ-30), methods of coping by Brief COPE-28 and demo-
graphics, stressors, resources and intimidation by Resident Physician Questionnaire. Simultaneously
we interviewed them one by one and it was kept anonymous.
Results: Thirty seven percentages of residents were under stress. Fifty fi ve percentages of those who
were in stress worked more than 80 hours per week. Being 1st year resident was associated with more
stress. No gender differences were seen in experience of stress. Fifty nine percent PGs reported intimi-
dation more than once. Residents mostly sought help from colleagues, chief residents and psychiatrists
when in stress. Those who were in stress more often used substance and self blame as coping skills.
Conclusion: Many residents experience signifi cant stressors and psychiatric morbidity. This study can
serve as a basis for future resource application, research and advocacy for overall improvements to
well-being during residency training.
Key words: stress, residents, coping
Introduction
Studies [5] have shown medical school training to
be a source of signifi cant stress. Stress is throughout
the medical training (i.e. under graduate [16, 17,
18] , internship [7], residency [4, 12, 13]). Given
the goals of residency training, some stress seems
inevitable[8] even favorable [9] yet scattered
studies suggest that residents experience high
rates of burnout, a severe stress reaction, and that
burnout may be associated with adverse mental
health and work performance. [4]
The expectations and responsibilities only increase
during residency training. [1] Also residents live
in their respective training hospitals and they are
expected to be profi cient clinicians, educators,
researchers and administrators by the time they
have completed their training. Many residents
report psychological symptoms during residency,
feelings of becoming less humanistic and more
cynical and “burning out”. [3, 4] Stress and Burn
out differs from depression in that they only
involve a person’s relationship to his or her work,
whereas depression globally affects a person’s life. [6]
Coping is often defi ned as cognitive and behavioral
efforts made in response to a threat. A common
model of coping set forth by Lazarus and Folkman
(1984) stresses that coping choices are dependent
on both the appraisal of the threat (primary
appraisal) and the appraisal of one’s resources to
address the threat (secondary appraisal). [20, 24]
The Resident Service Committee of the Association
of Program Directors in Internal Medicine,
Philadelphia, has grouped these stresses into
situational, personal and professional stressors. [28] Our hypothesis was to see if there is any
association between long working hours, stress and
the greater use of maladaptive coping strategies.
Also we have studied stress and its relation with
demographics, branch of post graduation and
Archives of Indian Psychiatry 13(2) October 201145
gender difference of coping style. Firstly, it might
be important to decrease the workload to enhance
the effi ciency of the residents. On the other hand,
it may be important to identify and discourage the
use of ‘negative’ coping mechanisms, which might
further contribute to the stress of these individuals,
rather than helping them in relieving it. These
measures would help improve patient’s safety as
well as in the training, quality of life and education
of the residents. [29, 30]
Aim
We investigated the psychiatric morbidity, stress-
ors and coping in resident doctors.
Material and Methods
Participants
Subjects were chosen who were working as
residents at tertiary care medical college affi liated
general hospital. It was cross sectional, voluntary
and anonymous study. Residents were interviewed
personally at their convenience and also asked to
fi ll selected questionnaires during the interview.
All the data were collected over a fi ve months
period (September through January 2008).
We have applied 3 different standardized tools.
Demographics, stressors, intimidation and
resources were assessed by Resident Physician
Questionnaire adopted from a Canadian study,
level of stress assessed by use of General Health
Questionnaire (GHQ-30), use of maladaptive
coping mechanisms through Brief Cope.
General Health Questionnaire (David Gold-
berg 1978)
The level of stress was measured through the
General Health Questionnaire (GHQ-30), which
is a measure of current mental health [14, 32].
The questionnaire was originally developed as a
60-item instrument but at present a range of other
shortened versions of the questionnaire including
the GHQ-28, GHQ-20 and GHQ-12 are available.
The scale asks whether the respondent has
experienced a particular symptom or behavior
recently. Each item is rated on a four-point scale
(less than usual, no more than usual, rather more
than usual, or much more than usual). A score of
0-0-1-1 for the 4 responses from left to right was
given. The cut off score would be between 3 and 4,
as described by Goldberg. All those who had scored
4 and above were considered to be suffering from
psychological morbidity due to stress, and were
labeled as “Stressed” and those with the score of 3
and below were labeled as “Not stressed”.
Brief COPE (Carver CS 1997)
The frequency of different coping strategies
employed by the residents in the past 2 weeks was
determined with the Brief COPE 28. [25] It has
28 items measuring 14 different coping styles (2
questions for each coping method). The responses
to these questions are measured on a 4-point
Likert-type scale with responses ranging from 1
(“I’ve not done this at all”) to 4 (“I’ve been doing
this a lot”). These include, for example, ‘Active
Coping’ (I’ve been taking action to try to make
the situation better), ‘Religion’ (I’ve been praying
or meditating), ‘Venting’ (I’ve been expressing
my negative feelings) and ‘Substance Use’ (I’ve
been using alcohol or other drugs to make myself
feel better). As evident, some of these coping
mechanisms will have a positive effect on the
individual’s life and can be termed positive. On
the other hand, others that can worsen the situation
can be termed as ‘maladaptive’. [26, 27]
Resident Physician Questionnaire (Happy doc
Jordan CS 2005)
Because the goals of the study were broad-based
and descriptive, a decision was made to cover a
large number of variables using single items and
small modules, rather than including a restricted
set of detailed “gold standard” measures. The
questionnaire was divided into fi ve sections: de-
mographics, stress, intimidation and harassment,
well-being and resources. Survey questions in-
cluded qualitative rating scales, multiple responses
and yes / no questions. To minimize a bias in rating
scales responses (response acquiescence bias); the
survey included a mixture of positively and nega-
tively stated items. [31] The purpose of this study
was to measure “perceived stress” which might
vary both quantitatively and qualitatively among
individuals. For similar reasons we did not defi ne
the term intimidation and harassment as this is also
perceived differently among individuals. We also
decided to keep the two terms (intimidation and
harassment) linked to avoid any confusion among
resident when completing the survey. In certain in-Archives of Indian Psychiatry 13(2) October 2011
46
stances group data was collapsed to increase num-
bers within response categories. As a part of the
study we have also included mental illness screen-
ing questions. To avoid any stigmatization and
thus decreased response rate, we did not further
screen these individuals for psychiatric diagnoses.
Resources questions focused on knowledge of cur-
rent resources (list provided of all resources and
residents were asked which resource they were aware of
prior to the survey) and perceived need for future resources.
Statistical analysis
The data was imported in Microsoft Excel and
Statistical Package for Social Sciences 10 (SPSS
10) for further analysis. All percentages reported
in this paper were rounded to the nearest whole
number. In addition, decimal points were rounded
to 1. p-values less then 0.05 were interpreted as
indicating statistical difference
Results
Demographic characteristics
The response rate for the survey was 40%
(145/365). Of those residents who completed the
survey, 68% (n = 98) were male and 32% (n = 47)
were female. The marital status among residents
revealed that 23% were married, 55% were
never married, and 22% were engaged. There is
signifi cant difference found between genders as
regard to marital status (p=0.006). More males
(25%) were found as engaged while females (41%)
were mostly married. The levels of residency were
as under: 34.5% were from fi rst year of post-
graduate training, 35% were in second year, 30.5%
were in third year. The average number of hours
worked per week among residents was 78 ± 15
hours. between the genders as regards experience
of stress. No specifi c specialty was associated with
higher stress.
First year of residents were in more stress than
second year and third year students (p=0.01).
Overall engaged were found to be in least stress
compared to the single or married residents
(p=0.01).
We found that those who worked for more than 80
hours in a week were in stress (p=0.002).
When asked to rank the most important thing
contributing to feelings of stress, both males and
females ranked time pressure as their number one
choice.
Intimidation and harassment
Ninety three (64%) residents reported experiencing
intimidation and harassment. Of those who
responded 59 per cent have reported it occurring
more than once. Residents reported intimidation
and harassment from many members of the
healthcare team. Intimidation and harraassment
was reported from nursing staff(38%), from
staff physician(27%), from residents of same
specialty(26%), % from residents of other
specialties(38%), patients and their relatives(22%)
Only 43% of residents who reported intimidation
and harassment were aware of the process to
address this issue and 32% of residents felt this
process was not adequate, fair and independent.
Also they have reported different forms of
intimidation and harassment (see Table-3).
Residents were questioned on what was the basis
for the reported intimidation and harassment.
Here also multiple responses were permissible.
The primary reported basis for intimidation and
harassment was language (n = 22/93).
Resources
Residents have reported very few available
resources to use when in stress. They explained
need mainly of resident support group (72%),
career counselor (70%), spiritual counselor (63%)
and family support program (47%).Whenasked to
rank the top three well-being resources a resident
in a situation where they were experiencing an
emotional or mental health problem, resident
colleagues were ranked as the number one
choice, followed by external psychiatrists and
psychologists and senior residents.
Coping with the stress
There is difference in styles of coping as per
stress profi le and gender. Among 14 different
coping styles negative styles were associated with
stress
Discussion
The number of participants were (40%) of all
Archives of Indian Psychiatry 13(2) October 201147
Table: 1 Stress in residency and Characteristics of Residents
GHQ
positive
N=53
n(%)
GHQ negative
N=92
n(%)
Signifi cance
Hours of
work
80 or less hrs work
>80 hrs work
28
25
71
21 p=0.002
Level of training
1st year
2nd year
3rd year
26
23
04
24
28
40p=4.3E-05(<0.05)
Marital status
Single
Engaged
Married
34
06
13
46
26
20p=0.01
Faculty
Surgical
Non surgical
21
32
27
65 p=0.2
Clinical
Para clinical
38
15
62
30 p=0.6
Gender Male
Female
33
20
65
27p=0.3
Table: 2 Top resident ranked contributors to
feelings of stress
Contributors Female
N=47
%
Male
N=98
%
Not enough time 9 24.1
Own work situation 8.3 17.9
Emotional problem 6.2 8.3
Residency program 4.1 4.8
Personal relationship 1.4 1.4
Physical problem 1.4 1.4
Financial problem 00 4.8
Discrimination 00 1.4
Table: 3 Forms of intimidation and harass-
ment reported by residents.
Forms
N=145
n, (%)
Inappropriate verbal command83 (57.2)
Work as punishment39 (26.9)
Privileges/opportunity taken
away
31(21.4)
Recrimination (i.e. accusation,
counter-attack)
16 (11.0)
Others (e.g. criticism, partiality
etc.)
8 (05.5)
Inappropriate or unwanted phy-
sical contact
1 (0.7)
Sexual harassment1 (0.7)
Archives of Indian Psychiatry 13(2) October 201148
Table: 4 Ways of coping and stress
Coping
Mechanism
GHQ Positive
N=53
Mean(SD)
GHQ Negative
N=92
Mean(SD) Signifi cance
Self Distraction 5.85(1.61) 6.33(1.18) p=0.03
Active Coping 5.91(1.47) 6.28(1.23) p=0.07
Denial 2.81(1.14) 2.60(0.90) p=0.1
Substance Use 2.38(1.02) 2.10(0.39) p=0.03
Emotional Support 5.75(1.54) 5.67(1.32) p=0.4
Instrumental Support 5.68(1.22) 5.91(1.20) p=0.1
Religion 5.08(1.83) 5.05(1.58) p=0.5
Behavioral Disengagement 3.43(1.37) 3.20(1.38) p=0.2
Venting 5.11(1.54) 4.92(1.57) p=0.4
Positive Reframing 5.74(1.57) 5.97(1.21) p=0.2
Planning 5.85(1.36) 6.11(1.18) p=0.1
Humor 3.62(1.46) 4.13(1.45) p=0.02
Acceptance 5.81(1.35) 5.72(1.14) p=0.3
Self Blame 3.81(1.89) 3.30(1.22) p=0.04
Table: 5 Ways of coping and gender
Coping
Mechanism
Female
N=47
Mean(SD)
Male
N=98
Mean(SD) Signifi cance
Self Distraction 6.13(1.3) 6.16(1.4) p=0.5
Active Coping 6.0(1.24) 6.2(1.4) P=0.4
Denial 3.0(1.1) 2.5(0.9) p=0.006
Substance Use 2.1(0.52) 2.24(0.8) p=0.08
Emotional Support 6.0(1.33) 5.6(1.41) p=0.006
Instrumental Support 5.6(1.3) 5.9(1.2) p=0.09
Behavioral Disengagement 3.6(1.2) 3.1(1.4) p=0.2
Venting 5.4(1.6) 4.8(1.5) p=0.02
Positive Reframing 5.9(1.2) 5.9(1.4) p=0.5
Planning 5.6(1.1) 6.2(1.3) p=0.002
Humor 3.7(1.4) 4.1(1.5) p=0.058
Acceptance 5.9(1.0) 5.7(1.3) p=0.2
Religion 5.1(1.9) 5.0(1.5) p=0.5
Self Blame 3.7(1.4) 3.4(1.6) p=0.1
Archives of Indian Psychiatry 13(2) October 201249
resident doctors working at the institute which is
comparable to other studies of this type. [5, 27, 12]
In our study 37% of 145 residents were found to be
under stress (GHQ>3). Jordan et al. [12] reported
34% and Pashtoon et al [27] reported 55% of
residents in stress in other similar studies.
As regards the socio demographic characteristics,
the results are similar to results found by Jenny
et al. [33] we found no difference in stress level
between genders. This fi nding is contrary to
fi ndings of Toews et al. and Jordan at el. [11,
12] This might be possibly explained by the fact
that other studies have used only self reporting
questionnaire sent impersonally by mail whereas
our questionnaire fi lling was preceded by a personal
psychiatric interview which probably made the
male counterpart equally psychological minded
and expressive as female students. Also we found
that there is signifi cant difference between genders
with respect to marital status. Here we identifi ed
signifi cant number of males being engaged while
signifi cant number of females was married. We
found that engaged were having least stress as
compared to married and singles. Studies have
shown thatfavorable life events help to reduce
stress. [19] So it might have facilitated to lower
gender difference in stress. As engagement period
has signifi cant occupancy in life of residents, we
have included it in our study. No study of such
kind has demonstrated stress in residents those
who were engaged. The probable explanation
for such difference might be that those who were
engaged might be getting dual benefi ts of less
family responsibility and a supportive affectionate
near one, facilitates to reduce stress. [7] Similar
results were found by Kerby et al, [12] Catherine
et al [22] and C van Ineveld et al. [23] Shanafelt et
al [2] and Lemakau et al [15] found no relationship
as regard to marital status.
No other signifi cant differences were found as
regard to socio demographic factors.
Regarding stress and work, more number of junior
residents reported stress than senior residents.
Residents that are more senior in training have
other stresses that may be equally concerning
(e.g. fi nal examinations, higher expectations) [12]
but we have not found them signifi cant. Similar
fi ndings were reported by DB Reuben [10] and
Garg G. [36] The possible stressors reported
for such difference might be less experience so
required constant learning, high skill work, work
load [34,36] hours of work [35,4] , reduced sleep [37, 47] and intimidation by senior residents and
nursing staff. [12, 39, 40] These stressors lead to
fatigue and poor care delivery. [5, 37, 41, 42] We
found that those who worked for more than 80
hours in a week were in more stress. Some studies
found higher incidence of emotional exhaustion,
depersonalization and ultimately reduced level of
satisfaction with more hours of work per week. [43] In our study we are able to provide estimates
of working hours of residents. We, however, from
the above results cannot suggest the limit for the
working hours but taking the 80 hours limit as
suggested by the American College of Graduate
Medical Education [27]; a lot of individuals were
overworked. Along with evidences from other
studies [42] we can say that work hours per week
were one of the major causes of stress in residents.
Also more working hours means less amount of
continuous refreshing sleep. Residents from some
specialty (i.e, gynecology, surgery, orthopedics)
found to work on scheduled call duty extending
from 24 to 48 hours in single stretch. This may
be the reason why working for long hours having
relationship with stress.
For over 10 years, the United Kingdom and
other Western countries have been substantially
reducing the work hours of “junior doctors”. [44] A
good review of the complex provisions in various
countries was prepared by the Australian Medical
Association. [45] In the United Kingdom, the
current weekly limit for “actual work” is 56 hours
(with an overall limit of 72 hours, including other
in-hospital activities). [42] Even more stringent
restrictions are mandated by the European Working
Time Directive, some had been implemented by
2004, and others to be by 2009. [46] Although such
changes are not easy to implement but certainly
it would be taken as a guide for making future
strategies. Some suggestions from residents were
recruitment of more postgraduates as per the load
of patients, nursing staff should work responsibly,
and equaldistribution of work as per skill and
clerical and paper work should be done by staff
other than post graduates. Studies have shown that
work hours, work load and sleep are dependent
on each other. [4, 21] So addressing one issue
Archives of Indian Psychiatry 13(2) October 201150
effectively would automatically bring changes to
others. [21]
In this study 93% (n=135/145) postgraduates
reported “learning new things” as stressor while
75% (n=109/145) reports “requirement of high
level of skill” as stressful. Geoffrey JR [21] also
reported so.
We have not found any signifi cant difference
between specialties for the level of stress. Contrary
fi ndings were reported by Thomas NK. [4] The
probable reasons may be stressors unique to
branches and tend to differ with it and as we have
measured perceived stress, there is more reliance
on subjectivity and resultant different sensitivity
to stressors.
Perception of intimidation at educational
environment is known to all. [38] In our study
“inappropriate verbal comments from seniors”
reported by 57% of residents in form of nagging
in presence of patient and their relatives, verbally
abusive and shouting at students. These fi ndings
are consistence with earlier studies. [12] Apart
from these “work as punishment” and “privilege
taken away” remain major causes.
The primary basis reported for the intimidation
and harassment was language n=22/145(15.2%)
a difference that could not attain statistical
signifi cance. Surprisingly, contrary to other
studies [12, 40] we found gender was less reported
as bases of intimidation in our studies.
The reason may be lying in cultural values and
customs as comparable studies are western. As
residents were also allowed to express other forms
of intimidations, they mostly reported jealousy,
arrogance, ego, seniority and incomplete work
in preset time frame as annoying. Most reported
senior residents and nursing staff as a source of
harassment. Intimidation and harassment occurred
often multiple times (more than once in 59% of
those responding to the study) in both genders.
Twenty one per cent of the residents felt that the
process to deal with it was not adequate, fair and
independent. This speaks to the need for further
educating all individuals in the healthcare system
on resident well-being. For bullying to be tackled,
trainees need a safe means of complaining. They
also need to be made aware of the impact that their
own behavior may have on colleagues. If some
of the behaviors that erode trainees’ professional
confi dence or self esteem attempted by trainers to
improve their performance then educational rather
than a punitive approach should be needed to help
trainers to develop effective ways of encouraging
better performance without becoming a source of
distress to junior colleagues. [48]
In time of stress most students prefer to contact
their own colleague or senior ones. Apart from
this they aware of family members and nearby
psychiatrist. They explain mainly need of resident
support group and career counselor .So more
education should be applied to this area as also
suggested by others. [12, 39, 40]
Here we have found those using positive coping
skills were in less stress than those
who were using maladaptive ones. Studies
have shown using positive coping skills help to
reduce stress. [49, 50] So help seeking should be
encouraged. There is also signifi cant difference
found in ways of coping used by males and
females. [24] We have found females using more
emotional support and venting while males mostly
use planning and humor.
Limitation of study
1. Residents from only a single center were
studied and the study was cross sectional.
2. All residents from studied specialties were not
willing to participate.
Conclusion
It is clear that there are signifi cant stressors incurred
during residency. Intimidation and harassment
occurs among many residents. It is also important
to recognize that a signifi cant amount of residents
are vulnerable to emotional and mental health
concerns. Residents need to be better informed
about well-being resources. Ensuring the education
of other healthcare professionals in the area of
well-being is needed, so that resident’s who ask
for help will be directed the correct sources.
Substantial reform is possible within the current
system of medical care. Focus should be made
towards identifying preventable factors of stress.
Archives of Indian Psychiatry 13(2) October 2011 51
Archives of Indian Psychiatry 13(2) October 201152
References
1. Woloschuk W, Harasym P, Mandin H. Implementing
a clinical presentation curriculum: impact on student
stress and workload. Teach Learn Med.1998; 10:44–50.
2. Shanafelt TD, Bradley KA, Wipf JE, Back AL. Burnout
and self-reported patient care in an internal medicine
residency program. Annals of Internal Medicine.2002;
136:358–367.
3. Hillhouse JJ, Adler CM, Walters DN. A simple model
of stress, burnout and symptomatology in medical
residents: a longitudinal study. Psych Health Med.2000;
5:63–73.
4. Thomas NK. Resident burnout. JAMA.2004;
292:2880–2889.
5. Lynda E, Len K. Coping strategies, depression, and
anxiety among Ontario family medicine residents.
Canadian family physician.2005; 51:80-83.
6. Tait DS, Katharine A, Bradley, Joyce E, Anthony L.
Burnout and Self-Reported Patient Care in an Internal
Medicine Residency Program Ann Intern Med.2002;
136:358-367.
7. Simon M Willcock, Michele G Daly, Christopher C
Tennant, Benjamin J Allard Burnout and psychiatric
morbidity in new medical graduates. MJA 2004; 181:
357–360.
8. Beckman JA, Fang JC. Resident burnout [letter]. Ann
Intern Med.2002; 137:698 700.
9. Levey RE. Sources of stress for residents and
recommendations for programs to assist them. Acad
Med.2001; 76:142-150.
10. DB Reuben. Depressive symptoms in medical house
offi cers. Effects of level of training and work rotation.
Arch Intern Med.1985; 145(2).
11. Toews JA, Lockyer JM, Dobson DJ, Simpson
E, Brownell AK, Brenneis F. Analysis of stress levels
among medical students, residents, and graduate
students at four Canadian schools of medicine. Acad
Med.1997; 72:997–1002.
12. Jordan SC and Scott P. Well-being in residency training:
a survey examining resident physician satisfaction both
within and outside of residency training and mental
health in Alberta.BMC Med Educ.2005; 5: 21.
13. Kirby H, Victor M. Prevalence of Depression and
Distress in a Large Sample of Canadian Residents,
Interns and Fellows. Am J Psychiatry.1987; 144:1561-
66.
14. TG Sriram, CR Chandrashekar, MK Isaac,
V. Shanmugham. The General Health Questionnaire
(GHQ). Comparison of the English version and a
translated Indian version. Social psychiatry and
psychiatric epidemiology.2005; 24:317-20.
15. Lemkau JP, Purdy RR, Rafferty JP, Rudisill JR.
Correlates of burnout among family practice residents.
J Med Educ.1988; 63:682-691.
16. Chandrashekhar TS, Pathiyil RS, VS Binu, Chiranjoy
M, Biswabina R, Ritesh GM. Psychological morbidity,
sources of stress and coping strategies among
undergraduate medical students of Nepal. BMC Medical
Education 2007; 7:26
17. Guthrie E, Black D, Bagalkote H, Shaw C, Campbell M,
Creed F. Psychological stress and burnout in medical
students: a fi ve-year prospective longitudinal study. J R
Soc Med 1998; 91:237-243.
18. Supe AN. A study of stress in medical students at Seth
G.S. Medical College. J Postgrad Med 1998; 44:1-6.
19. Holmes T. Life situations, emotions and diseases.
Psychosom Med.1978; 9:78.
20. John D, Catherine MacArthur, Shelley Taylor .Coping
Strategies. Summary prepared in collaboration with the
Psychosocial Working Group. Last revised July, 1998.
21. Geoffrey J Riley. Understanding the stresses and strains
of being a doctor.MJA.2004; 181:350–353.
22. Catherine M., Wayne S, Mary O, Harry R, Robert
L. Stress and Coping Among Orthopedic Surgery
Residents and Faculty. The American Journal of Bone
and Joint Surgery.2004 86:1579-1586.
23. C. van Ineveld. Stress in residency training: Symptom
management or active treatment? Can Med Assoc
J.1994; 150 (10).
24. Lisa KT. Sex Differences in Coping Behavior: A Meta-
Analytic Review and an Examination of Relative
Coping. Personality and Social Psychology Review
2002; 6(1):2-30.
25. Carver CS. You want to measure coping but your
protocol’s too long: consider the Brief COPE. Int J
Behav Med 1997; 4(1): 92-100.
26. Vosvick M, Koopman C, Gore-Felton C, Thoresen C,
Krumboltz J, Spiegel D. Relationship of functional
quality of life to strategies for coping with the stress of
living with HIV/AIDS. Psychosomatics 2003; 44(1):51-
8.
27. Pashtoon MK, Talha K, Farooq HK, Jawad GK, Umber
ZK, Hadi MK, Urooj BK, Musa R. Studying the
association between postgraduate trainees’ work hours,
stress and the use of maladaptive coping strategies. J
Ayub Med Coll Abbottabad 2007; 19(3):37-41.
28. Resident Services Committee, Association of Program
Directors in Internal Medicine. Stress and Impairment
during Residency Training: Strategies for Reduction,
Identifi cation and Management. Ann Int Med.1988;
109:154-61.
29. Steinbrook R. The Debate over Residents’ Work Hours.
N Engl J Med.2002; 347(16):1296-1302.
30. Gaba DM, Howard SK. Patient safety: Fatigue among
clinicians and the safety of patients. N Engl J Med 2002;
347(16):1249-55.
31. Guyatt GH, Cook DJ, King D, Norman GR, Kane SC,
van Ineveld C. Effect of the framing of questionnaire
items regarding satisfaction with training on resident
responses. Acad Med.1999; 74:192–194.
32. FA Huppert, DE Walters, NE Day, BJ Elliott. The factor
structure of the General Health Questionnaire (GHQ-
30). A reliability study on 6317 community residents.
The British Journal of Psychiatry.1989; 155:178-185.
33. Jenny FC, Leslie AM. Sources of stress and ways of
coping in junior house offi cers. Stress Medicine.1988; 5(2):121-126.
34. Ndom RJ, Makanjuola AB. Perceived stress factors among resident doctors in a Nigerian teaching hospital.
West Afr J Med.2004; 23(3):232-5.
35. Ravi Gopal, Jeffrey JG, Tom JM, Allan VP. Burnout and
Internal Medicine Resident Work-Hour Restrictions.
Arch Intern Med.2005; 165:2595-2600.
36. Garg G. Postgraduate lifestyle: Stress and satisfaction.
Indian J Pediatr 2005; 72:991-991.
37. Sigrid V, Raymond R, Barbara B, Ilene R, Judith O.
Sleep Loss and Fatigue in Residency Training: A
Reappraisal. JAMA.2002; 288:1116-1124.
38. Tibbo P, de Gara CJ, Blake TM, Steinberg C, Stonehocker
B. Perceptions of intimidation in the psychiatric
educational environment in Edmonton, Alberta. Can J
Psychiatry. 2002 Aug; 47(6):562-7.
39. Elisabeth P, Maryanne A, Anita H, Jenny FC. Bullying
among doctors in training: cross sectional questionnaire
survey. BMJ 2004; 329; 658-659.
40. 40. Lyn Quine. Workplace bullying in junior doctors:
questionnaire survey. BMJ 2002; 324:878–9.
41. 41. Chen-CL, Lawrence SW. Residents who stay late
at hospital and how they perform the following day.
Medical Education 2008; 42: 74–81.
42. David MG, Steven KH. Fatigue among clinicians and the
safety of patients. N Engl J Med.2002; 347(16):1249-
55.
43. Ravi Gopal, Jeffrey JG, Tom JM, Allan VP. Burnout and
Internal Medicine Resident Work-Hour Restrictions.
Arch Intern Med. 2005; 165:2595-2600.
44. Pickersgill T. The European working time directive for
doctors in training. BMJ 2001; 323:1266.
45. Overseas experience in regulating hours of work of
doctors in training. Australian Medical Association,1998.
(Accessed September 24, 2002, at http://www.domino.
ama.com.au/dir0103/IRRemun.nsf
46. Pickersgill T. The European working time directive for
doctors in training. BMJ. 2001; 323:1266.
47. A Bibliography of Articles on the Effect of Sleep Loss
on Performance. Compiled by Ingrid Philibert. Updated
March 2005.
48. Paice E, Firth-Cozens J. Who’s a bully then? BMJ 2003;
326(supple):S127. http://careerfocus. bmjjournals.com/
cgi/content/full/326/7393/S127
49. Pino Morales Socorro M, López-Ibor Aliño JJ.Stress
during post-graduate medical training. Actas Luso
Esp Neurol Psiquiatr Cienc Afi nes.1996 Mar-Apr;
24(2):75-80 [Article in Spanish].
50. Ratanawongsa N, Wright SM, Carrese JA. Well-being
in residency: a time for temporary imbalance? Med
Educ.2007; 41(3):273-80.
Sources of funding: None Confl ict of interet :None
Sohang Bhadania,MD,Resident
Minakshi Parikh,MD,Professor
G.K.Vankar,MD Professor and Head*
Dept.of Psychiatry
B.J.Medical College and
Civil Hospital
Ahmedabad 380016
*Correspondence
e-mail:[email protected]
cell:+919904160338
Archives of Indian Psychiatry 13(2) October 201153
Review Article
Who cares for caregivers?
Pooja Dangar
Parag S. Shah
Abstract
Legal, social and economic factors have changed the mental health care delivery system. Persons with
mental illness are now treated in the community and they live with or in close proximity to their fam-
ily. The burden, stress, and socio-economic effects on the people caring for them are not suffi ciently
appreciated. The societal neglect is evident from the absence of any established framework that pro-
tects their rights.
Health professionals and policy makers need to develop greater insight into the care giving experi-
ence, interact with caregivers more collaboratively, and implement strategies that facilitate better
outcomes for all family members. It is important to help them to protect their individuality, provide
support, respite care, legal protection, respect and consideration for the role they perform.
It is the need of the hour to realize that all individuals including caregivers have basic human rights
that need to be addressed.
Keywords: Mental health, Caregivers, Rights
Introduction
Historically, people who have mental disorders
were isolated from society in large stand-alone
mental institutions on the outskirts of cities. The
health care philosophy that sustained these large
institutions also perpetuated the image that these
people were dangerous and, therefore, unable to
live in society. In the 1960s, the shift from hospital
to community-based care was supposed to reduce
the load on hospitals, help early recovery and
prevent chronic disability among mentally ill
persons as well as effect substantial savings for
indoor hospital care. But this policy of treating
mentally ill people in the community could not
exist without the family members who almost
always bear the main burden of care.
Families play an increasingly important role in
facilitating the provision of mental health services
in the community. Carers are referred to in literature
as the “glue that holds the system together” [1] and
the “paraprofessionals who play a signifi cant role
in the service delivery system” [2].
Who are the carers?Caregivers are the person most closely engaged
in a patients experience with illness and also in-
volved in maintaining a person’s ability to live in-
dependently at home. In this care giving process,
carers life is in some way restricted by the need
to be responsible for the care of the patient. They
have been referred to as the “primary source of
care for persons with a severe illness…they pro-
vide housing, fi nancial aid, companionship and
emotional support”. [3]
Family members rarely become carers by making
a rational decision about what they are going to do
with their lives, rather, the caring role creeps up
on them, as it becomes clear that their relative is
going to need help for much longer. In a country
like India (a home of one sixth of world’s mentally
ill); families are the main caregivers for patients
suffering from psychiatric illness.
Burden of care
Individuals perform multiple roles in life such
as family role, occupational role and social role.
Becoming a caregiver introduces additional role
and therefore requires some rearrangement of pri-
orities and redirection of resources. The average
amount of time that caregivers spend on care giv-
ing is about 20 hours per week. Even more time is
required when the care receiver has multiple dis-
abilities. The presence of ill person at home affects
all family members, problems occur in the areas
of health, recreational activities, social and marital
Archives of Indian Psychiatry 13(2) October 201154
relations, along with fi nances.
Factors contributing to the burden are:
Changing Societal structure: the societal structure
has changed due to factors like small families,
nuclear families, working woman, increasing
rate of marital breakdown, migration to different
cities and social isolation. This combination leaves
smaller family units shouldering responsibilities
for increasing care-giving demands.[4,5].
Increased survival of patients: With latest advances
in medical science, survival of patients with illness
/ disabilities has risen. In Australia, one person in
fi ve will at some stage in their life experience the
burden of a major mental disorder [6]. About 1.25
million American families live with persistently
mentally ill members.
A fundamental lack of services in the community:
It leads to an over-reliance on family members.
Despite their central role in the healthcare of their
relative with an illness, only a minority of carers
receives services from health agencies [7]. World
Health Report 2001 stated that 40% of the countries
in the world don’t have mental health policy and
over 30% have no special programs related to
mental and behavioral disorders. Moreover, health
plans most often don’t cover mental and behavioral
disorders unlike other illness.
Literature mentions about both objective and
subjective burden placed on the caregivers.
Objective burden [8] can be:
Economic losses: Financial losses, inability
to maintain current employment status, loss of
newer opportunities and potential for professional
development are the problems faced by carers in
their routine life [1,2,9,10,11].
Impaired physical health: In a study conducted
by the NSF in 1997, over 70% of carers surveyed
felt that their health had suffered because of their
caring role.
Disruption of relationships: Familial
relationships can suffer due to patient’s behavior
which can be extremely diffi cult to cope with or
disruptive. [1,2,9,10].
Subjective burden [8] can be:
Subjective burden experienced by caregivers
is directly linked to the experience of objective
problems [12]. Caregivers’ burnout is suggested by
their feelings of anger, resentment, grief, guilt,
dependency and other distressing emotions. They
report their inability to care for a long time, wish that
they could just leave care giving to someone else,
unfavorable experiences in interactions with health
professionals, feeling that they have lost control of
their life and being uncertain about managing their
role [11]. In India, families have surprising reported
inactivity, not participating in household chores,
slowness and poor personal hygiene of the patient
as more distressful than aggressive and assaultive
behaviors [13]. Also, fi nancial costs were found to
be the most commonly experienced ‘felt burden’ [14].
Burden of care: unique in mental health
The care giving burden is unique to carers of
people with a mental illness. Physically ill people
are ordinarily deeply involved in getting well and
returning to their pre-sickness social roles. In
contrast, mentally ill people often cannot abide
by the usual rules of social settings, may engage
in behaviors considered socially repugnant,
sometimes deny that they are ill, and frequently
treat their caregivers with hostility instead of
gratitude [15]. Studies have found that the relatives
with chronic schizophrenia experienced severe
burden than those of patients with chronic lung
disease [16]. As a result, thousands of mentally sick
persons, some of them affl icted quite seriously, who
are neither on medication nor are institutionalized,
lead the life of a destitute. Discarded by their
families and uncared-for by any voluntary agency,
their existence is woefully dehumanized.
Factors increasing the burden:Instead of gaining community support, there is
reduction in social networks due to the disruption
of routine social and leisure activities as well as
stigmatization that occurs hand-in-hand with a
diagnosis of mental illness [1,2,9,10,].
Lowered self-esteem and increased isolation and
withdrawal due to stigma and lack of resources [9,17].
Grief on witnessing disintegration of personality
of a close family member who was on the verge
of realizing his/her potential in the formative years
(adulthood) and suffered from major disorder like
schizophrenia. There is a grieving that needs to be
done for this lost person, a grieving that will help
Archives of Indian Psychiatry 13(2) October 2011 55
the carer adjust to the new person the “sufferer has
become” [11].
For disorders like dementia, as the disease
progresses—the person may experience the
loss of self-esteem, dependency, and there can
be behavioural diffi culties such as agitation,
aggression or delusions. Carers need to spend
increasingly longer time providing care than do
carers with other disorders. The highest proportion
of time is spent in communicating, supervising,
and helping with eating and toileting [18].
Economic burden faced is extensive, which is
explained by the increased propensity for people
with disorders like dementia to use expensive
private medical care services, in addition to giving
up work to provide care [18]. A large majority
of persons with mental illness either remain
unemployed or underemployed, which further
adds to the fi nancial burden of the families. Direct
costs involve cost of traveling, taking time off
from work for both the patient (if employed) and
caregiver, and cost of psychotropic drugs (long
duration of treatment increases cost). Families
from the poorest countries are particularly likely to
have used expensive private medical services and
to be spending more than 10% of the per capita
GNP on health care [19].
Carers often become more vulnerable, with little
or no formal training in mental illness, lack of
information about the disorder and its medication,
lack of resources and treatment facilities and, at
times, a lack of responsive professional [2].
Caregivers or potential patients?
Caregivers’ stress is a daily fact of life for
many family members. Apart from sharing the
biological vulnerability, care giving itself acts
as a psychosocial stressor, enough to precipitate
depression and other psychiatric illnesses in the
caregivers. This is especially true if they do not
receive enough support from family, relatives,
friends, and the community as a whole. They
experience psychological distress in form of
worries, depression, anger, fear, guilt, and stigma.
Higher levels of both objective stress (care
receivers’ behavior problems) and subjective
stressors (caregivers feeling of overload) infl uence
the various dimensions of caregiver health, namely:
poorer self-reported health, more negative health
behaviors, and greater use of health care services.
Women, in turn are more vulnerable due to reduced
social and economic support. Studies show that
more than half of caregivers are women. Important
studies describing caregivers’ vulnerability to
suffer from mental disorders are:
A diagnosis of major depression in older medical
inpatients is independently associated with poor
mental health in their informal caregivers 6 months
later [20].
The potential compromise of caregiver’s health
brought on by demands of role is poignantly
exemplifi ed in well publicized Canadian story
known as “Latimer Case” where father had taken
extreme action of killing his child with disability.
The father’s explanation for committing this act
emphasized feelings of stress he experienced
relating to take care of his child, child’s degree of
pain and sense of helplessness regarding the same.
In a national survey of 1500 carers, 59% reported
their physical health was worse because of their
caring responsibilities, while 85% identifi ed
detrimental changes in their level of emotional
health [21]. Of the 1500, those caring for a family
member with a mental disorder reported that the
negative attitudes of health professionals and the
general public contributed to the deterioration in
their own health status.
Carers of mentally ill patients are much more likely
to have a common mental disorder than carers or
co-residents of controls [18].
Thus, as mental health professionals, it seems that
“we need to decide whether we want to provide
them Caregivers’ rights today or else Patients’
rights tomorrow”.
Caregivers’ (human) rights?
Universal declaration on human rights has
enshrined each individual with right to highest
attainable standard of physical and mental health.
Caregivers of mentally ill people are also human
beings with legitimate claim to human rights.
We have to take into account that caregivers are
important component of health care delivery
system and by recognizing their rights; we are
not helping only caregivers but also the patients
(indirectly) and society (largely) to cope up with
the increasing burden of mental illness.
Mental health professionals frequently encounter
in clinical settings, questions asked by care givers
Archives of Indian Psychiatry 13(2) October 201156
like these ones:
1. What if I get sick? Who will care whom?
2. Will I outlive the patient?
3. I need to choose between going to work to earn
money and spending time to take care of the
patient. Both seem indispensable. What should
I do?
4. What if I get physically, mentally or fi nancially
exhausted?
5. Is there an end to this (care)?
6. How long will this (care) continue?
7. When will I be able to live my life?
8. Do I have a right to live a healthy life?
The health care system needs to fi nd an answer to
these questions. We need to realize their (unmet)
needs. Health policy makers need to be sensitive
enough not only about the human rights of mental
health patients but also their caregivers.
Caregivers across globe have felt the need and
expressed desire to create a formal policy satisfying
their needs and protecting their rights (well
supported by mental health advocacy groups).
A formalized caregiver’s Bill of Rights has been
suggested by Wendy Lustbader, which asserts that
all individuals have needs that have to be fulfi lled.
It includes:
1. Protecting ones individuality, taking care of
self so that with more capability better care can
be given.
2. Seeking help from others & recognizing one’s
limits.
3. Getting angry, depressed, or expressing
themselves.
4. Receiving consideration, affection,
forgiveness, and acceptance in return to care
giving.
5. Deciding when to help and when not to help.
6. Finding the balance between family, work and
care giving.
7. Setting attainable goals.
8. Treating one self well, same as the person for
whom caring.
9. Accepting that their best is good enough.
Current Caregivers’ Policy Across
Globe [22]
In United States the National Family Caregiver
Support Program (NFCSP) provides direct
services (e.g., counseling, respite care), fi nancial
compensation to family caregivers, tax incentives,
etc. Caregivers are provided fi nancial help by
Federal Medicaid law (the “Cash and Counseling”
demonstration). Recently more laws were passed
providing workers with paid family leave.
In Australia extensive support is provided to
caregivers, including general community support
services, respite care, information and counseling,
employment-related initiatives, and cash benefi ts
by policies like the National Home and Community
Care Program (HACC) and National Respite for
Carers Program.
In Canada no formal national caregiver strategy
is in place for caregivers except some support
provided via the tax system. The Long-Term Care
Act of 1994 authorizes funding for caregiver
support services, including respite care.
In Germany and Japan no formal national caregiver
strategy is in place, but Long Term Care Insurance
(LTCI) Program builds in explicit policies to
recognize and support family caregivers.
In United Kingdom National Strategy for Carers
provides pension policy and social security
benefi ts.
In India government provides tax benefi t and free
bus and railway pass at a concession rate that
help to ease burden on patient’s family [18]. Under
Mental Health Act it is proposed that the costs of
maintenance of mentally ill persons detained as
in-patient in any Government Psychiatric hospital
or nursing home shall be borne by the State
Government concerned. This helps caregivers in
reducing fi nancial burden of care giving.
It is well evident that only few countries have
formulated special policies for carers of mentally
ill and that too insuffi cient.
Future Recommendations
At individual level
Caregivers should be regularly accessed with
regards to their capacities, stress levels, ability
to cope and available resources for the role they
perform. Carers should be taught self care and
making them realize its importance as they can
effectively help others only when they help
themselves.
Sensitizing health professionals regarding the
needs of carers, so that they have enough time,
Archives of Indian Psychiatry 13(2) October 2011 57
support and information for them along with that of
patients. It is of concern that sometimes caregivers
themselves are not receptive to treatment
interventions after discharge and it is therefore
important to try to engage these families during
the time of the patient’s hospitalization [23].
Education for caregivers about mental illness’
course and prognosis. Caregivers’ eventual
recognition that they cannot control their family
member’s illness allows them to decrease
involvement without guilt.
Special attention to be given to women caregivers
who are more vulnerable to the burden of care
giving.
Generation and dissipation of supportive resource
materials especially designed for caregivers
At community level
While implementing the policy of de-
institutionalization, giving utmost priority to
community resources developmental plans
including building up alternative social support
systems like half way homes, day care and
rehabilitation centers. This would not only divide
the care among various stakeholders but will also
help patients to reintegrate into the society, remain
gainfully employed as well as face less negative
expressed emotions.
Establishment of respite centers to help caregivers
cope with their role. Respite care enables caregivers
to be temporarily relieved from their care giving
responsibilities, providing them opportunity to
attend to other tasks such as shopping, running
errands, visiting doctor, relaxing or socializing.
Establishing and recognizing support groups
which help to decrease subjective burden of
caregivers by decreasing negative emotions (by
adaptation and acceptance), forming friendships,
re-establishing social networks, hope and positive
role models as well increasing the sense of
power and competence as self help groups have
the ability to infl uence legislation, redirecting
community services and infl uencing attitudes of
people who lack intimate contact with mentally
ill [24]. They also help in decreasing objective
burden by increasing knowledge about illness and
health services, demystifi cation of the illness and
shifting of illness attributions as well as increasing
their problem solving capacity and coping skills.
Provision of adequate and effective support
and services to caregivers, especially access to
emergency services.
Strengthening the community based interventions
by training mental health community workers.
Spreading awareness in general population about
mental disorders will help in decreasing caregivers’
burden by reducing stigma and increasing the level
of community support and understanding. We
should try to spread message that “mental health
is everybody’s business”. It’s a public health
concern.
Providing services that are easily available,
accessible, appropriate to the needs, culturally
acceptable and affordable to the caregivers.
Caregivers’ consideration of the affordability of
services should not only include fi nancial costs,
but also costs in terms of time, effort, potential loss
of confi dentiality, and potential family confl ict.
Introduction of a social support system like
specialized institutes for disabled, which can be
backed by government, public trends of charity or
organization.
At government level
Mental health care system should not over burden
caregivers by assigning them majority of the
responsibilities of managing the patient. Civil
commitment of patients, who are dangerous to
themselves or others or otherwise unmanageable,
should be the responsibility of the mental health
care system.
Mental disabilities should be well recognized and
benefi ted by social security schemes and social
insurances by the state.
Insurance schemes should recognize mental illness
and reimburse expenses not only on physical
ailments but also mental ailments.
Health policies and legislation should give
priority to caregivers of people with mental illness
considering the uniqueness of mental health care.
Considering caregivers as one of the important
component of treating team, they should be
included in all policy making and health care
service developmental plans [29].
Caregivers should unite on a common platform
(e.g. caregivers’ support groups, conferences,
associations) and express their issues in front of
the government, mental health care authorities and
others concerned.
Archives of Indian Psychiatry 13(2) October 201158
Conclusion
Thus society has yet a long way to go before
caregivers are fully recognized, their burdens and
challenges acknowledged and their needs met.
But with continued advocacy and a commitment to
raise caregivers’ voices, those who work on behalf
of care giving families - and caregivers themselves
- can continue to educate lawmakers about what
is needed to support family caregivers and to
affect change at the public policy level. . Health
professionals and policy makers need to develop
greater insight into the care giving experience,
interact with caregivers more collaboratively, and
implement strategies that facilitate better outcomes
for not only patients but also all members of the
family. It is important to help them to protect their
individuality, provide support, respite care, legal
protection, respect and consideration for the role
they perform.
References
1. Chapman, H. (1997). Self-help groups, family
carers and mental health. Australian & New
Zealand Journal of Mental Health Nursing,
6(4), 148-155.
2. Abramowitz, I.A., Coursey, R.D. (1989).
Impact of an educational support group on
family participants who take care of their
schizophrenic relatives. Journal of Consulting
and Clinical Psychology, 57(2), 232-236.
3. Pickett-Schenk, S.A., Cook, J.A., Laris, A.
(2000). Journey of Hope program outcomes,
Community Mental Health Journal, 36(4),
413-424.
4. Kapor, R.L. (1992) The family and
schizophreia: Priority areas for intervention
research in India. Indian Journal of Psychiatry,
34, 3-7
5. Sethi, B.B., Manchanda, R. (1978).
Socioeconomic, demographic and cultural
correlates of psychiatric disorder with special
reference to India. Indian Journal of Psychiatry,
20, 199- 211.
6. Australian Bureau of Statistics (1998). Mental
health and wellbeing: profi le of adults,
Australia, Canberra.
7. Dixon, L., Luckstead, A., Stewart, B., &
Delahanty, J. (2000). Therapists’ contact with
family members of persons with severe mental
illness in a community treatment program.
Psychiatric Services, 51(11), 1449-1451.
8. Reay-Young, R. (2000). Support groups for
relatives of people living with a serious mental
illness: An overview, International Journal of
Psychosocial Rehabilitation, 5, 56-80.
9. Brady, A. (1996). A Study on the Effects of
Schizophrenia on Carers (Thesis), Department
of Psychology, University of Dublin.
10. British Columbia Ministry of Health (1993).
Task Force of Families of People with a Mental
Illness: Families Sharing the Caring, Mental
Health Services Division, B.C. Ministry of
Health, Victoria, Canada.
11. Berry, D. (1997). Living with Schizophrenia,
Institute of Psychiatry, London.
12. Cuijpers, P. & Stam, H. (2000). Burnout among
relatives of psychiatric patients attending
psychoeducational support groups. Psychiatric
Services, 51(3), 375-379.
13. Gopinath, P.S., Chaturvedi, S.K. (1992).
Distressing behaviors of schizophrenia at
home. Acta Psychiatrica Scandinavica, 86,
185-188.
14. Pai, S., Kapur, R.L. (1981). The burden on the
family of a psychiatric patient. British Journal
of Psychiatry, 138, 332-335.
15. Karp, D.A., Tanarugsachock, V. (2000). Mental
illness, caregiving, and emotion management,
Qualitative Health Research, 10(1), 6-25.
16. Gautam, S., Nijhawan, M. (1984). Burden on
families of schizophrenia and chronic lung
disease patients. Indian Journal of Psychiatry,
26, 156-159.
17. Wintersteen, R.T., Young, L. (1988). Effective
professional collaboration with family support
groups. Psychosocial Rehabilitation Journal,
12(1), 19-31.
18. Varghese M., Patel V. (2004). The Graying
of India. In: Agarwal S, Goel D, Salhan R,
Ichhpujani R, Shrivastava S, editors. Mental
Health: an Indian Perspective 1946-2000. New
Delhi: Elsevier; 240-8.
19. Prince, M. J. (2009). The 10/66 dementia
research group - 10 years on. Indian Journal of
Psychiatry, 51(5), 8-15.
20. McCusker J., Latimer E., Cole M., Ciampi A.,
Sewitch M. (2007). Major depression among
medically ill elders contributes to sustained
poor mental health in their informal caregivers.
Archives of Indian Psychiatry 13(2) October 201159
Age Ageing, 36, 400-406.
21. Carers NSW (2003). Response to the
consultation paper on the National Mental
Health Plan 2003-2008. .
22. Montgomery, A., Feinberg, L.F. (2003). The
Road to Recognition: International Review of
Public Policies to Support Family and Informal
Caregiving. Issue Brief. San Francisco, CA:
Family Caregiver Alliance.
23. Heru, A., Ryan, C. (2002). Depressive Symptoms
and Family Functioning in the Caregivers of
Recently Hospitalized Patients with Chronic/
Recurrent Mood Disorders. International
Dr. Pooja Dangar,MBBS, Postgraduate student
Dr. Parag S. Shah,MD DNB, Assistant Professor*
Department of Psychiatry
Government Medical College & SSG Hospital
Baroda, India
A-404, Jeevandham Tower, Nr. Bimanagar,
Satellite Road, Ahmedabad – 380015, India
Tel: +91.9426138318
E-mail: [email protected]
Journal of Psychosocial Rehabilitation, 7, 53-
60.
24. Wyman K., Clarke S., McKenzie P., Gilbert
M. (2008). The Impact of Participation
in a support Group for Carers of a
Person with Schizophrenia: A Qualitative
Study. International Journal of Psychosocial
Rehabilitation, 12 (2), 97-109.
25. Gautam, S., Jain, S., Batra, L., Sharma,
R., Munshi, D. (2009). Human rights and
priveledges of mentally ill persons. IPS
Clinical Practice Guidelines, 2009.
Archives of Indian Psychiatry 13(2) October 201160
Case Report
Familial Hypokalemic Periodic Paralysis : A Case Report
Rajat Oswal
Devendra Chaudhari
Ritambhara Mehta
Abstract
Patients with motor weakness or paralysis are frequently sent for psychiatric evaluation especially
young female patients, without pain or paraesthesias, those who are fully alert, conscious and oriented.
Those who have episodic motor weakness with full recovery and have sensations over involved parts
with marked psychosocial stressor are readily diagnosed as suffering from Conversion Disorder. Some
neurological conditions may follow a similar symptom profi le and are often diffi cult to diagnose due to
its reversible nature and almost no signs and symptoms when the patient is well. Psychiatrists must have
thorough knowledge of such conditions to make a correct diagnosis of Conversion Disorder.
Here we discuss a case of Familial Hypokalemic Periodic Paralysis in a 45 years old male patient
referred to us with history of recurrent muscle weakness.
Key words: Familial Hypokalaemic Periodic Paralysis
Case Report
A case of 45 years old male married Hindu patient
who was referred from Department of Medicine
with history of episodes of weakness (paralysis)
in his limbs lasting from few hours to up to 3 days
for last 25 years.
These episodes of paralysis started at the age of
20 years. He would go to bed at night with no
weakness and wake at morning with inability to
move his upper or lower extremities. The episodes
would last for few hours to 3 days followed by
full recovery. During the episodes patient would
be fully conscious & oriented, alert and sensations
were also intact. These episodes vary in terms
of degree of inability to move limbs and time
duration of the episodes. He had no respiratory
or swallowing diffi culty and was able to move
his neck and facial muscles during most episodes.
But during severe episodes he had diffi culty even
in moving his eyelids, neck & facial muscles.
He denies of any pain or paraesthesia during the
episodes.
These episodes have been occurring 15-20 times
every year, more during winter, after physical
exertion and after eating sweets or sour foods.
Movement of fi ngers or other muscles helped him
recover from paralysis faster and it would take
longer time to recover if he were to sleep. Drinking
coconut water would also help him recover faster.
There were no major psychosocial stressors as-
sociated with or preceding the episodes. Patient
felt distressed by the fact that during the episodes
he would be functionally impaired. There was no
history of any substance abuse.
Family History
Patient has four brothers and one sister. Patient’s
father and father’s younger brother were suffering
from similar complains. Both died before 1 year
and patient’s uncledied during severe episode of
paralysis. Patients two brothers are also suffering
from similar complains. His youngest brother is
having more severe symptoms compared to other
brothers. He has more than 25 episodes every year
and his episodes usually last for 2-3 days.
A week after the initial consultation by the patient,
his younger brother had an episode of paralysis
after waking up from sleep. He was visited at his
home and examined. The weakness was bilateral
and involved both the proximal muscles of the
shoulders and hips as well as the distal extremities.
Patient was conscious and oriented to time, place
& person. Neurological examination of the
patient’s brother revealed fl accid type paralysis of
all extremities which involved the proximal and
distal muscles and included the hips and shoulders
but sensation was intact. Cranial nerve function
was grossly intact.
Routine blood investigation, liver enzymes and
Archives of Indian Psychiatry 13(2) October 201161
complete blood count were normal except for a
potassium level of 2mEq/lt (3.8-5.4mEq/lt). Thyroid
stimulating hormone (TSH), triiodothyronine (T3)
and thyroxine (T4) levels were also normal. Inter
episodic potassium levels were normal. General
examination & systemic examination did not
reveal any other abnormality. The patient was
diagnosed with Hypokalemic Periodic Paralysis.
Patient’s family started giving him coconut water
and it took him 2 days to recover fully from the
episode without any residual weakness.
Discussion
Conversion disorder is an illness of symptoms
or defi cits that affect voluntary motor or sensory
functions, which suggest another medical condition,
but that is judged to be caused by psychological
factors because the illness ispreceded by confl icts
or other stressors. (Sadock & Sadock, 2007)
The immediate cause of conversion disorder is a
stressful event or situation that leads the patient to
develop bodily symptoms as symbolic expressions
of a long-standing psychological confl ict or
problem. Associated features are evidence of
secondary gain,
The following disorders must be considered in the
differential diagnosis:
• multiple sclerosis (blindness resulting from
optic neuritis)
• myasthenia gravis (muscle weakness)
• myopathies (muscle weakness)
• polymyositis (muscle weakness)
• Guillain-Barré syndrome (motor and sensory
symptoms)
The usual pattern of inheritance is autosomal
dominant with reduced penetrance in women
(male-to-female ratio of 3 or 4 to 1). The typical
attack comes on during the second half of the
night or the early morning hours, after a day of
unusually strenuous exercise; a meal rich in
carbohydrates favours its development. Excessive
hunger or thirst, dry mouth, palpitation, sweating,
diarrhoea, nervousness, and a sense of weariness
or fatigue are mentioned as prodromal symptoms
but do not necessarily precede an attack. Usually
the patient awakens to discover a mild or severe
weakness of the limbs. However, diurnal attacks
also occur, especially after a nap that follows a
large meal. The attack evolves over minutes to
several hours; at its peak, it may render the patient
so helpless as to be unable to call for assistance.
Once established, the weakness lasts a few hours
if mild or several days if severe. The distribution
of the paralysis varies. Limbs are affected earlier
and often more severely than trunk muscles, and
proximal muscles are possibly more susceptible
than distal ones. The legs are often weakened
before the arms, but exceptionally the order is
reversed. The muscles most likely to escape are
those of the eyes, face, tongue, pharynx, larynx,
diaphragm, and sphincters, but on occasion even
these may be involved. When the attack is at its
peak, tendon refl exes are reduced or abolished and
cutaneous refl exes may also disappear. Sensation
is preserved. As the attack subsides, strength
generally returns fi rst to the muscles that were last
to be affected. Headache, exhaustion, diuresis,
and occasionally diarrhoea may follow the attack.
Myotonia is not seen; indeed, clinical or EMG
evidence of myotonia essentially excludes the
diagnosis of hypokalemic periodic paralysis.
Attacks of paralysis tend to occur every few weeks
and tend to lessen in frequency with advancing age.
Rarely, death may occur from respiratory paralysis
or derangements of the conducting system of the
heart. Mainly, such fatal cases were reported in the
era before modern intensive care.
Laboratory Findings
The attacks are accompanied by reduction in serum
potassium levels, as low as 1.8mEq/L, but usually
at levelsthat would not be associated with muscle
weakness in normal subjects. The fall in serum
potassium is associated with little or no increase
in urinary potassium excretion. Presumably, large
quantities of potassium enter the muscle fi bres
during an attack, but this explanation may not be
complete. Some episodes occur with near-normal
levels of potassium, and weakness persists for a
time after the serum level has been restored.. Rudel
and associates attribute the latter change to an
increased Na conductance. The ECG changes also
begin at levels of potassium that are slightly below
normal (about 3meq/L); they consist of prolonged
PR, QRS, and QT intervals and fl attening of T
waves.
Archives of Indian Psychiatry 13(2) October 2011 62
Diagnosis at a time when the patient is normal
may be facilitated by provocative tests. With the
patient carefully monitored, including the use of
an ECG, the oral administration of 50 to 100 g of
glucose or loading with 2 g of NaCl every hour for
seven doses, followed by vigorous exercise, brings
on an attack, which then can be terminated by 2 to
4 g of oral KCl (the opposite of what pertains in
hyperkalemic periodic paralysis).
Treatment
A low-sodium diet (160 mEq/day), avoidance
of large meals and exposure to cold, and
Acetazolamide 250 mg three times daily may be
helpful in preventing attacks. That Acetazolamide
reduces attacks is somewhat surprising since it is
kaluretic, but it may work through the production
of acidosis; a few patients have worsened with
the drug. Patients who are unresponsive to
Acetazolamide may be treated with the more potent
carbonic anhydrase inhibitor, Dichlorphenamide,
50 to 150 mg per day or with the potassium-
sparing diuretics Spironolactone or Triamterine
(both in doses of 25to 100 mg/day) but caution
must then be exercised with the simultaneous
administration of oral potassium supplements. The
daily administration of 5to 10 g of KCl orally in
an unsweetened aqueous solution prevents attacks
in many patients, and apparently this program can
be maintained indefi nitely. If this approach fails,
a low-carbohydrate, low-salt, high-potassium
diet combined with a slow-release potassium
preparation may be effective. For an acute attack,
0.25meq KCl/kg should be given orally or, if
this is not tolerated, some other potassium salt
may be tried. This dose may be insuffi cient, and
if there is no improvement in 1 or 2 h, KCl may
have to be given intravenously—0.05to 0.1 mEq/
kg intravenously initially in a bolus at a safe rate,
followed by 20 to 40 mEq KCl in 5% Mannitol,
avoiding glucose or NaCl as the carrier solution.
For the late progressive polymyopathy that follows
many severe attacks of periodic paralysis, Dalakas
and Engel report successful restoration of strength
by the long-term administration of the carbonic
anhydrase inhibitor Dichlorphenamide. Regular
exercise (not too strenuous) to keep the patient fi t
is desirable. (Ropper & Brown, 2005)
Conclusion
Periodic Paralysis is important to consider when
seeing a patient with sudden onset weakness or
paralysis, especially those with family history of
similar condition and no history or evidence of
other diseases and no signifi cant risk factors for
stroke.
Failure to properly diagnose and treat Periodic
Paralysis can be fatal, but rapid correction of
potassium abnormalities can resolve the symptoms
quickly and completely.
Bibliography
1. Encyclopedia of Mental Disorders. (n.d.). Retrieved from
http://www.minddisorders.com/Br-Del/Conversion-disorder.
html
2. Mastalgia, F. L., & Jones, D. H. (July 2007). Myopathies and
Muscle Diseases, Handbook of Clinical Neurology. Elsevier.
3. Ropper, A. H., & Brown, R. H. (2005). Adamsa and Victor’s
Principle of Neurology (8th ed.). McGraw-Hill.
4. Sadock, B. J., & Sadock, V. A. (2007). Synopsis of Psychiatry.
Philadelphia: Lippincott Williams & Wilkins.
Sources of support : None Confl ict of Interest : None
Dr. Rajat M. Oswal, Assistant Professor,
Dr. Devendra Chaudhari, M. D. Psychiatry
Dr. Ritambhara Y. Mehta, Professor & Head
Department of Psychiatry
Govt. Medical College,
Surat
Archives of Indian Psychiatry 13(2) October 201163
Quiz
1. Who gave “Biopsychosocial model” of disease?2. Who described temperamental di� erences among infants?3. What is “Anamnesis” in psychiatric history?4. Who escribed the term “Paranoid Pseudocommunity”- a perceived community of plotters,
for development of delusional disorders?5. For pedophilia to diagnose what should be the age of the victim according to DSM-IV-TR?6. Who � rst described the term Trichtillomania?7. Cytochrome P-450(CYP) enzymes act primarily on which cells of the body to a� ect drug
metabolism?8. Phase –III trials should be conducted in atleast how many patients to validate the � ndings
of the phase-II studies for new drug development?9. For neuroleptic induced Tardive Dyskinesia to diagnose according to DSM-IV-TR, what
should be the minimum exposure to neuroleptic medication?10. For a seizure to be e� ective in the course of ECT, it should be of how much duration?
Contributed byDr.Bhavesh Lakdawala MD
Asst.Prof of PsychiatryB.J.Medical CollegeAhmedabad
Archives of Indian Psychiatry 14(1) October 201164
Answers:
1. George Engel2. Stella Chess & Alexander ! omas3.Personal History4.Norman Cameron5.13 Years or younger6. Richard Asher7.Endoplasmic reticulum of hepatocytes8. 1000 to 3000 patients.9. 3 months (1 month if age 60 yrs or older)10. At least 25 seconds