02_pneumonias. pleural syndrome

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    Pneumonias. Pleural syndrome.

    Ethiology. Clinical pattern. Daignostics.

    Complications. Principles of treatment

    Ass-prof. N. Bilkevych

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    Acute inflammation of lung

    parenchyma with obvious

    involvement of alveoli.Usually is caused by bacteria or

    viruses

    Pneumonia(pneumonia)

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    Alveoli and lung cells that produce

    surfactant

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    Ethio logy:

    Not specific pathogenic

    or obligate-pathogenicmicrobes

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    Pathogenesis:

    Infection spread into theorganism through respiratory

    airways. Microbes appears and

    multiple on bronchial mucosa of

    upper airways and than spread

    down to bronchi and lung tissue

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    Classification

    Community-acquiredpneumonia.

    Nosocomia l(intrahospital) pneumonia acute

    infection of lower airways confirmed with X-ray, has being developed in 48 hrs after

    appearance of the patient in hospotal

    environment.

    Aspi rat ionpneumonia.

    Pneumoniain immunocomprom izwd pat ients

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    Croupous pneumonia Acu te inf lammat ion of lungs, which in

    mos t cases sp reads on all pulmonary

    lobe. That is why it is cal led lobar

    pneumonia (pneumonia lobar is) , but

    can be l imited to the affect ion of

    segment or a few segments.

    Synonims fibrinous pneumonia,

    pleuropneumonia

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    Pneumococc i

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    Pneumococci are typically

    asociated with pneumonia

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    Clin ical s tages:

    initial

    clinical manifestation

    (corresponds to red and greyhepatisation

    resolution

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    Lobar pneumonia: stage of

    onset Morphology. Congestion stage extensive serous

    exudation, vascular engorgement, rapid bacterial pro-liferation.

    Inspection. An increased respiratory rate is usuallyevident. Pain is a frequent accompaniment, and with itthe involved side shows a lag of respiratory motion.

    Palpation. Palpation confirms the findings on inspection.Tactile fremitus is normal or even slightly decreased, anda pleural friction rub may be present.

    Percussion. Impaired resonance may be elicited withlight percussion. This finding is extremely important.

    Auscultation. Although the breath sounds may bediminished, expiration is prolonged and crepitation(crepitus indux) is heard. With pleural involvement, apleural friction sound is determined.

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    Lobar pneumonia: stage of

    consolidation

    Morphology. Red hepatization stage airspaces are filledwith PMN cells, vascular congestion, extravasation of

    RBC. Grey hepatization stage

    accumulation of fibrin,inflammatory WBCs and RBCs in various stages ofdisintegration, alveolar spaces filled with inflammatoryexudate.

    Complaints. Coughing may be associated with i sharp painin the affected side. Mucoid sputum be comes rusty brown(prune juice color).

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    General inspection. Cyanosis of the lips and fin gers.When the fever is high, the face may be flushed The

    patient's nostrils dilate on inspiration, and expi ration isoften grunting.

    Inspection. Dyspnea is invariably present. Respi ratorymovements are generally decreased on the af fectedside.

    Palpation. Diminished respiratory excursions, i pleuralfriction rub may be felt. Tactile fremitus is in creased.

    Percussion. Dullness.

    Auscultation. Bronchial breathing, bronchophony,

    pectoriloquy and whispered bronchophony are evidentwith consolidation provided the bronchus to the in volvedarea is open. Rales are less numerous and dis tinct thanin the stages of engorgement or resolution,

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    Forced position

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    Lobar pneumonia: stage of

    resolution

    Morphology. Resolution stage resorption of theexudate.

    Inspection. The patient looks more comfortable and thecyanosis disappears. The dyspnea disappears and the

    affected lung begins to expand again. Palpation. The previously increased tactile fremitus

    becomes less marked and gradually findings becomenormal.

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    Percussion. The dullness gradually

    disappears and normal resonance returns.

    Auscultation. The bronchial breathing is

    gradually replaced by bronchovesicular

    breathing and later by normal vesicular

    breathing. Crepitation reappears (crepitus

    redux). Small and large moist rales areheard in increasing numbers.

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    Complications

    Lung abscess or gangroene

    Pleurisy

    Toxic shock

    Myocarditis Acute respiratory insufficiency

    Pneumosclerosis

    Atelectasis

    Sepsis

    Meningitis, encefalitis

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    Pleurisy

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    Pleurisy with effusion: 1

    Damoiseau's curve; 2

    Garland's triangle;

    3Rauchfuss-Grocco triangle.

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    Lung abscess

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    Lung abscess

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    Focal pneumonia

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    Focal pneumonia

    Focal pneumonia

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    Focal pneumonia

    The feature of these pneumon ias is

    an invo lvement o f separate lobules

    or g roups of lobules in the

    inf lammatory process . Therefore i t is

    named also lobu lar (pneumon ialobular is)

    Synonim: bronchopneumonia

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    Principles of treatment

    Antibiotics

    Expectorants

    Desintoxication

    Oxygen

    Antigistamine agents

    Symptomatic therapy

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    orrec t reg imen

    Bed mode

    Care of pat ients :

    proper lighting and ventilation(fresh air improve patientssleep and

    bronchial clearance)

    Care of oral cavity

    Diet

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    Diet

    about 2,5-3 litres of loquid per day(water with lemon juice, mineral or

    boiled water

    Fruit juices

    chicken clear soups

    food should be easly assimilable

    in some days diet 10 or 15.

    Diet enr iched w ith vi tam ins

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    Diet enr iched w ith vi tam ins

    Climatotherapy

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    pymild dry warmclimat

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    Symptomat ic means

    antitussives antipyretics

    Pain killers

    antiinflammmatory(nonsteroidal in pleural pain)

    cardiotonics

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