dr. rashi thermotherapy (2)

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Dr. Rashi Goel (PT) Assistant Professor DY Patil College of Physiotherapy Pimpri, Pune

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Page 1: Dr. Rashi Thermotherapy (2)

Dr. Rashi Goel (PT)Assistant Professor

DY Pati l Col lege of PhysiotherapyPimpri, Pune

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Temperature- measure of level of heat Eg.

Right hand- cold water + left hand- in hot water Then both hands in tepid water Perception in nervous system to recognize

contrasts

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Expansion to measure temperature Mercury- along a narrow tube

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2 fixed points-

Melting point of ice : 0 degrees Boiling point of water : 100 degrees 100 parts called degrees

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Celcius and Farenheit scale

Formula for conversion

Is there any same temperature in both scales??

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100 degrees C = F? 0 degrees C = F? 98.4 degrees F = C ? 0 degrees F = C?

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Celcius scale Farenheit scale Kelvin scale

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Lower limit where no motion occurs at all Absolute zero

-273.2 degrees C

In Kelvin scale: 0 Kelvin: 0 K

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Add 273 in Celcius scale value to get Kelvin scale value

Freezing point of water? In C? in K?

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Conduction- metals Convection- sweating Radiation-electromagnetic radiation lead to

heat when absorbed

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METHODS MODES OF TRANSFER

Superficial Heating Hot water bottle

Hot pack

Hydrocollator

Hot water bath

Wax bath

Electric heating pads

Fluidotherapy

Hot air / hairdryer

Hydrotherapy

Infra red radiation

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METHODS MODES OF TRANSFER

Deep Heating Ultrasound

SWD

MWD

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Causes of heat gain Causes of heat loss

Basal metabolism Radiation to environment

Muscle contraction

Digestion

Conduction from hotter objects

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1. Physical and chemical changes2. Physiological changes- to protect the body

from damage

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13% per 1 degrees C Temp rise- denatures proteins >45 degrees C protein damage occurs, cell

and tissue destruction Phagocytosis more Cellular growth more 5-6 degrees

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Any chemical change capable of being accelerated by heat is accelerated by a rise in temperature

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Collagen melts > 50 degrees Increased extensibility 40 – 45 degrees Reduces joint stiffness Softening of collagen

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Analgesic effects of local heating

Stimulates sensory receptors of the skin Gate control mechanism Heat receptors inhibits nociceptive impulses

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Hyperalgesia – localised- CNS phenomena subcortical level- short lived

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Erythema is produced- skin surface reddens Vasodilatation

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Reduced viscosity

Increased blood flow

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Increase in fluid exchange across capillary walls and cell membranes

pH falls Acidity rises Carbon dioxide increase

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Healing Pain relief Muscle spasm Sedative effect Joint ROM Prophylaxis of pressure sores Edema of extremities Skin diseases Precursor of other treatment

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Why healing?

Controversial in early stages of inflammation

Given in chronic and post traumatic conditions

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Collagen Cellular activity Metabolism Axon reflex- dilatation Cutaneous heat receptors- sedative &

analgesic

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Secondary afferent muscle spindle nerve endings get heat

Inhibition to motor neuron Muscle excitation decreases

Pain spasm cycle

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Heating afferent muscle spindle nerve endings

Reduces muscle spasm

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As a consequence of pain relief Reflex phenomenon Psychosomatic effect

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Analgesic effect Reduced viscosity and joint stiffness Increase collagen extensibility

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Blood flow Skin breakdown

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Chronic edema of hand and foot

Add- elevation Add- active exercises

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Fungal infections- IRR

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> 45 deg.

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Local burns if direct contact Ischaemic disease Deficient cardiovascular or heat regulatory

mechanism Infections- Athlete’s foot, paronychia Acute dermatitis or eczema Oedema

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