clinical presentations in respiratory disease
DESCRIPTION
Some common clinical presentations in respiratory system diseases.TRANSCRIPT
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Some Clinical PresentationsSome Clinical Presentations in Respiratory System Diseasesin Respiratory System Diseases
• Respiratory distress.
• Cough.
• Haemoptysis.
• Cyanosis
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((11 ) )RESPIRATORY DISTRESSRESPIRATORY DISTRESS
SubjectiveSubjective)Symptoms()Symptoms(
ObjectiveObjective)Signs()Signs(
• Exertional dyspnoea
• Orthopnoea
• Tachypnoea )> 20 /min(
• Inspiratory Retractions
• Active accessory muscles of respiration
• Adventitious sounds
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Dyspnoea: subjective feeling of difficulty in breathing due to increased respiratory effort.
Exertional: provoked or increased by physical activity
Types Physiologic: with more than usual daily activity
Pathologic- Psychogenic: mainly at rest, with frequent sighing
- Organic
Grades• Provoked by more than usual daily activity
• Provoked by usual daily activity• Provoked by less than usual daily activity
• Present at rest
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Progression to Grade 4 may occur over: Minutes )Acute Dyspnoea(: foreign body aspiration, bronchial asthma, pulmonary embolism Days: rapidly accumulating pleural effusion Months: interstitial lung disease Years: emphysema
OrthopnoeaIt is dyspnoea produced or aggravated on lying down,relieved )partially or completely( in the upright position.It may result from abdominal distension pushing the diaphragm upwards.
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Normal Breathing
Cheyne Stokes Breathing
Acidotic )Kussmaul( Breathing
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Suprasternal, Supraclavicular Retractions
Epigastric Retractions
Inetrcostal Retractions
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Active Accessory Muscles of Respiration
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Breath Sounds
Adventitious Sounds • Wheezes
• Crepitations
• Croup
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((22 ) )COUGHCOUGH
It is forced expiratory effort against a closed glottis which then suddenly opens with a jet of air expelled out, possibly along with secretions.
Types Dry )Irritant( Cough: The irritant stimulus may be obvious )smoke, dust, pharyngitis( and may not. Two common "concealed" causes of resistant
dry cough are post- nasal discharge and GERD )gastro-oesophageal reflux disease(.
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• Wet )Productive( Cough:
Frothy Pink: pulmonary oedema Mucoid: bronchial asthma, chronic bronchitis Mucopurulent, Purulent )yellowish, thick(: infections Rusty: lobar pneumonia Greenish/Bluish: Gram negative infection Foul Smelling: anaerobic infection Blackish: smoker, coal workers
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((33 ) )HAEMOPTYSISHAEMOPTYSIS
It is coughing of blood or blood tinged sputum due to bleeding from the respiratory tract below the vocal cords.
Bleeding originating above vocal cords )nose, mouth, larynx( may produce false haemoptysis.
HaemoptysisHaemoptysisHaematemesisHaematemesis
It isIt isCoughing of bloodCoughing of bloodVomiting of Vomiting of bloodblood
ColorColorBright redBright redDark redDark red
OdorOdor------soursour
ReactionReactionalkalinealkalineacidicacidic
Mixed Mixed withwith
Sputum, air (frothy)Sputum, air (frothy)foodfood
SputumSputumBlood tinged for 12 – 24 Blood tinged for 12 – 24 h after the attackh after the attack
normalnormal
StoolStoolnormalnormalmelenamelena
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((44 ) )CYANOSISCYANOSIS
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Cyanosis bluish discoloration of skin, mucous membranes due to presence of:
- > 5 gm deoxy Hb /100 mL blood Or - abnormal Hb )met or sulph Hb(
in surface capillaries.
Normal level of deoxy Hb /100 mL blood:- Arterial: 0.75 gm- Capillary 2.25 gm- Venous 3.75 gm
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Central CyanosisCentral Cyanosis1
23
4
1( Hypoventilation- alveolar PO2: eg, high altitude
- Obstructive lung diseases- Restrictive lung diseases
2( Shunt- Cardiac Rt to Lt shunt )congenital
cyanotic heart disease(: eg, Fallot tetralogy
- Pulmonary: pulmonary AV fistula
3( Diffusion Defect )Alveolo-Capillary Block(- Pulmonary fibrosis- Pulmonary oedema
4( Ventilation Perfusion )V/Q( Mismatch- Pulmonary embolism )ventilation > perfusion = dead space effect(
- Atelectasis/collapse )perfusion > ventilation = shunt effect(- Most pulmonary disorders produce hypoxia by more than one
mechanism. V/Q mismatch is the most common.
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Peripheral CyanosisPeripheral Cyanosis1( Generalized
stagnation )low COP(
i. HF
ii. Shock
2( Localized stagnation
i. Arterial
ii. Venous
Chemical CyanosisChemical Cyanosis
1( Met-haemoglobin
2( Sulph-haemoglobin
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Central CyanosisPeripheral Cyanosis
Cause O2 saturation of core
arterial blood ( O2
loading by cardiopulmonary circulation)
Stagnant circulation - peripheral arterial flow- O2 extraction by tissues
DistributionWarm areas (tongue, interior of lips), all over
Cold areas: tip of nose, lobule of ear, fingers, toes
TemperatureWarm Cold
Clubbing+-
Polycythaemia+-
Effect of- Exercise- Warming- Oxygen
May No effect
to variable extent, maximal in low atmospheric PO2 and minimal in shunt
No effect
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Hypoxia Low Oxygen Tension
Hypoxia TypeCyanosis
Hypoxic HypoxiaCentral
Stagnant HypoxiaPeripheral
Anaemic Hypoxia---
Histotoxic Hypoxia---