Sinoe Medical Association1
usmleDr.Danil Hammoudi
Sinoe Medical Association2
OSTEOARTHRITIS IS LEAST LIKELY TO BE ASSOCIATED WITH:
ULCERATIVE COLITIS
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OSTEOARTHRITIS ASSOCIATED WITH
GOUT HEMOPHILIA WILSON’S DISEASE EHLERS DALOS SYNDROME
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AMBIGOUS GENITALIA
ADRENAL HYPERPLASIA
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EYES MUSCLES ABDUCTING EYEBALL
INTERNAL RECTUS
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EXTRINSIC EYE MUSCLES AND ROLE
MUSCLE INNERVATION MOVEMENTLATERAL RECTUS ABDUCENS [VI] LATERAL
MEDIAL RECTUS OCULOMOTOR [III] MEDIAL
SUPERIOR RECTUS OCULOMOTOR SUPERIOR AND MEDIAL
INFERIOR RECTUS OCULOMOTOR INFERIOR AND MEDIAL
INFERIOR OBLIQUE OCULOMOTOR SUPERIOR AND LATERAL
SUPERIOR OBLIQUE TROCHLEAR [IV] INFERIOR AND LATERAL
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EXTERNAL MUSCULATURE OF THE EYE
LEVATOR PALPEBRAE SUPERIORIS MUSCLES OPEN THE UPPER EYELID
ORBICULARIS OCULI, A SPHINCTER LIKE MUSCLE, CLOSE EYELIDS
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INCUBATION PERIOD FOR STAPHYLOCOCCAL FOOD POISONING:
2 TO 4 HOURS
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KRABBE’S DISEASE
GLOBOID CELL LEUKODYSTROPHY ONSET IN INFANT WITH RIGIDITY TERMINAL BLINDNESS DEFICIENCY OF BETA
GALACTOSIDASE ACCUMULATION OF
GALACTOCEREBROSIDE.
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COBALT IS AN ESSENTIAL COMPONENT OF:
CYANOCOBOLAMIN [B12]
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HIRSUTISM ARE ASSOCIATED WITH
ADRENAL TUMOR HYPOTHYROIDISM DANAZAL THERAPY CYCLOSPORINE THERAPY
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INCREASED IN VMA IS USUALLY SEEN IN :INCREASED IN VMA IS USUALLY SEEN IN :
NEUROBLASTOMA PHEOCHROMOCYTOMA
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LANGHERAN’S CELL HISTIOCYTOSIS CLINICAL FORMS LETTER SIWE DISEASE HAND SCHBLIER CHRISTIAN DISEASE EOSINOPHILIC GRANULOMA HASHIMOTO PRITZKER DISEASE
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HISTIOCYTOSIS, LETTERER SIWE DISEASE
ONSET IN THE FIRST YEAR HAVE SEBORRHEIC RASH OTITIS MEDIA LYMPHADENOPATHY, PURPURA ANEMIA, HEPATOSPLENOMEGALY EXTENSIVE LYTIC SKULL LESION =RAINDROP SKULL UNKNOWN ETIOLOGY WORST PROGNOSIS
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HISTIOCYTOSIS X
INTENSE PROLIFERATION OF RETICULOHISTIOCYTIC CELLS
EOSINOPHILIC GRANULOMA HAND SCHULLER CHRISTIAN DISEASE LETTER SIWE DISEASE
INTENSE PROLIFERATION OF RETICULOHISTIOCYTIC CELLS
EOSINOPHILIC GRANULOMA HAND SCHULLER CHRISTIAN DISEASE LETTER SIWE DISEASE
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EOSINOPHILIC GRANULOMA: EG-60-80% OF HISTIOCYTOSIS X-AGE 5-10 YRS MOST COMMON-BEST PROGNOSIS
HAND SHULLER-CHRISTIAN DISEASE-AGE 1-3 YRS
LETTERER SIWE DISEASE-AGE 0-1YRS
-WORST PROGNOSIS : MORTALITY 70%]
-=MALIGNANT FORM OF HISTIOCYTOSIS
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HAND SCHUELLER CHRISTIAN DISEASE
1-3 YRS, MOST COMMON HISTIOCYTOSIS FORM SKULL – MANDIBULE LESION SEMILAR EOSINOPHIL GRANULOMAS
BUT MORE NUMEROUS GEOGRAPHIC SKULL “FLOATING TEETH” ADENOPATHY HEPATOSPLENOMEGALY SKIN LESION DIABETES INSIPIDUS EXOPHTALMOS LUNG DISEASE
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LETTERER SIWE DISEASE SYNONYMS
ACUTE DIFFUSIBLE HISTIOCYTOSIS ACUTE INFANTILE
RETICULOENDOTHELIOSIS ACUTE RETICULOSIS OF INFANCY GENERALIZED HISTIOCYTOSIS NON LIPID RETICULOENDOTHELIOSIS
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CURLING ULCER OF THE STOMACH /MARJOLIN OF THE SKIN ARE CAUSES BY:
BURNS
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CEREBROSPINAL FLUID IN VIRAL ENCEPHALITIS SHOWS:
VARIABLE PLEOCYTOSIS DECREASED SUGAR LEVEL INCREASED PROTEIN LEVEL
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MORE COMMON IN WOMEN THAN IN MEN
ASSOCIATED WITH PRURITUS, JAUNDICE,XANTHOMATOUS LESIONS
INCREASED ALKALINE PHOSPHATASE AND CHOLESTEROL
MITOCHONDRIAL ANTIBODIES AND INCREASED SERUM IgM.
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SABER SHIN OCCURS MAINLY IN CHILDREN WITH:
CONGENITAL SYPHILIS
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THE MOST COMMON INTRACRANIAL GERM TUMOR
GERMINOMA
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GERMINOMAS
HIGHLY RADIOSENSITIVE
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TYPES OF GLIOMAS
ASTROCYTOMAS EPENDYNOMAS GANGLIOMAS OLIGODENDROGLIOMAS MIXED GLIOMAS
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CHILDHOOD GLIOMAS
ASTROCYTOMAS EPENDYMOMAS GANGLIOGLIOMAS
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GERM CELL TUMORS [CGTs]
GERMINOMAS TERATOMAS CHORIOCARCINOMAS ENDODERMAL SINUS TUMORS EMBRYONAL CARCINOMAS MIXED GCTs
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PINEAL PARENCHYMAL TUMORS
PINEOBLASTOMAS PINEOCYTOMAS
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PINEAL GLIOMAS
ASTROCYTOMAS EPENDYMOMAS
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PINEAL NEOPLSMS
5% OF ALL PEDIATRICS BRAIN TUMORS
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OSGOOD SCHLATTER DISEASE USUALLY OCCURS IN CHILDREN AGED
11 TO 13 YEARS
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OSGOOD SCHLATTER
OSTEOCHONDROSIS PARTIAL AVULSION OF THE TIBIA TUBEROSITY, WITH NO
INVOLVEMENT OF THE TIBIA PHYSIS COMMON CAUSES CAUSES OF KNEE PAIN IN ADOLESCENT SWELLING AND TENDERNESS JUST BELOW THE KNEE OVER
THE SHIN BONE [TIBIA] MORE COMMON IN BOYS 8-13 YEARS GIRLS, 10-15 BOYS GROWTH SPURT AND MAY AFFECT BOTH KNEES ,
ENLARGEMENT OF THE TIBIAL TUBERCLE. GOES AWAY WITH TIME
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RED SCALY WITH A SHARP RAISED BORDER ON THE PERINEUM , THIGHS AND BUTTOCKS ARE SEEN IN TINEA CRURIS [JOCK ITCH]
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TINEA CRURIS= JOCK ITCH
ANY ITCHING IN THE GROIN RASH IN MEN WHEN CAUSED BY A FUNGUS = RASH=TINEA
CRURIS CAUSE OF TINEA CRURIS= FUNGUS
DERMATOPHYTES=RINGWORM FUNGI NOT CONTAGIOUS BILATERAL DO NOT INCLUDE PENIS AND SCROTUM MIGRATE TO THE BUTTOCK AND GLUTEAL CLEFT
AREA
ANY ITCHING IN THE GROIN RASH IN MEN WHEN CAUSED BY A FUNGUS = RASH=TINEA
CRURIS CAUSE OF TINEA CRURIS= FUNGUS
DERMATOPHYTES=RINGWORM FUNGI NOT CONTAGIOUS BILATERAL DO NOT INCLUDE PENIS AND SCROTUM MIGRATE TO THE BUTTOCK AND GLUTEAL CLEFT
AREA
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DERMATOPHYTES INFECTION IN TINEA CRURI
MICROSPORUM TRICHOPHYTON EPIDERMOPHYTON
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THE MOST COMMON CAUSE OF A VIGINITIS IN A 10 YEARS OLD GIRL IS
THE MOST COMMON CAUSE OF A VIGINITIS IN A 10 YEARS OLD GIRL IS
FOREIGN BODY
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RETINOBLASTOMA
MOST COMMON IN LESS THAN 3 YEARS OF AGE
SHOW WHITE CAT’S EYES REFLEX ON FUNDOSCOPY
ARE AUTOSOMAL DOMINANT BILATERAL 30% OF CASES ARE THE MOST INTRAOCULAR TUMOR
IN CHILDREN
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RETINOBLASTOMA
LEUKOCORIA [WHITE PUPIL REFLEX] STRABISMUS EXOTROPIA ESOTROPIA RED PAINFUL EYE POOR VISION , INFLAMMATION TISSUE AROUND THE EYE ENLARGED OR DILATATED PUPIL HETEROCHROMIA FAILURE TO THRIVE EXTRA FINGERS OR TOES MALFORMED EARS RETARDATION
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NOT A CLINICAL FEATURE OF ANOREXIA NERVOSA
HISTORY OF CHILDHOOD HYPERACTIVITY
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ANOREXIA NERVOSA
AMENORRHEA BRADYCARDIA ONSET BEFORE 25 YEARS OF AGE
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CHROMOSOMAL ANALYSIS IS USEFUL IN
TESTICULAR FEMINIZATION GONADAL DYSGENESIS
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DIFFERENTIAL DIAG OF TINEA CRURIS
INTERTRIGO ERYTHRASMA SEBORRHEIC DERMATITIS OF THE
GROIN PSORIASIS OF THE GROIN CANDIDIASIS OF THE GROIN
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SINDING LARSEN JOHANSSON SYNDROME
RESEMBLES OSGOOD SCHLOTTER DISEASE, EXCEPT SYMPTOMS LOCALIZED IN THE INFERIOR POLE OF PATELLA
CHRONIC OVERTENSION OF EXTENSOR MECHANISM
PAIN OSSIFICATION DISTURBANCE SWELLING WEAK QUAD REPETITIVE LOADED KNEE FLEXION ACTIVITIES:
LIGAMENT AVULSION SOFT TISSUE CALCIFICATION STRESS FRACTURE
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OSTEOCHONDROSIS
LEGG CALVE PERTHES DISEASES =CAPITAL FEMORAL EPIPHYSIS
KOHLER DISEASE =TARSAL NAVICULAR OSGOOD SHLATTER DISEASE=TIBIAL TUBEROSITY
SCHEUERMANN DISEASE=VERTEBRAL RING EPIPHYSES
FREIBER INFRACTION =METATARSAL HEAD SEVER DISEASE = APOPHYSIS OF OS CALCIS OSTEOCHONDRITIS DISSECANS
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OSTEOCHONDROSIS DISSECANS
SUBCHONDRAL FATIGUE FRACTURE COMMONLY SEEN IN ADOLESCENT CAPITELLUM OF THE ELBOW KNEE [MEDIAL FEMORAL CONDYLE
CLOSE TO FOSSA INTERCONDYLARIS] TALUS MOUSE= OSTEOCHONDROTIC FRAGMENT MOUSE BED= SCLEROSED PIT IN ARTICULAR SURFACE
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SEVER DISEASE
OSTEOCHONDROSIS APOPHYSIS OF THE OS CALCIS
[UNDERGOES FRAGMENTATION]
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FREIBERG INFRACTION
OSTEOCHONDROSIS
METATARSAL HEAD 2ND COMMON 3RD AND 1ST LESS COMMON INFARCTION OR STRESS FRACTURE LATE ADOLESCENCE A END ARTICULAR BECOMES FLATTENED SMALL OSSICLES MAY FORM AFTER HEALING DJD IS COMMONLY LATE COMPLICATION
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SCHMORL NODE
CHONDRIFICATION DEFECTS WHERE PERIOSTEAL VESSELS PENETRATE CARTILAGE PLATE OF DISC
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KOHLER DISEASE
OSTEOCHONDROSIS OF TARSAL NAVICULAR
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LEGG CALVE PERTHES DISEASE
OSTEOCHONDROTIS DEFORMANS COXA PLANA IDIOPATHIC AVASCULAR NECROSIS OF PROXIMAL FEMORAL
EPIPHYSIS MALE UNILATERAL 4-8 YRS OLD UNCOMMON BEFORE 3Y/O SELF LIMITED CAN PROGRESS TO COXA PLANA
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MEDIASTINAL ADENOPATHY IS NOT SEEN IN
IDIOPATHIC PULMONARY FIBROSIS
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MEDIASTINAL ADENOPATHY IS SEEN IN
SARCOIDOSIS MILARY TUBERCULOSIS PNEUMOCYSTIS CARNII PNEUMONIA
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ANTERIOR MEDIASTINAL MASSES
THYMOMA TERATOMA THYROID [ECTOPIC] LYMPHOMA
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MIDDLE MEDIASTINAL MASSES
ADENOPATHY: INFECTION [BACTERIAL, GRANULOMATOUS]
NEOPLASM : LEUKEMIA, LYMPHOMA METASTASES
BRONCHOPULMONARY FOREGUT MALFORMATIONS:
ESOPHAGAL DUPLICATION CYST BRONCHOGENIC CYST SEQUESTRATION
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POSTERIOR MEDIASTINAL MASSES
SYMPATHETIC GANGLION TUMORS MASSES] NEUROBLASTOMA GANGLIONEUROBLASTOMA GANGLIONEUROMA [95% OF POSTERIOR MEDIASTINAL MASSES
NEUROFIBROMAS NEURENTERIC CYST EXTRAMEDULLARY HEMATOPOESIS PARAVERTEBRAL SOFT TISSUE MASS FROM
INFECTION
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BEHAVIOR PSYCHOTHERAPY IS DISTINGUISHED FROM COGNITIVE PSYCHOTHERAPY BY:
THE VIEW OF BEHAVIOR AS A RESPONSE TO THE ENVIRONMENT
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CHILD WITH CIRCULAR AREA OF ALOPECIA THAT ARE WOOD’S LAMP POSITIVE
TINEA CAPITIS DUE TO : MICROSPORUM CANIS TRICHOPHYTON SPECIES [TOSURANS] MICROSPORUM SPECIES FLIORESCE A BRIGHT BLUE GREEN
COLOR EPIDERMOPHYTON FLOCCOSUM IS A CAUSE OF TINEA
CRURIS
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CANDIDA ALBICANS IS RESPONSIBLE FOR
ORAL RUSH DIAPER DERMATITIS IN INFANTS
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MUMPS IS A COMMON CAUSE OF:
VIRAL PAROTITIS
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ASSOCIATED WITH EYE PAIN
ACUTE ANTERIOR UVEITIS OPTIC NEURITIS ACUTE GLAUCOMA
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CENTRAL RETINAL VEIN OCCLUSION
SUDDEN, PAINLESS, UNILATERAL LOSS OF VISION IN PATIENTS WITH :
1/HYPERCOAGULABILITY STATE [POLYCUTHEMIA RUBRA VERA]
2/DIABETES MELLITUS 3/ GLAUCOMA
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RETINAL EXAM OF CENTRAL RETINAL VEIN OCCLUSION SWELLING OF THE OPTIC DISC VENOUS DILATATION TORTUOSITY WIDESPEAD RETINAL HEMORRHAGE COTTON WOOD EXUDATE
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HEPARIN
INCREASES EFFECTIVENESS OF ANTITHROMBIN III
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COUMARIN [WARFARIN]
CAUSE THE PLASMA LEVELS OF PROTHROMBINE, FACTORS VIII, IX, X TO FALL
WARFARIN COMPETE WITH VIT K
CAUSE THE PLASMA LEVELS OF PROTHROMBINE, FACTORS VIII, IX, X TO FALL
WARFARIN COMPETE WITH VIT K
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AUER ROD
ACUTE MYELOCYTIC LEUKEMIA
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REED STENDBERG CELLS
HODGKIN’S DISEASE MIXED CELLULARITY
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ERB DUCHENNE PALSY
INJURY TO C-5 AND C-6
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NEUROBLASTOMA
THE MOST COMMON LOCATION ABDOMEN
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NEUROBLASTOMA
IT ACCOUNT FOR 15% OF ALL CHILDHOOD CANCER DEATH
35% OF CASES APPEAR DURING THE FIRST YEAR OF LIFE
IT IS ONE OF THE MOST COMMON CHILDHOOD EXTRACRANIAL SOLID CANCER
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CHILDREN WITH NEUROBLASTOMA [THE MOST COMMON CONGENITAL TUMOR OCCURING DURING THE FIRST YEAR OF LIFE] USUALLY PRESENT:
PALPABLE ABDOMINAL MASS
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THE MOST COMMON SOLID MALIGNANT TUMOR IN CHILDREN UNDER THE AGE OF 4 YEARS IS
NEUROBLASTOMA NEUROBLASTOMA
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PHEOCHROMOCYTOMA AND NEUROBLASTOMA ARE ASSOCIATED
WITH INCREASED URINARY VANILLYLMANDELIC ACID [VMA]
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IN PATIENT WITH NEUROBLASTOMA , THE INCIDENCE OF METASTASES AT TIME OF INITIAL DIAGNOSIS IS 70
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SCREENING FOR NEUROBLASTOMA IS MADE BY VMA AND HVA [HOMOVANILLIC ACID]
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THE MOST PRIMARY SITE OF NEUROBLASTOMA IS:
ADRENAL GLAND
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IN NEUROBLASTOMA, CALCIFICATION IS PRESENT IN:
45%
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SPLENOMEGALY IS NOT A CHARACTERISTIC FEATURE OF
NEUROBLASTOMA
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SPLENOMEGALY CHARACTERISTICS OF
GALACTOSEMIA CYSTINOSIS PORPHYRIA GAUCHER’S DISEASE
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NEUROBLASTOMA IS ASSOCIATED WITH
SPONTANEOUS REGRESSION
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THE INFANT OF A DIABETIC MOTHER COMMONLY DEVELOPS :
HYPOGLYCEMIA HYPERBILIRUBINEMIA HYPOCALCEMIA POLYCYTHEMIA INCREASED INCIDENCE OF CONGENITAL
MALFORMATION [HEART AND SQUELETTAL DEFECT]
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ASSOCIATION OF HORNER’S SYNDROME WITH A NECK MASS IN CHILREN IS MOST LIKELYDUE TO: NEUROBLASTOMA
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A CHILD WITH A POSTERIOR MEDIASTINAL MASS THE MOST LIKELY DIAGNOSIS IS
NEUROBLASTOMA
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MEGALOBLASTIC ANEMIA
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OCCUR DURING NORMAL GROWTH IN THE FIRST 12 MONTHS OF LIFE AT LEAST A DOUBLING OF THE INITIAL BIRTH
WEIGHT TRIPLE THEIR WEIGHT BY 12 MONTHS CLOSURE OF THE POSTERIOR FONTANELLE [2-4
MONTHS] ANTERIOR FONTANELLE CLOSES AT 6-18 MONTHS ERUPTION OF AN AVERAGE OF 6-8 DECIDUOUS
TEETH INCREASE IN LENGTH OF 25 – 30 CM
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TODDLER 24 MONTHS CAN
LINK 3-4 WORDS TOGETHER IN SIMPLE SENTENCES
ASSIST IN UNDRESSING SELF HANDLE SPOON AND CUP WELL BUILD A TOWER OF A 5-6 CUBES
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HEMOPHILUS INFLUENZAE B
UNDER 2YRS OLD MENINGITIS SEPTIC ARTHRITIS BUCCAL CELLULITIS
OLDER CHILD
EPIGLOTTITIS PNEUMONIAE
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HEMOPHILUS INFLUENZAE B
SEPTIC ARTHRITIS IN A 4 MONTH OLD INFANT
MENINGITIS IN A 3 YRS OLD CHILD EPIGLOTTITIS IN A 6 YRS OLD CHILD BUCCAL CELLULITIS IN A 15 MONTH OLD
INFANT.
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PNEUMOCOCCAL SEPSIS IS A DITINCT POSSIBILITY IN PATIENTS WITH THE FOLLOWING UNDERLYING DISEASES
PATIENT WITH ASPLENIA 10 YRS OLD WITH SICKLE CELL ANEMIA 5YR OLD SURVIVOR OF HODGKIN’S
DISEASE FOLLOWING SPLENECTOMY 8 YRS OLD WITH CHRONIC
GRANULOMATOUS DISEASE
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ESOPHAGEAL ATRESIA
Excessive secretions noted in the new born nursery
Difficulty feeding with cyanotic episode Inability to pass a catheter into the
stomach A history of polyhydramnios Commonly associated with
tracheoesophageal fistula
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OTITIS MEDIA COMPLICATION
Hearing loss Brain abscess formation, meningitis, focal
encephalitis. Cholesteatoma Facial nerve paralysis Perforation of tympanic membrane
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Otitis media
S. pneumoniae , H.influtenzae are the most common causative agents
Most prevalence infectious diseases in the childhood.
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Congenital hypothyroidism
Newborn screening programs have been initiated in most states due to the difficulty in identifying the affected children
Delay in therapeutic can lead to brain damage Premature infants frequently have transiently low
levels of thyrotropin releasing factor The breast fed infants with hypothyroidism is
partially protected due to the presence of maternal thyroid hormone in the milk
Normal birth weight , length , head circumference
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The most common type of seizure seen in the new born period is:
Subtle seizures [eye deviations,sucking, posturing with the extremities]
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Most common manifestation of acute valvular disease during the initial stages of rheumatic fever:
Mitral regurgitation Tachycardia Cardiomegaly Pericardial effusion Complication later: valvular stenosis
Mitral regurgitation Tachycardia Cardiomegaly Pericardial effusion Complication later: valvular stenosis
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Normal hematologic values in the full term infant
Polycythemia with a normal hb of 16-18 and hct of 45-60 HT and HB relatively high declining by 7 weeks of age for
premature and 2- 3 months for for the term infant = physiologic anemia in infancy.
Predominance of fetal hemoglobin Mcv =mean corpuscular volume high during neonatal period
but declines during the later infancy. Normal white blood cell count ranging from 5000 to 30000 in
the first 48h after birth with a granulocytes predominance Midly prolonged prothrombin time [PT] and PTT
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New born anemia
Hemolytic diseases Acute blood loss Chronic blood loss Impaired red blood cell production
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New born anemia etiology
I/ hemolytic disease of the new born =erythroblastosis fetalis
Blood group incompatibility