02_pneumonias. pleural syndrome
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Pneumonias. Pleural syndrome.
Ethiology. Clinical pattern. Daignostics.
Complications. Principles of treatment
Ass-prof. N. Bilkevych
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Acute inflammation of lung
parenchyma with obvious
involvement of alveoli.Usually is caused by bacteria or
viruses
Pneumonia(pneumonia)
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Alveoli and lung cells that produce
surfactant
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Ethio logy:
Not specific pathogenic
or obligate-pathogenicmicrobes
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Pathogenesis:
Infection spread into theorganism through respiratory
airways. Microbes appears and
multiple on bronchial mucosa of
upper airways and than spread
down to bronchi and lung tissue
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Classification
Community-acquiredpneumonia.
Nosocomia l(intrahospital) pneumonia acute
infection of lower airways confirmed with X-ray, has being developed in 48 hrs after
appearance of the patient in hospotal
environment.
Aspi rat ionpneumonia.
Pneumoniain immunocomprom izwd pat ients
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Croupous pneumonia Acu te inf lammat ion of lungs, which in
mos t cases sp reads on all pulmonary
lobe. That is why it is cal led lobar
pneumonia (pneumonia lobar is) , but
can be l imited to the affect ion of
segment or a few segments.
Synonims fibrinous pneumonia,
pleuropneumonia
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Pneumococc i
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Pneumococci are typically
asociated with pneumonia
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Clin ical s tages:
initial
clinical manifestation
(corresponds to red and greyhepatisation
resolution
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Lobar pneumonia: stage of
onset Morphology. Congestion stage extensive serous
exudation, vascular engorgement, rapid bacterial pro-liferation.
Inspection. An increased respiratory rate is usuallyevident. Pain is a frequent accompaniment, and with itthe involved side shows a lag of respiratory motion.
Palpation. Palpation confirms the findings on inspection.Tactile fremitus is normal or even slightly decreased, anda pleural friction rub may be present.
Percussion. Impaired resonance may be elicited withlight percussion. This finding is extremely important.
Auscultation. Although the breath sounds may bediminished, expiration is prolonged and crepitation(crepitus indux) is heard. With pleural involvement, apleural friction sound is determined.
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Lobar pneumonia: stage of
consolidation
Morphology. Red hepatization stage airspaces are filledwith PMN cells, vascular congestion, extravasation of
RBC. Grey hepatization stage
accumulation of fibrin,inflammatory WBCs and RBCs in various stages ofdisintegration, alveolar spaces filled with inflammatoryexudate.
Complaints. Coughing may be associated with i sharp painin the affected side. Mucoid sputum be comes rusty brown(prune juice color).
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General inspection. Cyanosis of the lips and fin gers.When the fever is high, the face may be flushed The
patient's nostrils dilate on inspiration, and expi ration isoften grunting.
Inspection. Dyspnea is invariably present. Respi ratorymovements are generally decreased on the af fectedside.
Palpation. Diminished respiratory excursions, i pleuralfriction rub may be felt. Tactile fremitus is in creased.
Percussion. Dullness.
Auscultation. Bronchial breathing, bronchophony,
pectoriloquy and whispered bronchophony are evidentwith consolidation provided the bronchus to the in volvedarea is open. Rales are less numerous and dis tinct thanin the stages of engorgement or resolution,
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Forced position
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Lobar pneumonia: stage of
resolution
Morphology. Resolution stage resorption of theexudate.
Inspection. The patient looks more comfortable and thecyanosis disappears. The dyspnea disappears and the
affected lung begins to expand again. Palpation. The previously increased tactile fremitus
becomes less marked and gradually findings becomenormal.
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Percussion. The dullness gradually
disappears and normal resonance returns.
Auscultation. The bronchial breathing is
gradually replaced by bronchovesicular
breathing and later by normal vesicular
breathing. Crepitation reappears (crepitus
redux). Small and large moist rales areheard in increasing numbers.
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Complications
Lung abscess or gangroene
Pleurisy
Toxic shock
Myocarditis Acute respiratory insufficiency
Pneumosclerosis
Atelectasis
Sepsis
Meningitis, encefalitis
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Pleurisy
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Pleurisy with effusion: 1
Damoiseau's curve; 2
Garland's triangle;
3Rauchfuss-Grocco triangle.
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Lung abscess
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Lung abscess
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Focal pneumonia
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Focal pneumonia
Focal pneumonia
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Focal pneumonia
The feature of these pneumon ias is
an invo lvement o f separate lobules
or g roups of lobules in the
inf lammatory process . Therefore i t is
named also lobu lar (pneumon ialobular is)
Synonim: bronchopneumonia
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Principles of treatment
Antibiotics
Expectorants
Desintoxication
Oxygen
Antigistamine agents
Symptomatic therapy
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orrec t reg imen
Bed mode
Care of pat ients :
proper lighting and ventilation(fresh air improve patientssleep and
bronchial clearance)
Care of oral cavity
Diet
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Diet
about 2,5-3 litres of loquid per day(water with lemon juice, mineral or
boiled water
Fruit juices
chicken clear soups
food should be easly assimilable
in some days diet 10 or 15.
Diet enr iched w ith vi tam ins
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Diet enr iched w ith vi tam ins
Climatotherapy
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pymild dry warmclimat
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Symptomat ic means
antitussives antipyretics
Pain killers
antiinflammmatory(nonsteroidal in pleural pain)
cardiotonics
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