2020 self study #4 course - college of dentistrycimetidine, and those with hepatic diseases patients...

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2020 Course #4 SelfStudy Course Contact Us: Phone 614‐292‐6737 Toll Free 1‐888‐476‐7678 Fax 614‐292‐8752 E mail [email protected] Web dentistry.osu.edu/sms The Ohio State University College of Dentistry 305 W. 12th Avenue Columbus, OH 43210 The Ohio State University College of Dentistry is a recognized provider for ADA CERP credit. ADA CERP is a service of the American Dental Association to assist dental professionals in identifying quality providers of continuing dental education. ADA CERP does not approve or endorse individual courses or instructors, nor does it imply acceptance of credit hours by boards of dentistry. Concerns or complaints about a CE provider may be directed to the provider or to the Commission for Continuing Education Provider Recognition at www.ada.org/cerp. The Ohio State University College of Dentistry is approved by the Ohio State Dental Board as a permanent sponsor of continuing dental education. This continuing education activity has been planned and implemented in accordance with the standards of the ADA Continuing Education Recognition Program (ADA CERP) through joint efforts between The Ohio State University College of Dentistry Office of Continuing Dental Education and the Sterilization Monitoring Service (SMS). Course Instructions: Read and review the course materials. Complete the 15 question test. A total of 12 questions must be answered correctly for credit. Submit your answers online at: http://dentistry.osu.edu/sms‐ continuing‐education Check your email for your CE certification of completion (please check your junk/spam folder as well). About SMS CE courses: TWO CREDIT HOURS are issued for successful completion of this self‐study course for the OSDB 2019‐2021 biennium totals. CERTIFICATE of COMPLETION is used to document your CE credit and is emailed to each course participant. ALLOW 2 WEEKS for processing of your certificate. Frequently Asked Questions: Q: Who can earn FREE CE credits? A: EVERYONE All dental professionals in your office may earn free CE credits. Each person must read the course materials and submit an online answer form independently with their OWN email address. Q: Where can I find my SMS number? A: Your SMS number can be found in the upper right hand corner of your monthly reports, or, imprinted on the back of your test envelopes. The SMS number is the account number for your office only, and is the same for everyone in the office. Q: How often are these courses available? A: Four times per year (8 CE credits).

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  • 2020Course#4Self‐Study

    Course

    ContactUs:Phone

    614‐292‐6737TollFree

    1‐888‐476‐7678Fax

    614‐292‐8752E‐mail

    [email protected]

    dentistry.osu.edu/sms

    TheOhioStateUniversityCollegeofDentistry305W.12thAvenueColumbus,OH43210

    TheOhioStateUniversityCollegeofDentistryisarecognizedproviderforADACERPcredit.ADACERPisaserviceoftheAmericanDentalAssociationtoassistdentalprofessionalsinidentifyingqualityprovidersofcontinuingdentaleducation.ADACERPdoesnotapproveorendorseindividualcoursesorinstructors,nordoesitimplyacceptanceofcredithoursbyboardsofdentistry.ConcernsorcomplaintsaboutaCEprovidermaybedirectedtotheproviderortotheCommissionforContinuingEducationProviderRecognitionatwww.ada.org/cerp.

    TheOhioStateUniversityCollegeofDentistryisapprovedbytheOhioStateDentalBoardasapermanentsponsorofcontinuingdentaleducation.ThiscontinuingeducationactivityhasbeenplannedandimplementedinaccordancewiththestandardsoftheADAContinuingEducationRecognitionProgram(ADACERP)throughjointeffortsbetweenTheOhioStateUniversityCollegeofDentistryOfficeofContinuingDentalEducationandtheSterilizationMonitoringService(SMS).

    CourseInstructions:

    Readandreviewthecoursematerials.

    Completethe15questiontest.Atotalof12questionsmustbeansweredcorrectlyforcredit.

    Submityouranswersonlineat:http://dentistry.osu.edu/sms‐continuing‐education

    CheckyouremailforyourCEcertificationofcompletion(pleasecheckyourjunk/spamfolderaswell).

    AboutSMSCEcourses: TWOCREDITHOURSareissued

    forsuccessfulcompletionofthisself‐studycoursefortheOSDB2019‐2021bienniumtotals.

    CERTIFICATEofCOMPLETIONisusedtodocumentyourCEcreditandisemailed toeachcourseparticipant.

    ALLOW2WEEKS forprocessingofyourcertificate.

    FrequentlyAskedQuestions:

    Q:WhocanearnFREECEcredits?

    A:EVERYONE‐ AlldentalprofessionalsinyourofficemayearnfreeCEcredits.EachpersonmustreadthecoursematerialsandsubmitanonlineanswerformindependentlywiththeirOWNemailaddress.

    Q:WherecanIfindmySMSnumber?

    A:YourSMSnumbercanbefoundintheupperrighthandcornerofyourmonthlyreports,or,imprintedonthebackofyourtestenvelopes.TheSMSnumberistheaccountnumberforyourofficeonly,andisthesameforeveryoneintheoffice.Q:Howoftenarethesecoursesavailable?

    A:Fourtimesperyear(8CEcredits).

  • Learning Objectives:

    a. Discuss neurophysiology and action potentialb. List and describe the composition of local anesthetic agents c. Discuss the properties, precautions, and contraindications

    for the following local anesthetics: lidocaine, mepivacaine, prilocaine, articaine, bupivacaine

    d. Discuss the importance of obtaining patient’s medical/dental history

    e. List the steps to administering successful local anesthesia f. Correlating the maxillary and mandibular injections to the

    teeth and areas anesthetized

    2020Course#4

    OriginalAuthors:IrinaNovopoltseva,RDH,

    MS

    ReleaseDate:December7,2020

    8:30amEST

    LastDaytoTakeCourseFreeofCharge:January7,2021

    4:30pmEST

    LocalAnesthesia:RefresherfortheDental

    Professional

    ThisisanOSDBCategoryB:Supervisedself‐instructioncourse

    About the AuthorIrina Novopoltseva, RDH, MSAssistant Professor- Clinical

    College of Dentistry Division of Dental Hygiene

    Neither I nor my immediate family have any financial interests that would create a conflict of interest or restrict my judgement with regard to the

    content of this course.

    2

  • 3

    One of the most important skills required of dental practitioners is the ability to provide

    safe and effective local anesthesia. The agents and armamentarium available today

    provide the clinician with an array of options to effectively manage the pain associated

    with dental procedures. We came a long way from the time when the numbing

    properties of the cocaine were first recognized, to the development of the amide

    anesthetics implemented today into the dental care of our patients.1

    Local anesthetics work by diffusing into a nerve and blocking Na+ channels, preventing

    nerve impulses from reaching the brain. As long as the concentration of local

    anesthetic outside of the nerve is greater than that inside, the drug will diffuse into the

    nerve. Once the equilibrium is achieved, the diffusion stops. Blood vessels inside and

    outside the nerve continue to ‘carry away’ local anesthetic.2 The concentration of local

    anesthetic inside the nerve exceeds the concentration of it outside the nerve, thus the

    diffusion of the drug reverses. Consequently, the local anesthetic diffuses out of the

    nerve. As the local anesthetic concentration in the nerve decreases, a point is reached

    at which nerve impulses begin to propagate and reach the brain.2,3

    Local anesthetics used in dentistry are manufactured in a single-use cartridge. The

    cartridge is a glass cylinder containing the local anesthetic drug, along with other

    ingredients.3 In the United States, each cartridge of local anesthetic is designed to

    contain 2.0ml of solution. However, by the time the silicone rubber stopper is added to

    the cartridge, it can contain anywhere between 1.7ml-1.76 ml.2

    Local anesthetics are vasodilators, regardless of the rate of absorption, the blood

    vessels around injection side will immediately begin to absorb the anesthetic by

    causing vasodilation of the blood vessels. This physiological process results in the

    increased blood flow to the injection site.3 Consequently causing higher blood levels of

    local anesthetic, increasing the risk of systemic toxicity, and rate of anesthetic

    absorption into the bloodstream. The anesthetic solution is carried away from the

    injection site, causing decrease in duration and rapid rate of diffusion. This chemical

    process increases bleeding at the injection site.2,3

  • In addition to the local anesthetic drug, the dental cartridge contains several other

    ingredients: vasoconstrictor, preservative, reducing agent, and fungicide.2

    Vasoconstrictors are combined with the anesthetic solution to counteract the vasodilating

    properties of local anesthetics. The vasoconstrictor function includes constricting the

    blood vessels leading to a decrease in blood flow to the injection site.4 All while

    supporting the increased duration of action, and reduction of bleeding at the injection site.3

    Since vasoconstrictors are unstable with a short shelf life, the addition of a preservative

    delays these properties. Thus, only the local anesthetic agents with vasoconstrictors

    contain the preservatives. Sodium bisulfite, metabisulfite, or acetone sodium bisulfite are

    the vasoconstrictor preservatives only added to the local anesthetic agents with

    vasoconstrictors.3 As a chemical property, sodium bisulfite dissociates in water into

    bisulfite and sodium ions, decreasing the pH of the agent.5

    Local anesthesia is the temporary loss of sensation or pain in one part of the body

    produced by a topically applied or injected agent without depressing the level of

    consciousness. Dental anesthetics fall into two groups: amides (lidocaine, mepivacaine,

    prilocaine and articaine), which are metabolized in the liver, and esters (procaine,

    benzocaine), metabolized in the blood.2 Due to the high degree of hypersensitivity to

    injectable esters, all injectable local anesthetics manufactured in a single-use dental

    cartridge are of amide group. Procaine is available in the multidose vials and is used in

    medicine.3 Typically, procaine and the longer-acting related compound, procaine amide,

    have been most widely employed for the treatment of the various ventricular arrhythmias

    occurring during cardiac operations.6 Benzocaine is one of the most commonly used

    topical anesthetics available in various forms (i.e. gel, spray, ointment, and patch). It is

    preferred due to rapid onset, slow absorption into circulation, therefore having a desired

    low systemic toxicity. However, methemoglobinemia has been reported with higher

    concentrations of 14% to 20% spray.3,7

    Supraperiosteal injection anesthetizes a small area by depositing anesthetic near the

    terminal nerve endings and is referred to as infiltration.3 Whereas with a nerve block, an

    injection of local anesthetic is in the area of a nerve trunk.2,3

    4

  • 5

    Available Formulations

    Lidocaine 2% plain Lidocaine 2% 1:50,000 epinephrine 

    Lidocaine 2% 1:100,000 epinephrine 

    ADA Color coding band

    Light Blue Green Red

    Onset of action (in minutes)

    2‐3 2‐3 2‐3

    Duration category Very short Intermediate IntermediateDuration pulpal tissues (in minutes)

    5‐10 60 60

    Duration soft tissues (in minutes)

    60‐120 180‐300 180‐300

    Precautions with Vasoconstrictors 

    N/A Patients with significant cardiovascular disease, elderly patients with sensitivity to epi.Patients taking non‐selective beta‐blockers, CNS stimulants, tricyclic antidepressants, and/or allergic to sodium bisulfite

    Patients with significant cardiovascular disease, elderly patients with sensitivity to epi.Patients taking non‐selective beta‐blockers, CNS stimulants, tricyclic antidepressants, and/or allergic to sodium bisulfite

    Precautions with Local Anesthetic

    Patients taking non‐selective beta‐blockers, central nervous system depressants, cimetidine, and those with hepatic diseases 

    Patients taking non‐selective beta‐blockers, central nervous system depressants, cimetidine, and those with hepatic diseases and renal dysfunction

    Patients taking non‐selective beta‐blockers, central nervous system depressants, cimetidine, and those with hepatic diseases and renal dysfunction

    Helpful Tips  Not commonly used Low risk of systemic toxicityBest choice for bleeding control (infiltrate small amnt. into area requiring hemostasis)

    Most used anestheticLow risk of systemic toxicity Best choice for pregnancy (category B)Can be used for significant cardiovascular disease at decreased dose of 0.04mg (2.2 cartridges) per appt.

    The tables that follow describe the properties, precautions, and contraindications of each

    amide group local anesthetic mentioned above.

  • 6

    Available Formulations

    Mepivacaine 3%Plain

    Mepivacaine 2% 1:20,000 levonordefrin  

    ADA Color coding band

    Tan Brown

    Onset of action (in minutes)

    2‐4 1.5‐2

    Duration category Short IntermediateDuration pulpal tissues (in minutes)

    20 (Supraperiosteal)40 (Block)

    60

    Duration soft tissues (in minutes)

    120‐180 180‐300

    Precautions with Vasoconstrictors 

    N/A Patients with significant cardiovascular disease, elderly patients with sensitivity to epi.Patients taking non‐selective beta‐blockers, CNS stimulants, tricyclic antidepressants, and/or allergic to sodium bisulfite 

    Precautions with Local Anesthetic

    Patients taking beta‐blockers, central nervous system depressantsSevere renal dysfunctionHepatic diseaseRisk of systemic toxicity Use with caution in pediatric patients

    Patients taking non‐selective beta‐blockers, central nervous system depressantshepatic diseases renal dysfunction

    Helpful Tips Good choice when a vasoconstrictor is contraindicated utilizing nerve block

    Low risk of systemic toxicityCan be used for patients with significant cardiovascular disease at 0.2 mg (2.2. cartridges) per appt.

  • 7

    Available Formulations

    Prilocaine 4% Plain Prilocaine 4% 1:200,000 epinephrine 

    ADA Color coding band

    Black Yellow

    Onset of action (in minutes)

    2‐4 2‐4

    Duration category Short (Supraperiosteal) Intermediate (Block)

    Intermediate

    Duration pulpal tissues (in minutes)

    10‐15 (Supraperiosteal)   

    40‐60 (Block)

    60‐90

    Duration soft tissues (in minutes)

    90‐120 (Supraperiosteal)

    120‐240 (Block)

    180‐480

    Precautions with Vasoconstrictors 

    N/A Patients with significant cardiovascular disease, elderly patients with sensitivity to epi.Patients taking non‐selective beta‐blockers, CNS stimulants, tricyclic antidepressants, and/or allergic to sodium bisulfite

    Precautions with Local Anesthetic

    Risk for systemic toxicity 

    Avoid using on patients with sickle cell anemia

    Risk of methemoglobinemia 

    Patients taking central nervous system depressants

    Avoid using on patients with sickle cell anemia

    Risk of methemoglobinemiaHelpful Tips Least vasodilatory properties of all 

    amide anesthetics

    Metabolizes in lungs and liver, ؞patients with hepatic disease less of a concern 

    Best choice for pregnancy (category B)

    Least toxic of all amide local anesthetics

    Good choice for elderly patients, hyper‐sensitive to epinephrine

    Best choice for patients with significant cardiovascular disease, or those needing modifications due to epinephrine

  • 8

    Available Formulations

    Articaine 4% 1:100,000 epinephrine 

    Articaine 4% 1:200,000 epinephrine 

    Bupivacaine 0.5% 1:200,000 epinephrine 

    ADA Color coding band

    Gold Silver Blue

    Onset of action (in minutes)

    1‐2 (Supraperiosteal)

    2‐2.5 (Block)

    1‐2 (Supraperiosteal)

    2‐3 (Block)

    6‐10

    Duration category Intermediate Intermediate LongDuration pulpal tissues (in minutes)

    60‐75 45‐60 90‐180

    Duration soft tissues (in minutes)

    180‐360 120‐300 240‐540

    Precautions with Vasoconstrictors 

    Patients with significant cardiovascular disease, elderly patients with sensitivity to epi.Patients taking non‐selective beta‐blockers, CNS stimulants, tricyclic antidepressants, and/or allergic to sodium bisulfite

    Patients with significant cardiovascular disease, elderly patients with sensitivity to epi.Patients taking non‐selective beta‐blockers, CNS stimulants, tricyclic antidepressants, and/or allergic to sodium bisulfite

    Patients with significant cardiovascular disease, elderly patients with sensitivity to epi.Patients taking non‐selective beta‐blockers, CNS stimulants, tricyclic antidepressants, and/or allergic to sodium bisulfite 

    Precautions with Local Anesthetic

    Patients taking central nervous system depressants

    Patients with myasthenia gravis

    Patients taking central nervous system depressants

    Patients with myasthenia gravis

    Patients taking non‐selective beta‐blockers

    Patients taking central nervous system depressants

    Patients prone to self‐mutilation (special needs/young children)

    Helpful Tips Safer choice for patients with hepatic disease 

    Safer drug to re‐administer later in the appointment

    Best choice when mandibular supraperiosteal injection is needed 

    Safer choice for patients with hepatic disease 

    Good choice for elderly patients hyper‐sensitive to epinephrine

    Safer drug to re‐administer later in the appointment (due to short half‐life)

    Best choice when mandibular supraperiosteal injection is needed

    Procedures requiring long duration of anesthesia

    Greater risk of systemic toxicity 

    Good alternative when profound anesthesia cannot be achieved with other anesthetics

  • Over time, the administration of local anesthesia can become mundane to the clinician, as

    the patient is often viewing it as a stressful experience and part of their dental visit. For

    many of our patients, the anxiety and anticipation of receiving a ‘shot’ could generate an

    emergency situation.3 Syncope, is the most common medical emergency observed in the

    dental office, and it is most frequently linked to the administration of local anesthesia.6

    Dental professionals must be mindful of not allowing the administration of the local

    anesthesia to become mundane and repetitive. Therefore, establishing effective

    communication and psychological support is essential in developing patient rapport and

    instilling confidence thus reducing the likelihood for an emergency. Implementing the

    following steps will assist each provider to administer safe, effective, and will provide a

    comfortable patient experience.

    The dental provider must evaluate a patient by completing a thorough medical and dental

    history, including physical and psychological evaluations. If necessary, a medical clearance

    evaluation should be requested and before any dental treatment rendered the vital signs

    should be taken and recorded.7 The care plan must be tactfully developed in collaboration

    with the patient involving their risk assessments and expectations. It should be viewed as a

    mutual road map for the clinician and the patient. The benefits and risks of all the

    treatments should be discussed including the administration of local anesthesia. One

    cannot overlook the importance of discussing the risk associated with not receiving

    treatment. The clinician in detail should discuss the treatment including the type of

    anesthetic, injection(s), and any post-operative instructions.8 Prior to treatment, the

    informed consent should be presented, and any questions need to be addressed, after

    which, the treatment can be accepted by the patient. The written agreement of the care

    plan becomes a legal binding contract between the patient and the provider.4 Based on the

    care plan, the clinician should select the appropriate injection(s) and the agent(s). In this

    selection process, the dental provider ought to determine the need for pulpal and/or soft

    tissue anesthesia and select an anesthetizing agent taking into consideration the

    medical/dental history, hemostasis, duration of the procedure, and a potential for self-

    mutilation.6

  • 10

    Ideally, the preparation of all the equipment and supplies should be completed prior to

    patient arriving to the operatory. The selection of the syringe is important, considering

    the clinician’s hand size, as it relates to be able to properly aspirate. The view of the

    syringe, needle, and cartridges can be unsettling for a patient, therefore, assembled

    armamentarium along with any additional supplies should be kept out of patient’s

    sight. The supine position, with the patient’s feet slightly elevated, is the recommended

    position for the administration of local anesthesia. This is the best position to treat

    vasodepressor syncope, the most common dental emergency due to local

    anesthesia.3,4 The injection site should be evaluated for any anatomical abnormalities

    and/or trauma and dried prior to the application of a topical anesthetic. Topical

    anesthetics are designed to provide soft tissue anesthesia prior to the needle insertion.

    They do not contain vasoconstrictors, therefore rapidly absorb when applied to the

    mucous membrane. You would want to follow the manufacturers recommendations

    and guidelines for usage, but typically the application of the topical anesthetic is

    indicated anywhere between 1 – 2 minutes.12

    During this window of time, it is advised to provide supportive communication to the

    patient, to help elevate any anxiety. Following the application of topical anesthetic,

    redry the injection site to remove any excess and offer a dry area effective for

    visualization of the injection site. Palpate the anatomical landmark to determine the

    insertion site.2-4

    As the clinician picks up the syringe, the following stipulations must be evaluated. The

    orientation of the bevel, should be oriented toward the bone to ensure the proximity to

    the periosteum. The dental professional should hold the syringe palm up, with the

    large window facing upward. It is suggested to expel few drops of the local anesthetic

    to guarantee a free flow of the solution, and ensure the harpoon is fully engaged in the

    rubber stopper. The clinician should double check the safety protocol such as the

    needle sheath protector, patient protective eyewear.2,3

  • 11

    Establishing a fulcrum is essential in administering safe and comfortable injections. This is

    also critical during the aspiration in two planes, as it will ensure the minimum movement

    of the needle. Retracting the tissue taut at the injection site serves two purposes, it allows

    for greater clinical visibility, and less trauma with needle insertion. Concurrently, the

    clinician has to provide encouraging and reassuring words to the patient while observing

    the facial expressions and overall well-being.13

    Aspiration is one of the most critical steps in the prevention of administrating the local

    anesthetic directly into the blood vessel. Once the desired location is reached, aspirate by

    pulling back on the thumb ring gently for about 1-2mm. Following the successful negative

    aspiration, rotate the barrel of the syringe 45° and aspirate the second time.2-4 Aspirating

    in two planes ensures that the needle is not located in the blood vessel, and that first

    aspiration is not a false negative. A negative aspiration is when there is no blood in the

    cartridge, rather a clear air bubble. Whereas a positive aspiration of a slight reddish

    discoloration at the diaphragm indicated venous penetration. The clinician should

    reposition the needle, re-aspirate, and proceed if aspiration is negative. The bright red

    blood rapidly filling the cartridge is indicative of arterial penetration. Thus, the clinician

    needs to remove the syringe, replace the cartridge, and repeat the procedure.2

    Once the clinician successfully aspirated in two planes, the anesthetic solution should be

    deposited at a slow rate of 1 mL per minute. Therefore, a cartridge of 1.8mL should be

    deposited in approximately 2 minutes for patient comfort, and most importantly the safety.

    The slow injection prevents the solution from tearing the tissue at the injection site.2,3

    Some studies have linked the slow deposition of the anesthetic solution for the Inferior

    Alveolar Block providing a faster onset with greater efficacy.14 Following the completion of

    the injection, the clinician should slowly withdraw the needle from the tissue. The needle

    must be recapped following safety protocol to prevent needle punctures by the clinicians.

    Succeeding, the proper safety protocol, the clinician should monitor the patient for

    possible adverse reactions to the anesthetic. Under no circumstance, the patient should

    be left unattended after administration of local anesthetic. The delivery of the local

    anesthesia must be documented and becomes a part of the patient’s permanent record.6

  • 12

    Maxillary Anesthesia In effort to provide patients with successful anesthesia, dental professionals must

    determine how the trigeminal (V) nerve along with its branches can be anesthetized in

    various ways with clinical effectiveness.2 The local anesthesia on the maxillary arch is

    typically more effective due to the anatomical considerations. The facial cortical plate of the

    maxillae is less dense and more porous in comparison to the mandible. In addition, the

    maxillae presents with less variations in relations of anatomical structures and nerves

    associated with the local anesthesia landmarks.2

    Maxillary nerve blocks anesthetize various maxillary nerve branches and have high level of

    clinical effectiveness when administered correctly. To accomplish the highest level of

    patient comfort with the maxillary facial nerve blocks, a clinician must avoid any bony

    contact with a needle during the injection. The exception to this concept is the infraorbital

    block. In addition, a clinician is required to establish a stable fulcrum in effort to minimize

    the needle movement within the tissue during a deposition. There is no significant evidence

    supporting the shaking motion of the upper lip during the administration of a local

    anesthetic as a patient management strategy.2,5 During patient evaluation, a dental

    professional must access the patient’s anatomy. For example, the site of deposition for the

    maxillary facial nerve blocks is superior to the apex of each target tooth. The topical

    anesthetic is yet another means to increase patient comfort during the administration of the

    local anesthetic. It should be applied in the vestibule at the height of the maxillary

    mucobuccal fold, with the focus on the redder, soft tissue of the superior alveolar mucosa.2

    The following set of charts will discuss maxillary facial and palatal nerve blocks including

    areas anesthetized, landmarks, and potential complications for each injection.

  • 13

    INJECTION Middle Superior Alveolar Block (MSA)

    Branch of Trigeminal Nerve Middle Superior Alveolar branch of infraobital nerve. (V2)

    Area Anesthetized (Teeth &/orSoft Tissue)

    Field block that includes maxillary premolars & MB root of Max. 1stmolar, periosteum and buccal soft tissue of same area. Absent in a large % of the population.

    Needle 25, 27 or 30 short

    Landmarks Apex Max 2nd premolar

    Insertion/ Penetration Site Height of muccobuccal fold over Max. 2nd premolar.

    *Approximate Depth of Penetration 

    4‐10mm 1/4 – 1/2”

    Deposition/ Target Site  Para‐periosteal through the mucous membrane over apex of 2ndpremolar. 

    *Volume of Anesthetic 1.0‐1.2 (1/2‐2/3 cartridge)Potential Complications/ Additional Considerations

    Pain if periosteum is scraped. Ballooning of tissue possible.Some patients do not have MSA.

    INJECTION Posterior Superior Alveolar Block (PSA)

    Branch of Trigeminal Nerve Posterior Superior Alveolar Max. Division (V2)

    Area Anesthetized (Teeth &/or Soft Tissue)

    3rd, 2nd, 1st max. molars (except mesio‐buccal root of 1st perm. molar if MSA is present) periosteum, and buccal soft tissue of same area.

    Needle 25 or 27 short

    Landmarks Maxillary Tuberosity Alveolar canal openings Zygomatic Process of maxilla

    Insertion/ Penetration Site Height of mucobuccal fold in concavity distal to zygomatic buttress, distal of 2nd molar, 45° angle from occlusal plane

    *Approximate Depth of Penetration 

    (3/4”) 16mm(3‐4mm from hub of short needle)

    Deposition/ Target Site  Posterior, superior, & medial to max. tuberosity; 45° angle to occlusal plane; 45° angle to midsaggital plane 

    *Volume of Anesthetic 1‐1.8ml (3/4 –1 cartridge)

    Potential Complications/ Additional Considerations

    Mandibular anesthesia if anesthetic is deposited too far laterally.No bony landmark, risk of hematoma; hemorrhage, may be problem for some patient. May require 2nd injection for MB root of 1st molar. 

  • 14

    INJECTION Infraorbital Block (IO) 

    Branch of Trigeminal Nerve Infraorbital is a branch of the Maxillary nerve (V2)Area Anesthetized (Teeth &/orSoft Tissue)

    Field block that includes maxillary central, lateral, canine, periosteum, and facial/labial soft tissue of same area (to midline). 

    Needle 27 or 25 gauge needle is recommended ‐ 27 gauge short most commonly used

    • if larger individual, may need a long needleLandmarks Extraoral: IO rim, IO foramen, zygomaticomaxillary suture

    Intraoral: Maxillary first premolar, maxillary mucobuccal fold

    height of IO foramen varies based on facial size, vestibular depth, and age

    • adult 8‐10 mm below the IO ridge

    • children ‐ shorter distanceInsertion/ Penetration Site height of the mucobuccal fold directly over the first premolar*Approximate Depth of Penetration 

    16mm or ¾ of a short needle or ½ of a long needle 

    Deposition/ Target Site  Depth adequate to reach the foramen

    *Volume of Anesthetic 0.9‐1.2 ml or ½ ‐ 2/3 of a cartridge 

    Potential Complications/ Additional Considerations

    Pain if periosteum is scraped. Ballooning of tissue possible.

    INJECTION (Anterior Superior Alveolar Block) ASA

    Branch of Trigeminal Nerve Anterior Superior Alveolar branch of infraorbital n. (V2)

    Area Anesthetized (Teeth &/or Soft Tissue)

    Field block that includes maxillary central, lateral, canine, periosteum, and facial/labial soft tissue of same area (to midline). 

    Needle 25, 27, or 30 short

    Landmarks Canine fossae, located between lateral and canine.Infraorbital foramen vs. Apex of canine

    Insertion/ Penetration Site Height of MB fold mesial to root of canine at canine fossa.*Approximate Depth of Penetration 

    4‐6mm 1/4”

    Deposition/ Target Site  Para‐periosteal through the mucous membrane mesial to the apex of max. canine. 

    *Volume of Anesthetic 1.0‐1.2 (1/2‐2/3 cartridge)Potential Complications/ Additional Considerations

    Pain if periosteum is scraped. Ballooning of tissue possible.Often central innervation overlap; may require additional infiltration over central.

  • 15

    INJECTION Nasopalatine Nerve Block (NP)

    Branch of Trigeminal Nerve Nasopalatine nerve, branch of pterygo‐palatine nerve (V2)

    Area Anesthetized (Teeth &/orSoft Tissue)

    No teeth.

    Anterior 1/3 of hard palate, lingual tissues from cuspid to cuspid. 

    Needle 25, 27 or 30 short

    Landmarks Max. centrals, incisive papilla

    Insertion/ Penetration Site Para‐periosteal through the mucous membrane next to incisive papilla at 45 degree angle

    *Approximate Depth of Penetration 

    1/8 – 1/4 “ (usually 3‐6mm)

    Deposition/ Target Site  At the incisive foramen. One injection will anesthetize both right & left NP nerves.

    *Volume of Anesthetic .2‐.45 ml 1/8‐1/4 cartridge Potential Complications/ Additional Considerations

    Necrosis of soft tissue from vasoconstrictor is possible.

    No hemostasis except in injection area, traumatic injection

    INJECTION Greater Palatine Nerve Block (GP)

    Branch of Trigeminal Nerve Greater (or Anterior) Palatine, branch of pterygopalatine n. (V2)

    Area Anesthetized (Teeth &/orSoft Tissue)

    No teeth

    Hard palate & lingual tissue posterior to 1st premolar & medial to midline

    Needle 25, 27 or 30 short

    Landmarks Vertical and horizontal processes of maxillae & palatine bones. 

    Insertion/ Penetration Site Anterior to Greater Palatine foramen & junction of Max. alveolar process and palatine bone. 

    *Approximate Depth of Penetration 

    1/8 – 1/4 “

    (usually 3‐6mm)Deposition/ Target Site  Slightly anterior to greater (anterior) palatine foramen. 

    *Volume of Anesthetic 0.45 to 0.6 ml (1/4 to 1/3 cartridge)Potential Complications/ Additional Considerations

    No hemostasis except in injection are

  • 16

    Mandibular Anesthesia

    Local anesthesia on the mandibular arch is achieved by anesthetizing the mandibular

    nerve of the trigeminal or cranial nerve (V) along with its branches. The mandible is less

    porous and dense than the maxillae, this anatomical difference can be evaluated on the

    panoramic radiograph. Therefore, nerve blocks are favored to supraperiosteal injections

    on the mandibular arch.2,3

    The following set of charts will discuss mandibular nerve blocks including areas

    anesthetized, landmarks, and potential complications for each injection.

    INJECTION Inferior Alveolar Nerve Black (IA/LINGUAL)

    Branch of Trigeminal Nerve Inferior Alveolar nerve: branch of posterior root of Mandibular (V3)

    Lingual nerve: branch of posterior root of Mandibular (V3).Area Anesthetized (Teeth &/or Soft Tissue)

    I.A. nerve: Mandibular teeth to midline (molars, premolars, cuspid, central, lateral); body of mandible, all buccal soft tissue except buccal area of molars 

    Lingual nerve:  No teeth; lingual gingival of mandibular quadrant (Central to 3rdmolar), anterior 2/3 of tongue, floor of mouth.

    The lingual nerve is commonly anesthetized during the IA injection but can be done separately.

    Needle 25 or 27 longLandmarks Coronoid notch, lingula, pterygomandibular raphe, occlusal plane of MN premolar 

    teeth & commissure on contralateral side.

    Insertion/ Penetration Site Medial to internal & external oblique ridges. Height of coronoid notch. Lateral to pterygomandibular raphe. The syringe barrel placed at the corner of the mouth, usually corresponding to the contralateral premolars.

    The lingual nerve will be anesthetized during the same insertion for IA injection*Approximate Depth of Penetration  IA:  20‐25 mm 2/3‐3/4 length of long needle. 

    Lingual:  1/2 distance of IA.Deposition/ Target Site  IA:  Directly above mandibular foramen. 

    Lingual:  Half the distance to mandibular foramen at lingual nerve.

    *Volume of Anesthetic IA:   1.5‐1.8ml (3/4 ‐ 1 cartridge)

    Lingual:   25‐.5ml (1/8 cartridge)Potential Complications/ Additional Considerations

    Too far medially:  medial pterygoid muscle; trismus. Too deep: facial nerve paralysis if anesthetic is deposited in parotid gland. Wide area anesthesia; lower lip anesthesia. Warn patients to not bite lip or tongue. Shocking pain if lingual nerve touched.

  • 17

    INJECTION Buccal Nerve Block (B)

    Branch of Trigeminal Nerve Long buccal nerve, branch of anterior root of Mandibular (V3)Area Anesthetized (Teeth &/orSoft Tissue)

    No teeth.

    Soft tissues & periosteum buccal to MD molars

    Needle 25 or 27 long because it follows IALandmarks External oblique ridge and 2nd molar; retromolar fossa.

    Insertion/ Penetration Site Mucobuccal fold distal & buccal to last molar; parallel with occlusal plane

    Border of ramus / External oblique ridge / distal & buccal to last molar*Approximate Depth of Penetration 

    3 ‐ 6mm (1/8 – 1/4”)

    Deposition/ Target Site  Medial to external oblique ridge; distal and buccal to last molar. 

    *Volume of Anesthetic .25‐.5ml (1/8 cartridge)Potential Complications/ Additional Considerations

    Uncomfortable if needle contacts periosteum

    INJECTION MENTAL BLOCK

    Branch of Trigeminal Nerve Terminal branch of the inferior alveolar nerveArea Anesthetized (Teeth &/orSoft Tissue)

    No teeth. 

    Buccal soft tissues from mental foramen to midline and the soft tissues of the lower lip and chin.

    Needle 25 or 27 shortLandmarks Mental foramen‐usually located between the two premolars. 

    However, it may be either anterior or posterior to this site.

    Insertion/ Penetration Site Mental foramen usually between apices of 1st and 2nd premolars, Mucobuccal fold

    *Approximate Depth of Penetration 

    4‐6mm (1/8‐1/4”)

    Deposition/ Target Site  Directly over the mental foramen*Volume of Anesthetic .5‐1.0ml (1/3‐1/2 cartridge)Potential Complications/ Additional Considerations

    Possibility of hematoma

  • 18

    Since 1884, when cocaine was first used by William Halsted as a nerve block, local anesthetics came a long way.15 With the improved efficacy and safety for our patients, dental providers can administer these anesthetics in effort to provide painless procedures. Nevertheless, the medical and dental histories, along with the patients’ previous exposures and procedures at hand must be carefully evaluated in the selection of the local anesthetic. Successfully administered local anesthesia can elevate the anxiety of the procedure and create an overall positive experience for the patient, leading to a trustworthy relationship between the patient and healthcare provider.

    End

  • 19

    References

    1. Moore PA, Hersh EV, Boynes SG. Preface update of dental local anesthesia. Dent Clin North Am. 2010 Oct;54(4):xiii-xiv. doi: 10.1016/j.cden.2010.07.001. PMID: 20831922.

    2. Malamed S: Handbook of local anesthesia, ed 7, St Louis, 2020, Elsevier. 3. Logothetis, DD: Local Anesthesia for the Dental Hygienist, ed 2, St Louis, 2017,

    Elsevier. 4. Darby M, Walsh M: Dental hygiene theory and practice, ed 4, St Louis, 2014,

    Saunders.5. Sodium Bisulfite https://pubchem.ncbi.nlm.nih.gov/compound/Sodium-bisulfite6. Malamed SF: Medical emergencies in the dental office, ed 7, St Louis, 2015, Mosby. 7. Becker, D. E., & Reed, K. L. (2006). Essentials of local anesthetic

    pharmacology. Anesthesia progress, 53(3), 98–110. https://doi.org/10.2344/0003-3006(2006)53[98:EOLAP]2.0.CO;2

    8. Little JW, Falace DA, Miller CS, Rhodus NL: Dental management of the medically compromised patient, ed 8, St Louis, 2013, Mosby.

    9. Frank, S. G., & Lalonde, D. H. (2012). How acidic is the lidocaine we are injecting, and how much bicarbonate should we add?. The Canadian journal of plastic surgery = Journal canadien de chirurgie plastique, 20(2), 71–73. https://doi.org/10.1177/229255031202000207

    10. Orrett E. Ogle DDS and Ghazal Mahjoubi DMD Dental Clinics of North America, 2012-01-01, Volume 56, Issue 1, Pages 133-148, Copyright © 2012 Elsevier Inc

    11. The Antiarrhythmic Properties of Lidocaine and Procaine Amide Clinical and Physiologic Studies of Their Cardiovascular Effects in Man By DONALD C. HARRISON, M.D., J. HENRY SPROUSE, M.D., AND ANDREW G. MORROW, M.D. https://www.ahajournals.org/doi/pdf/10.1161/01.CIR.28.4.486

    12. Nusstein JM, Beck M. Effectiveness of 20% benzocaine as a topical anesthetic for intraoral injections. Anesth Prog. 2003;50(4):159-63. PMID: 14959903; PMCID: PMC2007446.

    13. Malamed SF. Emergency medicine: preparation and basics of management. Dent Today. 2001;20(64):66-67

    14. Kanaa MD, Meechen JG, Corbett IR, Whitworth JM. Speed of injection influences efficacy of inferior alveolar nerve blocks: a double-blind randomized controlled trial in volunteers. J Endod. 2006;32:919-923.

    15. Redman M. Cocaine: What is the Crack? A Brief History of the Use of Cocaine as an Anesthetic. Anesth Pain Med. 2011 Fall;1(2):95-7. doi: 10.5812/kowsar.22287523.1890. Epub 2011 Sep 26. PMID: 25729664; PMCID: PMC4335732.